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Lung
Examination:
Abnormal
MAGDI AWAD SASI
LMB LIMU WHO
7TH OCTOPER
HOSPITAL
Inspection of the chest
Scars
Median sternotomy scar:
AIM-Used for cardiac valve replacement and coronary
artery bypass grafts (CABG).
Axillary thoracotomy scar: located between the
posterior border of the pectoralis major and anterior
border of latissimus dorsi muscles, through the 4th
or 5th intercostal space.
AIM-Used for the insertion of chest drains.
Chest wall deformities
Asymmetry:
Pneumonectomy (e.g.
lung cancer) and
thoracoplasty (e.g.
tuberculosis).
Pectus excavatum:
Pectus carinatum:
Hyperexpansion
‘barrel chest’)
Barrel chest
Kyphosis
Scoliosis
Assess tracheal position
Gently assess the position of the trachea, which should
be central in healthy individuals (this can be uncomfortable, so warn
the patient in advance):
1. Ensure patient’s neck musculature is relaxed by asking them to
position their chin slightly downwards.
2. Dip your index finger into the thorax beside the trachea.
3. Gently apply side pressure to locate the border of the trachea.
4. Compare this space to the other side of the trachea using the
same process.
5. A difference in the amount of space between the sides suggests
the presence of tracheal deviation.
Tracheal Position: Mediastinum
Any deviation of the mediastinum is abnormal
Lateral shift: The mediastinum can be either
pulled or pushed away from the lesion
– Pull: Loss of lung volume (Atelectasis, fibrosis,
agenesis, surgical resection, pleural fibrosis)
– Push: Space occupying lesions (pleural effusion,
pneumothorax, large mass lesions)
– Mediastinal masses and thyroid tumors
Palpate the apex beat
1. Palpate the apex beat with your fingers placed
horizontally across the chest.
2. In healthy individuals, it is typically located in the 5th
intercostal space in the midclavicular line.
Causes of a displaced apex beat
Respiratory causes of a displaced apex beat:
Right ventricular hypertrophy (e.g. pulmonary
hypertension, COPD, interstitial lung disease)
Large pleural effusion
Tension pneumothorax
Assess chest expansion
1. Place your hands on the patient’s chest, inferior to the
nipples.
2. Wrap your fingers around either side of the chest.
3. Bring your thumbs together in the midline, so that they
touch.
4. Ask the patient to take a deep breath in.
5. Observe the movement of your thumbs (in healthy
individuals they should move symmetrically
upwards/outwards during inspiration and symmetrically
downwards/inwards during expiration ).
6. Reduced movement of one of your thumbs indicates
reduced chest expansion on that side.
Respiratory causes of reduced chest
expansion
Symmetrical: pulmonary fibrosis reduces
lung elasticity, restricting overall chest
expansion.
Asymmetrical: pneumothorax,
pneumonia and pleural effusion would all
cause ipsilateral reduced chest expansion.
Percussion of the chest
Percussion of the chest involves
listening to the volume and pitch of
percussion notes across the chest to
identify underlying pathology.
Correct technique is essential to
generating effective percussion notes.
Percussion technique
1. Place your non-dominant hand on the patient’s chest
wall.
2. Position your middle finger over the area you want to
percuss, firmly pressed against the chest wall.
3. With your dominant hand’s middle finger, strike the
middle phalanx of your non-dominant hand’s middle
finger using a swinging movement of the wrist.
4. The striking finger should be removed quickly,
otherwise, you may muffle the resulting percussion note.
Areas to percuss
Percuss the following areas of the chest,
comparing side to side as you progress (see
image example below):
Supraclavicular region: lung apices
Infraclavicular region
Chest wall: percuss over 3-4 locations
bilaterally
Axilla
Types of percussion note
Resonant: a normal finding
Dullness: suggests increased tissue density (e.g. cardiac
dullness, consolidation, tumour, lobar collapse).
Stony dullness: pleural effusion.
