Wolff–Parkinson–White syndrome (WPW) is one of several disorders of the conduction system of the heart that are commonly referred to as pre-excitation syndromes. WPW is caused by the presence of an abnormal accessory electrical conduction pathway between the atria and the ventricles. Electrical signals travelling down this abnormal pathway (known as the bundle of Kent) may stimulate the ventricles to contract prematurely, resulting in a unique type of supraventricular tachycardia referred to as an atrioventricular reciprocating tachycardia.The incidence of WPW is between 0.1% and 0.3% in the general population.Sudden cardiac death in people with WPW is rare (incidence of less than 0.6%), and is usually caused by the propagation of an atrial tachydysrhythmia (rapid and abnormal heart rate) to the ventricles by the abnormal accessory pathway.
Wolff–Parkinson–White syndrome (WPW) is one of several disorders of the conduction system of the heart that are commonly referred to as pre-excitation syndromes. WPW is caused by the presence of an abnormal accessory electrical conduction pathway between the atria and the ventricles. Electrical signals travelling down this abnormal pathway (known as the bundle of Kent) may stimulate the ventricles to contract prematurely, resulting in a unique type of supraventricular tachycardia referred to as an atrioventricular reciprocating tachycardia.The incidence of WPW is between 0.1% and 0.3% in the general population.Sudden cardiac death in people with WPW is rare (incidence of less than 0.6%), and is usually caused by the propagation of an atrial tachydysrhythmia (rapid and abnormal heart rate) to the ventricles by the abnormal accessory pathway.
Wolff–Parkinson–White syndrome (WPW) is one of several disorders of the conduction system of the heart that are commonly referred to as pre-excitation syndromes. WPW is caused by the presence of an abnormal accessory electrical conduction pathway between the atria and the ventricles. Electrical signals traveling down this abnormal pathway (known as the bundle of Kent) may stimulate the ventricles to contract prematurely, resulting in a unique type of supra-ventricular tachycardia referred to as an atrio-ventricular reciprocating tachycardia.
Chronic Stable Angina- Diagnosis & management
By Dr Awadhesh Kumar Sharma
Dr. Awadhesh kumar sharma is a young, diligent and dynamic interventional cardiologist. He did his graduation from GSVM Medical College Kanpur and MD in Internal Medicine from MLB Medical college jhansi. Then he did his superspecilisation degree DM in Cardiology from PGIMER & DR Ram Manoher Lohia Hospital Delhi. He had excellent academic record with Gold medal in MBBS,MD and first class in DM.He was also awarded chief ministers medal in 2009 for his academic excellence by former chief minister of UP Smt Mayawati in 2009.He is also receiver of GEMS international award.He had many national & international publications.He is also in editorial board of international journal- Journal of clinical medicine & research(JCMR).He is also active member of reviewer board of many journals.He is also trainee fellow of American college of cardiology. He is currently working in NABH Approved Gracian Superspeciality Hospital Mohali as Consultant Cardiologist.
Wolff–Parkinson–White syndrome (WPW) is one of several disorders of the conduction system of the heart that are commonly referred to as pre-excitation syndromes. WPW is caused by the presence of an abnormal accessory electrical conduction pathway between the atria and the ventricles. Electrical signals traveling down this abnormal pathway (known as the bundle of Kent) may stimulate the ventricles to contract prematurely, resulting in a unique type of supra-ventricular tachycardia referred to as an atrio-ventricular reciprocating tachycardia.
Chronic Stable Angina- Diagnosis & management
By Dr Awadhesh Kumar Sharma
Dr. Awadhesh kumar sharma is a young, diligent and dynamic interventional cardiologist. He did his graduation from GSVM Medical College Kanpur and MD in Internal Medicine from MLB Medical college jhansi. Then he did his superspecilisation degree DM in Cardiology from PGIMER & DR Ram Manoher Lohia Hospital Delhi. He had excellent academic record with Gold medal in MBBS,MD and first class in DM.He was also awarded chief ministers medal in 2009 for his academic excellence by former chief minister of UP Smt Mayawati in 2009.He is also receiver of GEMS international award.He had many national & international publications.He is also in editorial board of international journal- Journal of clinical medicine & research(JCMR).He is also active member of reviewer board of many journals.He is also trainee fellow of American college of cardiology. He is currently working in NABH Approved Gracian Superspeciality Hospital Mohali as Consultant Cardiologist.
