28,000 ankle sprains occur daily in the US (Kaminski 2013)
Ankle is the 2nd most commonly injured body site. (Ferran 2006)
Ankle sprains are the most common type of ankle injury. (Ferran 2006)
A sprained ankle can happen to athletes and non-athletes,
children and adults.
Inversion injury most common mechanism (Ferran 2006)
Only risk factor is previous ankle sprain (Ferran 2006)
Sex , generalized joint laxity or anatomical foot types are
not risk factors. (Beynnon et al. 2002 )
MCL. LCL.ALL injuries
To understand the relevant anatomy of the side ligaments of the knee
To study the mechanism of injury of each ligament and how to diagnose such injury
To highlight the different treatment options in acute or chronic situations
28,000 ankle sprains occur daily in the US (Kaminski 2013)
Ankle is the 2nd most commonly injured body site. (Ferran 2006)
Ankle sprains are the most common type of ankle injury. (Ferran 2006)
A sprained ankle can happen to athletes and non-athletes,
children and adults.
Inversion injury most common mechanism (Ferran 2006)
Only risk factor is previous ankle sprain (Ferran 2006)
Sex , generalized joint laxity or anatomical foot types are
not risk factors. (Beynnon et al. 2002 )
MCL. LCL.ALL injuries
To understand the relevant anatomy of the side ligaments of the knee
To study the mechanism of injury of each ligament and how to diagnose such injury
To highlight the different treatment options in acute or chronic situations
This is the presentation which was delivered to third year Bachelor of Physiotherapy students at Kathmandu University School of Medical Sciences (KUSMS), Dhulikhel, Nepal. Different soft tissue injuries are the part of curriculum for the undergraduate students at KUSMS.
Dr.MD.Monsur Rahman,PT
MPT-Musculoskeletal Disorders
Maharishi Markandeshwar Institute Of Physiotherapy And Rehabilitation, Maharishi Markandeshwar (Deemed to be University), Mullana - Ambala,133-207 (Haryana)
This is the presentation which was delivered to third year Bachelor of Physiotherapy students at Kathmandu University School of Medical Sciences (KUSMS), Dhulikhel, Nepal. Different soft tissue injuries are the part of curriculum for the undergraduate students at KUSMS.
Dr.MD.Monsur Rahman,PT
MPT-Musculoskeletal Disorders
Maharishi Markandeshwar Institute Of Physiotherapy And Rehabilitation, Maharishi Markandeshwar (Deemed to be University), Mullana - Ambala,133-207 (Haryana)
This is the Presentation on the topic "Pathomechanics of Knee Joint".
The presentation includes images and a clip for proper understanding. The sentences are framed in the way that you can learn it in a easy way.
meniscus injury explained with treatment and videos to help understand the use of MRI to help understand injury to meniscus and help diagnose meniscal tear
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
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NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
2. INTRODUCTION
Soft tissue injuries of the knee are some of the most
common and clinically challenging musculoskeletal
disorders in patients presenting to the ED.
soft tissue structures that stabilize and cushion the
knee joint including
ligaments
muscles
tendons
menisci
3. SPRAIN VS STRAIN
Definitions of sprains and strains
Sprains:
characterized by the stretching or tearing of non-
contractile structures, such as the investing ligaments
or of the joint capsule itself.
strain :
characterized by stretching or severing along the
course of muscles or tendons.
*Both collateral ligament and cruciate ligament
sprains, as well as muscular strains, are
relatively common.
4. GRADES OF SPRAINS AND STRAINS
Grade I sprain
Stretching but no tearing of the ligament, local tenderness,
minimal edema, no gross instability with stress testing, firm end
point
Grade II sprain
Partial tears of the ligaments, moderate local tenderness, mild
instability with stress testing (but firm end point), moderately
incapacitating
Grade III sprain
- Complete tear, discomfort with manipulation but less than
expected for degree of injury, variable amount of edema
(ranging from negligible to grossly conspicuous), clear
instability with stress testing (expressing a mushy end point),
severe disability
6. Of the all ligaments there are 4 ligaments which are
commonly injured , they can be classified as following
:
1-intra-articular
-anterior cruciate ligament ACL
-posterior cruciate ligament PCL
2-extra-articular
-medial (tibial ) collateral ligament MCL
-lateral (fibular) collateral ligament LCL
7.
