Fracture diseases
Dr. Arhab Alshabi
Resident Orthopaedic Surgery
Definition:
it is the complication of fracture treatments due to
prolong immobilisation .
Principle of fracture treatment:
- anatomical reduction
- rigid fixation
-preservation of soft tissue
-Early Active Mobilisation
The fracture Diseases include:
- Musculoskeletal.
-Cardiovascular.
Musculoskeletal:
-muscle weakness, atrophy
- Soft tissue Contracture.
- Osteoporosis
- Joint Stiffness.
- Skin Atrophy
Cardiovascular:
- Increase the hart rate ,circulatory dysfunction.
- Orthostatic hypotension.
- venous Thromboembolism, pulmonary problem.
Muscle weakness:
- it is the most obvious effect of prolonged Immobilisation
,muscle at complete rest loss about 10-15 % of it’s strength
in each week.
- Half of normal strength loss within 3-5 weeks of
immobilisation .
- First muscle become weak and atrophy are those of lower
extremities .
- muscle strength can be maintain without loss or gain with
daily muscle contraction.
- Unfortunately the rate of recovery from weakness is slower
than rate of loss.
Muscle Atrophy :
-It is the loss of muscle mass.
normal muscle at rest loss half of their bulk after 2 months .
Soft tissue Contracture :
The connective tissue contain collagen fibbers that maintain
the muscle length if frequently stretching , but shorten if
immobilised which lead to contracture.
Disuse Osteoporosis:
Like connective tissue ,bone is dynamic tissue ,the constant
equilibrium is maintain between the bone formation and desorption of
bone density depend on force that act upon bone.
Joint stiffness:
- it’s limitation of movement , after periods of inactivity.
Skin atrophy :
- decrease circulation in skin due to pressure in immobilisation
patient then ischemia ,skin breakdown and ulcer .
Cardiovascular:
Prolonged immobilisation stimulate sympathetic nerve system
activity ,increase the heart rate ,less diastolic filling time ,shortened
systolic ejection end by circulatory dysfunction.
venous thromboembolism and pulmonary
problem :
Due to venous stasis in the leg following decrease contraction of
Gastrocnemius ,solus muscle the emboli formation.
may occur within 7-10 days of immobilisation.
characteristic by pain, tenderness, swelling, venous distension, in
the calf muscle and confirm by Doppler study .
Prevention:
-Proper fixation (rigid fixation)
-free pain early mobilisation and as soon as pt.'s can.
-Daily muscle stretching ,contraction for 20-30 seconds.
- ROM of adjacent joint below and above the fracture.
-leg stocking to prevent DVT
-anticoagulant ,antiplatelet drugs
-change patient position every 2H.
Thank You
Prepared by :Arhab Alshabi

Fracture disease

  • 1.
    Fracture diseases Dr. ArhabAlshabi Resident Orthopaedic Surgery
  • 2.
    Definition: it is thecomplication of fracture treatments due to prolong immobilisation .
  • 3.
    Principle of fracturetreatment: - anatomical reduction - rigid fixation -preservation of soft tissue -Early Active Mobilisation
  • 5.
    The fracture Diseasesinclude: - Musculoskeletal. -Cardiovascular.
  • 6.
    Musculoskeletal: -muscle weakness, atrophy -Soft tissue Contracture. - Osteoporosis - Joint Stiffness. - Skin Atrophy Cardiovascular: - Increase the hart rate ,circulatory dysfunction. - Orthostatic hypotension. - venous Thromboembolism, pulmonary problem.
  • 7.
    Muscle weakness: - itis the most obvious effect of prolonged Immobilisation ,muscle at complete rest loss about 10-15 % of it’s strength in each week. - Half of normal strength loss within 3-5 weeks of immobilisation . - First muscle become weak and atrophy are those of lower extremities . - muscle strength can be maintain without loss or gain with daily muscle contraction. - Unfortunately the rate of recovery from weakness is slower than rate of loss.
  • 8.
    Muscle Atrophy : -Itis the loss of muscle mass. normal muscle at rest loss half of their bulk after 2 months . Soft tissue Contracture : The connective tissue contain collagen fibbers that maintain the muscle length if frequently stretching , but shorten if immobilised which lead to contracture.
  • 10.
    Disuse Osteoporosis: Like connectivetissue ,bone is dynamic tissue ,the constant equilibrium is maintain between the bone formation and desorption of bone density depend on force that act upon bone. Joint stiffness: - it’s limitation of movement , after periods of inactivity. Skin atrophy : - decrease circulation in skin due to pressure in immobilisation patient then ischemia ,skin breakdown and ulcer .
  • 12.
    Cardiovascular: Prolonged immobilisation stimulatesympathetic nerve system activity ,increase the heart rate ,less diastolic filling time ,shortened systolic ejection end by circulatory dysfunction. venous thromboembolism and pulmonary problem : Due to venous stasis in the leg following decrease contraction of Gastrocnemius ,solus muscle the emboli formation. may occur within 7-10 days of immobilisation. characteristic by pain, tenderness, swelling, venous distension, in the calf muscle and confirm by Doppler study .
  • 14.
    Prevention: -Proper fixation (rigidfixation) -free pain early mobilisation and as soon as pt.'s can. -Daily muscle stretching ,contraction for 20-30 seconds. - ROM of adjacent joint below and above the fracture. -leg stocking to prevent DVT -anticoagulant ,antiplatelet drugs -change patient position every 2H.
  • 16.
    Thank You Prepared by:Arhab Alshabi