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The Roman Empire, a vast and enduring power, stands as one of history's most remarkable civilizations, leaving an indelible imprint on the world. It emerged from the Roman Republic, transitioning into an imperial powerhouse under the leadership of Augustus Caesar in 27 BCE. This transformation marked the beginning of an era defined by unprecedented territorial expansion, architectural marvels, and profound cultural influence.
The empire's roots lie in the city of Rome, founded, according to legend, by Romulus in 753 BCE. Over centuries, Rome evolved from a small settlement to a formidable republic, characterized by a complex political system with elected officials and checks on power. However, internal strife, class conflicts, and military ambitions paved the way for the end of the Republic. Julius Caesar’s dictatorship and subsequent assassination in 44 BCE created a power vacuum, leading to a civil war. Octavian, later Augustus, emerged victorious, heralding the Roman Empire’s birth.
Under Augustus, the empire experienced the Pax Romana, a 200-year period of relative peace and stability. Augustus reformed the military, established efficient administrative systems, and initiated grand construction projects. The empire's borders expanded, encompassing territories from Britain to Egypt and from Spain to the Euphrates. Roman legions, renowned for their discipline and engineering prowess, secured and maintained these vast territories, building roads, fortifications, and cities that facilitated control and integration.
The Roman Empire’s society was hierarchical, with a rigid class system. At the top were the patricians, wealthy elites who held significant political power. Below them were the plebeians, free citizens with limited political influence, and the vast numbers of slaves who formed the backbone of the economy. The family unit was central, governed by the paterfamilias, the male head who held absolute authority.
Culturally, the Romans were eclectic, absorbing and adapting elements from the civilizations they encountered, particularly the Greeks. Roman art, literature, and philosophy reflected this synthesis, creating a rich cultural tapestry. Latin, the Roman language, became the lingua franca of the Western world, influencing numerous modern languages.
Roman architecture and engineering achievements were monumental. They perfected the arch, vault, and dome, constructing enduring structures like the Colosseum, Pantheon, and aqueducts. These engineering marvels not only showcased Roman ingenuity but also served practical purposes, from public entertainment to water supply.
The French Revolution, which began in 1789, was a period of radical social and political upheaval in France. It marked the decline of absolute monarchies, the rise of secular and democratic republics, and the eventual rise of Napoleon Bonaparte. This revolutionary period is crucial in understanding the transition from feudalism to modernity in Europe.
For more information, visit-www.vavaclasses.com
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Artificial Intelligence (AI) technologies such as Generative AI, Image Generators and Large Language Models have had a dramatic impact on teaching, learning and assessment over the past 18 months. The most immediate threat AI posed was to Academic Integrity with Higher Education Institutes (HEIs) focusing their efforts on combating the use of GenAI in assessment. Guidelines were developed for staff and students, policies put in place too. Innovative educators have forged paths in the use of Generative AI for teaching, learning and assessments leading to pockets of transformation springing up across HEIs, often with little or no top-down guidance, support or direction.
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Operation “Blue Star” is the only event in the history of Independent India where the state went into war with its own people. Even after about 40 years it is not clear if it was culmination of states anger over people of the region, a political game of power or start of dictatorial chapter in the democratic setup.
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3. DEFINITION
•
Sick sinus syndrome describes
dysfunction of the intrinsic
pacemaker of heart, the
sinoatrial node. As a result, the
cardiac rhythm becomes
abnormal characterized by:
Sinus bradycardia…slow
heart rate
Tachycardia…fast heart rate
Bradycardia-tachycardia…
alternating slow and fast
heart rhythms.
4. MECHANISM
Normally, the sinuatrial node produces a steady pace of regular
electrical impulses. In sick sinus syndrome, these signals are
abnormally paced. A person with sick sinus syndrome may have
heart rhythm that are too fast, too slow, punctuated by long pause or
.
an alternating combination of all of these rhythm problems
TYPES AND CAUSES
Types of sick sinus syndrome and their causes include:
SINUS BLOCK:
Electrical signals move too slowly through sinus node, causing
abnormally slow heart rate.
SINUS ARREST:
The sinus node activity pauses.
