This document provides an overview of chest radiograph interpretation for interns, covering normal anatomy, common pathologies, and technical factors. It summarizes how to evaluate for adequate penetration, inspiration, rotation, magnification, and angulation. Common pathologies like pleural effusion, pneumothorax, pneumonia, and pulmonary tuberculosis are described with examples. Normal pediatric and adult chest x-ray features are outlined along with how to read and interpret the major anatomical structures visible.
Describes cross sectional anatomy of the mediastinum , and lobar and segmental anatomy of the lung with teaching points and radiological guidelines and multiple examples of lobar and segmental pathologies and how we localize these pathologies .Also the types of chest CT images and indications of chest CT.
Concise overview of all the information that a Medico must know for his knowledge as well as to appear for entrance exams as well as for physicians for their routine practice.
Describes cross sectional anatomy of the mediastinum , and lobar and segmental anatomy of the lung with teaching points and radiological guidelines and multiple examples of lobar and segmental pathologies and how we localize these pathologies .Also the types of chest CT images and indications of chest CT.
Concise overview of all the information that a Medico must know for his knowledge as well as to appear for entrance exams as well as for physicians for their routine practice.
This free eBook was written to help Radiologic Technologists improve their image quality and understand some of the latest tools and techniques available with Computed and Digital Radiography equipment.
This presentation is almost a complete Pictoral view of Radiograph chest.
This presentation will help radiologist in daily reporting.
This presentation will help physicians, surgeons, anesthetist and almost all medical professionals in diagnosing commonly presenting cardiac diseases.
This will also help all in preparaing TOACS examination.
Lecture on SexEd for Grade 7 female students
Instead of the usually awkward sex ed lecture, I included self-care in all aspects of health: physical, mental, emotional, social, and spiritual.
List of vaccines available in the market
This list includes trade name, manufacturer, common abbreviation, type and route of administration, and primary and booster doses for pediatric population. It also includes link to the vaccines' product information.
This is helpful especially for starting pediatricians
Presentation on Prevention and Management of Infants With Suspected or Proven Neonatal Sepsis
References:
American Academy of Pediatrics. Prevention and Management of Infants With Suspected or Proven Neonatal Sepsis, 2013.
American Academy of Pediatrics. Management of Neonates With Suspected or Proven Early-Onset Bacterial Sepsis, 2012.
This is Cristal Laquindanum’s 20 yr marketing plan, project submitted to her Marketing subject. She is currently a Year Level 8 (clinical clerk) in Ateneo School of Medicine and Public Health taking up double degree in Medicine and Masters in Business Administration.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
1. Chest Radiograph For Interns Reported by: Cristal Ann G. Laquindanum ASMPH Class of 2012
2. References Brant and Helm’s Paul and Juhls Learningradiology.com Clinical Radiology Made Ridiculously Simple School lectures
3. Outline Normal Chest Radiograph – adult and pediatric Requirement for chest xray film Anatomy Common pathologies – pleural effusion, pneumothorax, pneumonia, PTB
9. Underpenetration > Left hemidiaphragm may not be visible on the frontal film; left lung base may appear opaque > Pulmonary markings may appear more prominent Overpenetration > Lung markings may seem decreased or absent
11. Rotation If the spinous process of the vertebral body is equidistant from the medial ends of each clavicle, there is NO rotation
12. If the spinous process appears closer to the right clavicle (redarrow), the patient is rotated toward their own left side If the spinous process appears closer to the left clavicle (redarrow), the patient is rotated toward their own right side
13. Severe rotation may make the pulmonary arteries appear larger on the side farther from the film
17. Angulation If the x-ray beam is angled toward the head (mostly because the patient is semi-recumbent), the film so obtained is called an “apical lordotic” view
19. Penetration see spine through the heart Inspiration at least 8-9 posterior ribs Rotation spinous process between clavicles Magnification AP films will slightly magnify the heart Angulation clavicle over 3rd rib Factors to Evaluate:
20. How to read a Chest Xray: Basics Technically Adequate ☐ Anatomy
21.
22.
23. Trachea Upper Lobes Aortic knob Left Pulmonary Artery Right Atrium Left Ventricle Lower Lobes
24.
