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ECG interpretation
Dr Sudhir Dev
House Officer
Dept. Of GP and Emergency Medicine
Objectives
• Justify the reasons for performing an ECG
• Develop a structured approach to interpreting
an ECG
• Practice interpreting ECGs
The ECG
“The ECG (electrocardiogram) is a transthoracic
interpretation of the electrical activity of the
heart.”
The ECG
SA node -> atrial muscle -> AV node -> bundle of His ->
Left and Right Bundle Branches -> Ventricular muscle
Lead Placement
EKG Distributions
• Anteroseptal: V1, V2, V3, V4
• Anterior: V1–V4
• Anterolateral: V4–V6, I, aVL
• Lateral: I and aVL
• Inferior: II, III, and aVF
• Inferolateral: II, III, aVF, and V5 and V6
Why perform an ECG?
• It’s part of the admission bundle
• Indicated by the patient’s symptoms
- symptoms of IHD/MI
- symptoms associated with dysrhythmias
• Indicated by the patient’s examination findings
- cardiac murmur
ECG Interpretation
• Quality of ECG?
• Rate
• Rhythm
• Axis
• P wave
• PR interval
• QRS duration
• QRS morphology
• Abnormal Q waves
• ST segment
• T wave
• QT interval
Quality of the ECG
• Patient name
• Date of the ECG
• Is there any interference?
• Is there electrical activity from all 12 leads?
Calibration
• Standard calibration of the ECG is 10mm/mV and normal speed 25mm/sec.
At this calibration, 1 miliVolt calibration signal is expected to produce a
rectangle of 10 mm height and 5 mm width.
• When ECG waves are tall, the R or S waves may extend into the QRS
complexes above or below them. To prevent this superimposition, the whole
ECG may be calibrated at 5mm/mV .
LOW VOLTAGE
Low voltage of the limb leads is present when the amplitude of the QRS
complex in each of the three standard limb leads (I, II, and III) is <5 mm . Low
voltage of all leads is diagnosed when the average voltage in the limb leads is
<5 mm and the average voltage in the chest leads is <10 mm.
An example of correct calibration at 10 mm/mV and speed of 25 mm/sec : The
calibration signal is a rectangle making 90 degrees of angles.
ECG interpretation
• Quality of ECG?
• Rate
• Rhythm
• Axis
• P wave
• PR interval
• QRS duration
• QRS morphology
• Abnormal Q waves
• ST segment
• T wave
• QT interval
Rate
• The 6 Second Rule: All but very slow heart rates can be determined by counting
the number of cycles that occur within 6 seconds, and then multiplying that
number by 10.
• The rule of 300 : 300/number of big squares between R waves
• Rate is either:
- Normal
– Bradycardia – HR < 40bpm
– Tachycardia HR > 120bpm
Rate
Rhythm
• Look for:
• Are there P waves?
• Are they regular?
• Does one precede every QRS complex?
Rythm could either be of sinus or non sinus in origin . Having P wave is sinus origin.
Without P wave or buried with preceeding T wave is either Venticular or Supraventricular
origin.
Axis
Definition:
the mean direction of electrical forces in the frontal plane ( limb leads) as measured
from the zero reference point (lead 1)
Normal values
P wave: 0 to 75 degrees
QRS complex: -30 to 90 degress
T wave: QRS-T angle <45 degrees frontal or <60 degrees precordial
The Quadrant Approach
• QRS up in I and up in aVF = Normal
Axis
Positive in I and II
= NORMAL
Positive in I and
negative in II = LAD
Negative in I and
positive in II = RAD
D/D of Right Axis Deviation (RAD)
• Differential diagnosis Right Ventricular Hypertrophy (RVH) — most common
• Left Posterior Fascicular Block (LPFB) — diagnosis of exclusion
• Lateral and apical MI
• Acute Right Heart Strain, e.g. acute lung disease such as pulmonary embolus
• Chronic lung disease, e.g. COPD
• Dextrocardia
• Ventricular pre-excitation (WPW) — LV free wall accessory pathway
• Ventricular ectopy
• Hyperkalemia
• Sodium-channel blockade, e.g. tricyclic toxicity
• Normal in infants and children
• Normal young or slender adults with a horizontally positioned heart can also
demonstrate a rightward QRS axis on the ECG.
D/D ofLeft Axis Deviation (LAD)
• left ventricular hypertrophy (LVH)
• Left Anterior Fascicular Block (LAFB) — diagnosis
of exclusion
• LBBB
• inferior MI
• ventricular ectopy
• paced beats
• Ventricular pre-excitation (WPW)
ECG interpretation
• Quality of ECG?
