mucogingival surgery or plastic surgery of muco-gingival tissue is a surgical procedure targeted to correct and eliminate anatomic, developmental and traumatic alterations of gingiva.
A brief description of all topics to recent advances,SDD, host modulation and diabetes, host modulation in smokers, chemically modified tetracyclines, bisphosphonates
Certains medications have been associated with gingival enlargement.
the seminar gives a complete analysis of etilogy and pathogenesis involved in digo as well as sequlae of it
Periodontitis is a complex infection initiated by bacteria –tissue destruction.
Host: the organism from which a parasite obtains its nourishment/ an individual who receives a graft
Modulation: the alteration of function or status of something in response to a stimulus or an altered physical or chemical environment
REFERENCES TAKEN FROM CARRANZA'S TEXTBOOK OF CLINICAL PERIODONTOLOGY AND LINDHE'S TEXTBOOK OF CLINICAL PERIODONTOLOGY AND IMPLANT DENTISTRY. CONTAINS ENOUGH AND MORE DETAILS OF THIS TOPIC FOR BDS STUDENTS.HOPE THIS PRESENTATION WILL HELP U GAIN SOME KNOWLEDGE ABOUT PERIODONTAL PLASTIC AND ESTHETIC DENTISTRY.
Furcation involvement is a common sequela of severe chronic periodontal disease. Its effective management has a profound influence on the outcome of periodontal therapy.
The periodontal dressing is a physical barrier that is placed in the surgical site to protect the healing tissues from the forces produced during mastication, for comfort and close adaptation.
Classification of peri-implant diseases and condition, implant failures causes, Peri-implant mucositis and its management, Peri-implantitis and its classification, clinical features, Treatment (Surgical and Non-surgical) And management. Implant success rate and conclusion.
mucogingival surgery or plastic surgery of muco-gingival tissue is a surgical procedure targeted to correct and eliminate anatomic, developmental and traumatic alterations of gingiva.
A brief description of all topics to recent advances,SDD, host modulation and diabetes, host modulation in smokers, chemically modified tetracyclines, bisphosphonates
Certains medications have been associated with gingival enlargement.
the seminar gives a complete analysis of etilogy and pathogenesis involved in digo as well as sequlae of it
Periodontitis is a complex infection initiated by bacteria –tissue destruction.
Host: the organism from which a parasite obtains its nourishment/ an individual who receives a graft
Modulation: the alteration of function or status of something in response to a stimulus or an altered physical or chemical environment
REFERENCES TAKEN FROM CARRANZA'S TEXTBOOK OF CLINICAL PERIODONTOLOGY AND LINDHE'S TEXTBOOK OF CLINICAL PERIODONTOLOGY AND IMPLANT DENTISTRY. CONTAINS ENOUGH AND MORE DETAILS OF THIS TOPIC FOR BDS STUDENTS.HOPE THIS PRESENTATION WILL HELP U GAIN SOME KNOWLEDGE ABOUT PERIODONTAL PLASTIC AND ESTHETIC DENTISTRY.
Furcation involvement is a common sequela of severe chronic periodontal disease. Its effective management has a profound influence on the outcome of periodontal therapy.
The periodontal dressing is a physical barrier that is placed in the surgical site to protect the healing tissues from the forces produced during mastication, for comfort and close adaptation.
Classification of peri-implant diseases and condition, implant failures causes, Peri-implant mucositis and its management, Peri-implantitis and its classification, clinical features, Treatment (Surgical and Non-surgical) And management. Implant success rate and conclusion.
Dr. Rahul VC Tiwari - Fellowship In Orthognathic Surgery - Jubilee Mission Medical College Hospital and Research Center, Thrissur, Kerala - 31st publication IJAR 1st name
This seminar deals with implant-related complications that lead to implant failure.this also discus diagnostic criteria and preventive methods for an implant failure.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
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2. Definition
Peri-implant disease- Peri-implant disease is a collective
term for inflammatory processes in the tissues surrounding
an implant - Albrektsson & Isidor 1993, 1st EWP, Switzerland.
Definition of Mombelli A, Lang N.P 1998- pathological
inflammatory changes that take place in the tissue
surrounding a load bearing implant
5. Peri-implant mucositis
Albrektsson & Isidor (1994) reversible inflammatory reactions in
the soft tissues surrounding a functioning implant.
According to Kostovillis (2008) Inflammatory changes which are
confined to the soft tissue surrounding an implant with no signs of
loss of supporting bone.
Roos-Jansaker AM et al. prevalence is 48% of implants followed
from 9 to 14 years
7. Histopathology
Response to early plaque formation
Pontoriero et al. (1994)- Inflammation and probing
depth change over a period of 3 weeks
Zitzmann et al. (2001) also concluded that at the end
of 3 weeks of plaque built-up, increase in size of
peri-implant mucosa from 0.03 mm2 at baseline to
0.2 mm2
Proportions of neutrophills increased in CT
8. Response to long standing plaque formation
Ericsson et al.- Inflammatory response same
In gingival tissues amount of tissue breakdown that
occurs during the 3 month interval is more or less
fully compensated by the tissue built up during the
subsequent phases of repair.
