The document discusses the anatomy and clinical features of spinal fractures. It begins with the anatomy of the vertebral column and its supporting ligaments. It then discusses the classification, mechanisms of injury, and clinical features of spinal fractures. Diagnosis involves history, physical exam including neurological exam, and imaging studies like x-rays, CT scans, and MRI to identify fractures and spinal cord injuries. Management aims to prevent secondary injury through immobilization of the spine.
The presentation is for the use of Physiotherapy students. It covers a brief introduction, classification, clinical features and general principles of management.
Presentation of common upper limb fractures and dislocations. Covering all the injuries from many sides (Definition - Classification - Mechanisms of injury - Clinical features - Radiological studies - Management - Complications)
The presentation is for the use of Physiotherapy students. It covers a brief introduction, classification, clinical features and general principles of management.
Presentation of common upper limb fractures and dislocations. Covering all the injuries from many sides (Definition - Classification - Mechanisms of injury - Clinical features - Radiological studies - Management - Complications)
Spinal stenosis is a degenrative spine disorder in which the AP and transverse diameter are decreased causing neural compression and symptoms of chronic & acute nerve compression
Lubar Arthritis : Lumbar Stenosis by Pablo Pazmino, MDPablo Pazmino
This video explains Lumbar Stenosis. When arthritis begins to encroach around the spinal cord and neural elements this is called Lumbar Stenosis. This video highlights the history, epidemiology, and treatment options both conservative and surgical. If you or someone you know needs to be seen in regards to Lumbar Stenosis/Arthritis feel free to look us up online www.beverlyspine.com or www.santamonicaspine.com OR call toll free 1-8SPINECAL-1
Spinal cord injuries complete topic about it and how to make good rehabilitation for the patient with spinal cord injuries .
wish it help people
my pleasure :)
Mostafa shakshak
Radiological evaluation of Lower Limb in acute ED setting !!Runal Shah
Radiological evaluation of Lower Limb in acute ED setting !!
How to evaluate lower limb injuries in ED by primary look out... How to assess simple bony injuries ! A simple radiological approach for ED physicians..
Spine and extremity injuries are common among people of all ages and can have a significant impact on mobility and quality of life. This PowerPoint presentation provides a comprehensive overview of spine and extremity injuries, including the causes, symptoms, and treatment options.
Through powerful images and personal stories, we showcase the impact of spine and extremity injuries on individuals, families, and communities. We highlight the challenges of accessing healthcare and rehabilitation services, particularly in low-resource settings, and the importance of early intervention and treatment.
The presentation provides detailed information about the various types of spine and extremity injuries, including fractures, dislocations, and soft tissue injuries. We also discuss the diagnostic procedures, including imaging tests and physical exams, and the treatment options, such as surgery, physical therapy, and pain management.
In addition, we explore the efforts being made to prevent and manage spine and extremity injuries. We highlight the importance of safety precautions, such as proper equipment use and ergonomic work practices, and the role of rehabilitation services in promoting recovery and restoring function.
Through this PowerPoint presentation, we aim to raise awareness about spine and extremity injuries and the importance of early diagnosis and treatment. We showcase the latest research and innovations in injury prevention and treatment, and the importance of collaboration and partnership to address the disease.