• Rate
• Rhythm
• Axis
• P wave
• PR interval
• QRS duration
• QRS morphology
• Abnormal Q waves
• ST segment
• T wave
• QT interval
P wave
• The P wave is the first positive deflection on the ECG
• It represents atrial depolarisation
Characteristics of the Normal Sinus P Wave
• Morphology
• Smooth contour
• Monophasic in lead II
• Biphasic in V1
• Axis
• Normal P wave axis is between 0° and +75°
• P waves should be upright in leads I and II, inverted in aVR
• Duration
• < 120 ms
• Amplitude
• < 2.5 mm in the limb leads,
• < 1.5 mm in the precordial leads
P wave abnormalities
Common P wave abnormalities include:
• P mitrale (bifid P waves), seen with left atrial
enlargement.
• P pulmonale (peaked P waves), seen with right atrial
enlargement.
• P wave inversion, seen with ectopic atrial and
junctional rhythms.
• Variable P wave morphology, seen in multifocal atrial
rhythms.
P mitrale
P pulmonale
PR interval
• The PR interval is the time from the onset of the P wave to the
start of the QRS complex.
• It reflects conduction through the AV node.
• Start of P wave to start of QRS complex
• Normal = 0.12 - 0.2 seconds (3-5 small squares)
• Decreased = can indicate an accessory pathway
• Increased = indicates AV block (1st
/2nd
/3rd
)
ECG interpretation
• Quality of ECG?
• Rate
• Rhythm
• Axis
• P wave
• PR interval
• QRS duration
• QRS morphology
• Abnormal Q waves
• ST segment
• T wave
• QT interval
QRS complex
Main Features to Consider
• Width of the complexes: Narrow versus broad.
• Voltage (height) of the complexes.
• Spot diagnoses: Specific morphology patterns that are important to
recognise.
• Normal = <0.12 seconds
• Narrow complexes (QRS < 100 ms) are supraventricular in origin.
• Broad complexes (QRS > 100 ms) may be either ventricular in origin, or
may be due to aberrant conduction of supraventricular complexes (e.g.
due to bundle branch block, hyperkalaemia or sodium-channel blockade)
Low Voltage QRS
• Low Voltage
• The QRS is said to be low voltage when:
• The amplitudes of all the QRS complexes in
the limb leads are < 5 mm; or
• The amplitudes of all the QRS complexes in
the precordial leads are < 10 mm
QRS complex
• Is there LVH?
• Probably the most commonly used are the Sokolov-Lyon criteria (S wave
depth in V1 + tallest R wave height in V5-V6 > 35 mm).
>35mm is suggestive of LVH
Q waves
• Q waves are allowed in V1, aVR & III
• Pathological Q waves can indicate previous MI
ECG interpretation
• Quality of ECG?
• Rate
• Rhythm
• Axis
• P wave
• PR interval
• QRS duration
• QRS morphology
• Abnormal Q waves
• ST segment
• T wave
• QT interval
ST segment
• The ST segment is the flat, isoelectric section of the ECG between the end of the S
wave and the beginning of the T wave.
• It represents the interval between ventricular depolarisation and repolarisation.
• The most important cause of ST segment abnormality (elevation or depression) is
myocardial ischaemia / infarction.
• ST depression (upsloping, horizontal and downsloping)
- downsloping or horizontal = ABNORMAL
• ST elevation
- infarction
- pericarditis (widespread)
ST segment depression
ST segment elevation
T wave
• Small = hypokalaemia
• Tall = hyperkalaemia
• Inverted/biphasic = ischaemia/previous infarct
• Hyperkalemia: Potassium reduces myocardial excitability, with
depression of pacemaking and conducting tissues.
• K+ >5.5 meq/l is a/w repolarization abnormalities causing peaked T waves
(earliest sign of hyperkalemia)
• K+>6.5 a/w progressive paralysis of artia , ECG shows wide and flat P
wave, incerase PR and P wave eventually dissapear.
• K+ more than 7 causes conduction abnormalities and bradycardia and
eventually asystole.
