The term ‘‘aniseikonia” comes from the Greek words ‘‘an” (not) ‘‘is” (equal) & ‘‘eikon” (icon or image) so aniseikonia is a binocular condition in which the apparent sizes of the images seen with the two eyes are unequal.
Whenever refractive ametropias in the two eyes of a person are different (i.e., when there is an anisometropia), the corrected retinal images of the two eyes, and consequently the two visual images, differ in size.
This condition has been termed aniseikonia
Optical aniseikonia
Retinal aniseikonia
Cortical aniseikonia
Presbyopia/ Methods of Presbyopic Addition Determination (healthkura.com)Bikash Sapkota
DIRECT DOWNLOAD LINK ❤❤https://healthkura.com/presbyopia-near-addition/❤❤
Dear viewers Check Out my other piece of works at ❤❤❤ https://healthkura.com ❤❤❤
Presbyopia and techniques of measurement
A fantastic presentation in the topic "Presbyopia and techniques of measurement"
A detailed information about presbyopia, techniques of presbyopic add determination and different correction methods.
Informative slide presentation on presbyopia for ophthalmology residents, ophthalmologists, optometrists, ophthalmic assistants, ophthalmic technicians, ophthalmic nurses, medical students, medical professors, teaching guides.
Presentation Contents:
--Introduction to presbyopia
-Types of presbyopia
-Risk factors
-Symptoms and signs
-Refractive error and presbyopia
-Methods of determining near add.
-Management of presbyopia
In a nutshell..
- The evaluation and management of presbyopia are important because significant functional deficits can occur when the condition is left untreated
- Undercorrected or uncorrected presbyopia can cause significant visual disability and have a negative impact on the pt.'s quality of life
- Finally, every tentative addition should be adjusted according to the particular needs of the patient
For Further Reading:
-Clinical Procedures in Optometry by J.D. Bartlett, J.B. Eskridge, J.F. Amos
-Primary Care Optometry by Theodere Grosvenor
-Borish’s Clinical Refraction by W.J. Benjamin
-Clinical Procedures for Ocular examination by Carlson et al
-American Academy of Ophthalmology
-Optometric Clinical Practice Guideline by American Optometric Association
-Internet
Follow me to get in touch with optometric and ophthalmic updates.
The term ‘‘aniseikonia” comes from the Greek words ‘‘an” (not) ‘‘is” (equal) & ‘‘eikon” (icon or image) so aniseikonia is a binocular condition in which the apparent sizes of the images seen with the two eyes are unequal.
Whenever refractive ametropias in the two eyes of a person are different (i.e., when there is an anisometropia), the corrected retinal images of the two eyes, and consequently the two visual images, differ in size.
This condition has been termed aniseikonia
Optical aniseikonia
Retinal aniseikonia
Cortical aniseikonia
Presbyopia/ Methods of Presbyopic Addition Determination (healthkura.com)Bikash Sapkota
DIRECT DOWNLOAD LINK ❤❤https://healthkura.com/presbyopia-near-addition/❤❤
Dear viewers Check Out my other piece of works at ❤❤❤ https://healthkura.com ❤❤❤
Presbyopia and techniques of measurement
A fantastic presentation in the topic "Presbyopia and techniques of measurement"
A detailed information about presbyopia, techniques of presbyopic add determination and different correction methods.
Informative slide presentation on presbyopia for ophthalmology residents, ophthalmologists, optometrists, ophthalmic assistants, ophthalmic technicians, ophthalmic nurses, medical students, medical professors, teaching guides.
Presentation Contents:
--Introduction to presbyopia
-Types of presbyopia
-Risk factors
-Symptoms and signs
-Refractive error and presbyopia
-Methods of determining near add.
-Management of presbyopia
In a nutshell..
- The evaluation and management of presbyopia are important because significant functional deficits can occur when the condition is left untreated
- Undercorrected or uncorrected presbyopia can cause significant visual disability and have a negative impact on the pt.'s quality of life
- Finally, every tentative addition should be adjusted according to the particular needs of the patient
For Further Reading:
-Clinical Procedures in Optometry by J.D. Bartlett, J.B. Eskridge, J.F. Amos
-Primary Care Optometry by Theodere Grosvenor
-Borish’s Clinical Refraction by W.J. Benjamin
-Clinical Procedures for Ocular examination by Carlson et al
-American Academy of Ophthalmology
-Optometric Clinical Practice Guideline by American Optometric Association
-Internet
Follow me to get in touch with optometric and ophthalmic updates.
Real subjective refraction in astigmatismBipin Koirala
hope it will be beneficial for the students in eye care system . please like it and share it if you think it is beneficial for your studies. It will motivate me to upload more slides ..
Retinoscope is an objective refraction instrument used to
determine the spherocylindrical refractive error, as well as
observe optical aberrations, irregularities, and opacities.
The technique is called Retinoscopy/Skiascopy/Shadow Test
Correction of Ametropia is very basic topic in Optometry background. Hope the SlideShare may help you. This PPT will help Bachelor students (B.optoms).
Aniseikonia [ophthalmology description for medical students ]Madhuri Kureti
concise description of aniseikonia which is a condition wherein the images projected to the visual cortex from the two retinae are abnormally unequal in size and /or shape
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
2. Ametropia is defined as a state of
refraction wherein the parallel rays of
light coming from infinity are focussed
either in front or behind the retina which
therefore receives a blurred image.
It includes-
Myopia
Hyperopia
Astigmatism
Aphakia
3. HYPERMETROPIA Long sightedness.
Images are focussed behind retina.
ie. Posterior focal point is behind retina.
Mechanisms-
1. AXIAL: 1mm shortening of AP diameter of eye causes 3D of
hypermetropia.
2. CURVATURAL: curvature of cornea or lens or both is flatter
than normal. 1mm decrease in radius of curvature causes
6D of hypermetropia.
3. INDEX: due to change in refractive index of lens in old age
and in diabetics.
4. POSITIONAL: posteriorly placed lens.
5. ABSENCE OF CRYSTALLINE LENS.
4. CLINICAL TYPES:
1) SIMPLE HYPERMETROPIA: due to biological variation in size and
shape of eyeball. Could be axial or curvatural.
2) PATHOLOGICAL:
A) CONGENITAL: Associated with microphthalmos, micro cornea
congenial posterior subluxation of lens or congenital aphakia.
B)ACQUIRED:
i. SENILE: could be curvatural or index (due to cortical
sclerosis)
ii. POSITIONAL: subluxation of lens
iii. APHAKIA.
iv. CONSECUTIVE:
v. ORBITAL MASS: tumors or edema may push the retina
forward.
C) FUNCTIONAL: due to paralysis of accommodation as in 3rd
nerve palsy.
5. COMPONENTS OF HYPERMETROPIA:
TOTAL
MANIFEST
FACULTATIVE
ABSOLUTE
LATENT
Total- after complete cycloplegia with atropine.
Latent- about 1D of hyperopia that is corrected by
inherent tone of ciliary muscles.
Manifest- remainder of hyperopia.
Facultative- can be corrected by patient’s accommodative
effort.
Absolute- residual of manifest hyperopia.
6. MYOPIA Short sightedness.
Parallel rays coming from infinity focus in
front of retina with accommodation at rest.
MECHANISMS:
1. AXIAL: increased axial length of the eye ball.
Commonest form.
2. CURVATURAL: increased curvature of cornea or lens
or both.
3. INDEX: increased refractive index of lens with nuclear
sclerosis.
4. EXCESSIVE ACCOMMODATION: in spasm of
accommodation.
7. In myopia the image of a distant object is formed of the
divergent beam.
Far point of the myopic eye is at a finite point in front of
the eye.
Nodal point of the eye is further away from the retina.
Hence the image of the object formed is larger than
that of the emmetropic eye or spectacle corrected eye.
This compensates for visual acuity to some extent.
They do not need to accommodate. Hence it is not well
developed and they may suffer from convergence
insufficiency, exophoria or early presbyopia.
8. ASTIGMATISM
Astigmatism is a refractive error in which the
refraction varies in different meridia.
ETIOLOGY:
CORNEAL: due to abnormalities in the curvature of cornea.
Maybe congenital or acquired (often irregular).
LENTICULAR:
CURVATURAL: due to abnormal curvature of the lens. eg- lenticonus.
POSITIONAL: due to oblique placement or tilting of the lens eg-in
subluxation.
INDEX: due to difference of refractive index of the in different
meridia.
RETINAL: due to oblique placement of macula.
9. ASTIGMATISM Regular astigmatism – principal meridians are
perpendicular.
With-the-rule astigmatism – the vertical meridian is
steepest (a rugby ball or American football lying on its
side).
Against-the-rule astigmatism – the horizontal meridian is
steepest (a rugby ball or American football standing on
its end).
Oblique astigmatism – the steepest curve lies in between
120 and 150 degrees and 30 and 60 degrees.
Irregular astigmatism – principal meridians are not
perpendicular.
10. ASTIGMATISM Simple astigmatism
Simple myopic astigmatism – first focal line is in front
of the retina, while the second is on the retina.
Simple hyperopic astigmatism – first focal line is on
retina, while the second is located behind the retina.
Compound astigmatism
Compound myopic astigmatism – both focal lines are
located in front of the retina.
Compound hyperopic astigmatism – both focal lines
are located behind the retina.
Mixed astigmatism – focal lines are on both sides
of the retina (straddling the retina)
11. FARPOINT
Far point of the eye is the position of
an object such that its image falls on
the retina of the relaxed eye (ie. With
accommodation relaxed).
For emmetropia it is at infinity.
For myopia it lies at a finite
distance in front of the eye.
In hypermetropia it is virtual (as
only converging light can focus on
the retina in hyperope).
15. EFFECTIVEPOWEROFLENSES In uncorrected hyperopia
the image of an object falls
behind the retina.
The purpose of convex lens
is to bring the image
forward.
If the correcting lens is itself
moved forward the image
will move still forward.ie- the
effectivity of the lens is
increased.
Thus a weaker lens is
required to project the
image onto the retina.
Similarly in uncorrected myopia
the image falls in front of the
retina.
The purpose of the concave
lens is to bring the image
behind.
If the correcting lens is itself
moved forward the image
moves still forward.ie- the
effectivity of the lens is
reduced.
Thus a stronger lens is required
to project the image onto the
retina
16. EFFECTIVEPOWEROFLENSES Thus the convex lens in
hypermetropia has to be made
weaker and the concave lens in
myopia has to be made stronger
when the lens is moved further
away from the eye
Hence aphakics or high
hyperopes pull their glasses
down their nose to read.
While myopes do not like their
glasses slipping down.
17. EFFECTIVEPOWEROFLENSES
Formula to calculate the new focal length of lens at the new distance-
F2= 1/ f1- d or F2= F1/ 1- dF1
Where, F1= power of the original lens in diopters
F2= power of lens in diopters at new position
f1= focal length in meters of original lens
d= distance moved in meters. It is taken positive if
moved toward the eye and negative if moved away from
the eye.
18. BACKVERTEXDISTANCE
For any prescription greater than 5D especially in aphakics the
refractionist must state how far the trial frame was placed, to
adjust the power of contact lens is used or if the glasses are to
be worn at a different distance.
The distance between the back of the lens and the cornea
must be measured.
Measurement can be made with a ruler held parallel to the arm
of the trial frame or slipped through a steanopic slit till it
touches thee closed lid. 2mm should be added to correct for
the thickness of the lid.
19. BACKVERTEXDISTANCE
Example 1: A patient has been prescribed glasses
with +16.00D sphere at a BVD of 14mm. He selects a
frame that fits him at a BVD of 16mm. What is the
power of the new lens?
Ans: +15.50D
20. BACKVERTEXDISTANCE
Example 2: A aphakic patient requires a +10.00D lens
at BVD 15mm. He now wants a contact lens. What
should be the power of the contact lens?
Ans- +11.75D
21. BACKVERTEXDISTANCE
Example 3: A patient was given a prescription of -
16.00D at a BVD of 14mm. He selects a spectacle
frame of BVD 16mm. What will be the power of the
new lens?
Ans- -16.50D
22. BACKVERTEXDISTANCE
Example 4: A high myope whose spectacle correction
is -10.00D at BVD 14mm requires a contact lens. What
is the power of the contact lens?
Ans- -8.75 D
23. SPECTACLEMAGNIFICATION The optical correction of ametropia is associated with in a
change in the retinal image size.
Spectacle magnification = corrected image size
uncorrected image size
Relative sp. magnification = corrected image size
emmetropic image size
24. SPECTACLEMAGNIFICATION
In axial ametropia, if the correcting lens is placed at the anterior focal
point of the eye then the image size is same as that of emetropia.
But in refractive ametropia the image size differs even if lens is placed at
the anterior focal point.
In refractive hypermetropia the image size is increased. RSM>1
While in refractive myopia the image size is reduced.RSM<1
As the distance of the lens approaches the eye the image size
approaches the emmetropic size
25. SPECTACLEMAGNIFICATION RSM= 1.36 for aphakia with lens at anterior focal point ie 23.2mm
RSM= 1.33 for aphakia with lens placed at 12-15mm
RSM= 1.1 for contact lenses.
normal
contact lens
spectacles
26. OPTICALPROBLEMSINAPHAKIA
1. SPECTACLE MAGNIFICATION:
The spectacle magnification produced by
aphakic glasses is 1.33. thus the image is one
third times larger than emmetropes.
The patient thus tends to misjudge distances.
Objects appear closer to the eye than they are.
Leads to enhanced performance in visual acuity
tests.
27. OPTICALPROBLEMSINAPHAKIA
2. DISTORTION OF IMAGES DUE TO
ABERRATIONS:
Straight lines appear curved except
through a small central portion of the lens.
At the periphery of the lens the lines
appear to be more curved- pincushion
effect.
Thus the environment appears as curves as
the patient moves his eyes across different
parts of the lens. Patients adapt to this by
moving their head rather than eyes.
28. OPTICALPROBLEMSINAPHAKIA 3) PRISMATIC EFFECT OF LENS:
The prismatic effect increases towards the
periphery of the lens.
It produces a troublesome ring scotoma at
the edge of the lens. Hence they can trip
over unseen objects.
The direction of the ring scotoma changes
and objects disappear into the scotoma and
appear to reappear out of it- jack in the box
phenomenon.
29. OPTICALPROBLEMSINAPHAKIA
4) DUE TO WEIGHT OF THE GLASSES:
Aphakic glasses are very heavy and tend to slip
down the nose.
Plastic glasses are lighter but less scratch resistant.
Lenticular form of lenses reduce weight but also
reduce field of vision.
30. OPTICALPROBLEMSINAPHAKIA
5) UNILATERAL APHAKIA WITH
NORMAL FELLOW EYE:
The image in aphakic eye is one third larger hence
causes aniseikonia. Patient is unable to fuse these
images and hence suffers from diplopia.
The use of contact lenses and intra ocular
implants reduce this effect.
Aniseikonic glasses though available are very
heavy and costly.