A basic overview on the management of intra-operative bronchospasm: the risk factors, triggers, diagnosis, prevention and management. Includes a case scenario – discussion.
A basic overview on the management of intra-operative bronchospasm: the risk factors, triggers, diagnosis, prevention and management. Includes a case scenario – discussion.
Tonsillitis slideshare for medical students NehaNupur8
complete and detail information about tonsillits , that is the inflammation of the tonsils ,present in the oral cavity , disease of oral cavity contains introduction, definition, types, causes, risk factors,pathophysiology , treatment , medical management, nursing management, nurses role, patient teaching sign and symptoms , drug therapy, diet management,
Airway management is the cornerstone of resuscitation and is a defining skill for the specialty of emergency medicine. The emergency clinician has primary airway management responsibility, and all airway techniques lie within the domain of emergency medicine. Although rapid sequence intubation (RSI) is the most commonly used method for emergent tracheal intubation, emergency airway management includes various intubation techniques and devices, approaches to the difficult airway, and rescue tech- niques when intubation fails.
The decision to intubate should be based on careful patient assessment and appraisal of the clinical presentation with respect to three essential criteria: (1) failure to maintain or protect the airway; (2) failure of ventilation or oxygenation; and (3) the patient’s anticipated clinical course and likelihood of deterioration.
In most patients, intubation is technically easy and straightfor- ward. Although early ED-based observational registries reported cricothyrotomy rates of about 1% for all intubations, more recent studies have shown a lower rate, less than 0.5%.3 As would be expected with an unselected, unscheduled patient population, the ED cricothyrotomy rate is greater than in the operating room, which occurs in approximately 1 in 200 to 2000 elective general anesthesia cases.4 Bag-mask ventilation (BMV) is difficult in approximately 1 in 50 general anesthesia patients and impossible in approximately 1 in 600. BMV is difficult, however, in up to one-third of patients in whom intubation failure occurs, and dif- ficult BMV makes the likelihood of difficult intubation four times higher and the likelihood of impossible intubation 12 times higher. The combination of failure of intubation, BMV, and oxy- genation in elective anesthesia practice is estimated to be exceed- ingly rare, roughly 1 in 30,000 elective anesthesia patients.4 These numbers cannot be extrapolated to populations of ED patients who are acutely ill or injured and for whom intubation is urgent and unavoidable. Although patient selection cannot occur, as with a preanesthetic visit, a preintubation analysis of factors predicting difficult intubation gives the provider the information necessary to formulate a safe and effective plan for intubation.
Preintubation assessment should evaluate the patient for potential difficult intubation and difficult BMV, placement of and ventilation with an extraglottic device (EGD; and cricothyrotomy. Knowledge of all four domains is crucial to successful planning. A patient who exhibits obvious difficult airway characteristics is highly predictive of a challenging intuba- tion, although the emergency clinician should always be ready for a difficult to manage airway, because some difficult airways may not be identified by a bedside assessment.
Airway difficulty exists on a spectrum and is contextual to the provider’s experience, environment, and armamentarium of devices.
Please share your valuable opinions.
Obstructive sleep apnea (OSA) is a prevalent chronic disease characterized by pharyngeal collapse during sleep.
Sleep disorder that involves cessation or significant decrease in airflow through the upper airway in the presence of breathing effort.
Obstructive sleep apnea is the second most common sleep disorder, insomnia being the most common.
Associated with recurrent oxyhemoglobin desaturations and arousals from sleep
Apnea index- no. of apneas /hr of total sleep time.
AHI (APNEA-HYPOPNEA INDEX)- No of apneas and hypoapneas/hr of total sleep time.
RDI (Respiratory Disturbance Index) – no. of apneas, hypoapneas and respiratory effort related arousals(RERA)/hr of total sleep time.
A detailed and accurate presentation on the Dental Management of Respiratory and Adrenal Disorders.
Presentation deals specifically what a dental health care professional should care for himself and the patient while managing Respiratory and Adrenal disorders.
presentation by Dr. Ishaan Adhaulia
Journal club covid vaccine neurological complications ZIKRULLAH MALLICK
the risks of adverse neurological events following SARS-CoV-2 infection are much greater than those associated with vaccinations, highlighting the benefits of ongoing vaccination programs.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
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The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
3. Waldeyer’s tonsillar ring
The tonsils are areas of lymphoid tissue on
either side of the throat
Tubal, palatine and lingual tonsils : 3 pairs
The adenoid tissue is in the midline of the
posterior nasopharyngeal wall
8. Venous drainage
Plexus surrounding the tonsil drains into
paratonsillar vein which joins common
facial vein and pharyngeal plexus.