Hyper-resonance: (e.g. pneumothorax).
Hyper resonance: Increased resonance can be noted either due to
lung distention as seen in asthma, emphysema, bullous disease or
due to Pneumothorax
Tactile vocal fremitus
Assessing tactile vocal fremitus involves
palpating over different areas of the chest
wall whilst the patient repeats a word or
number consistently (e.g. “ninety-nine”).
The presence of increased tissue density or
fluid affects the strength at which the
patient’s speech is transmitted as vibrations
through the chest wall to the examiner’s
hands.
Technique
1. Ask the patient to say “99” repeatedly at
the same volume and in the same tone.
2. Palpate the chest wall on both sides,
using the ulnar border of your hand.
3. Cover all major regions of the chest
wall, comparing each side at each
location.
Abnormal tactile vocal fremitus
Increased vibration over an area
suggests increased tissue density (e.g.
consolidation, tumour, lobar collapse).
Decreased vibration over an area
suggests the presence of fluid or air
outside of the lung (e.g. pleural effusion,
pneumothorax).
An alternative method of assessment
Vocal resonance is an alternative method of assessing
the conduction of sound through lung tissue and involves
auscultation over different areas of the chest wall whilst
the patient repeats a word or number consistently.
The presence of increased tissue density or fluid affects
the volume at which the patient’s speech is transmitted
to the diaphragm of the stethoscope.
Given both tests assess the same thing, there is no
reason to perform both vocal resonance and tactile vocal
fremitus in the same examination.
Auscultation of the chest
Technique
1. Ask the patient to relax and breathe deeply in and out
through their mouth (prolonged deep breathing should,
however, be avoided).
2. Position the diaphragm of the stethoscope over each of
the relevant locations on the chest wall to ensure all lung
regions have been assessed and listen to the breathing
sounds during inspiration and expiration. Assess
the quality and volume of breath sounds and note
any added sounds.
3. Auscultate each side of the chest at each location to allow
for direct comparison and increased sensitivity at detecting
local abnormalities.
Auscultate the chest
Volume of breath sounds
Quiet breath sounds: suggest reduced air entry (e.g
pleural effusion, pneumothorax).
When presenting your findings, state ‘reduced breath
sounds’, rather than ‘reduced air entry’.
Added sounds
Auscultate the chest
Stridor: a high-pitched extra-thoracic breath
sound resulting from turbulent airflow through narrowed
upper airways.
Coarse crackles: discontinuous, brief, popping lung
sounds typically associated with pneumonia,
bronchiectasis and pulmonary oedema.
Fine end-inspiratory crackles: often described as
sounding similar to the noise generated when separating
velcro. Fine end-inspiratory crackles are associated with
pulmonary fibrosis.
Bronchial
Bronchial breathing anywhere other than over
the trachea, right clavicle or right inter-scapular
space is abnormal.
In consolidation, the bronchial breathing is low
pitched and sticky and is termed tubular type of
bronchial breathing.
In cavitary disease, it is high pitched and hollow
and is called cavernous breathing. You can
simulate this sound by blowing over an empty
coke bottle.
Rhonchi
Rhonchi are long continuous adventitious
sounds, generated by obstruction to
airways.
Generalized or localized, during inspiration
or expiration, and the pitch.
Diffused rhonchi = asthma or COPD.
Pleural Rub
Normal parietal and visceral pleura glide
smoothly during respiration.
If the pleura is roughened due to any reason, a
scratching, grating sound, related to respiration
is heard.
You can hear the sound by compressing harder
with the stethoscope and making the patient
take deep breaths.
It is localized and can be palpable.
Crackles
Interrupted adventitious sounds
Timing and Intensity Crackles heard only at the
end of inspiration are called fine crackles.
– When the surfactant is depleted, the alveoli collapse.
Air enters the alveoli at the end of inspiration.
– This sound is generated as the alveoli pop open from
it's collapsed state.
