This document discusses bone destruction patterns caused by periodontal disease. It identifies the main causes of bone destruction as the extension of gingival inflammation, trauma from occlusion, and systemic disorders. It describes several patterns of bone loss seen in periodontal disease, including horizontal, vertical, osseous craters, bulbous contours, reversed architecture, ledges, and furcation involvement. The rate and episodic nature of bone destruction in periodontal disease is also covered.

1. BY-
J.RAHUL RAGHAVENDER
IV YEAR

2. INTRODUCTIO
N- The height and density of alveolar bone are
normally maintained by an equilibrium.
 Regulated by local and systemic
influences , between bone formation and
resorption.
 When resorption exceeds formation, both
bone height and density is reduced

3. Causes of bone destruction-
(in periodontal disease)
1) Extension Of Gingival Inflammation
2) Trauma from occlusion(TFO)
3) Systemic disorders

4. 1) Bone Destruction Caused By
Extension Of Gingival
Inflammation
 “Periodontitis is always preceded by gingivitis,
but not all gingivitis progress to periodontitis”
 Most common cause
 Extension of inflammation
from marginal gingiva to
supporting tissues.

5.  The transition from gingivitis to
periodontitis is associated with changes
in composition of bacterial plaque.
GINGIVITIS PERIODONTITIS
COMP. OF
PLAQUE
Coccoid rods ,
straight rods
spirochetes
CELLS
(By Heijl)
Plasma cells PMNs

6.  HISTOPATHOLOGY :
Area of inflammation extending from
gingiva into suprabony area.
course : along collagen bundle fibres,
blood vessels, loosely arranged tissues.

7. PATHWAYS OF
INFLAMMATION :
Facially and
Lingually,
1) Gingiva to outer
periosteum
2) Periosteum to
bone
3) Gingiva to PDL

8. After inflammation reaches the bone, it spreads into
the marrow spaces and replaces the marrow with a
leukocytic and fluid exudate, new blood vessels and
proliferating fibroblasts.
Multinuclear osteoclasts and mononuclear
phagocytes increase in number, and the bone
surfaces appear, lined with Howship lacunae.
In the marrow spaces, resorption proceeds from
within, causing a thinning of the surrounding bony
trabeculae and enlargement of the marrow spaces,
followed by destruction of the bone and a reduction
in bone height .

9. RADIUS OF ACTION-

10. RATE OF BONE LOSS
 Loe et al in 1986 , found rate of
bone loss to average about
# 0.2 mm/year for facial surfaces
# 0.3 mm/year for proximal
when periodontal disease was
allowed to progress untreated .

11. PERIODS OF DESTRUCTION-
Periodontal destruction occurs in
episodic and intermittent manner.
Periods of inactivity &
destruction.
Destructive activity , results in,
loss of collagen & alveolar bone.
Followed by an advanced host
defense that controls the attack.

12. MECHANISM OF BONE
DESTRUCTION-
Bone destruction
Bacterial
Differentiation of
bone progenitor
cells into
osteoclasts
Inhibit action of
osteoblasts
Host-mediated
Releases PGE2,
IL-1α,IL-1β,TNF-α

13. 2) BONE DESTRUCTION
CAUSED BY TRAUMA FROM
OCCLUSION -
 Periodontal response to the
external force.
 TFO can occur in presence or
absence of inflammation.
 In the absence, effects on
alveolar bone ranges from
resorption to necrosis.
 Persistent TFO results in
angular defects of the bone.

14.  When combined with inflammation,
i.e. ZONE OF CO-DESTRUCTION,
 Plaque induced inflammation entering into the zone of
trauma from occlusion(supporting structures).
 Results in angular bone defects, bizarre bone pattern.

15. 3) BONE DESTRUCTION
CAUSED BY SYSTEMIC
DISORDERS -
 Possible relationship between periodontal bone
loss and systemic disorders.
 OSTEOPOROSIS : loss of bone mineral content
and structural bone changes. Risk factors-
ageing,smoking,etc
 OSTEOPENIA : tooth mobility and tooth loss
 Hyperparathyroidism, leukopenia

16. FACTORS DETERMINING BONE
MORPHOLOGY IN PERIODONTAL
DISEASE-
1) Normal variation of alveolar bone :
a) thickness,width,crestal angulation of
interdental septa
b) thickness of facial & lingual septa
c) presence of fenestrations &
dehiscences

17. 2) Exostoses :
a) overgrowths of bone
b) they can occur as small or large
nodules, sharp ridges , spike-like
projections.

18. 3) TFO :
a) thickening of cervical margin of
alveolar bone.
b) angular defects or buttressing bone.

19. c) buttressing bone formation occurs
during the repair phase of TFO
d) host reinforces thin trabeculae with
new bone
e) when it occurs within the jaw , it is
central buttressing bone formation.
f) when it occurs on external surface,
peripheral buttressing bone formation.
g) results in bulbous bone
contours(lipping) and osseous craters.

20. 4) Food impaction :
a) interdental bone defects occur
when there is abnormal or absence of
proximal contact.
b) food impaction here , results in
inverted bone architecture.
5) Aggressive periodontitis :
a) vertical or angular bone defects.

21. BONE DESTRUCTION
PATTERN -
1) Vertical or angular defects
2) Osseous craters
3) Bulbous bone contours
4) Reverse architecture
5) Ledges
6) Furcation involvement

22.  Horizontal bone loss :
a) most common pattern
b) bone height reduced, but margin
remains perpendicular to tooth surface.

23.  Vertical bone loss :
a) angular defects , occur in an oblique
direction
b) leads to hollowed-out trough in the bone
alongside root.
c) Depending on number of walls present ,
angular defects were classified by Goldman
and Cohen (1958) as,

24. (i) Three osseous walls
(ii) Two osseous walls
(iii) One osseous wall
(iv) Combination

25.  Osseous craters :
a) concavities in the crest of interdental
bone confined within faciolingal walls.
b) Reasons :
(i) plaque accumulation and difficulty to
clean.
(ii) normal concavity in lower molars
(iii) vascular patterns from gingiva to
crest, a pathway for
inflammation

26.  Bulbous bone contours :
a) bony enlargement
b) an adaptation to Exostoses
c) adaptation to function or buttressing
bone formation.

27. o Reversed architecture :
produced by loss of interdental bone,
facial and lingual plates without concomitant
loss of radicular bone.

28.  Furcation involvement :
Invasion of bifurcation or trifurcation of
multirooted teeth by periodontal disease.
Clinical features-
(i) Grade 1 : incipient bone loss
(ii) Grade 2 : partial bone loss
(iii) Grade 3 : total bone loss with through
and through opening of furcation
(iv) Grade 4 : similar to grade 3,with gingival
recession exposing the furcation to view.

30.  Ledges :
(a) plateau-like bony margins
(b) caused by resorption of thickened
bony plates

31. CONCLUSIO
N-
 Although periodontitis is an infectious
disease of the gingival tissue , changes that
occur in bone are crucial because
destruction of bone is responsible for tooth
loss.
 Bone loss patterns associated with
periodontal disease is varied and the type of
management depends upon the type of loss.