TACHYCARDIA-BRADYCARDIA SYNDROME:
The heart rate alternates between abnormally fast and slow
rhythms, often with a long pause (asystole) between heart beats.
5. It may be brought on by use of drugs like digitalis, calcium channel
blockers, beta blockers and antiarrhythmics.
Sick sinus syndrome usually occurs in people older than 50,in whom
the cause is often a non specific, scar like degeneration of the heart’s
conduction system like amyloidosis,sarcoidosis,chagas disease and
cardiomyopathies.
• In children, a common cause of sick sinus syndrome is heart
surgery especially on heart chambers.
• In coronary artery disease, high blood pressure, aortic and
mitral valve diseases may be associated with sick sinus
syndrome.
6. HEART BLOCKS
•
Interference with the conduction process of the heart causes the
phenomena called heart block or atrioventricular block.
Heart block is classified according to the level of impairment:
First Degree Heart Block
Second Degree Heart Block
Third Degree (complete) Heart Block
FIRST DEGREE HEART BLOCK:
First degree heart block or first degree atrioventricular block is
defined as prolongation of the PR interval on the ECG to more than
200msec.First degree heart block is considered “marked” when the
PR exceeds 300msec.While the conduction is slowed, there are no
missed beats.
7. MECHANISM
With first degree atrioventricular (AV) block every atrial impulse is
transmitted to the ventricles ,resulting in regular ventricular rate.
This type of AV block can arise from delays in the conduction
system in the AV node itself, the His Purkinjie system or a
combination of both. Overall, dysfunction at the AV node is much
more common than dysfunction at the His Purkinjie system.
If the QRS complex is of normal width and morphology on the ECG
than the conduction delay is almost always at the level of the AV
node.
If however, the QRS demonstrates a bundle branch morphology
than the level of the conduction delay is often localized to His
Purkinjie system
8. CAUSES
Following are the most common causes of first degree
(atrioventricular block).
Intrinsic AV nodal disease.
Enhanced vagal tone.
Acute MI particularly acute inferior wall MI.
Myocarditis.
)
Electrolyte disturbances (e.g hypokalemia, hypomagnesemia
Drugs(esp.those drugs that increase the refractory time of the AV
node, there by slowing conduction).
Drugs that most commonly causes first degree AV block include
following:
Class 1c antiarrhythmias (e.g Quinidine, procainamide,
disopyramide)
Class2c antiarrhythmias (e.g Flecainide, encainide)
Class3c antiarrhythmias (Beta blockers)
9. SECOND DEGREE HEART BLOCK
Second degree heart block implies intermittent conduction, some impulses
from the atria are conducted to ventricles whereas others are not.
CAUSES:
Inferior wall MI.
Drugs like digitalis, beta blockers, calcium antagonists.
Hyperkalemia in well trained athelets during sleep.
Myocarditis
TYPES OF SECOND DEGREE HEART BLOCK:
There are three types of second degree heart block.
Mobitz type 1
Mobitz type 2
2:1 block
10. MOBITZ TYPE 1
In this condition, there is progressive lengthening of successive PR
intervals followed by a dropped beat (non conducted P). This is also
known as wenckebach’s phenomenon.
In this AV block, there is conduction defect in AV node and AV
conduction time (PR interval) progressively lengthens before
blocked beat pulse is clinically irregular.
Prognosis is good in first degree and in Mobitz type 1,since reliable
alternative pacemaker arise from AV junction below the block, if
complete heart block develops.
Site of block is AV node. QRS complex is normal in morphology
because there is no delay in interventricular depolarization .
11. MOBITZ TYPE 2
In this condition, the PR interval of the conducted impulses remain
constant but some P waves are not conducted (i.e more P waves
than QRS complexes)
Site of block is infranodal in location and QRS complexes are wide.
Mobitz type 2 AV block is abrupt and is not preceded by
lengthening of AV conduction time. It is usually due to block within
the bundle of His.
Mobitz 2 block is almost always due to organic heart disease, in
case it proceeds to complete heart block.
2:1 BLOCK
It may represent as either type for type 2 AV block in which there
are two P waves to each QRS complex and therefore, called 2:1
block.
If PR interval is prolonged and QRS complex is narrow then it is
type1 second degree heart block.
If PR interval is normal then QRS complex is wide, it is called
second degree AV block
12.
