It describes about the procedure of Hess charting. it serves as a great tool to understand the concepts involved. Suitable for optometry course. This is not a routine procedure but an important procedure which is used in diagnosis.
Contact lens for congenital aphakia and other eye conditions for infants and toddlers. The slide presentation encompasses indications for CL fitting in paediatric, contact lens options, fitting techniques, challenges and contact lens as myopia control.
Presbyopia/ Methods of Presbyopic Addition Determination (healthkura.com)Bikash Sapkota
DIRECT DOWNLOAD LINK ❤❤https://healthkura.com/presbyopia-near-addition/❤❤
Dear viewers Check Out my other piece of works at ❤❤❤ https://healthkura.com ❤❤❤
Presbyopia and techniques of measurement
A fantastic presentation in the topic "Presbyopia and techniques of measurement"
A detailed information about presbyopia, techniques of presbyopic add determination and different correction methods.
Informative slide presentation on presbyopia for ophthalmology residents, ophthalmologists, optometrists, ophthalmic assistants, ophthalmic technicians, ophthalmic nurses, medical students, medical professors, teaching guides.
Presentation Contents:
--Introduction to presbyopia
-Types of presbyopia
-Risk factors
-Symptoms and signs
-Refractive error and presbyopia
-Methods of determining near add.
-Management of presbyopia
In a nutshell..
- The evaluation and management of presbyopia are important because significant functional deficits can occur when the condition is left untreated
- Undercorrected or uncorrected presbyopia can cause significant visual disability and have a negative impact on the pt.'s quality of life
- Finally, every tentative addition should be adjusted according to the particular needs of the patient
For Further Reading:
-Clinical Procedures in Optometry by J.D. Bartlett, J.B. Eskridge, J.F. Amos
-Primary Care Optometry by Theodere Grosvenor
-Borish’s Clinical Refraction by W.J. Benjamin
-Clinical Procedures for Ocular examination by Carlson et al
-American Academy of Ophthalmology
-Optometric Clinical Practice Guideline by American Optometric Association
-Internet
Follow me to get in touch with optometric and ophthalmic updates.
It describes about the procedure of Hess charting. it serves as a great tool to understand the concepts involved. Suitable for optometry course. This is not a routine procedure but an important procedure which is used in diagnosis.
Contact lens for congenital aphakia and other eye conditions for infants and toddlers. The slide presentation encompasses indications for CL fitting in paediatric, contact lens options, fitting techniques, challenges and contact lens as myopia control.
Presbyopia/ Methods of Presbyopic Addition Determination (healthkura.com)Bikash Sapkota
DIRECT DOWNLOAD LINK ❤❤https://healthkura.com/presbyopia-near-addition/❤❤
Dear viewers Check Out my other piece of works at ❤❤❤ https://healthkura.com ❤❤❤
Presbyopia and techniques of measurement
A fantastic presentation in the topic "Presbyopia and techniques of measurement"
A detailed information about presbyopia, techniques of presbyopic add determination and different correction methods.
Informative slide presentation on presbyopia for ophthalmology residents, ophthalmologists, optometrists, ophthalmic assistants, ophthalmic technicians, ophthalmic nurses, medical students, medical professors, teaching guides.
Presentation Contents:
--Introduction to presbyopia
-Types of presbyopia
-Risk factors
-Symptoms and signs
-Refractive error and presbyopia
-Methods of determining near add.
-Management of presbyopia
In a nutshell..
- The evaluation and management of presbyopia are important because significant functional deficits can occur when the condition is left untreated
- Undercorrected or uncorrected presbyopia can cause significant visual disability and have a negative impact on the pt.'s quality of life
- Finally, every tentative addition should be adjusted according to the particular needs of the patient
For Further Reading:
-Clinical Procedures in Optometry by J.D. Bartlett, J.B. Eskridge, J.F. Amos
-Primary Care Optometry by Theodere Grosvenor
-Borish’s Clinical Refraction by W.J. Benjamin
-Clinical Procedures for Ocular examination by Carlson et al
-American Academy of Ophthalmology
-Optometric Clinical Practice Guideline by American Optometric Association
-Internet
Follow me to get in touch with optometric and ophthalmic updates.
