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Eye Emergencies
Dr Andrew White
BMedSci(hons) MBBS PhD FRANZCO
Clinical Senior Lecturer
Discipline of Ophthalmology and Eye Health
Glaucoma Specialist, Westmead Hospital
A Useful Resource
http://www.aci.health.nsw.gov.au/__data/assets/pdf_file/0013/155011/eye_manual.pdf
Anatomy
Anatomy
The Basic Eye Exam
• You can learn a lot from pinhole acuity!
• Better with pinhole: refractive/anterior
segment problem
• Not better: posterior segment problem
• Dilate (Tropicamide)! The risk of angle
closure is tiny and you are in a hospital!
• Pupil exam will tell you a lot.
• Look for RAPD
– Optic nerve problem
• Look for sluggish pupil
– Uveitis
– Angle closure glaucoma
Common Benign Things
• Pingueculum
• Pterygium
• Trabeculectomy bleb
Lids
Chalazion
• Often a child but also
young adults
• Small lump on the upper
or lower lid with some
swelling
• Usually sent in by LMO
as periorbital cellulitis but
rarely is
• Treated by warm
compresses and chlorsig
• Often spontaneously
discharges
Periorbital and Orbital Cellulitis
• Usually in young children who
are fairly well
• May start from a small scratch
or chalazion
• Usually a 48hr trial of oral
antibiotics before moving to IV
if needed
• Don’t need to do a CT straight
off
• Children with orbital cellulitis
look sick.
• CT will show full sinuses and
ENT need to be involved to
drain it +IV antibiotics
Dacryocystitis
• Usually a young to middle
aged adult with localised
cellulitis over the lacrimal
sac and a fever
• Needs IV antibiotics and
warm compresses
• A pimple may form that
will discharge
spontaneously or can be
incised by eye team
Lid Trauma
• We are more concerned
about medial canthus
injuries than lateral
canthus due to location of
lacrimal system
• Lid margin injuries need
specialised repair
• Make sure the eye and
orbit are intact
Cornea
Corneal Abrasion
• Commonly from a finger
in the eye
• Very painful
• Treat with chlorsig and a
double firm pad
• QID chlorsig drops 7 days
• Intensive chlorsig is
totally useless
• Look under the lids for
tarsal foreign body!
• Can lead to recurrent
erosions
Corneal Foreign Body
• Typically from angle
grinding or small piece of
wood
• Usually can be lifted off
with a cotton bud or
25/23G needle
• Rust ring can be removed
later in clinic
• Chlorsig QID 1 week
Subconjunctival Haemorrhage
• Looks worse than it is
• Trivial
• Idiopathic
• Not related to INR.
Sometimes high BP
• Will get better in 2
weeks
• May need lubricants
for comfort
Conjunctiva
Bacterial Conjunctivitis
• Unilateral
• Mucous discharge
• ?Previous foreign
body
• Swab the discharge
and give chlorsig 7
days
• Clinic f/u if no better
Viral Conjunctivitis
• Fairly common and goes in runs
• Characterised by watery discharge and
itch
• 2 main forms:
• Adenoviral
• Herpes (simplex and zoster)
Adenoviral Conjunctivitis
• Probably the most contagious
thing on the planet
• Wash everything after contact!!!
• Often an flu like prodrome
• Accompanied by chemosis and lid
swelling
• Characterised by conjunctival
follicles
• Typically goes from one eye to the
next over a few days
• Self limiting but can persist for 2-3
weeks.
• Patient is contagious for 2 weeks
• Lubricants and cool compresses
help
Herpes Conjunctivitis
• Either HSV 1, 2 or Zoster
• Unilateral
• Usually fairly mild
symptoms
• May be accompanied by
a dendritic ulcer
• Treatment is zovirax
ointment 5x a day 10
days
• Check the retina!
Herpes Zoster Ophthalmicus
• Only possible if the V1 division
is involved.
• Usually only a mild
conjunctivitis is involved
treated with lubricants and cool
compresses
• There may be some corneal
ulceration
• Main concern is retinal
involvement – check!