Right lung
abscess
Chest examination magdi sasi2021

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Chest examination magdi sasi2021

  • 1. Lung Examination: Abnormal MAGDI AWAD SASI LMB LIMU WHO 7TH OCTOPER HOSPITAL
  • 2.
  • 3.
  • 4. Inspection of the chest Scars Median sternotomy scar: AIM-Used for cardiac valve replacement and coronary artery bypass grafts (CABG). Axillary thoracotomy scar: located between the posterior border of the pectoralis major and anterior border of latissimus dorsi muscles, through the 4th or 5th intercostal space. AIM-Used for the insertion of chest drains.
  • 5.
  • 6. Chest wall deformities Asymmetry: Pneumonectomy (e.g. lung cancer) and thoracoplasty (e.g. tuberculosis). Pectus excavatum: Pectus carinatum: Hyperexpansion ‘barrel chest’) Barrel chest Kyphosis Scoliosis
  • 7. Assess tracheal position Gently assess the position of the trachea, which should be central in healthy individuals (this can be uncomfortable, so warn the patient in advance): 1. Ensure patient’s neck musculature is relaxed by asking them to position their chin slightly downwards. 2. Dip your index finger into the thorax beside the trachea. 3. Gently apply side pressure to locate the border of the trachea. 4. Compare this space to the other side of the trachea using the same process. 5. A difference in the amount of space between the sides suggests the presence of tracheal deviation.
  • 8. Tracheal Position: Mediastinum Any deviation of the mediastinum is abnormal Lateral shift: The mediastinum can be either pulled or pushed away from the lesion – Pull: Loss of lung volume (Atelectasis, fibrosis, agenesis, surgical resection, pleural fibrosis) – Push: Space occupying lesions (pleural effusion, pneumothorax, large mass lesions) – Mediastinal masses and thyroid tumors
  • 9. Palpate the apex beat 1. Palpate the apex beat with your fingers placed horizontally across the chest. 2. In healthy individuals, it is typically located in the 5th intercostal space in the midclavicular line. Causes of a displaced apex beat Respiratory causes of a displaced apex beat: Right ventricular hypertrophy (e.g. pulmonary hypertension, COPD, interstitial lung disease) Large pleural effusion Tension pneumothorax
  • 10. Assess chest expansion 1. Place your hands on the patient’s chest, inferior to the nipples. 2. Wrap your fingers around either side of the chest. 3. Bring your thumbs together in the midline, so that they touch. 4. Ask the patient to take a deep breath in. 5. Observe the movement of your thumbs (in healthy individuals they should move symmetrically upwards/outwards during inspiration and symmetrically downwards/inwards during expiration ). 6. Reduced movement of one of your thumbs indicates reduced chest expansion on that side.
  • 11. Respiratory causes of reduced chest expansion Symmetrical: pulmonary fibrosis reduces lung elasticity, restricting overall chest expansion. Asymmetrical: pneumothorax, pneumonia and pleural effusion would all cause ipsilateral reduced chest expansion.
  • 12. Percussion of the chest Percussion of the chest involves listening to the volume and pitch of percussion notes across the chest to identify underlying pathology. Correct technique is essential to generating effective percussion notes.
  • 13.
  • 14. Percussion technique 1. Place your non-dominant hand on the patient’s chest wall. 2. Position your middle finger over the area you want to percuss, firmly pressed against the chest wall. 3. With your dominant hand’s middle finger, strike the middle phalanx of your non-dominant hand’s middle finger using a swinging movement of the wrist. 4. The striking finger should be removed quickly, otherwise, you may muffle the resulting percussion note.
  • 15. Areas to percuss Percuss the following areas of the chest, comparing side to side as you progress (see image example below): Supraclavicular region: lung apices Infraclavicular region Chest wall: percuss over 3-4 locations bilaterally Axilla
  • 16.
  • 17.