We urge the audience to take action in the fight against spine and extremity injuries, whether it be through spreading awareness, supporting organizations working on the ground, or advocating for policy change. Let us come together to create a world where everyone has access to the care and support they need to recover from spine and extremity injuries and live healthy, fulfilling lives.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
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Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
1. Done by malika hameed
Supervision by dr - Anwer Naif
Group D
Spine
fractur
e
2. Clinical Anatomy
• Vertebral Column:
– Extends from skull
to the pelvis
– 33 total vertebrae:
• Superiorly: 24
individual vertebrae
(separated by
intervertebral discs)
• Inferiorly: 9 fuse to
form 2 composite
bones
– Sacrum (5)
– Coccyx (4)
3. Clinical Anatomy
• Vertebral Column:
– Functions:
1. Transmits weight
of the trunk to the
lower limbs
2. Surrounds/protect
s spinal cord
3. Attachment point
for the ribs and
muscles of neck
and back
8. • Intervertebral Discs:
– 23 intervertebral discs
– No disc between skull and C1 or between
C1-C2
– Discs are thickest in the lumbar vertebrae
and cervical regions (enhances flexibility)
• Functions:
– Shock absorbers
• walking, jumping, running
– Allow spine to bend
– At points of compression, the discs flatten
out and bulge out a bit between the
vertebrae
Vertebral Anatomy
10. Clinical Anatomy
• Major Supporting
Ligaments
– Anterior Longitudinal
Ligament
– runs vertically along
anterior surface of
vertebral bodies
• Neck - Sacrum
• Attaches strongly to both
vertebrae and
intervertebral discs (very
wide)
• Prevents back
hyperextension
11. Clinical Anatomy
• Major Supporting
Ligaments
– *Posterior Longitudinal
Ligament
– - runs vertically along
posterior surfaces of
vertebral bodies
• Narrower, weaker
• Attaches to
intervertebral discs
• Prevents hyperflexion
12. • Major Supporting Ligaments
– Ligamentum Flavum - strong
ligament that connects the
laminae of the vertebrae
• Protects the neural elements
and the spinal cord
• Stabilizes the spine to prevent
excessive vertebral body
motion
• Strongest of the spinal
ligaments
• Forms the posterior wall of the
spinal canal with the laminae
• Stretches with forward
bending / recoils in erect
position
Clinical Anatomy
13. Clinical Anatomy
sporting Ligaments
– Intertransverse Ligament
- located between the
transverse processes
• Cervical region:
consist of a few
irregular, scattered
fibers
• Thoracic region:
rounded cords
connected with deep
muscles of the back
• Lumbar region: thin
and membranous
15. Clinical Anatomy
Supporting Ligaments
– Supraspinal Ligament
-connects together the
apexes of the spinous
processes
• Extends from 7th
cervical vertebra to
sacrum
• Strong fibrous cord
• At points of attachment
(tips of the spinous
processes) fibrocartilage
is developed in the
ligament
Supraspinal
Ligament
18. Significance
Unstable
if middle column + either Anterior or Posterior column
is damaged
Rupture of interspinous ligament is :
- associated with avulsion of spinous process
- Unstable spine
- Further flexion increases neurological injury
19. • Cervical 40%
• Thoracic 10%
• Lumbar 3%
• Dorso lumbar
35%
Incidence :-
-More in males 20-40 (more
exposure to mechanism of
injury) .
Most frequently injured spinal regions
C5-C7 (Mid cervical)
C1-C2 (Upper cervical)
T12-L2 (Dorso-lumbar)
22. :
A. Hyper-flexion trauma:
-Fall in bent position.
-Deceleration.
II-indirect Trauma
-Wedge fracture
-Stable
-No spinal cord
injury
23. B- Hyper-extension injuries
-sudden acceleration
Blow on forehead-
INCLUDE
hangman’s #
# of arch of atlas and axis
ant. Longitudinal tear
-stable except hangman fracture-
-Spinal cord may be damage
24. C- compression trauma
-fall from height on foot .
-Fall on head
-fall something on head
-burst fracture
-Stable #
-No spinal nerve
injury
25. D-Shearing (specially rotation)
-rotation cause ligaments Damage
, usually associated with flexion.
As:
-Fall from height with the body twisted
Or
Weight fall asymmetrically onto the back
**Wedge shape # with shearing
of posterior ligament.
spinal usually affect
unstable #
28. I- according to morphology
1. Wedge compression fracture:
Hyper-flexion trauma crushes the vertebral body into the shape of a wedge.
2. Fracture dislocation:
Commonly there is damage to the spinal cord and nerve roots.
3. Dislocation:
Pure dislocation without fracture is common in cervical vertebrae because its
articular processes are rather horizontal.
4. Comminuted (burst) fracture:
This is an uncommon injury due to vertical compression of the straight spine
.
5. Avulsion fractures of transverse and spinous
processes:
Not accompanied by neurological injury and require no special
treatment.
29. II- according to stability
1-Stable fractures: Have intact posterior
ligaments, e.g., wedge compression,
comminuted and avulsion fracture of the
transverse and spinous processes.
2-Unstable fractures: Have torn posterior
ligaments and are liable to injure the
cord and nerve roots, these include fracture
dislocation and pure dislocations
31. classification
I- According to injury
1-Primary injuries
Occur at time of trauma.