Screening guidelines for BP, Dental, Hearing and Vision evaluation in children. Also injury prevention, anemia screening and newborn screening in brief.
Eye survival for medical students, a narrated presentation.TheEyeExpert
This narrated powerpoint presentation will cover all the information a medical student should know about ophthalmology in relation to common conditions and rare but important conditions.
Similar to Vision Assessment and Vision Screening in Children, Refractive Error and Spectacle Correction, Amblyopia, Strabismus and Nystagmus (20)
250+ High Frequency MCQs in Optometry and OphthalmologyRabindraAdhikary
The collection of high-ranked, top-rated high frequency multiple-choice questions suitable for any examination of optometry, ophthalmology and ophthalmic sciences with their answers for FREE. No Log in, No Pay!!
Contact lens care and maintenance
RGP care
Soft Contact lens care
Silicon Hydrogel Care
Contact Lens Disinfection
Thermal disinfection
Chemical Disinfection
Oxidative chemical disinfection
Troubleshooting bifocals and Market Availability in Nepal
Bifocals in Anisometropia
Prismatic Effect in Bifocal
Bifocal Prescription
Bifocals in High Astigmatism
Variables: Types and their Operational Definitions
Unit III: Problem identification formulation of research objectives and hypothesis (as part of M.Optom Curriculum of Pokhara University, Nepal)
Cholinergic agent: Autonomic Drugs
According to the M. Optom curriculum, we have prepared a concise presentation on Cholinergic or parasympathomimetic or cholinomimetic drugs
Multiple Choice Questions (MCQs) for Masters of Optometry Entrance Examinatio...RabindraAdhikary
Multiple Choice Questions (MCQs) for Masters of Optometry Entrance Examination, Pokhara University NEPAL
MCQs Optometry Nepal
Here we have included syllabus of entrance examinations for Master of Optometry in Pokhara University, entry requirements of candidate for the master of optometry course and multiple choice questions that appeared in the entrance examinations of 2019.
Prepared by: Rabindra Adhikary
for more MCQs:
http://ravinems.blogspot.com/2019/05/multiple-choice-questions-mcqs-for.html
Visual Implication in Diabetes Mellitus
These slides talk in detail about the visual implications of Diabetes Mellitus and how to address them systematically
Systemic Diseases and the Eye
Various systemic diseases affect the eye and it's functioning. Here we present those common systemic diseases that are responsible to cause effects in the eye.
What are the diseases that affect eye?
Eye is affected by the following diseases:
Systemic Hypertension (Increased blood pressure of the body)
Diabetes Melitus (Increased blood sugar level)
Systemic Lupus Erythromatosus (SLE)
AIDS and other Venereal Diseases like Syphilis
Sickle Cell Anemia,
Eales Disease and many more.
Look at the slides.
Ophthalmic Prisms: Prismatic Effects and DecentrationRabindraAdhikary
Ophthalmic Prisms: Prismatic Effects and Decentration
here we discuss about the ophthalmic prisms, the prismatic effects as caused by the decentration( moving the optical center away from the visual axis)
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Triangles of Neck and Clinical Correlation by Dr. RIG.pptx
Vision Assessment and Vision Screening in Children, Refractive Error and Spectacle Correction, Amblyopia, Strabismus and Nystagmus
1. Vision Assessment and Vision Screening in
Children
Refractive error and spectacle correction
Amblyopia
Strabismus
Nystagmus
Rabindra Adhikary
M. Optom 1st Batch
Tilganga Institute of ophthalmology (TIO)
Pokhara University
Facilitators: Dr Srijana Adhikari, Dr Rojeeta Parajuli
Date: 4th of June, 2019
2. Pediatric Ophthalmology
– Layout
• Vision Assessment and Vision Screening in
Children
• Refractive error and spectacle correction
• