INTRODUCTION
DEFINITION
TYPES OF TRAUMA FROM OCCLUSION
GLICKMAN CONCEPT
WAERHAUG CONCEPT
STAGES OF TISSUE RESPONSE TO INJURY
CLINICAL AND RADIOGRAPHIC FEATURES OF TFO
CLINICAL DIAGNOSIS OF TFO
TFO AND IMPLANTS
TREATMENT OF TFO
CONCLUSION
REFRENCES
Periodontitis is a complex infection initiated by bacteria –tissue destruction.
Host: the organism from which a parasite obtains its nourishment/ an individual who receives a graft
Modulation: the alteration of function or status of something in response to a stimulus or an altered physical or chemical environment
INTRODUCTION
DEFINITION
TYPES OF TRAUMA FROM OCCLUSION
GLICKMAN CONCEPT
WAERHAUG CONCEPT
STAGES OF TISSUE RESPONSE TO INJURY
CLINICAL AND RADIOGRAPHIC FEATURES OF TFO
CLINICAL DIAGNOSIS OF TFO
TFO AND IMPLANTS
TREATMENT OF TFO
CONCLUSION
REFRENCES
Periodontitis is a complex infection initiated by bacteria –tissue destruction.
Host: the organism from which a parasite obtains its nourishment/ an individual who receives a graft
Modulation: the alteration of function or status of something in response to a stimulus or an altered physical or chemical environment
Phase I periodontal therapy is the first in the chronologic sequence of procedures that constitute periodontal treatment. It is also referred to as cause related therapy or non-surgical periodontal therapy.
A brief description of all topics to recent advances,SDD, host modulation and diabetes, host modulation in smokers, chemically modified tetracyclines, bisphosphonates
Phase I periodontal therapy is the first in the chronologic sequence of procedures that constitute periodontal treatment. It is also referred to as cause related therapy or non-surgical periodontal therapy.
A brief description of all topics to recent advances,SDD, host modulation and diabetes, host modulation in smokers, chemically modified tetracyclines, bisphosphonates
Surgical v/s Non surgical periodontal therapy Achi Joshi
Both surgical and nonsurgical therapy produced improvement in the periodontal health.
Treatment approach was based on the comfort level of the practitioner.
In the late 60’s and continuing into the 70’s and 80’s, many series of longitudinal studies were conducted, aimed to document the immediate and most importantly long term clinical results following several types of periodontal therapy.
This presentation focusses on definition, history, goals and objectives of SPT, patient compliance, ways to improve patient's compliance, parts of SPT, SPT in daily practice, classification of post treatment patients, AAP Guidelines for periodontist and dentist and studies related to SPT.
THE EFFECTS OF PERIODONTAL TREATMENT ON DIABETIC PATIENTS: THE DIAPERIO RANDO...sDJKFhjh
While many systemic diseases have been considered to be associated with periodontitis, the link between diabetes and periodontitis has been the subject of the most intensive research over the past decades.
Both periodontitis and diabetes are highly prevalent in modern societies and have a negative impact on quality of life.
Periodontal therapy might help to reduce local inflammation, and consequently, the putative systemic entry of bacterial by- products (e.g., lipopolysaccharides from Gram - negative bacterial walls) and pro- inflammatory molecules.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
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The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
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2. Definition
• Procedures performed at selected intervals to assist the
periodontal patient in maintaining oral health. -GPT 2001.
• Therapeutic measures to support the patient’s own efforts to
control and to avoid re‐infection. - Jan Lindhe.
4. Rationale for SPT
1. Limitation of mechanical subgingival debridement
2. Recolonization of pocket
3. Long JE (weak attachment)
4. Subgingival scaling alters the microflora of periodontal
pockets
5. To prevent interception
5. Goals Of SPT
• 3 main goals according to the AAP position paper
(1998)
1. To prevent or minimize the recurrence and
progression of periodontal disease in patients who
have been previously treated for gingivitis,
periodontitis and for peri-implantitis.
6. Goals Of SPT
2. To prevent or reduce the incidence of tooth loss by
monitoring the dentition
3. To locate and treat other diseases or conditions found in the
oral cavity in a timely manner
7. Objectives of SPT
Periodontal
• Preservation of bone
support
• Maintenance of stable
CAL.
• Reinforcement of proper
home care.
• Maintenance of a healthy
and functional oral
environment
General
• Assessment of the general
health status
• Encouragement of
patients oral hygiene
efforts
• Continuation of patient
education
• Establishment of the
future maintenance
regime
8. Types of SPT • Schallhorn and Snider (1981)
1. Preventive maintenance therapy
• Periodontally healthy individuals.
2. Trial maintenance therapy
• Mild to moderate periodontitis
3. Compromised maintenance therapy
• Medically compromised patients where active
therapy is not possible.
4. Post-maintenance treatment therapy
• maintenance for prevention of recurrence of
disease
9. Maintenance Program (Carranza)
SPT
1. Examination and
Evaluation (14 mins.)
2. Maintenance Rx and
oral hygiene
reinforcement (36 mins.)
3. Report, Cleanup, and
Scheduling (10 mins.)