25. Trachea Upper Lobes Aortic knob Carina Left Pulmonary Artery Right Atrium Left Ventricle Lower Lobes Costophrenic Angle Gastric bubble
39. Lungs more radiolucent Thymus is often large, Widening of superior mediastinum Ribs angulate downward Heart is globular and large Left ventricle more prominent with age Diaphragm is higher Left > Right
41. Pleural Effusion complete opacification of the right mid and lower zones is due to fluid in the pleural cavity meniscus sign - concavity of the fluid level due to surface tension with the pleura Blunting of costophrenic angle may be due to a small pleural effusion or focal pleural thickening. May coexist with pneumothorax or entrapped within fissures mimicking a tumor. Right pleural effusion in a patient with nephrotic syndrome The flattened and laterally displaced curvature of the right hemidiaphragm indicates presence of subpulmonic pleural fluid
42. Pleural Effusion Patient cannot stand? lateral decubitus Ultrasound or CT thorax as alternative modalities for early detection of small pleural effusion. Ultrasound advantage: no radiation; can be used to guide drainage CT advantage: evaluate the underlying lung and mediastinal structures to identify the cause of the effusion
43. Pneumothorax Pneumothorax represents abnormal air accumulation within pleural cavity. This may be due to trauma (accidental or iatrogenic), underlying pulmonary disease (e.g. asthma) or idiopathic in origin Erect chest radiograph in full expiration is diagnostic in majority of cases If the patient is unable to stand erect, lateral decubitus view may be helpful
44. Pneumothorax Radiologic Findings: Contralateral mediastinalshift Depression of ipsilateralhemi-diaphragm Compressive atelectasis of adjacent normal lung presence of significant increased intrathoracic pressure Role of imaging in patients with pneumothorax: 1. Confirm the clinical diagnosis 2. Assess extent of pneumothorax 3. Detect signs of tension 4. Follow-up examination to monitor resolution of pneumothorax after drainage
45. The right lung (white open arrows) has been pushed medially. The mediastinum is shifted to the left (black arrow). This appearance is typical of tension pneumothorax. Magnified view of a PA chest radiograph of a right pneumothorax. The visceral pleura (arrow heads) is seen as a thin white line.
46. Pneumonia Role of imaging in patients with pneumonia 1. Confirm the clinical diagnosis 2. Detect possible complications such as pleural effusion / empyema or lung abscess if clinically not responsive to appropriate antibiotic treatment 3. Follow-up CXR to monitor response to treatment may take 4-6 weeks for consolidative changes to resolve Radiologic improvement usually lags behind clinical improvement If radiologic signs still present after adequate treatment, underlying predisposing factors have to be excluded (e.g. central obstructive carcinoma in elderly patients)
52. Pneumonia Pneumonia caused by certain organisms may produce characteristic radiologic features Unilateral lobar involvement in streptococcus infection Bilateral patchy involvement sometimes with cavitation in staphylococcus pneumonia Upper lobe involvement with cavitation in pulmonary TB Bilateral symmetrical perihilar distribution which progresses rapidly over 3-5 days in PCP pneumonia in immunocompromised patients
57. Pulmonary Tuberculosis Radiologic Findings in PTB Cavitation and air-fluid level- the opacity represents caseous necrosis in tuberculosis Enlarged hilum– representinggranulomatous inflammation of lymph nodes, usually in primary TB Fibrocalcificchanges in lung apex usually representing healing of previous TB infection Multi-focal air-space opacities representing bronchogenic spread of infection Tiny miliary nodules in both lungs representing miliary TB due to haematogenous spread of infection
61. AM, 5year old female CC: fever and cough HPI: 6 days PTC – on and off fever (Tmax 39) + headache 3 days PTC – abdominal pain, consult done and was given Cefaclor and Ventolin expectorant Morning PTC – symptoms persisted, one episode of post-tussive vomiting, decreased appetite admission HISTORY
62. PAST MEDICAL HISTORY TMC – Dengue and UTI – Oct 2010 VRMC – Pneumonia 2006 BIRTH HISTORY Born full term via NSD to a 30 year old G2P2 (2002) no fetomaternal complications VACCINATION HISTORY Only BCG, DPT, HepB, MMR x1, no HiB