• Treatment is systemic oral
antivirals (acyclovir. Valtrex
etc).
• Trigeminal neuralgia may be a
long term problem
Allergic Conjunctivitis
• Can be seasonal, drugs or
contact
• Frequently bilateral but can be
unilateral with mild lid swelling
• History is usually the giveaway
• Papillae under the lids
• Remove the stimulus, lubricate
• Call us if contemplating
steroids
• DO NOT GIVE STERIODS
INDEPENDENTLY
• There are a number of over
the counter topical
antihistamines (eg Lomide,
Zatiden)
Inflammatory Conditions
Uveitis
• Essentially an arthritis of the eye
• Usually young, unilateral red eye
with photophobia
• Flare and cells in the anterior
chamber +/- hypopion and
irregular sluggish pupil
• May be HLAB27 +ve (ankylosing
spondylitis)
• Other causes: Drugs, HSV,
Syphilis, Sarcoid, TB, Bartonella,
Lyme disease, LYMPHOMA –
beware the elderly patient
• Treated with dilating drops and
intensive topical steroids (by
ophthalmology)
Scleritis and Episcleritis
• Episcleritis: Young, localised
area of redness over the
conjunctiva that blanches with
phenylephrine 2.5%
• Self limiting. Gets better with
NSIADS and weak topical
steroids
• Scleritis: Old, rheumatoid
patients but also seen in
infections like syphilis
• Redness does not blanch
• Extremely painful.
• Treated with oral steroids and
NSAIDS
• Risk of perforation
Blinding Conditions
Contact Lens Keratitis
• Painful, rapidly
progressive
• Almost always
pseudomonas
• Characterised by white
corneal infiltrates
• Can progress to
blindness in 24hrs
• Needs intensive ciloxan
eye drops (hourly)
• Chlorsig will do nothing!
Chemical Burns
• Acid and chemical burns
will damage superficial
cornea then stop
• Alkali burns will continue
to penetrate until
neutralised
• Blindness results not only
from initial damage but
limbal stem cell failure
• Irrigate, irrigate, irrigate!
ASAP
• Check pH of all chemical
injuries at presentation
and after irrigation
Endophthalmitis
• Will be surgical or endogenous
• Surgical typically presents day
1-7 post surgery with a rapidly
progressive history of pain and
redness (it is rare to present
months to years later but it
does happen)
• Endogenous is found in the
immunosuppressed, moribund
and IV drug users
• Blindness is rapid if untreated
(hours)
• Needs a vitreous tap and
intravitreal
antibiotics/antifungals/antivirals
as determined by
ophthalmology
Acute Glaucoma
• Will be open or closed angle
• Open angle usually has a long
history of glaucoma and a
slowly progressive history
• Angle closure glaucoma is
typically an elderly, dark iris
patient who presents at night
(when the eye dilates)
• Acute glaucoma is unilateral,
painful, often accompanied by
nausea
• The iris is usually stuck to the
cornea and the eye will feel
relatively firm
• The pupil will not react
Sudden Painless Loss of Vision
Optic Neuritis
• Typically young sudden loss of vision
accompanied by pain on eye movement
• May or may not have a background of MS
• Will have a RAPD
• Otherwise normal exam though there may be
swelling of the optic disc
• There is no role for oral steroids
• IV methylprednisone shortens the duration of the
attack but not the severity or risk of recurrence
• Needs MRI to look for plaques
Ischemic Optic Neuritis
• Will be arteritic or non
arteritic
• May or may not have
optic disc swelling
• Check ESR and CRP as
GCA symptoms are
vague and GCA patients
tend to be poor historians
• Carotid dopplers need to
be done via LMO
Central Retinal Vein Occlusion
• Typically large hypertensive
50+ year old women
• Dilated exam reveals
widespread haemorrhage and
swelling (chronic diabetics look
the same but the vision loss is
not acute)
• Can be ischemic or non
ischemic
• May progress to
neovascularisation and
rubeosis if untreated (the 100
day glaucoma) so followup is
important
Central Retinal Artery Occlusion
• Presents with a very
pale retina (look at
the other side) and
you may or may not
see an embolus
• Needs aggressive
IOP lowering if
presents within 6 hrs
but patients rarely do
• Needs carotid
dopplers via LMO
Wet ARMD
• Usually an elderly
patient with known
ARMD with sudden
loss of central vision
• Needs f/u for
intravitreal Lucentis
(rooms)
Retinal Detachment
• Usually presents with a
few days of
flashes/floaters then a
slowly progressive
shadow or cobweb in 1
eye that doesn’t move or
go away
• Usually sent in by
optometrists who may be
wrong
• Needs VR review/repair
Vitreous Haemorrhage
• Often confused with
detachment as symptoms
are similar
• May be a known diabetic
who bleeds from
neovascularisation or a
traumatic vitreous
detachement
• You won’t be able to see
the retina on dilated exam
and neither will we.