2-Secondary injuries
Occur later due to:
-Swelling
-Ischemia
-Movement of bony fragments
33. II – According to neurology DAMAGE
Complete Cord Lesions –
Death --aboveC4
-Quadriplegia below C4
-Paraplegia thoracic or lumbar level
Incomplete Cord Lesions -
mixed loss
- Anterior sc syndrome
- Posterior sc syndrome
- Central cord syndrome
- Brown sequard’s syndrome
- Cauda equina syndrome
34. Incomplete cord lesion:
1-Central cord syndrome
Flaccid weakness of arm and spastic
weakness of the leg
2- anterior cord syndrome
complete loss of muscle strength below the
level of injury
loss pain and temperature
3-brown sequard syndrome
Motor deficit on side of lesion while sensory
deficit on the other side
4-Posterior cord syndrome
Loose of light touch and
proprioception
36. History
• Mechanism of injury
• Position of the patient when found
• Transient motor or sensory loss
• Paradoxical breathing
• Seat belt
37. Physical examination
• A- General examination:
- -Vital sign
- -Look for missed injury
- -look for associated fracture
38. B-Local examination:
Inspection:
Abrasion - hematoma- deformity
Soft tissue swelling and bruising
Palpation :
Point of spinal tenderness
Gap or Step-off
Spasm of associated muscles
DON’T MOVE
C- neurological
examination
1-Dermatome and
myotome.
2- superficial and deep
reflex .
39. Is the patient awake or
“unexaminable”?
• What’s the difference ?
– Awake
• ask/answer question
• pain/tenderness
• motor/sensory exam
– Not awake
• you can ask (but they won’t answer)
• can’t assess tenderness
• no motor/sensory exam
OW!
------
41. Neurogenic Shock
• Temporary loss of autonomic function of the
cord at the level of injury
– results from cervical or high thoracic injury
• Presentation
– Flaccid paralysis distal to injury site
– Loss of autonomic function
• hypotension
• vasodilatation
• loss of bladder and bowel control
• loss of thermoregulation
• warm, pink, dry below injury site
• bradycardia
42. 42
Neurogenic Hypovolemic
Etiology Loss of sympathetic
outflow
Loss of blood volume
Blood
pressure
Hypotension Hypotension
Heart rate Bradycardia Tachycardia
Skin
temperature
Warm Cold
Urine
output
Normal Low
Comparison of neurogenic and
hypovolemic shock
43. Cervical Spine Imaging Options
– Plain films
• AP, lateral and open mouth view
– Optional: Oblique and Swimmer’s
– CT
• Better for occult fractures
– MRI
• Very good for spinal cord, soft tissue and
ligamentous injuries
– Flexion-Extension Plain Films
• to determine stability
45. Adequacy
• Must visualize entire C-spine
• A film that does not show the
upper border of T1 is
inadequate
• Caudal traction on the arms
may help
• If can not, get swimmer’s view
or CT
47. Alignment
• The anterior vertebral line,
posterior vertebral line, and
spinolaminar line should
have a smooth curve with
no steps or discontinuities
• Malalignment of the
posterior vertebral bodies is
more significant than that
anteriorly, which may be
due to rotation
• A step-off of >3.5mm is
significant anywhere
48. Lateral Cervical Spine X-Ray
• Anterior subluxation of
one vertebra on another
indicates facet
dislocation
–< 50% of the width of a
vertebral body
unilateral facet
dislocation
–> 50% bilateral facet
dislocation
50. Disc
• Disc Spaces
– Should be
uniform
• Assess spaces
between the
spinous
processes
51. Soft tissue
• Nasopharyngeal space
(C1)
– 10 mm (adult)
• Retropharyngeal space
(C2-C4)
– 5-7 mm
• Retrotracheal space
(C5-C7)
– 14 mm (children)
– 22 mm (adults)
52. AP C-spine Films
• Spinous processes
should line up
• Disc space should be
uniform
• Vertebral body height
should be uniform.
Check for oblique
fractures.