Venous haemorrhage is mostly responsible
for bleeding following tonsillectomy .
12. The tonsils act as part of the immune system
to help protect against infection.
Involved in helping fight off pharyngeal and
upper respiratory tract infections.
13. What is the Classification of Acute
Tonsillitis?
18. GRADE I: Congested but within fossa
GRADE II : till the brim of tonsillar fossa
GRADE III : beyond the pillars but doesn’t touch
each other.
GRADE IV : kissing tonsils
20. Pain in the throat
Dyphagia
Mouth breathing
Failure to thrive/repeated infection—pain
fever, tachycardia.
Cervical adenopathy.
Visibly inflamed tonsil which may have
discharge.
22. 1. Upper airway obstruction, dysphagia and
obstructive sleep apnea.
2. Peritonsillar abscess, not responding to
adequate medical management and surgical
drainage.
3. The requirement of biopsy to confirm tissue
pathology in suspected neoplastic causes.
23. 4. Recurrent tonsillitis that is unresponsive to
medical treatment.
7 or more episodes in 1 year.
5 episodes per year for 2years.
3 episodes per year for 3 years.
2 weeks or more of lost school or work in 1 year
5. Persistent bad-breath and taste in mouth due to
chronic tonsillitis.
24. 6. Persistent tonsillitis in streptococcus carrier,
which is unresponsive to antibiotics.
25. As a part of another operation
Palatopharyngoplasty for sleep apnoea
Glossopharyngeal neurectomy
Removal of styloid process
26. What are the Contraindications
for tonsillectomy?
27. Presence of acute infection in Upper Respiratory
Tract even acute tonsillitis
Haemoglobin level less than 10 g%
Children under 3 years
Overt or sub mucous cleft palate
28. Bleeding disorders ( leukemia , purpura , aplastic
anemia or haemophilia)
At the time of epidemic polio
Uncontrolled systemic disease
Tonsillectomy is avoided during period of menses
29. What are the procedures available
for tonsillectomy?
31. Hot methods
Bipolar Radio frequency
Electrocautery
LASER tonsillectomy (CO2 or KTP-512 )
Coblation tonsillectomy
32. What are the relevant histories that
should be taken before tonsillectomy?
33. In pediatric ; milestone development and
vaccination.
Repeat episodes of fever, throat pain, dysphagia.
History of any easy bruising, bleeding gums,
epistaxis, menorrhagia
Family history of any bleeding disorders
Recent ingestion of Aspirin, NSAIDs
34. Mouth breathing
The triad of hyponasality, snoring, and mouth
breathing normally indicates enlarged, obstructing
adenoids
Other symptoms of adenoid disease include
rhinorrhea, postnasal drip, chronic cough and
headache
History of possible allergies, GERD, and sinusitis.
35. What are the signs and symptoms of
Obstructive sleep apnoea ?
36. In children, adenotonsillar hypertrophy is the most
common cause of obstructive sleep apnoea.
The signs and symptoms :
chronic hypoxemia manifesting itself as
polycythemia and right ventricular strain.
Snoring, apneic episodes followed by grunting and
restlessness occurring during sleep.
37. The daytime symptoms include headaches,
excessive daytime somnolence and not feeling
fresh in the morning.
Diagnosed by polysomnography
OSA syndrome : AHI > 5 with symptoms or AHI>15
regardless of symptoms
38. What are the things that should be
included in examination of a case of
tonsillitis?
39. Routine examination in a pediatric patient
Loose/missing teeth:
Patency of oral and nasal cavity
Patients may have “adenoid facies” (long face,
flattened midface, open mouth) and hyponasal
speech
Enlarged (> 2 cm) or tender cervical adenopathy
Tonsillar or pharyngeal exudates.
43. An antisialogogue and a narcotic.
Barbiturates will be of little use in short upper
airway surgery which requires quick return of
protective airway reflexes.
Sedatives should not be used if there is history
suggestive of obstructive sleep apnoea.
44. What are the anaesthetic
considerations in a case of tonsillitis?
