This document discusses acute infectious diarrhea. It defines it as an alteration of normal gastrointestinal function caused by ingested microorganisms or their toxins. The mechanisms of disease include osmotic load, intestinal secretion, malabsorption, and altered motility. Common causes are discussed such as ETEC, Salmonella, Campylobacter, Shigella, and viruses. Symptoms, diagnostic testing, treatment, and prevention are covered. Management involves rehydration and use of antimicrobials in some cases.
Let's learn the pharmacology related to nephrotic syndrome - features of nephrotic syndrome with underlying mechanisms, objectives of treatment, and management of the nephrotic syndrome.
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CHRONIC DYSPEPSIA
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Pancreatitis is an inflammatory condition of the pancreas. Two major forms : acute pancreatitis (is reversible) and chronic pancreatitis(is irreversible).
Acute infectious diarrhea
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Seminar Prepared by :-
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Alaa Shamil
Guldan Hameed
Internal Medicine
College of Medicine - University of Kirkuk
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Acute infectious diarrhea
Seminar Prepared by :-
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Mohammed Saadi
Hussein Jassam
Mahmoud Ahmed
Meran Salih
Internal Medicine
College of Medicine - University of Kirkuk
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Approach oriented presentation for some of the emergencies of clinical gastroenterology, including upper GI bleeding, hepatic encephalopathy, acute severe attack of IBD and acute pancreatitis.
Cholera is devastating diarrheal disease caused by V. Cholerae that has been responsible for seven global pandemics.
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https://forms.gle/apSH6sR5mRTwRvjJ6
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Dear Dr. Kornbluth and Mr. Gorenberg,
The US House of Representatives is deeply concerned by ongoing and pervasive acts of antisemitic
harassment and intimidation at the Massachusetts Institute of Technology (MIT). Failing to act decisively to ensure a safe learning environment for all students would be a grave dereliction of your responsibilities as President of MIT and Chair of the MIT Corporation.
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• The Committee on Education and the Workforce has been investigating your institution since December 7, 2023. The Committee has broad jurisdiction over postsecondary education, including its compliance with Title VI of the Civil Rights Act, campus safety concerns over disruptions to the learning environment, and the awarding of federal student aid under the Higher Education Act.
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12. SYMTOMS & SIGNS
TOXIGENIC INFLAMMATORY VIRAL
No systemic upset Systemic upsetAbdminal Systemic upset
Small number pain, tenesmus, fever . URTI
Large volume Stool Fever nausea
Stool no RBCs no Large number Frequent Myalgia
leukocytes. Small volume blood -stain Stool
mucus, pus cells Voluminous, watery
Mechanism Mechanism
Invasion of enterocytes Osmatic or secretory
leading to mucosal cell
death and inflammatory
response
13. APROACH TOwith diarrhea
Approach to pt PATIENTS
( entrotoxin ( a febrile- 1
potentially invading( Fever + blood or – 2
( dysentery
completely invading ( fever + bactreamia- 3
Diarrhea with GI bleeding- 4
Investigation
Rectal swab, stool general , Stool c/s , toxin-
assay ,elisa, PCR
14. entrotoxin ( a febrile ) - 1
2 – potentially
invading( Fever + blood or
dysentery )
3- completely invading ( fever
+ bactreamia
4- Diarrhea with GI bleeding
Investigation
- Rectal swab, stool general ,
Stool c/s , toxin assay
,elisa, PCR
15. DIARRHEA CLINICS
PRIMARY CARE CENTER-
SEASONAL CENTER-
HOSPITALS-
EACH CLINIC-
Doctor
Nurse
Health supervisor
Necessary requirements
Supervision -
Regional Infection control Committee + Preventive -
.Medicine Dept
16. OUT PATIENT MANAGEMENT
FOR MILD DIARRHEA
SAUDI -
RESIDENTS -
HAJI WITH PROPER EASILY TRACED -
ADDRESS
18. HAJ NON HAJ
:Name
SURVEY SHEET OF HOSPITALIZED INFECTIOUS DIARRHEA CASES IN
.MEDINA MUNAWARAH DURING HAJ
:Age Years Sex: M/F Nationality
:History of
Fever Nausea Vomiting
Tenesmus Headache Colic
( Mayalgia Other (specify
:Stools
Frequent >6/day Volume Large Actual amount >1/2 liter
day small >1/2 liter/6 <
Blood Mucus Consistency Formed
Semi formed
Watery
(Other( specify
Contacts: One person involved
Group with common food source
:Examination
.Temp. Pulse BP Resp
Dehydration Mild Level of consciousness Drowsy
Moderate Semiconscious
Severe Comatosed
21. TREATMENT OF MOST COMMON
.GASTROINTESTINAL PATHOGENS
S. aureus Hydration Self limited
B. cereus Hydration Self limited
C. perfringeus Hydration Self limited
V.cholrae Tetrcycline’s Ampicilin & TMP-SMX
So effective
Salmonella TMP-SMX Ampicilin
Shigella TMP-SMX Quinolon
,Yersinia TMP-SMX Tetracyclines
.minoglycosides in severe cases
Campylobacter Erythromycin Quinolones
E. coli Hydration Self limiting
v. parahaemolyticus Tetracyclines TMP-SMX
Rotavirus Hydration No specific treatment
Norwalk virus Hydration No specific treatment
TNIDAZOLE Me tronidazole G. lambia
E. histolytica Me tronidazole
22. حال ت الهسهال منطقة المدينة المنورة
1341 --- 2341 حتى شعبان
23. حال ت الهسهال منطقة المدينة
المنورة
2341 حتى شعبان
6747
312
24. MANAGEMENT
MANAGEMENT OF INFECTIOUS
DIARRHEA
1. REHYDRATION REHYDRATION 1.
ORS ORS
IV IV FLUIDS
FLUIDS SHOCK SHOCK
SEVERE VOMITING SEVERE VOMITING
DEPRESSED MENTAL STATE
DEPRESSED MENTAL
2. INDICATIONS FOR ANTIMICROBIALS STATE
a( Parasitic infections E. histililytica
INDICATIONS FOR ANTIMICROBIALS. 2
a) Parasitic infections G. lambia
E. histililytica
Other parasitic infection
G. lambia
b(. V.cholerae Cases
Other parasitic infection
Carriers
b). V.cholerae Cases
discharge after 3 consecutive negative post treatment samples
c(. Carriers
Salmonella Infants
discharge after 3 consecutive negative post treatment samples
c). Salmonella Infants