10. Part- 1: Examination and Evaluation
• Patient greeting
• Medical history changes
• Oral hygiene status
• Gingival changes
• Pocket depth changes
• Mobility changes
• Occlusal changes
• Dental caries
• Oral pathologic examination
• Restorative, prosthetic, and implant status
Examination of
prosthesis/abutment
components;
Evaluation of
implant stability;
Occlusal
examination;
Other signs and
symptoms of
disease activity.
11. Radiographic Examination of Recall Patients
Clinical caries and no high-risk factors
for caries.
Posterior bite-wing examination at
24 to 36-month intervals.
Clinical caries or high-risk
factors for caries
Posterior bite-wing examination at
12 to 18-month intervals.
History of periodontal treatment with
disease under good control.
Bite-wing examination every 24 to 36 months; full-
mouth series every 5 years.
Periodontal disease not under good
control.
Periapical and/or vertical bite-wing radiographs of
problem areas every 12 to 24 months; full-mouth
series every 3 to 5 years
Root form
dental implants
Periapical or vertical bite-wing
radiographs at 6, 12, and 36
months after prosthetic placement,
then every 36 months unless
clinical problems arise.
13. Part-2: Maintenance treatment and
Oral hygiene reinforcement
• Oral hygiene reinforcement
• Scaling
• Polishing
• Chemical irrigation or site-specific antimicrobial placement
14. Part-3: Report, Cleanup, and Scheduling
• Write report in chart.
• Discuss report with patient.
• Clean and disinfect operatory.
• Schedule next recall visit.
• Schedule further periodontal treatment.
• Schedule or refer for restorative or prosthetic treatment.
15. Frequency of SPT
• Ramfjord et al. (1993) For most patients with gingivitis but no
previous attachment loss, supportive periodontal treatment
twice a year.
• For patients with a previous history of periodontitis, 3 months
interval (less than 6 months)
• Various clinical trails suggest- four times a year.
20. Effectiveness of SPT
(for patients with Gingivitis)
• Axelsson and Lindhe (1974-1976): demonstrated that
gingivitis could be prevented by regular professional cleaning in
children aged 7 to 14 years.
• Axelsson and Lindhe (1981): reduced caries and BOP
• Badersten et al.(1990): Routine mechanical subgingival
debridement of shallow bleeding sites at SPT visits results in
attachment loss.
21. • Gingival conditions improved by 60% and tooth loss
was reduced by about 50% (Lovdal et al. 1961)
• The mean loss of probing attachment was only 0.08
mm per surface as opposed to 0.3 mm in the
control group (Suomi et al. 1971).
22. Effectiveness of SPT
(for patients with Periodontitis)
• Nyman et al. (1977) reported on 25 patients for 2 years (No SPT)
• Regular SPT at 3-4 times a year allows
disease monitoring
• Early detection and Rx Nyman et al.
(1975)
Test
OHI and SPT
2 weeks
Control
OHI and SPT
6 months
23. • Dahlen et al. (1992) Professionally delivered supragingival
toothcleaning, in combination with self-performed plaque
control for 2-year period effectively change the quantity and the
composition of the subgingival microbiota
• Hellstrom et al. (1996) same protocol has a significant effect
on the subgingival microbiota of moderate to deep periodontal
pockets clinically.
24. • (Lindhe & Nyman 1984)
• 75 patients with extremely advanced periodontitis,
• recurrent infection occurred in only very few sites during a
14‐year period of effective SPT.
• recurrent periodontitis was noted at completely unpredictable
time intervals, in about 25% of the patient population (15 of 61)
25. 30‐years plaque‐control-based maintenance program
• Axelsson et al. 2004
375 test and 180 control
The test group- prophylactic visits every second month
for the first 2 years and every 3–12 months (according
to their individual needs) over 3–30 years
very few teeth were lost (0.4–1.8)
1.2–2.1 new carious lesions (>80% secondary caries)
2–4% of all sites exhibited attachment loss of ≥2 mm.
26. Compliance affecting SPT
• Wilson stated that “Patients
who comply to suggested
recall visits are periodontally
healthy and keep their teeth
longer.”
27. Improving compliance
• Wilson suggests:
• Counselling them about their condition,
the role of treatment and the importance of compliance
• Simplify instructions to patients
• Teach them self performed plaque control
• Acccommodating patient needs
• Positive reinforcement
30. Maintenance intervals for implant patients
1. Patients with both teeth and implants should see the
periodontist as often as necessary to keep the
periodontium and peri-implant tissues healthy.
2. Totally edentulous patients with implants should be
seen at least once per year.
31. Conclusion and recommendations
• SPT should be based on profile of risk assessment
• No scientific evidence for subgingival debridement of sites with
BOP without concomitant increase in pocket depth. (it should
better be avoided)
• SPT minimizes the risk of periodontal disease progression and
tooth mortality
• In absence of long-term evaluation of SPT for dental implants,
same principles of SPT that is used for periodontitis is also
apropriate
32. Recommendation for research
• Studies are needed:
1. to evaluate the efficacy of supragingival Rx alone as
compared to subgingival debridement
2. To assess the value of antibiotics as adjunct and stand alone
Rx during SPT
3. Patient based factors must be considered in analysis
4. Prospective and multicenter studies to findout efficacy of SPT