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Morning Educational Activity
Rashid A. M. Abuelhassan
R4, Emergency Medicine
Early Repolarization
CRETERIA
• wide spread ST elevation (90% < 2 mm
in precordial leads, and < 0.5 mm in
limb leads) with precordial > limb leads
• J point elevation
• concave initial upsloping of ST segment
• notching/irregular contour of J point
• prominent concordant T waves
• stability of ECG over time
• 38, m, Chest pain
Acute pericarditis
• Widespread concave ST ↑ and PR ↓
throughout most of the limb and
precordial leads.
• Reciprocal ST ↓ and PR ↑ in lead aVR
• Sinus tachycardia is common in acute
pericarditis due to pain /effusion.
Early Repolarization Vs. PERICARDITIS
ER PERICARDITIS
ST ↑ limited to the precordial leads Generalised ST ↑
Absence of PR ↓ Presence of PR ↓
Prominent T waves Normal T wave amplitude
ST segment / T wave ratio < 0.25 ST segment / T wave ratio > 0.25
“fish-hook” appearance in V4 Absence of “fish hook” appearance in V4
ECG changes relatively stable over time ECG changes evolve over time
49 y man 1/2hour chest pain.
Hyperacute T waves
•FAT
• w i d e
• with a blunt peak
• poor R-wave progression (especially V3).
• Sometimes loss of precordial T-wave balance (when the upright T
wave is larger than that in V6).
De Winter ST/T-Wave Complexes
2% of total proximal LAD lesion
• Precordial ST-segment depression at the J-point
• Tall, peaked, symmetric T waves in the precordial leads
• Lead aVR shows slight ST-segment elevation in most cases
Posterior MI
• ST-segment depression
• Prominent and broad R wave (>30ms)
• Relative tall R waves in precordial leads
• R/S wave ratio >1.0 in lead V2
• Prominent, upright T wave
• Combination of horizontal ST-segment depression with
upright T wave
LMCA Occlusion
occlusion of the proximal left anterior descending artery and severe
multivessel coronary artery disease
* findings
ST elevation in aVR ≥ 1mm
ST elevation in aVR ≥ V1
ST depression typically seen in lateral
69years, F, c/o sever chest pain,
Pt is saying
“ Doctor I cant speak , give me something
for the pain “
• What will you give ?
Why is it important to differentiate
• ECG Findings:
• ST elevation in right-sided V leads
(V4 R, V5 R)
• ST elevation greater in lead III than
lead II suggests RV MI
• ST elevation in the normally
obtained V1 also strongly suggests
RV MI
• Often associated with inferior MI
and/or posterior MI
What is this
Thank you

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What about this ST and T waves ( ECG sessions )

  • 1. Morning Educational Activity Rashid A. M. Abuelhassan R4, Emergency Medicine
  • 2.
  • 3. Early Repolarization CRETERIA • wide spread ST elevation (90% < 2 mm in precordial leads, and < 0.5 mm in limb leads) with precordial > limb leads • J point elevation • concave initial upsloping of ST segment • notching/irregular contour of J point • prominent concordant T waves • stability of ECG over time
  • 4. • 38, m, Chest pain
  • 5. Acute pericarditis • Widespread concave ST ↑ and PR ↓ throughout most of the limb and precordial leads. • Reciprocal ST ↓ and PR ↑ in lead aVR • Sinus tachycardia is common in acute pericarditis due to pain /effusion.
  • 6. Early Repolarization Vs. PERICARDITIS ER PERICARDITIS ST ↑ limited to the precordial leads Generalised ST ↑ Absence of PR ↓ Presence of PR ↓ Prominent T waves Normal T wave amplitude ST segment / T wave ratio < 0.25 ST segment / T wave ratio > 0.25 “fish-hook” appearance in V4 Absence of “fish hook” appearance in V4 ECG changes relatively stable over time ECG changes evolve over time
  • 7. 49 y man 1/2hour chest pain.
  • 8. Hyperacute T waves •FAT • w i d e • with a blunt peak • poor R-wave progression (especially V3). • Sometimes loss of precordial T-wave balance (when the upright T wave is larger than that in V6).
  • 9.
  • 10.
  • 11. De Winter ST/T-Wave Complexes 2% of total proximal LAD lesion • Precordial ST-segment depression at the J-point • Tall, peaked, symmetric T waves in the precordial leads • Lead aVR shows slight ST-segment elevation in most cases
  • 12.
  • 13. Posterior MI • ST-segment depression • Prominent and broad R wave (>30ms) • Relative tall R waves in precordial leads • R/S wave ratio >1.0 in lead V2 • Prominent, upright T wave • Combination of horizontal ST-segment depression with upright T wave
  • 14.
  • 15. LMCA Occlusion occlusion of the proximal left anterior descending artery and severe multivessel coronary artery disease * findings ST elevation in aVR ≥ 1mm ST elevation in aVR ≥ V1 ST depression typically seen in lateral
  • 16.
  • 17. 69years, F, c/o sever chest pain,
  • 18. Pt is saying “ Doctor I cant speak , give me something for the pain “ • What will you give ?
  • 19. Why is it important to differentiate • ECG Findings: • ST elevation in right-sided V leads (V4 R, V5 R) • ST elevation greater in lead III than lead II suggests RV MI • ST elevation in the normally obtained V1 also strongly suggests RV MI • Often associated with inferior MI and/or posterior MI
  • 21.

Editor's Notes

  1. CRETERIA wide spread ST elevation (90% < 2 mm in precordial leads, and < 0.5 mm in limb leads) with precordial > limb leads J point elevation concave initial upsloping of ST segment notching/irregular contour of J point prominent concordant T waves stability of ECG over time
  2. Widespread concave ST ↑ and PR ↓ throughout most of the limb and precordial leads. Reciprocal ST ↓ and PR ↑ in lead aVR Sinus tachycardia is common in acute pericarditis due to pain /effusion.
  3. Stage 1 – widespread STE and PR depression with reciprocal changes in aVR (occurs during the first two weeks) Stage 2 – normalization of ST changes; generalized T wave flattening (1 to 3 weeks) Stage 3– Flattened T waves become inverted (3 to several weeks) Stage 4 – ECG returns to normal (several weeks onwards)
  4. FAT w i d e with a blunt peak poor R-wave progression (especially V3). Sometimes loss of precordial T-wave balance (when the upright T wave is larger than that in V6).
  5. NOTE: The normal T wave in V1 is inverted. An upright T wave in V1 is considered abnormal — especially if it is tall (TTV1), and especially if it is new (NTTV1).
  6. Hyperkalemia
  7. Precordial ST-segment depression at the J-point Tall, peaked, symmetric T waves in the precordial leads Lead aVR shows slight ST-segment
  8. LMCA Occlusion findings ST elevation in aVR ≥ 1mm ST elevation in aVR ≥ V1 ST depression typically seen in lateral
  9. Right side MI
  10. nferolateral STEMI. Posterior extension is suggested by: Horizontal ST depression in V1-3 Tall, broad R waves (> 30ms) in V2-3 Dominant R wave (R/S ratio > 1) in V2 Upright T waves in V2-3
  11. Marked ST elevation in V7-9 with Q-wave formation confirms involvement of the posterior wall, making this an inferior-lateral-posterior STEMI (= big territory infarct!).