T wave
T wave
T wave
QT interval
• Start of QRS to end of T wave
• Needs to be corrected for HR
• Normal QTc = < 400ms
• Long QT can be genetic or iatrogenic
QT interval
Blocks
AV blocks
First degree block
PR interval fixed and > 0.2 sec
Second degree block, Mobitz type 1
PR gradually lengthened, then drop QRS
Second degree block, Mobitz type 2
PR fixed, but drop QRS randomly
Type 3 block
PR and QRS dissociated
Some important ECG examples
Lateral MI
Reciprocal changes
Inferolateral MI
ST elevation II, III, aVF
ST depression in aVL, V1-V3 are reciprocal changes
Anterolateral / Inferior Ischemia
LVH, AV junctional rhythm, bradycardia
Left Bundle Branch Block
Monophasic R wave in I and V6, QRS > 0.12 sec
Loss of R wave in precordial leads
QRS T wave discordance I, V1, V6
Consider cardiac ischemia if a new finding
Right Bundle Branch Block
V1: RSR prime pattern with inverted T wave
V6: Wide deep slurred S wave
First Degree Heart Block, Mobitz Type I (Wenckebach)
PR progressively lengthens until QRS drops
Supraventricular Tachycardia
Narrow complex, regular; retrograde P waves, rate <220
Retrograde P waves
Right Ventricular Myocardial Infarction
Found in 1/3 of patients with inferior MI
Increased morbidity and mortality
ST elevation in V4-V6 of Right-sided EKG
Ventricular Tachycardia
Prolonged QT
QT > 450 ms
Inferior and anterolateral ischemia
Second Degree Heart Block, Mobitz Type II
PR interval fixed, QRS dropped intermittently
Acute Pulmonary Embolism
SIQIIITIII in 10-15%
T-wave inversions, especially occurring in
inferior and anteroseptal simultaneously
RAD
Wolff-Parkinson-White Syndrome
Short PR interval <0.12 sec
Prolonged QRS >0.10 sec
Delta wave
Can simulate ventricular hypertrophy, BBB and previous MI
Hypokalemia
U waves
Can also see PVCs, ST depression, small T waves
THANK YOU

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Ecg interpretation

  • 1. ECG interpretation Dr Sudhir Dev House Officer Dept. Of GP and Emergency Medicine
  • 2. Objectives • Justify the reasons for performing an ECG • Develop a structured approach to interpreting an ECG • Practice interpreting ECGs
  • 3. The ECG “The ECG (electrocardiogram) is a transthoracic interpretation of the electrical activity of the heart.”
  • 4. The ECG SA node -> atrial muscle -> AV node -> bundle of His -> Left and Right Bundle Branches -> Ventricular muscle
  • 5.
  • 7. EKG Distributions • Anteroseptal: V1, V2, V3, V4 • Anterior: V1–V4 • Anterolateral: V4–V6, I, aVL • Lateral: I and aVL • Inferior: II, III, and aVF • Inferolateral: II, III, aVF, and V5 and V6
  • 8. Why perform an ECG? • It’s part of the admission bundle • Indicated by the patient’s symptoms - symptoms of IHD/MI - symptoms associated with dysrhythmias • Indicated by the patient’s examination findings - cardiac murmur
  • 9. ECG Interpretation • Quality of ECG? • Rate • Rhythm • Axis • P wave • PR interval • QRS duration • QRS morphology • Abnormal Q waves • ST segment • T wave • QT interval
  • 10. Quality of the ECG • Patient name • Date of the ECG • Is there any interference? • Is there electrical activity from all 12 leads?
  • 11. Calibration • Standard calibration of the ECG is 10mm/mV and normal speed 25mm/sec. At this calibration, 1 miliVolt calibration signal is expected to produce a rectangle of 10 mm height and 5 mm width. • When ECG waves are tall, the R or S waves may extend into the QRS complexes above or below them. To prevent this superimposition, the whole ECG may be calibrated at 5mm/mV . LOW VOLTAGE Low voltage of the limb leads is present when the amplitude of the QRS complex in each of the three standard limb leads (I, II, and III) is <5 mm . Low voltage of all leads is diagnosed when the average voltage in the limb leads is <5 mm and the average voltage in the chest leads is <10 mm.
  • 12. An example of correct calibration at 10 mm/mV and speed of 25 mm/sec : The calibration signal is a rectangle making 90 degrees of angles.