THIRD DEGREE(COMPLETE) HEART
BLOCK
Third degree heart block is an advanced form of block. No impulse
from atria reaches the ventricles. Cardiac action is maintained by an
escape rhythm.
CAUSES:
CONGENITAL
ACQUIRED
Idiopathic fibrosis
MI/Ischemia
INFECTIONS: Infective endocarditis, Chagas disease, Lyme’s
disease.
INFILTRATION: Sarcoidosis, Amyloidosis, Neoplasia
DRUGS: Digioxin, Beta blockers, Amiodaron
TRAUMA: Cardiac surgery.
CONNECTIVE TISSUE DISEASE: SLE,RA
13. MECHANISM
Escape rhythm arising in the bundle of His produces narrow QRS
complex at the rate of 50-60beats/min.Escape rhythm arising below
the His bundle produces broad complexes and at rate of 1540beats/min.
It is more advanced form of heart block due to lesion at the level of
bundle of His or more often distally in Purkinjie system and
associated with bilateral bundle branch block. QRS complex is wide
and ventricular rate is slower. Transmission of atrial pulses through
AV node is completely blocked and a ventricular rate, usually less
than 45beats/min.
In chronic complete heart block, pulse is slow (30-40min) regular
and does not vary with exercise
14. Stokes-Adams Attacks
Episodes of ventricular asystole may occur during periods of
transition from partial to complete heart block lasting several
seconds to minutes. These episodes may cause cardiac syncope
called Stokes-Adams attacks.
These attacks often occur without warning, there is rapid loss of
consciousness and pt. may fall.
Convulsions may occur, if heart does not begin to beat within about
.
10sec and death will result, if arrest is prolonged
16. BUNDLE BRANCH BLOCK
In normal heart, each electrical impulses from the atria is
conducted through the AV node to the bundle of His, from which it
is transmitted to the ventricles by the right and left bundle branches.
TYPES
Left bundle branch block
Right bundle branch block
17. LEFT BUNDLE BRANCH BLOCK
In LBBB, left bundle branch no longer conducts electrical activity. The
electrical impulse thus enters right bundle branch block and is carried to
right ventricle. From there, it finally spreads to left ventricle. The two
ventricles no longer receive the electrical impulse simultaneously. First, the
Rt. ventricle receives the electrical impulse, then left.
CAUSES
Hypertrophy, dilatation or fibrosis LV myocardium.
Ischemic heart disease.
Cardiomyopathies.
Advanced valvular heart disease.
Toxic, inflammatory changes.
Hyperkalemia.
Digitalis toxicity.
Degenerative disease of conducting system (Lenegre disease).
18. TYPES OF LBBB
•
The LBBB can be divided into two fascicles:
Anterior Fascicles.
Posterior Fascicles.
Conduction system is composed of three fascicles:
Right Bundle Fascicle.
Left Anterior Fascicle.
Left Posterior Fascicle.
19. RIGHT BUNDLE BRANCH BLOCK
RBBB results from a defect in heart’s electrical conducting system.
There is a delay in or failure of transmission of electrical impulses
down the right bundle of heart. As a result , the Rt. Ventricle
depolarizes by means of cell- to- cell conduction. These impulses
spread more slowly than usual from inter ventricular septum to Rt.
Ventricle.This delay in conduction results in characteristic ECG
pattern, which is wide and notched QRS. Although conduction down
the Rt. branch is delayed ,conduction down the Lt. branch is normal.
As a result, the interventricular septum and Lt. ventricle in normal
fashion.
TYPES
There are two types.
Complete RBBB.
Incomplete RBBB.
20. CAUSES OF RBBB
Following are the causes of RBBB.
After repair of VSD.
After Rt.ventriculotomy.
Right Venticular hypertrophy
Ebstein’s anomaly.
.
.
Large ASD or AV cushion defect
Right ventricular dysplasia.
Brugada syndrome.
Congenital absence or atrophy of bundle branch.
After CABG and in transplanted heart.
21. HEMIBLOCK
DEFINITION:
When there is blockage in anterior or posterior division of left
bundle branch, it is called hemiblock.
BIFASCICULAR BLOCK:
RBBB plus left anterior or posterior hemiblock.
In bifascicular and trifascicular block there are more chances to
progress to complete heart block.
TRIFASCICULAR BLOCK:
RBBB + Hemiblock + First degree heart block.