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Group D
Talipes Equinovarus
TalipesEquinovarus
*Talipes: derived from talus ( L = ankle bone )
and pes ( L = foot ).
* Equinovarus: one of the several different
deformities of talipes and means that the
entire hindfoot is in varus and the mid &
forefoot are adducted and supinated.
* Others:Talipes calcaneus &Talipes valgus.
Incidence
* 1-2 every 1000 live births.
* Boys : Girls → 2 : 1 .
* Sporadic but may be autosomal
dominant trait.
* Bilateral in 50% of patients or in one
third of cases.
Causes/Mechanisms
1- Genetic defect : primary germ plasm defect in
the talus causes continued planter flexion and
inversion of this bone and soft tissue
changes.
2- Form of arrested development.
3- Neuromuscular disorder : primary soft tissue
abnormalities within neuromuscular units
cause secondary bony changes , e.g : neural
tube defect.
What part of the foot is
affected?
Clubfoot primarily affects three bones:
calcaneus, talus and navicular.
The deformity can affect the growth of the entire
foot to some degree so other bones may be
involved as well.
PathologicalAnatomy
①Talus :
- Neck : downwards , deviated medially.
- Body : rotated slightly outwards in relation to
calcaneus and ankle mortise.
② Calcaneus :
- Posterior part is held close to fibula by a tight
calcaneo-fibular ligament and tilted into
equinus and medially rotated beneath the
ankle.
③ Navicular & the entire forefoot :
- Shifted medially and rotated into supination.
④ Metatarsals : adducted & deviated at
tarsometatarsal joints.
ClinicalPicture
1- Foot is turned & twisted inwards , so the sole
faces posteromedially with high medial arch (
cavus ) & smaller than the other normal foot.
2- Ankle is in equinus.
3- Forefoot is adducted & supinated.
4- Skin & soft tissues of the calf &medial side of
the foot are short & underdeveloped.
5-The heel is small & high.
6- Deep creases appear posteriorly & medially.
Examination
* In infants :
examine associated disorders as congenital hip
dislocation & spina bifida.
* 3 Basic components of club foot :
equinus , varus , adduction , & may be cavus.
RoentgenographicEvaluation
* Before , during & after treatment.
*To assess progress after treatment.
* 3 views:
- AP with foot 30° planter flexion & tube
angled 30°.
- Stress dorsiflexion lateral view of both heels.
- Lateral standing view : in older children.
* Important angels :
1-Talocalcaneal angle.
2-Talo-1st metatarsal angle.
APfilm
- Line is drawn through the long axis of talus
parallel to its medial border & through the
calcaneus parallel to its lateral border ,
normally cross at angle 20°- 40° “ Kite’s angle
“, in club foot , the two lines may be parallel.
LateralFilm
* Foot is forced dorsiflexion.
* Line is drawn through the mid-longitudinal
axis of talus & lower border of calcaneus ,
normally the angle is 40°.
* If the angle is less than 20° >>> club foot.
* Angles normally changes with age.
* Club foot can’t be dorsiflexed , but if so , it will
be broken at the midtarsal level producing
“Rocker – Bottom deformity”.
Treatment
- Aim : to produce & maintain a plantigrade ,
replase may occur in neuromuscular disorders
babies.
① Non-operative : the initial treatment.
- Repeated manipulation & adhesive strapping.
-Weekly serial manipulation & light plaster cast
during the first 6 weeks of life followed by
manipulation & casting every the other week
untill the foot clinically & roentgenohraphic
corrected.
*With experience , predict which feet will respond to non-
surgical treatment.
*The more the rigid the initial deformity , more likely surgical
treatment.
*The order of correction :
1- Correction of forefoot adduction : rotational alignment
with hindfoot , by ↑ supination deformity.
2- Correction of hindfoot & forefoot for varus & supination by
keeping fulcrum on the lateral side of head of talus.
3- Correction of hindfoot equinus by bringing the heel down
& dorsiflexing the ankle & percutaneous tendo Achillis
lengthening.
*The aim of the order is to prevent “Rocker Bottom Deformity”
by dorsiflexing the foot from the hindfoot rather than
midfoot , but if occurs, forefoot placed back in planter
flexion , casting & surgery is important here.
* Casting by Kite , modified by Lovell & Hancock.
* Manipulation & casting success rate is 15-80%.
* Resistant cases declare after 8-12 weeks of serial
manipulation & strapping , so early surgery or continued
conservative treatment is indicated.
* Delaying surgery untill child walking >>> surgery is easier &
maintenance of the correction. It is suitable for severe ,
rigid deformities.