53. Open mouth view
• Adequacy: all of: all of
the dens andthe dens and
lateral borders oflateral borders of
C1 & C2C1 & C2
• Alignment: lateral: lateral
masses of C1 andmasses of C1 and
C2C2
• Bone: Inspect dens
for lucent fracture
lines
54. CT Scan
• Thin cut CT scan should
be used to evaluate
abnormal, suspicious or
poorly visualized areas
on plain film
• The combination of plain
film and directed CT scan
provides a false negative
rate of less than 0.1%
55. MRI
• Ideally all patients
with abnormal
neurological
examination
should be
evaluated with MRI
scan
56. Management
• Primary Goal
– Prevent secondary injury
• Immobilization of the spine begins in the initial
assessment
– Treat the spine as a long bone
• Secure joint above and below
– Caution with “partial” spine splinting
58. Goal of spine trauma care
• Protect further injury during evaluation and
management
• Identify spine injury or document absence of
spine injury
• Optimize conditions for maximal neurologic
recovery
59. Goal of spine trauma care
• Maintain or restore spinal alignment
• Minimize loss of spinal mobility
• Obtain healed & stable spine
• Facilitate rehabilitation
60. Pre-hospital management
1- ensure the ventilation and circulation
:2- CAREFUL transportation OF PATIENT if
we Suspect Spinal Injury
When should we Suspect Spinal Injury
-High speed crash
-Unconscious
-Multiple injuries
-Neurological deficit
-Spinal pain/tenderness
61. • PROTECTION PRIORITY
• Detection Secondary
• Up to 15% of spinal injuries have a second
(possibly non adjacent) fracture elsewhere in the
spine
• Ideally, whole spine should be immobilized in
neutral position on a firm surface
62.
63.
64.
65.
66.
67. Clinical assessment
• Advance Trauma Life Support (ATLS)
guidelines
• Primary and secondary surveys
• Adequate airway and ventilation are the
most important factors
• Supplemental oxygenation
• Early intubation is critical to limit secondary
injury from hypoxia
70. Stable # :
-no reduction
-immobilization with cervical
collar or
plaster jacket for 4-6 weeks .
71. Unstable #
Unstable cervical #
1- reduction
-close reduction : by skull traction –skin traction
or manipulation
Open reduction
2- fixation:
By cervical collar or halo vest(6-12 weeks)
or internal fixation
or post fusion.
72.
73. Unstable lumbar #
1- reduction:
By gradual postural reduction by use of pillow or
sand bags under appropriate spinal level for 12 weeks
or
Open reduction
2- immoilization:
By plaster jacket or
By internal fixation
74.
75.
76.
77. Local management
-Restore alignment
-Decompress
neural damage
-Stabilize the spine
by fixation and
fusion
-Allow early
mobilization
-Restore alignment by traction
Decompress not retard - recovery
or decrease further injury
-No need for stabilization most
injury stable
-Mobilization achieved by active
physiotherapy in confined bed pt
78. General management
1- care of skin:
-Trophic ulcer
-Skin keep dry by regular washing and powder
2- care of bladder:
Decompress by catheter-
Antibiotics if infection occurred
3- care of bowel:
By evacuation by enema and giving low residual diet
4- care of muscles and joints:
By passive movement twice daily
79. complication
• 1- Spinal cord and/or root injury including roots
of cauda spina
• 2- Shock may be:
• a- Spinal shock
• - Sympathatic affection leading to loss of
vascular tone & bradycardia
• - loss of muscle tone ) venous pooling &
hypovolemia
• b- Hemorrhage lead to true hypovolemia
81. Jefferson Fracture
• Burst fracture of C1 ring
• Unstable fracture
• Increased lateral ADI on
lateral film if ruptured
transverse ligament and
displacement of C1 lateral
masses on open mouth view
• Need CT scan
83. Burst Fracture
• Fracture of C3-C7 from
axial loading
• Spinal cord injury is
common from posterior
displacement of fragments
into the spinal canal
• Unstable
85. Flexion Teardrop Fracture
• Flexion injury causing a
fracture of the
• anteroinferior portion
of the vertebral body
• Unstable because
usually associated with
posterior ligamentous
injury
86. Bilateral Facet Dislocation
• Flexion injury
• Subluxation of dislocated
vertebra of greater than
½ the AP diameter of the
vertebral body below it
• High incidence of spinal
cord injury
• Extremely unstable
87. Hangman’s Fracture
• Extension injury
• Bilateral fractures of
C2 pedicles
(white arrow)
• Anterior dislocation of
C2 vertebral body
(red arrow)
• Unstable
88.
89. Odontoid Fractures
• Complex mechanism of injury
• Generally unstable
• Type 1 fracture through the tip
– Rare
• Type 2 fracture through the base
– Most common
• Type 3 fracture through the base into body
of axis
– Best prognosis
90. Odontoid fracture Type I
fracture in superior tip of
the odontoid.
potentially unstable.
It is a relatively rare
fracture.
94. • This is an example of dens type III fracture.
The first image is an odontoid view, which
shows the fracture line extending beyond the
base of the dens.
• The second image is a CT that confirms the
fracture in the body of C2
95. Question
Which of the following fractures is caused by
hyperextension?
Jefferson fracture.
Hangman's fracture.
Teardrop fracture.