8. ANTERIOR CRUCIATE LIGAMENT
The anterior cruciate ligament (ACL) , it resists the anterior translation
of the tibia relative to the
femur.
- it originates on the anterior intercondylar area of the tibia and passes
upward backward and laterally to be inserts to posteromedial aspect of
the lateral condyle of the femur
- ACL includes two functional bundles:
1- anteromedial bundle, which tightens in flexion,
2-posterolateral bundle, which tightens in extension
-ACL prevents posterior displacement of the femur on the tibia -with
the knee joint flexed the ACL prevents the tibia from being pulled
anterior -tight in hyperextension of the knee.
10. The posterior cruciate ligament (PCL) resists against
posterior translation of tibia over femur .
It originates on the posterior intercondylar area of the tibia
and passes upward forward and medially and insert to the
anterolateral aspect of the medial femoral
condyle
-The PCL also is made up of two functional bundles. 1-
anterior meniscofemoral 2-
posterior meniscofemoral ligament originate from the
posterior horn of the lateral meniscus and contribute to the
function of the PCL.
- PCL prevent anterior displacement of femur on tibia -
with the knee joint flexed the PCL prevent the tibia from
being pulled posteriorly - tight in hyperflexion
14. Complete tearpartial tear
a complete tear the
patient may have little or no pain,
a partial
tear the knee is painful.
Swelling is less with complete tearSwelling also is worse with
partial tears
Abnormal movement of a complete
tear is often painless or
prevented by spasm
attempted movement is always
painful
16. A- lockman test
*position of the patient :
supine
*position of the examiner:
- he or she stand on the side of affected
limb
-the proximal hand attaches to the patient
femur and stabilizes it.
- the distal hand attaches to proximal part
of the tibia position it in 20 of flexion and
push it forward .
- not forget to examine the other limb for
comparison .
*results:
positive test indicated by noticing
abnormal ant displacement of the tibia
forward .
17. B- PIVOT SHIFT TEST
*position of the patient:
supine
*position of examiner :
. The examiner should lift the tested leg off the
table with the knee fully extended. Place the
heel of one hand behind the fibular head of the
patient. Use the other hand to grasp the tibia,
while palpating the medial joint line. While
maintaining a valgus force and internal rotation
of the tibia throughout the test, slowly flex the
patient's knee (note: the test starts by putting
the tibia in the abnormal position!).
*results:
if there is an anterior subluxation felt during
extension the test is positive for instability
18. C-ANTERIOR DRAWER TEST
The patient lies supine on a plinth with
their hips flexed to 45degrees, his/her
knees flexed to 90degress and feet flat
on the plinth. The examiner sits on the
toes of the tested extremity to help
stabilize it. The examiner grasps the
proximal lower leg, just below the tibial
plateau or tibiofemoral joint line, and
attempts to translate the lower leg
anteriorly. The test is considered positive
if there is a lack of end feel or excessive
anterior translation relative to the
contralateral side.
20. 2-POSTERIOR DRAWER TEST
The patient lies supine on a plinth with
their hips flexed to 45degrees, his/her
knees flexed to 90degress and feet flat on
the plinth. The examiner sits on the toes of
the tested extremity to help stabilize it. The
examiner grasps the proximal lower leg,
just below the tibial plateau or tibiofemoral
joint line, and attempts to translate the
lower leg posteriorly . The test is
considered positive if there is a excessive
posterior translation relative to the
contralateral side.
21. 1-POSTERIOR SAG TEST
*position of the patient :
supine
*position of the examiner:
- the examiner stand on the side of the patient
and passively bring the hip and knee to 90 of
flexion ,and compare the level of tibial
tubersiteies of both knee.
*results:
a positive test is indicated when posterior
displacement of tibal tuberosity is more in the
affected limb .
22. 2- IMAGING
* knee x ray
Although x-rays do not show meniscal tears, they
may show other causes of knee pain, such
osteoarthritis and demonstrate bone avulsion if
present .
*Magnetic resonance imaging (MRI)
This study can create better images of the soft
tissues of your knee joint, like a meniscus
26. TREATMENT
A- conservative
RICE. The RICE protocol is effective for most sports-related
injuries. RICE stands for Rest, Ice, Compression, and
Elevation.
Aspiration of hemartharosis to relive pain .
Exercises and physiotherapy must start since thediagnosis
is approved to prevent adhesion .
using knee brace until pain is disappear .
27. B- SURGICAL TREATMENT
Unfortunately, the cruciate ligaments -- ACL and PCL
-- cannot be repaired.
Once they are completely torn or stretched beyond
their limits, The only option a reconstruction.
In this procedure, tendons are taken from other parts
of your leg or a cadaver to replace the torn ligament.
31. Medial collateral ligamentis
flat band and attached above to the medial epicondyle
of the femur and below to the shift of the tibia it is
firmly attached to the edge of the medial meniscus -
(MCL) is the primary restraint to valgus stress.
*The MCL is the most commonly injured knee
ligament.
32. LATERAL COLLATERAL LIGAMENT
is cordlike and is attached above to the lateral
condyle of the femur and below to the head of fibula
the tendon of the popliteus muscle intervenes
between the ligament and the lateral meniscus -
(LCL) is the primary restraint to varus stress
33. MECHANISM OF INJURY
Medial collateral ligament
tears often occur as a result of a direct blow to the
outside of the knee. This pushes the knee inwards
(toward the other knee).
lateral collateral ligament
tears often occur as a result of a direct Blows to the
inside of the knee that push the knee outwards.
35. CLINICAL PICTURE:
1-Pain at the sides of knee.
If there is an MCL injury, the pain is on the
inside of the knee; an LCL injury may cause pain
on the outside of the knee.
2-Swelling over the site of the injury
3-Instability : the feeling that your knee is giving
way.
37. A-BY PHYSICAL EXAMINATION
Valgus vs varus stress tests
*position of the patient:
supine
*position of the patient leg :
hip is abducted and knee flexed with 20 degree
*position of the examiner:
-the examiner stands on the side of the affected leg
,with one hand on the medial and lateral
(respectively for LCL,MCL) line of the knee and the
other hand on the lateral aspect of the ankle .
1- a varus force pushing toward the Medline is
applied to the ankle for testing LCL injury
2- a valgus force pushing away from Medline is
applied through the ankle for testing MCL injury
38.
39. RESULTS OF THE TESTS
1- in MCL injury
pain and excessive gaping is positive indicator for the
injury
40. 2- in LCL injury
pain and excessive gaping is positive indicator for the
injury
45. WHAT IS KNEE MENISCI ??
they are fibrocartilagenous structures
present In the intercondylar fossa between
.femur and tibia condyles
.
- functions:
1- disperse the Wight of the body
2-stabalization of knee joint
3- reduce friction between articular surfaces
of tibia and femur condyle.
4-shock absorber
48. What is a meniscus injury?
Patients describe meniscal tears in a
variety of ways.
Knowing where and how a meniscus
was torn helps the doctor determine the
best treatment.
Location :- A tear may be located in the
anterior horn, body, or posterior horn. A
posterior horn tear is the most common.
The meniscus is broken down into the
outer, middle, and inner thirds. The third
in which the tear is located will
determine the ability of the tear to heal,
since blood supply in that area is critical
to the healing process. Tears in the
outer 1/3 have the best chance of
healing.
49. Pattern -
Meniscal tears come in many
shapes. The pattern of the tear
influences the doctor's decision
on treatment. Examples of the
various patterns are:
longitudinal
bucket-handle
displaced bucket handle
parrot beak
radial
displaced flap
50. CLINICAL PICTURE
1- severe pain
2-joint locking
3-limited movement of knee joint
4-swelling
5-inablity to stand on affected limb
6-popping or clicking within knee
51. DIAGNOSIS
•A – clinically by :
1-physical examination ,including the
following tests
a- muc Murray's test
b- Thessaly's test
c- apley’s test
52. HOW TO INSURE UR DIAGNOSIS BY
EXAMINATION ??
1-muc Murray's test
* Position of the patient
supine
*position Of the examiner:
-stand on the side of the patient , the proximal
hand on knee joint and the distal one on the heel
of the same limb
- the knee should be fully flexed ,the examiner
passively rotate the tibia and extend the knee for
examining medial menisci , internal rotation and
extension of the tibia for examining the lateral
menisci
*results
a positive test indicated by hearing a crepitus
associated with pain .
53. 2-THESSALY’S TEST
*position of the patient :
stand on the affected leg
*position of the examiner:
stand in front of the patient and provide his or her
hands for stability
*principle:
- knee is flexed 5 and femur is rotate medially and
laterally for 3 times
- the same step is repeated with knee flexed 20
*results :
a positive test is indicated if there is locking of
movement .