In the lesion within the peri-implant mucosa, the
tissue breakdown is not fully recovered by reparative
events. (reduced tissue built up)
Histopathology
9. Concluding remark
Shares similarity with gingivitis in terms of host
response and development of clinical signs.
it represents an obvious precursor to peri-implantitis.
Early detection is essential.
10. Peri-implantitis
The term “Peri-implantitis” was introduced in the late
1980s (Mombelli et al. 1987) and was subsequently
defined as “an inflammatory process affecting the
soft and hard tissues around a functioning
osseointegrated implant, resulting in loss of
supporting bone” (Albrektsson & Isidor 1994).
11. In the consensus report from the 6th European
Workshop on Periodontology- peri-implant
mucositis was an inflammatory lesion that resides
in the mucosa, while peri-implantitis also affects
the supporting bone”. (Lindhe & Meyle 2008,
Zitzmann & Berglundh 2008).
Peri-implantitis- progressive loss of supporting
bone beyond biological bone remodeling.
Consensus report of Working Group JCP 2012
13. Etiopathogenesis
Biofilm formation
Staph. Aureus for initiation and host response is
overwhelmed by gram –ve bacteria.
The connective exhibit B-lymphocytes and plasma
cell infiltration
14. Etiopath…
The rate of disease progression and the severity of
inflammatory signs different than periodontitis
The increased susceptibility for bone loss around
implants may be related to the absence of inserting
collagen fibers into the implant
spontaneous continuous progression of the disease
with additional bone loss
17. Classification
Newman and Flemming (1992) have proposed a
classification of non successful implants, based on the
severity of peri-implantitis:
1. “Compromised successful implant” characterized by
inflammation, hyperplasia, fistula formation occurring
near an otherwise fully osseointegrated implant.
2. “Failing implant” characterized by progressive bone
resorption, but the implant remains functional.
3. “Failed implant” in which infection persists around an
implant whose function is compromised.
19. Based on radiographic presentation of peri-
implant bone loss as 5 main types:
Zhang L, Geraets W, Zhou Y, et al, 2014
20. Diagnosis of peri-implantitis
The examination of peri-implant tissues should
include:-
1. Evaluation of oral hygiene standard.
Modified plaque index- Mombelli et al.
2. Evaluation of peri-implant marginal tissues.
Probing, bleeding and suppuration
3. Evaluation of bone-implant interface
Radiographs and mobility
21. Probing around implants
Initial probing immediately before installing final
restoration using 0.25N probing force
Gentle probing resulting in bleeding suggests the
presence of soft tissue inflammation
presence of suppuration/exudate indicates
pathological changes
Increasing probing depth and bleeding are
indicators for additional radiographic examination
22. Radiographs
IOPA following placement and then following the
prosthesis installation should function as the
baseline
Bone loss can have a number of nonbacterial
causes including surgical technique, implant
design, implant position, crestal thickness of bone,
loose prosthesis/abutment, and excessive occlusal
force
24. Treatment
Objectives in the therapy of peri-implantitis:
1. The removal of bacterial plaque within the peri-
implant pocket.
2. The decontamination and conditioning of the
implant surface.
3. Elimination of the sites that cannot be maintained
plaque-free by oral-hygiene procedures.
4. The establishment of an effective maintenance
program.
25. CIST
Cummulative interceptive and supportive therapy
Cumulative therapy depending on the clinical and
radiographic diagnosis
26. Nonsurgical approach
1. Mechanical debridement
Hand Instruments coated with titanium, carbon fiber,
polytetrafluoroethylene, plastic, polyetheretherketone,
or silicon.
Ultrasonic tips or polishing cups coated with carbon
fiber or plastic
Air abrasive systems that use low abrasive amino acid,
glycine powder
27. 2. Occlusal therapy
An analysis of the fit of the prosthesis
Prosthesis design changes, improvement in
implant number and occlusal equilibration can
contribute to the arrest of peri-implant tissue
breakdown progression
29. Laser
• The commonly used lasers for the decontamination of
the implant surface are:
Nd:YAG (1064 nm),
Erbium:yttrium-aluminium garnet(Er:YAG)(2940 nm),
Diode (660 nm), and
Carbon dioxide (10600 nm) lasers
• Er:YAG laser could remove the bacterial-contaminated
titanium oxide layer, thus promoting reosseointegration
Nevins M, Nevins ML, Yamamoto A, et al. 2014
30. Photodynamic therapy
• The activation of these dyes, such as toluidine
blue-O, using specific wavelength of light
(630– 700 nm) causes the release of oxygen
radicals that will decimate periodontal
pathogens.