  • 13. ECG interpretation • Quality of ECG? • Rate • Rhythm • Axis • P wave • PR interval • QRS duration • QRS morphology • Abnormal Q waves • ST segment • T wave • QT interval
  • 14. Rate • The 6 Second Rule: All but very slow heart rates can be determined by counting the number of cycles that occur within 6 seconds, and then multiplying that number by 10. • The rule of 300 : 300/number of big squares between R waves • Rate is either: - Normal – Bradycardia – HR < 40bpm – Tachycardia HR > 120bpm
  • 15. Rate
  • 16. Rhythm • Look for: • Are there P waves? • Are they regular? • Does one precede every QRS complex? Rythm could either be of sinus or non sinus in origin . Having P wave is sinus origin. Without P wave or buried with preceeding T wave is either Venticular or Supraventricular origin.
  • 17. Axis Definition: the mean direction of electrical forces in the frontal plane ( limb leads) as measured from the zero reference point (lead 1) Normal values P wave: 0 to 75 degrees QRS complex: -30 to 90 degress T wave: QRS-T angle <45 degrees frontal or <60 degrees precordial
  • 18. The Quadrant Approach • QRS up in I and up in aVF = Normal
  • 19. Axis Positive in I and II = NORMAL Positive in I and negative in II = LAD Negative in I and positive in II = RAD
  • 20. D/D of Right Axis Deviation (RAD) • Differential diagnosis Right Ventricular Hypertrophy (RVH) — most common • Left Posterior Fascicular Block (LPFB) — diagnosis of exclusion • Lateral and apical MI • Acute Right Heart Strain, e.g. acute lung disease such as pulmonary embolus • Chronic lung disease, e.g. COPD • Dextrocardia • Ventricular pre-excitation (WPW) — LV free wall accessory pathway • Ventricular ectopy • Hyperkalemia • Sodium-channel blockade, e.g. tricyclic toxicity • Normal in infants and children • Normal young or slender adults with a horizontally positioned heart can also demonstrate a rightward QRS axis on the ECG.
  • 21. D/D ofLeft Axis Deviation (LAD) • left ventricular hypertrophy (LVH) • Left Anterior Fascicular Block (LAFB) — diagnosis of exclusion • LBBB • inferior MI • ventricular ectopy • paced beats • Ventricular pre-excitation (WPW)
  • 22. ECG interpretation • Quality of ECG? • Rate • Rhythm • Axis • P wave • PR interval • QRS duration • QRS morphology • Abnormal Q waves • ST segment • T wave • QT interval
  • 23. P wave • The P wave is the first positive deflection on the ECG • It represents atrial depolarisation Characteristics of the Normal Sinus P Wave • Morphology • Smooth contour • Monophasic in lead II • Biphasic in V1 • Axis • Normal P wave axis is between 0° and +75° • P waves should be upright in leads I and II, inverted in aVR • Duration • < 120 ms • Amplitude • < 2.5 mm in the limb leads, • < 1.5 mm in the precordial leads
  • 24. P wave abnormalities Common P wave abnormalities include: • P mitrale (bifid P waves), seen with left atrial enlargement. • P pulmonale (peaked P waves), seen with right atrial enlargement. • P wave inversion, seen with ectopic atrial and junctional rhythms. • Variable P wave morphology, seen in multifocal atrial rhythms.
  • 25.
  • 28. PR interval • The PR interval is the time from the onset of the P wave to the start of the QRS complex. • It reflects conduction through the AV node. • Start of P wave to start of QRS complex • Normal = 0.12 - 0.2 seconds (3-5 small squares) • Decreased = can indicate an accessory pathway • Increased = indicates AV block (1st /2nd /3rd )
  • 29. ECG interpretation • Quality of ECG? • Rate • Rhythm • Axis • P wave • PR interval • QRS duration • QRS morphology • Abnormal Q waves • ST segment • T wave • QT interval
  • 30. QRS complex Main Features to Consider • Width of the complexes: Narrow versus broad. • Voltage (height) of the complexes. • Spot diagnoses: Specific morphology patterns that are important to recognise. • Normal = <0.12 seconds • Narrow complexes (QRS < 100 ms) are supraventricular in origin. • Broad complexes (QRS > 100 ms) may be either ventricular in origin, or may be due to aberrant conduction of supraventricular complexes (e.g. due to bundle branch block, hyperkalaemia or sodium-channel blockade)
  • 31. Low Voltage QRS • Low Voltage • The QRS is said to be low voltage when: • The amplitudes of all the QRS complexes in the limb leads are < 5 mm; or • The amplitudes of all the QRS complexes in the precordial leads are < 10 mm