* Less severe cases >>> operation at 6 months.
OperativeTreatment②
OperativeTreatment
* Indication :
1- No response to conservative treatment often
in children with significant rigid deformity.
2- Deformity has recurred.
3-The forefoot has been corrected by
conservative treatment but hindfoot remains
fixed in both varus & equinus.
* Surgical treatment is according to the age of
child & deformity to be corrected.
* Objectives :
1- Complete release of joint tethers.
2- Lengthening of tendons.
Incisions
1- Posteromedial incision >>> Turco.
2- Posterior curved transverse , extended
anteriorly on both sides medial & lateral >>>
Cincinatti – Crawford.
3- Posterolateral with separate curved medial
incision >>> Caroll.
*To correct equinus :
1- Achillis tendon & tibialis posterior tendon are
lengthened through Z-divisions.
2- Posterior capsules of ankle & subtalar joints are
divided.
*To correct cavus :
1- Release contractures around talonavicular &
calcanocuboid joints.
2- Origin of intrinsic muscles & planter fascia from
calcaneum are divided.
*The foot , in its corrected position , is
immobilized in a plaster cast.
* Kirshner wires >>> across talonavicular &
subtalar joint.
*Wires & cast are removed at 6-8 weeks.
* After immobilization , hobble boots “ Dennis
Browne “.
* Stretching exercises are continued.
Dennis Browne “
DAFOs for Clubfoot:
designed for soft, comfortable repositioning of
clubfoot or maintaining post-surgical positions
Lateorrelapsedclubfoot
* Late >>> comes with secondary bony changes.
* Relapsed >>> scarring of previous surgery.
*Youngs ( 4-7 ) years >>> calcaneo-cuboid fusion or
cuboid enucleation ( Dilwyn Evans ).
* Calcaneal osteotomies >>> for varus.
*Tendon transfer.
* Circular external fixator ( Ilizarov method ) >>>
gradual correction.
Metatarsusadductus
*Varies from slightly curved forefoot to mild club
foot.
* Management :
- 90% improve spontaneously.
- corrective casts followed by straight last shoes.
- Extensive capsulectomy of tarsometatarsal
joints. No splintage to avoid early degenerative
arthritis.
- Dilwyn Evans procedure + Basal metatarsal
osteotomy.
TalipesCalcaneovalgus
Idiopathic club foot

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Idiopathic club foot

  • 1. Done by malika hameed Group D
  • 3. TalipesEquinovarus *Talipes: derived from talus ( L = ankle bone ) and pes ( L = foot ). * Equinovarus: one of the several different deformities of talipes and means that the entire hindfoot is in varus and the mid & forefoot are adducted and supinated. * Others:Talipes calcaneus &Talipes valgus.
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  • 5. Incidence * 1-2 every 1000 live births. * Boys : Girls → 2 : 1 . * Sporadic but may be autosomal dominant trait. * Bilateral in 50% of patients or in one third of cases.
  • 6. Causes/Mechanisms 1- Genetic defect : primary germ plasm defect in the talus causes continued planter flexion and inversion of this bone and soft tissue changes. 2- Form of arrested development. 3- Neuromuscular disorder : primary soft tissue abnormalities within neuromuscular units cause secondary bony changes , e.g : neural tube defect.
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  • 10. What part of the foot is affected? Clubfoot primarily affects three bones: calcaneus, talus and navicular. The deformity can affect the growth of the entire foot to some degree so other bones may be involved as well.
  • 11. PathologicalAnatomy ①Talus : - Neck : downwards , deviated medially. - Body : rotated slightly outwards in relation to calcaneus and ankle mortise. ② Calcaneus : - Posterior part is held close to fibula by a tight calcaneo-fibular ligament and tilted into equinus and medially rotated beneath the ankle.
  • 12. ③ Navicular & the entire forefoot : - Shifted medially and rotated into supination. ④ Metatarsals : adducted & deviated at tarsometatarsal joints.
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  • 15. ClinicalPicture 1- Foot is turned & twisted inwards , so the sole faces posteromedially with high medial arch ( cavus ) & smaller than the other normal foot. 2- Ankle is in equinus. 3- Forefoot is adducted & supinated. 4- Skin & soft tissues of the calf &medial side of the foot are short & underdeveloped. 5-The heel is small & high. 6- Deep creases appear posteriorly & medially.
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  • 17. Examination * In infants : examine associated disorders as congenital hip dislocation & spina bifida. * 3 Basic components of club foot : equinus , varus , adduction , & may be cavus.