  • 18. Types of percussion note Resonant: a normal finding Dullness: suggests increased tissue density (e.g. cardiac dullness, consolidation, tumour, lobar collapse). Stony dullness: pleural effusion. Hyper-resonance: (e.g. pneumothorax). Hyper resonance: Increased resonance can be noted either due to lung distention as seen in asthma, emphysema, bullous disease or due to Pneumothorax
  • 19. Tactile vocal fremitus Assessing tactile vocal fremitus involves palpating over different areas of the chest wall whilst the patient repeats a word or number consistently (e.g. “ninety-nine”). The presence of increased tissue density or fluid affects the strength at which the patient’s speech is transmitted as vibrations through the chest wall to the examiner’s hands.
  • 20. Technique 1. Ask the patient to say “99” repeatedly at the same volume and in the same tone. 2. Palpate the chest wall on both sides, using the ulnar border of your hand. 3. Cover all major regions of the chest wall, comparing each side at each location.
  • 21. Abnormal tactile vocal fremitus Increased vibration over an area suggests increased tissue density (e.g. consolidation, tumour, lobar collapse). Decreased vibration over an area suggests the presence of fluid or air outside of the lung (e.g. pleural effusion, pneumothorax).
  • 22. An alternative method of assessment Vocal resonance is an alternative method of assessing the conduction of sound through lung tissue and involves auscultation over different areas of the chest wall whilst the patient repeats a word or number consistently. The presence of increased tissue density or fluid affects the volume at which the patient’s speech is transmitted to the diaphragm of the stethoscope. Given both tests assess the same thing, there is no reason to perform both vocal resonance and tactile vocal fremitus in the same examination.
  • 23. Auscultation of the chest Technique 1. Ask the patient to relax and breathe deeply in and out through their mouth (prolonged deep breathing should, however, be avoided). 2. Position the diaphragm of the stethoscope over each of the relevant locations on the chest wall to ensure all lung regions have been assessed and listen to the breathing sounds during inspiration and expiration. Assess the quality and volume of breath sounds and note any added sounds. 3. Auscultate each side of the chest at each location to allow for direct comparison and increased sensitivity at detecting local abnormalities.
  • 24. Auscultate the chest Volume of breath sounds Quiet breath sounds: suggest reduced air entry (e.g pleural effusion, pneumothorax). When presenting your findings, state ‘reduced breath sounds’, rather than ‘reduced air entry’. Added sounds
  • 25. Auscultate the chest Stridor: a high-pitched extra-thoracic breath sound resulting from turbulent airflow through narrowed upper airways. Coarse crackles: discontinuous, brief, popping lung sounds typically associated with pneumonia, bronchiectasis and pulmonary oedema. Fine end-inspiratory crackles: often described as sounding similar to the noise generated when separating velcro. Fine end-inspiratory crackles are associated with pulmonary fibrosis.
  • 26. Bronchial Bronchial breathing anywhere other than over the trachea, right clavicle or right inter-scapular space is abnormal. In consolidation, the bronchial breathing is low pitched and sticky and is termed tubular type of bronchial breathing. In cavitary disease, it is high pitched and hollow and is called cavernous breathing. You can simulate this sound by blowing over an empty coke bottle.
  • 27. Rhonchi Rhonchi are long continuous adventitious sounds, generated by obstruction to airways. Generalized or localized, during inspiration or expiration, and the pitch. Diffused rhonchi = asthma or COPD.
  • 28. Pleural Rub Normal parietal and visceral pleura glide smoothly during respiration. If the pleura is roughened due to any reason, a scratching, grating sound, related to respiration is heard. You can hear the sound by compressing harder with the stethoscope and making the patient take deep breaths. It is localized and can be palpable.
  • 29. Crackles Interrupted adventitious sounds Timing and Intensity Crackles heard only at the end of inspiration are called fine crackles. – When the surfactant is depleted, the alveoli collapse. Air enters the alveoli at the end of inspiration. – This sound is generated as the alveoli pop open from it's collapsed state.
  • 30.
  • 31.