  • 32. QRS complex • Is there LVH? • Probably the most commonly used are the Sokolov-Lyon criteria (S wave depth in V1 + tallest R wave height in V5-V6 > 35 mm). >35mm is suggestive of LVH
  • 33. Q waves • Q waves are allowed in V1, aVR & III • Pathological Q waves can indicate previous MI
  • 34. ECG interpretation • Quality of ECG? • Rate • Rhythm • Axis • P wave • PR interval • QRS duration • QRS morphology • Abnormal Q waves • ST segment • T wave • QT interval
  • 35. ST segment • The ST segment is the flat, isoelectric section of the ECG between the end of the S wave and the beginning of the T wave. • It represents the interval between ventricular depolarisation and repolarisation. • The most important cause of ST segment abnormality (elevation or depression) is myocardial ischaemia / infarction. • ST depression (upsloping, horizontal and downsloping) - downsloping or horizontal = ABNORMAL • ST elevation - infarction - pericarditis (widespread)
  • 38. T wave • Small = hypokalaemia • Tall = hyperkalaemia • Inverted/biphasic = ischaemia/previous infarct • Hyperkalemia: Potassium reduces myocardial excitability, with depression of pacemaking and conducting tissues. • K+ >5.5 meq/l is a/w repolarization abnormalities causing peaked T waves (earliest sign of hyperkalemia) • K+>6.5 a/w progressive paralysis of artia , ECG shows wide and flat P wave, incerase PR and P wave eventually dissapear. • K+ more than 7 causes conduction abnormalities and bradycardia and eventually asystole.
  • 42. QT interval • Start of QRS to end of T wave • Needs to be corrected for HR • Normal QTc = < 400ms • Long QT can be genetic or iatrogenic
  • 44. Blocks AV blocks First degree block PR interval fixed and > 0.2 sec Second degree block, Mobitz type 1 PR gradually lengthened, then drop QRS Second degree block, Mobitz type 2 PR fixed, but drop QRS randomly Type 3 block PR and QRS dissociated
  • 45. Some important ECG examples
  • 47. Inferolateral MI ST elevation II, III, aVF ST depression in aVL, V1-V3 are reciprocal changes
  • 48. Anterolateral / Inferior Ischemia LVH, AV junctional rhythm, bradycardia
  • 49. Left Bundle Branch Block Monophasic R wave in I and V6, QRS > 0.12 sec Loss of R wave in precordial leads QRS T wave discordance I, V1, V6 Consider cardiac ischemia if a new finding
  • 50. Right Bundle Branch Block V1: RSR prime pattern with inverted T wave V6: Wide deep slurred S wave
  • 51. First Degree Heart Block, Mobitz Type I (Wenckebach) PR progressively lengthens until QRS drops
  • 52. Supraventricular Tachycardia Narrow complex, regular; retrograde P waves, rate <220 Retrograde P waves
  • 53. Right Ventricular Myocardial Infarction Found in 1/3 of patients with inferior MI Increased morbidity and mortality ST elevation in V4-V6 of Right-sided EKG
  • 55. Prolonged QT QT > 450 ms Inferior and anterolateral ischemia
  • 56. Second Degree Heart Block, Mobitz Type II PR interval fixed, QRS dropped intermittently
  • 57. Acute Pulmonary Embolism SIQIIITIII in 10-15% T-wave inversions, especially occurring in inferior and anteroseptal simultaneously RAD
  • 58. Wolff-Parkinson-White Syndrome Short PR interval <0.12 sec Prolonged QRS >0.10 sec Delta wave Can simulate ventricular hypertrophy, BBB and previous MI
  • 59. Hypokalemia U waves Can also see PVCs, ST depression, small T waves

Editor's Notes

  1. Cardiac conducting system
  2. It’s vital to have a system in place to interpret the ECG.
  3. Small square 0.04s; Large square 0.2s
  4. It’s vital to have a system in place to interpret the ECG.
  5. Can use lead II
  6. The normal axis is around 60 degrees.
  7. It’s vital to have a system in place to interpret the ECG.
  8. Not very useful signs.
  9. Short PR interval can be accessory pathway or can be normal
  10. It’s vital to have a system in place to interpret the ECG.
  11. It’s vital to have a system in place to interpret the ECG.
  12. Tall = can be normal young man
  13. T wave more than 10mm size= hyperkalemia
  14. Long QT syndrome. Amiodarone, sotalol.
  15. Long QT syndrome is associated with Torsades de pointes
  16. Resiprocal: Reciprocal changes are simple the opposite deflection in an opposing lead, for example a posterior infarction causing ST depression in the anterior leads.
  17. Mobitz type I, RBBB, LVH