Konopka K, Goslinski T. 2007
31. Surgical interventions
1. ACCESS FLAP
The objective of the access flap is to gain
access to submucosal implant surface for
debridement and decontamination
32. 2. Implantoplasty
• Clinical trial reported that implants treated with implantoplasty had
a higher implant survival rate compared with those that were treated
with an apically positioned flap only
Romeo E, Ghisolfi M, Murgolo N, et al, 2005
33. • 2 gm amox 1 hr prior to surgery
• FTF to expose the area
• Debride the defect with titanium or plastic curettes
• Air powder abrasive (Bicarbonate powder) for 60 Sec
• 60 sec irrigation with sterile saline
• 60 sec application of tetracycline
• Defect filled with Bone Graft
• Membranes are placed to cover all surfaces
• Flap released and coronally advanced and sutured.
REGENERATIVE APPROACH
34.
35. • The effectiveness of 4 surgical procedures (access
flap and debridement alone, Surgical resection,
regeneration with bone grafts, and guided bone
regeneration) were studied in a systematic review
and meta-analysis
• Each of the 4 procedures yielded roughly 2 to 3
mm PD reduction
• 2-mm increase in bone height was associated with
the regenerative procedures in a systematic review
Chan HL, Lin GH, Suarez F, et al 2014
36. MAINTENANCE
Needs to be individually determined
Needs to be enforced by doctor and Hygienist
Patient need to assume responsibility
Low Risk Patients
-Highly motivated
-Excellent oral hygiene
-One or two implants
-No associative risk
factors
Moderate Risk
Patients
-Loss of motivation
-Fair oral hygiene
-3-6 implants
-Moderate smoker
(half pack)
-Controlled medical
issues
High Risk Patients
-Unmotivated
-Poor oral hygiene
-Previous periodontitis
->6 implants
-Smokers more than
half pack
-Poorly controlled
systemic diseases
37.
38. There is no single superior antiinfective method available.
Surgical interventions achieved greater probing depth
reduction and clinical attachment gain compared with
nonsurgical
Access flap surgery shows resolution in only 58% of the
lesions.
The combination of resective and regenerative surgical
techniques seemed to have favorable treatment outcomes in
the management of periimplantitis.
Reosseointegration of a previously contaminated implant
surface is possible but highly variable and unpredictable.
39. Summery & Conclusion
Similarity between periodontal and peri-implant
diseases
Early diagnosis of peri-implantitis is imperative
Several risk factors exist for the development of peri-
implantitis, which can guide patient selection and
treatment planning.
Treatment of peri-implantitis should be tailored to the
severity of the lesion (as outlined by the CIST
protocol), which ranges from non surgical to surgical
approach
40.
41.
42. Refrences
1. Rosen P, Clem D, Cochran D, et al. Peri-implant mucositis and peri-
implantitis: a current understanding of their diagnoses and clinical
implications. J Periodontol 2013;84(4):436–43.
2. Froum SJ, Rosen PS. A proposed classification for peri-implantitis.
Int J Periodontics Restorative Dent 2012;32(5):533–40.
3. Schwarz F, Herten M, Sager M, et al. Comparison of naturally
occurring and ligature-induced peri-implantitis bone defects in
humans and dogs. Clin Oral Implants Res 2007;18(2):161–70.
4. Zhang L, Geraets W, Zhou Y, et al. A new classification of peri-
implant bone morphology: a radiographic study of patients with
lower implant-supported mandibular overdentures. Clin Oral
Implants Res 2014;25(8):905–9.
43. 5. Padial-Molina M, Suarez F, Rios HF, et al. Guidelines for the
diagnosis and treatment of peri-implant diseases. Int J
Periodontics Restorative Dent 2014;34(6):e102–11.
5. Saaby M, Karring E, Schou S, et al. Factors influencing severity
of peri-implantitis. Clin Oral Implants Res 2014.
6. Heitz-Mayfield LJ. Peri-implant diseases: diagnosis and risk
indicators. J Clin Periodontol 2008;35(8 Suppl):292–304.
7. Jia-Hui Fu, Hom-Lay Wang. Can Periimplantitis Be Treated?
Dent Clin N Am.2015:59;951–980.
Editor's Notes
Retrograde peri-implantitis is defined as a radiographically diagnosed, periapical, lucent lesion that is symptomatic and that develops shortly after implant placement.
self-limiting” process existing in the tissues
Boneloss on the buccal and lingual surfaces of implant
Protocol of access flap
Elevation of full thickness flap
Debridement of the implant surface Decontamination
Postoperative antimicrobial and antiseptic mouth rinses.
Increase in bone height may not be reosseointegrated bone but merely bone filling a space. Even nonosseointegrated bone in a treated lesion could be an achievable treatment goal that would provide for more favorable maintenance, esthetics, and implant stability compared with a lack of new bone.