54. 3-APLEY’S TEST
For this test, the patient is
positioned prone, with his or
her knee flexed. Compression
and external or internal
rotation may be painful,
showing that the medial or
the lateral meniscus are torn.
This test is always checked,
by performing rotation without
compression
55. B- IMAGING
* knee x ray
Although x-rays do not show meniscal tears, they
may show other causes of knee pain, such
osteoarthritis.
*Magnetic resonance imaging (MRI)
This study can create better images of the soft
tissues of your knee joint, like a meniscus.
56.
57. TREATMENT
Conservative Treatment
RICE. The RICE protocol is effective for most sports-
related injuries. RICE stands for Rest, Ice, Compression,
and Elevation.
If the joint is not locked, it is reasonable to hope that the
tear is peripheral and can therefore heal spontaneously.
After an acute episode, the joint is held straight in a
plaster backslab for 3–4 weeks; the patient uses crutches
and quadriceps exercises are encouraged .
58. by arthroscopy :
Meniscectomy
In this procedure, the damaged meniscal tissue is
trimmed away.
Meniscus repair. Some meniscal tears can be
repaired by suturing (stitching) the torn pieces
together
SURGICAL TREATMENT
59.
60. RUPTURE OF QUADRICEPS TENDON
The four quadriceps muscles
meet just above the kneecap
(patella) to form the quadriceps
tendon. Tendons attach
muscles to bones. The
quadriceps tendon attaches the
quadriceps muscles to the
patella. The patella is attached
to the shinbone (tibia) by its
tendon, the patellar tendon.
Working together, the
quadriceps muscles, quadriceps
tendon and patellar tendon
straighten the knee.
61. QUADRICEPS TENDON TEARS
Quadriceps tendon tears more common in people more
40 year , can be either partial or complete.
Partial tears. Many tears do not completely disrupt the
soft tissue. This is similar to a rope stretched so far that
some of the fibers are torn, but the rope is still in one
piece.
Complete tears. A complete tear will split the soft tissue
into two pieces.
When the quadriceps tendon completely tears, the muscle
is no longer anchored to the kneecap. Without this
attachment, the knee cannot straighten when the
quadriceps muscles contract.
62. Cause
Injury
A quadriceps tear often occurs when there is a heavy
load on the leg with the foot planted and the knee
partially bent. Think of an awkward landing from a
jump while playing basketball. The force of the
landing is too much for the tendon and it tears.
Tears can also be caused by falls, direct force to the
front of the knee, and lacerations (cuts).
Tendon Weakness
A weakened quadriceps tendon is more likely to tear.
63. CLINICAL PICTURE
1-The typical injury is followed by tearing pain and
giving way of the knee.
3-There is bruising and local tenderness;
3-Active knee extension is either impossible
(suggesting a complete rupture) or weak (partial
rupture)
65. DIAGNOSIS
X-rays.
The kneecap moves out of place when the
quadriceps tendon tears. This is often very obvious
on a "sideways" X-ray view of the knee. Complete
tears can often be identified with these X-rays
alone.
MRI
diagnosis can be confirmed by MRI.
68. TREATMENT
Partial tears
Non-operative treatment with plaster cylinder is
applied for 6 weeks, followed by physiotherapy that
concentrates on restoring knee.
Complete tears
Early operation is needed, End-to-end suturing can
be reinforced by turning down a partial-thickness
triangular flap of quadriceps tendon proximal to the
repair (Scuderi).
69.
70. RUPTURE OF PATELLAR LIGAMENT
This is an uncommon injury; it is usually seen in
young athletes and the tear is almost always at the
proximal or distal attachment of the ligament.
71. CLINICAL PICTURE
* The patient gives a history of :
1-sudden pain on forced extension of
the knee
2- bruising
3-swelling
4- tenderness at the lower edge of the
patella or more distally.
74. TREATMENT
Partial tears : can be treated by applying a plaster cylinder.
Complete tears : need operative repair or reattachment to
bone, and keep the knee in extension position and use knee
immobilizer for 4-6 weeks Immobilization in full extension
may precipitate stiffness – after all, it is a joint injury – and it
may be better to support the knee in a hinged brace with
limits to the amount of flexion permitted. This range can be
gradually increased after 6 weeks.