63. NUTRITIONAL HISTORY Breastfed until 2 months, on milk formula until 15 months Weaning at months, food preference fish FAMILY HISTORY unremarkable
64. BP 90/60 , RR 30 , HR 118 , Temp 39.5 Harsh breath sounds, equal chest expansion, rales R>L, no wheezes, no alar flaring, no retractions Hyperactive bowel sounds, epigastric tenderness Tachycardic, normal rhythm PHYSICAL EXAMINATION
67. Patches of hazed densities are noted in the right middle lobe, bordered superiorly by the minor fissure. Hazy densities are likewise seen in the posterior segment of the right lower lobe. The pulmonary vascular pattern is within normal. Cardiac shadow is normal in size and configuration. The retrosternal and retrocardiac spaces are intact. The diaphragm, costophrenic sulci, and the bony thorax are unremarkable. Consider Pneumonia with beginning consolidation, right
68. Patches of hazed densities are noted in the right middle lobe, bordered superiorly by the minor fissure. Hazy densities are likewise seen in the posterior segment of the right lower lobe. The pulmonary vascular pattern is within normal. Cardiac shadow is normal in size and configuration. The retrosternal and retrocardiac spaces are intact. The diaphragm, costophrenic sulci, and the bony thorax are unremarkable. Consider Pneumonia with beginning consolidation, right
69. Admitted VS q4 DAT Monitor I&O D5Nm 1 L x 69-70 cc/hr (M+10) Paracetamol 250mg/5mL q4 Amoxicillin 250mg/5mL at 40mkd Currently on 3rd hospital stay MANAGEMENT
The less dense an object is, the fewer xrays it absorbs, and the blacker it wll appearAir attenuates very little of the xray beam, allowing nearly full force of the beam to blacken the image. Fat, and soft tissues attenuate interediate amounts of the xray beam , resulting in proportional degrees of image blackening (shades of gray) radiation than thin structures iBone, metal, and radiographic contrast agents attenuate a large proportion of the xray beam, allowing very little radiation through to blacken the image. Thus, bone and metallic objects and structures opacified by xray contrast agents appear white on radiographs. Bone is densest naturally occurring tissue. It absorbs the greatest amount of xray and appears white on radiographs.Metal is even denser than bone and essentially absorbs all xrays, but unless you have bullets or artifical hip replacements, metal doesn’t naturally occur in humans
Underpenetration – mimic or hide true disease of left lower lung field (lower lobe pneumonia, left pleural effusion)Overpenetration – mistakenly think patient has emphysema or pneumothorax; pulmonary nodule may be invisible
In an AP film, the heart is farther from the film and is more magnifiedPortable chest x-rays are almost always done AP
In an AP film, the heart is farther from the film and is more magnifiedPortable chest x-rays are almost always done AP
Anterior structures (like the clavicles) will be projected higher on the film than posterior structures
A film which is apical lordotic (beam is angled up toward head) will have an unusually shaped heart and the sharp border of the left hemidiaphragm will be absentAnterior structures (like the clavicles) will be projected higher on the film than posterior structureshis projectionresults in clear visualization of the lung apices because the clavicle and first rib are projected above the pulmonary apex
Right – minor fissure separates upper and middle lobe. Major fissure middle lobe and lower lobeRight upper lobe – anterior apical and posteriorMiddle lobe – medial and lateralRight lower lobe – anterior, lateral, posterior and medialLeft – upper and lower lobes by left major fissure Left upper lobe is analogous to the combined right upper and middle , anterior, apicoposterior, superior and inferior lingular segmentsLeft lower lobe – anteromedial, lateral and posterior
Airways, including hilaradenopathy or enlargementBreast shadowsBones, e.g. rib fractures and lytic bone lesionsCardiac silhoutte, detecting cardiac enlargementCostophrenic angles, including pleural effusionsDiaphragm, e.g. evidence of free airEdges, e.g. apices for fibrosis, pneumothorax, pleural thickening or plaquesExtrathoracic tissuesFields (lung parenchyma), being evidence of alveolar fillingFailure, e.g. alveolar air space disease with prominent vascularity with or without pleural effusions