• Needs f/u for a B scan
U/S to ensure no
detachment
Hyphema
• Typically a history of blunt
trauma to the eye and
sudden loss of vision
• The AC may be cloudy
before a blood level
settles in the AC
(microhyphema)
• IOP is usually low but can
go high
• Needs dilating drops,
topical steroids and strict
bed rest
Orbital fractures
• Usually from an assault or
sports injury
• Usually medial wall or floor
• Needs CT to check for globe
rupture
• True entrapment of rectus
muscles is rare, usually in
young teens and they will
vomit/be unwell
• They need oral antibiotics and
are not to blow nose
• Maxfax/plastics need to be
involved.
• If acuity is OK, eye review can
be within the week as an
outpatient
Major Blunt Trauma
• MVAs etc
• High
impact/deceleration
injuries can shear the
optic nerve
• The patients are
usually intubated
Perforating Eye Injury
• Something flies into the eye at
high speed
• Vision is down
• Pupil will look irregular and will
point towards the perforation
• Leave the eye alone
• No pressure on the eye
• Give ADT and an IV
cephalosporin
• Get whatever imaging you can
for intraocular foreign body
(CT is best)
• Keep NBM
Ruptured Globe
• Occurs in high speed
MVAs and assaults
involving bats and
crowbars
• Fists are not usually
enough
• Lids may be so swollen
the eye can’t open – get a
CT
• No pressure on the eye,
ADT and an IV
cephalosporin
• Keep NBM

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Opthalmology in the ED - Dr Andrew White (June 2013)

  • 1. Eye Emergencies Dr Andrew White BMedSci(hons) MBBS PhD FRANZCO Clinical Senior Lecturer Discipline of Ophthalmology and Eye Health Glaucoma Specialist, Westmead Hospital
  • 6. • You can learn a lot from pinhole acuity! • Better with pinhole: refractive/anterior segment problem • Not better: posterior segment problem • Dilate (Tropicamide)! The risk of angle closure is tiny and you are in a hospital!