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  • 19. RoentgenographicEvaluation * Before , during & after treatment. *To assess progress after treatment. * 3 views: - AP with foot 30° planter flexion & tube angled 30°. - Stress dorsiflexion lateral view of both heels. - Lateral standing view : in older children.
  • 20. * Important angels : 1-Talocalcaneal angle. 2-Talo-1st metatarsal angle.
  • 21. APfilm - Line is drawn through the long axis of talus parallel to its medial border & through the calcaneus parallel to its lateral border , normally cross at angle 20°- 40° “ Kite’s angle “, in club foot , the two lines may be parallel.
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  • 23. LateralFilm * Foot is forced dorsiflexion. * Line is drawn through the mid-longitudinal axis of talus & lower border of calcaneus , normally the angle is 40°. * If the angle is less than 20° >>> club foot. * Angles normally changes with age. * Club foot can’t be dorsiflexed , but if so , it will be broken at the midtarsal level producing “Rocker – Bottom deformity”.
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  • 25. Treatment - Aim : to produce & maintain a plantigrade , replase may occur in neuromuscular disorders babies. ① Non-operative : the initial treatment. - Repeated manipulation & adhesive strapping. -Weekly serial manipulation & light plaster cast during the first 6 weeks of life followed by manipulation & casting every the other week untill the foot clinically & roentgenohraphic corrected.
  • 26. *With experience , predict which feet will respond to non- surgical treatment. *The more the rigid the initial deformity , more likely surgical treatment. *The order of correction : 1- Correction of forefoot adduction : rotational alignment with hindfoot , by ↑ supination deformity. 2- Correction of hindfoot & forefoot for varus & supination by keeping fulcrum on the lateral side of head of talus. 3- Correction of hindfoot equinus by bringing the heel down & dorsiflexing the ankle & percutaneous tendo Achillis lengthening.
  • 27. *The aim of the order is to prevent “Rocker Bottom Deformity” by dorsiflexing the foot from the hindfoot rather than midfoot , but if occurs, forefoot placed back in planter flexion , casting & surgery is important here. * Casting by Kite , modified by Lovell & Hancock. * Manipulation & casting success rate is 15-80%. * Resistant cases declare after 8-12 weeks of serial manipulation & strapping , so early surgery or continued conservative treatment is indicated. * Delaying surgery untill child walking >>> surgery is easier & maintenance of the correction. It is suitable for severe , rigid deformities. * Less severe cases >>> operation at 6 months.
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  • 35. OperativeTreatment * Indication : 1- No response to conservative treatment often in children with significant rigid deformity. 2- Deformity has recurred. 3-The forefoot has been corrected by conservative treatment but hindfoot remains fixed in both varus & equinus. * Surgical treatment is according to the age of child & deformity to be corrected.
  • 36. * Objectives : 1- Complete release of joint tethers. 2- Lengthening of tendons.
  • 37. Incisions 1- Posteromedial incision >>> Turco. 2- Posterior curved transverse , extended anteriorly on both sides medial & lateral >>> Cincinatti – Crawford. 3- Posterolateral with separate curved medial incision >>> Caroll.
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  • 40. *To correct equinus : 1- Achillis tendon & tibialis posterior tendon are lengthened through Z-divisions. 2- Posterior capsules of ankle & subtalar joints are divided. *To correct cavus : 1- Release contractures around talonavicular & calcanocuboid joints. 2- Origin of intrinsic muscles & planter fascia from calcaneum are divided.
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  • 42. *The foot , in its corrected position , is immobilized in a plaster cast. * Kirshner wires >>> across talonavicular & subtalar joint. *Wires & cast are removed at 6-8 weeks.
  • 43. * After immobilization , hobble boots “ Dennis Browne “. * Stretching exercises are continued.
  • 45. DAFOs for Clubfoot: designed for soft, comfortable repositioning of clubfoot or maintaining post-surgical positions
  • 46. Lateorrelapsedclubfoot * Late >>> comes with secondary bony changes. * Relapsed >>> scarring of previous surgery. *Youngs ( 4-7 ) years >>> calcaneo-cuboid fusion or cuboid enucleation ( Dilwyn Evans ). * Calcaneal osteotomies >>> for varus. *Tendon transfer. * Circular external fixator ( Ilizarov method ) >>> gradual correction.
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  • 50. *Varies from slightly curved forefoot to mild club foot. * Management : - 90% improve spontaneously. - corrective casts followed by straight last shoes. - Extensive capsulectomy of tarsometatarsal joints. No splintage to avoid early degenerative arthritis. - Dilwyn Evans procedure + Basal metatarsal osteotomy.