Airways, including hilaradenopathy or enlargementBreast shadowsBones, e.g. rib fractures and lytic bone lesionsCardiac silhoutte, detecting cardiac enlargementCostophrenic angles, including pleural effusionsDiaphragm, e.g. evidence of free airEdges, e.g. apices for fibrosis, pneumothorax, pleural thickening or plaquesExtrathoracic tissuesFields (lung parenchyma), being evidence of alveolar fillingFailure, e.g. alveolar air space disease with prominent vascularity with or without pleural effusions
Airways, including hilaradenopathy or enlargementBreast shadowsBones, e.g. rib fractures and lytic bone lesionsCardiac silhoutte, detecting cardiac enlargementCostophrenic angles, including pleural effusionsDiaphragm, e.g. evidence of free airEdges, e.g. apices for fibrosis, pneumothorax, pleural thickening or plaquesExtrathoracic tissuesFields (lung parenchyma), being evidence of alveolar fillingFailure, e.g. alveolar air space disease with prominent vascularity with or without pleural effusions
DeviationFBET Tube
Airways, including hilaradenopathy or enlargementBreast shadowsBones, e.g. rib fractures and lytic bone lesionsCardiac silhoutte, detecting cardiac enlargementCostophrenic angles, including pleural effusionsDiaphragm, e.g. evidence of free airEdges, e.g. apices for fibrosis, pneumothorax, pleural thickening or plaquesExtrathoracic tissuesFields (lung parenchyma), being evidence of alveolar fillingFailure, e.g. alveolar air space disease with prominent vascularity with or without pleural effusions
Airways, including hilaradenopathy or enlargementBreast shadowsBones, e.g. rib fractures and lytic bone lesionsCardiac silhoutte, detecting cardiac enlargementCostophrenic angles, including pleural effusionsDiaphragm, e.g. evidence of free airEdges, e.g. apices for fibrosis, pneumothorax, pleural thickening or plaquesExtrathoracic tissuesFields (lung parenchyma), being evidence of alveolar fillingFailure, e.g. alveolar air space disease with prominent vascularity with or without pleural effusions
Airways, including hilaradenopathy or enlargementBreast shadowsBones, e.g. rib fractures and lytic bone lesionsCardiac silhoutte, detecting cardiac enlargementCostophrenic angles, including pleural effusionsDiaphragm, e.g. evidence of free airEdges, e.g. apices for fibrosis, pneumothorax, pleural thickening or plaquesExtrathoracic tissuesFields (lung parenchyma), being evidence of alveolar fillingFailure, e.g. alveolar air space disease with prominent vascularity with or without pleural effusions
Airways, including hilaradenopathy or enlargementBreast shadowsBones, e.g. rib fractures and lytic bone lesionsCardiac silhoutte, detecting cardiac enlargementCostophrenic angles, including pleural effusionsDiaphragm, e.g. evidence of free airEdges, e.g. apices for fibrosis, pneumothorax, pleural thickening or plaquesExtrathoracic tissuesFields (lung parenchyma), being evidence of alveolar fillingFailure, e.g. alveolar air space disease with prominent vascularity with or without pleural effusions
Newborn infantAP diameter of the thorax is greater compared with the tranverse diameter than in adultsDiaphragm is higher, vertical diameter of the thoracic cavity relatively less than in the adultWith growth, chest becomes narrower and ribs gradually angulate downward from horizontal position Thymus is often large enough in early infancy to produce widening of the superior mediastinum -bilobed structure located in the anterior mediastinum that can cause considerable confusion simulate cardiomegaly, upper-lobe pneumonia, and atelectasis. Additionally, it can appear as a pathologic mass if it is aberrant in locationHeart is globular and is relatively large in comparison with the diameter of the chest than in adultsLeft ventricle becomes more prominent with age, resulting in downward displacement of the apex, and the relatively heart size gradually decreasesLungs more radiolucent than in the adult because the pulmonary interstitium usually is not visible., tracheal bifurcation gradually descends and reaches the adult level (T5) at about 10 yrs old The hilar shadows are relatively high and usually are situated at the level of the third thoracic vertebra.Diaphragm tends to be higher in infancy and childhood than in adult life; opposite to adult that left hemidiaphragm higher than the right (stomach is distended with air)
Two substances of the same density, in direct contact, cannot be differentiated from each other on x-rayCommon locationsLower lobes-diaphragmsRight heart border – RMLLeft heart border – LingulaLeft diaphragm – Heart (on lateral view)