  • 7. • Pupil exam will tell you a lot. • Look for RAPD – Optic nerve problem • Look for sluggish pupil – Uveitis – Angle closure glaucoma
  • 8. Common Benign Things • Pingueculum • Pterygium • Trabeculectomy bleb
  • 10. Chalazion • Often a child but also young adults • Small lump on the upper or lower lid with some swelling • Usually sent in by LMO as periorbital cellulitis but rarely is • Treated by warm compresses and chlorsig • Often spontaneously discharges
  • 11. Periorbital and Orbital Cellulitis • Usually in young children who are fairly well • May start from a small scratch or chalazion • Usually a 48hr trial of oral antibiotics before moving to IV if needed • Don’t need to do a CT straight off • Children with orbital cellulitis look sick. • CT will show full sinuses and ENT need to be involved to drain it +IV antibiotics
  • 12. Dacryocystitis • Usually a young to middle aged adult with localised cellulitis over the lacrimal sac and a fever • Needs IV antibiotics and warm compresses • A pimple may form that will discharge spontaneously or can be incised by eye team
  • 13. Lid Trauma • We are more concerned about medial canthus injuries than lateral canthus due to location of lacrimal system • Lid margin injuries need specialised repair • Make sure the eye and orbit are intact
  • 15. Corneal Abrasion • Commonly from a finger in the eye • Very painful • Treat with chlorsig and a double firm pad • QID chlorsig drops 7 days • Intensive chlorsig is totally useless • Look under the lids for tarsal foreign body! • Can lead to recurrent erosions
  • 16. Corneal Foreign Body • Typically from angle grinding or small piece of wood • Usually can be lifted off with a cotton bud or 25/23G needle • Rust ring can be removed later in clinic • Chlorsig QID 1 week
  • 17. Subconjunctival Haemorrhage • Looks worse than it is • Trivial • Idiopathic • Not related to INR. Sometimes high BP • Will get better in 2 weeks • May need lubricants for comfort
  • 19. Bacterial Conjunctivitis • Unilateral • Mucous discharge • ?Previous foreign body • Swab the discharge and give chlorsig 7 days • Clinic f/u if no better
  • 20. Viral Conjunctivitis • Fairly common and goes in runs • Characterised by watery discharge and itch • 2 main forms: • Adenoviral • Herpes (simplex and zoster)
  • 21. Adenoviral Conjunctivitis • Probably the most contagious thing on the planet • Wash everything after contact!!! • Often an flu like prodrome • Accompanied by chemosis and lid swelling • Characterised by conjunctival follicles • Typically goes from one eye to the next over a few days • Self limiting but can persist for 2-3 weeks. • Patient is contagious for 2 weeks • Lubricants and cool compresses help
  • 22. Herpes Conjunctivitis • Either HSV 1, 2 or Zoster • Unilateral • Usually fairly mild symptoms • May be accompanied by a dendritic ulcer • Treatment is zovirax ointment 5x a day 10 days • Check the retina!
  • 23. Herpes Zoster Ophthalmicus • Only possible if the V1 division is involved. • Usually only a mild conjunctivitis is involved treated with lubricants and cool compresses • There may be some corneal ulceration • Main concern is retinal involvement – check! • Treatment is systemic oral antivirals (acyclovir. Valtrex etc). • Trigeminal neuralgia may be a long term problem
  • 24. Allergic Conjunctivitis • Can be seasonal, drugs or contact • Frequently bilateral but can be unilateral with mild lid swelling • History is usually the giveaway • Papillae under the lids • Remove the stimulus, lubricate • Call us if contemplating steroids • DO NOT GIVE STERIODS INDEPENDENTLY • There are a number of over the counter topical antihistamines (eg Lomide, Zatiden)
  • 26. Uveitis • Essentially an arthritis of the eye • Usually young, unilateral red eye with photophobia • Flare and cells in the anterior chamber +/- hypopion and irregular sluggish pupil • May be HLAB27 +ve (ankylosing spondylitis) • Other causes: Drugs, HSV, Syphilis, Sarcoid, TB, Bartonella, Lyme disease, LYMPHOMA – beware the elderly patient • Treated with dilating drops and intensive topical steroids (by ophthalmology)
  • 27. Scleritis and Episcleritis • Episcleritis: Young, localised area of redness over the conjunctiva that blanches with phenylephrine 2.5% • Self limiting. Gets better with NSIADS and weak topical steroids • Scleritis: Old, rheumatoid patients but also seen in infections like syphilis • Redness does not blanch • Extremely painful. • Treated with oral steroids and NSAIDS • Risk of perforation
  • 29. Contact Lens Keratitis • Painful, rapidly progressive • Almost always pseudomonas • Characterised by white corneal infiltrates • Can progress to blindness in 24hrs • Needs intensive ciloxan eye drops (hourly) • Chlorsig will do nothing!