45. Maintain deep general anaesthesia that prevents
reflex-induced hypertension, tachycardia or
arrhythmias.
Muscle relaxation is required to allow placement
of the mouth gag and prevent any bucking,
coughing or straining.
A rapid recovery of consciousness and return of
protective airway reflexes is also desired.
47. Inhalational induction with sevoflurane is
preferred in small children especially when IV
line is not inserted and in OSA patients.
If IV line is present Thiopentone or propofol can
be given.
48. What are the considerations in intubation in
a case of tonsillitis?
49. Intubation under deep inhalational or muscle
relaxant assisted anesthesia is preferred.
Regular tube/RAE tube may be passed by
orotracheal route.
Throat should be well packed especially when
uncuffed tubes are used to prevent aspiration of
blood and secretions
50. Tube can either be fixed in the midline or fixed
on one side at the angle of the mouth and the
side changed once the tonsillectomy is done and
hemostasis achieved for removal of the opposite
tonsil.
When only tonsillectomy and no adenoidectomy
is planned one can also insert a nasotracheal
tube.
51. Intubation could be difficult if the tonsils
are very large and approximating in the
midline (kissing tonsils).
53. Nasal patency should be checked before
Nasal decongestant drops should be instilled 15
min before procedure
Antisialagogue like glycopyrolate can be given
10min before surgery
One size lesser than the predicted ETT is
preferred to avoid injury
54. Lignocaine jelly is applied over tube to lessen
trauma.
Put the ETT in warm water to make it soft.
Magill’s forceps, Laryngoscope/ fiberoptic should
be ready
55. What are the methods available for
nasal intubation?
56. Conventional laryngoscopy with Magill’s forceps
With help of video laryngoscopes like ‘King vision’
Nasal fiberoptic intubation
With help of Light wand
Blind nasal intubation
58. Flexible LMA may be used for adenotonsilletomy
surgeries and is routinely used in some centres.
It requires lighter plane of anaesthesia, and there
is no need for muscle relaxants; with resultant
rapid induction and smooth recovery.
LMA is not removed until full return of reflexes.
59. Disadvantage is if airway is lost during surgery, it
can be difficult to rectify the situation.
60. What all things should we consider
during maintenance of anaesthesia?
61. inhaled anaesthetics and short-acting opioids like
fentanyl using spontaneous ventilation
Or muscle relaxants with controlled ventilation
Adequate depth should be maintained to prevent
any reflex-induced hypertension, tachycardia and
arrhythmias and avoid bucking , coughing or
straining during surgery
62. Blood loss during tonsillectomy may be difficult to
estimate and may reach up to 5 % of the blood
volume.
Blood transfusion may be required in some cases.
Local anesthetic plus adrenaline applied in the
tonsillar fossa gives the advantages of bloodless
dissection, reduced operative time and reduced
postoperative pain.
63. If large volumes of L.A are injected, it can give rise
to respiratory obstruction once the patient is
extubated because of bilateral glossopharyngeal
nerve block.
64. As it is shared airway ,should be very vigilant
about accidental extubation or aspiration of blood
and secretion if the throat pack is displaced under
GA when uncuffed tubes are used.
At the end of surgery, pack removal and good
pharyngeal and laryngeal suction under vision is
essential.
65. What is the role of anti-emetics in
tonsillectomy?
66. Patients undergoing tonsillectomy are prone to
develop PONV.
Antiemetic should be given prior to reversal.
Ondansetron (0.1 mg/kg) or dexamethasone
(0.1–0.2 mg/kg) or a combination of both can be
considered.
68. Extubated only when awake and there is return of
protective airway reflexes.
Extubation should be smooth thereby preventing
rise in blood pressure which can cause bleeding.
70. Patient should be transported in tonsillar position
with oxygen supplementation
Tonsillar position : left lateral position, with one
knee flexed and the hand under the face along
with a slight head low position.
This allows the blood and secretion to drain out
rather than flow back onto the vocal cords
73. ‘Rose position’
Both the head and neck are extended.
This is done by keeping a sand bag under the
patient's shoulder blade.
Its contraindicated in patients with Down’s
syndrome owing to atlanto-axial instability
The operator has a direct view of the tonsils and
there is the added advantage of the posterior part
of the pharynx forming a sump into which the
blood may drain, below the level of the glottis.