  • 30. Chemical Burns • Acid and chemical burns will damage superficial cornea then stop • Alkali burns will continue to penetrate until neutralised • Blindness results not only from initial damage but limbal stem cell failure • Irrigate, irrigate, irrigate! ASAP • Check pH of all chemical injuries at presentation and after irrigation
  • 31. Endophthalmitis • Will be surgical or endogenous • Surgical typically presents day 1-7 post surgery with a rapidly progressive history of pain and redness (it is rare to present months to years later but it does happen) • Endogenous is found in the immunosuppressed, moribund and IV drug users • Blindness is rapid if untreated (hours) • Needs a vitreous tap and intravitreal antibiotics/antifungals/antivirals as determined by ophthalmology
  • 32. Acute Glaucoma • Will be open or closed angle • Open angle usually has a long history of glaucoma and a slowly progressive history • Angle closure glaucoma is typically an elderly, dark iris patient who presents at night (when the eye dilates) • Acute glaucoma is unilateral, painful, often accompanied by nausea • The iris is usually stuck to the cornea and the eye will feel relatively firm • The pupil will not react
  • 33. Sudden Painless Loss of Vision
  • 34. Optic Neuritis • Typically young sudden loss of vision accompanied by pain on eye movement • May or may not have a background of MS • Will have a RAPD • Otherwise normal exam though there may be swelling of the optic disc • There is no role for oral steroids • IV methylprednisone shortens the duration of the attack but not the severity or risk of recurrence • Needs MRI to look for plaques
  • 35. Ischemic Optic Neuritis • Will be arteritic or non arteritic • May or may not have optic disc swelling • Check ESR and CRP as GCA symptoms are vague and GCA patients tend to be poor historians • Carotid dopplers need to be done via LMO
  • 36. Central Retinal Vein Occlusion • Typically large hypertensive 50+ year old women • Dilated exam reveals widespread haemorrhage and swelling (chronic diabetics look the same but the vision loss is not acute) • Can be ischemic or non ischemic • May progress to neovascularisation and rubeosis if untreated (the 100 day glaucoma) so followup is important
  • 37. Central Retinal Artery Occlusion • Presents with a very pale retina (look at the other side) and you may or may not see an embolus • Needs aggressive IOP lowering if presents within 6 hrs but patients rarely do • Needs carotid dopplers via LMO
  • 38. Wet ARMD • Usually an elderly patient with known ARMD with sudden loss of central vision • Needs f/u for intravitreal Lucentis (rooms)
  • 39. Retinal Detachment • Usually presents with a few days of flashes/floaters then a slowly progressive shadow or cobweb in 1 eye that doesn’t move or go away • Usually sent in by optometrists who may be wrong • Needs VR review/repair
  • 40. Vitreous Haemorrhage • Often confused with detachment as symptoms are similar • May be a known diabetic who bleeds from neovascularisation or a traumatic vitreous detachement • You won’t be able to see the retina on dilated exam and neither will we. • Needs f/u for a B scan U/S to ensure no detachment
  • 41. Hyphema • Typically a history of blunt trauma to the eye and sudden loss of vision • The AC may be cloudy before a blood level settles in the AC (microhyphema) • IOP is usually low but can go high • Needs dilating drops, topical steroids and strict bed rest
  • 42. Orbital fractures • Usually from an assault or sports injury • Usually medial wall or floor • Needs CT to check for globe rupture • True entrapment of rectus muscles is rare, usually in young teens and they will vomit/be unwell • They need oral antibiotics and are not to blow nose • Maxfax/plastics need to be involved. • If acuity is OK, eye review can be within the week as an outpatient
  • 43. Major Blunt Trauma • MVAs etc • High impact/deceleration injuries can shear the optic nerve • The patients are usually intubated
  • 44. Perforating Eye Injury • Something flies into the eye at high speed • Vision is down • Pupil will look irregular and will point towards the perforation • Leave the eye alone • No pressure on the eye • Give ADT and an IV cephalosporin • Get whatever imaging you can for intraocular foreign body (CT is best) • Keep NBM
  • 45. Ruptured Globe • Occurs in high speed MVAs and assaults involving bats and crowbars • Fists are not usually enough • Lids may be so swollen the eye can’t open – get a CT • No pressure on the eye, ADT and an IV cephalosporin • Keep NBM