76. Post-tonsillectomy bleeding
Airway obstruction because of upper airway
edema, presence of blood and secretions and
laryngospasm
Postoperative nausea and vomiting during first
24 hours (as high as 70%) because of pharyngeal
mucosal irritation from surgery and swallowed
blood and secretions.
77. Pain and sore throat lasts for 3–4 days.
Postoperative respiratory complications.
Negative pressure pulmonary edema due to
sudden release of upper airway obstruction,
but very rare.
78. What is the classification of Post
tonsillectomy bleeding?
79. Primary :
within 24 hours
Bleeding from adenoid bed is more commen in
first 4 hours.
Bleeding from tonsillar bed is more common in
first 6-8 hours
80. Secondary :
24 hours to 28days
May be due to:
Sloughing of the eschar (dead tissue) overlying
the tonsillar bed
Loosened vessel ties
Infection from underlying chronic tonsillitis
81. What are the Risk factors for post
tonsillectomy haemorrhage?
82. The risk of haemorrhage increases with age
Higher in males.
The surgical technique also influences the
incidence of bleeding.
Hot surgical technique (diathermy or
radiofrequency coblation) has 3 times risk
compared to cold steel tonsillectomy (traditional)
83. What are the Anaesthetic considerations
for re-exploration ?
84. Child may loose large amounts of blood and
become hypovolemic and even progress to shock
in a short time.
Immediate resuscitation with colloid and
crystalloid while waiting for blood to become
available.
Intravenous boluses of fluid, 20 ml/kg stat,
repeated if necessary after reassessment of the
cardiovascular system.
85. Preoperative sedation should be avoided
Adequate preoxygenation
IV induction agent depending on hemodynamic
stability
Child should be considered as full stomach as
large amount of blood and secretions may be
swallowed.
86. A rapid sequence intubation with cricoid pressure
and cuffed ETT using succinyl choline is
warranted.
Two good working suctions should be ready at the
head end in case of vomiting
Reintubation may be difficult if bleeding is
obscuring the view or due to edema from
previous airway instrumentation and surgery.
87. A smaller size ETT than the previous anaesthetic
should be ready.
Hypothermia should be avoided as it exacerbates
coagulopathy
Decompression of stomach prior to extubation
Extubation should be done in lateral position and
only if the child is fully awake with normal gag,
cough reflex and is stable hemodynamically
89. Quinsy is term for Peritonsillar abscess
Situated outside tonsillar capsule
Tonsil is pushed medially
90. What are the anaesthetic
considerations in Quinsy?
91. Aggravation of a preexisting respiratory
obstruction
Even with relaxation, trismus may not resolve,
making laryngoscopy and intubation difficult.
Abscess may rupture at any time during
induction or intubation and there is a risk of
aspiration of purulent material.
92. GA is induced with inhalational agent in oxygen or
intravenous induction agents like propofol along
with sevoflurane.
Patient is kept in head low position with the head
turned toward the affected side.
Under deep plane of anaesthesia laryngoscopy is
done extremely carefully for fear of rupturing the
abscess
93. What are the Preparations to be
done for LASER surgery?
94. The biggest concern here is prevention of an airway
fire.
A plan to deal effectively with such a disaster if
occurs
O T staff must wear protective eye gear and laser
masks when working around the laser.
95. Clear PVC plastic tubes seem to catch fire much
more easily than older red rubber tubes .
Red rubber tubes seem to lead to less toxic
combustion products once ignited.
In conventional PVC tubes safer is to guard it with
reflective tape( ‘Al’ & ‘Cu’ or FDA approved
Merocel Laser Guard.)
96. Fill the cuff with an indicator dye (e.g., methylene
blue in normal saline) to detect a break early.
The cuff should also be covered from above with
wet gauze or neurosurgical sponges to retard
heating.
The tube diameter should be chosen 1 to 2 mm
smaller than usual.
97. Wrapping should start at the distal end and be
continued up to the level of the uvula.
The distal end of the tape should be cut at a 60-
degree angle
The tube should then be wrapped in a spiral with
~ 30% overlap, avoiding sharp edges and leaving
no PVC exposed.
101. Extract : ETT and other combustible materials
Eliminate : O2 supply disconnection
Extinguish residual fire
Evaluate injury using direct laryngoscopy and
rigid bronchoscopy
Continue oxygenation with mask
If severe injury consider low tracheostomy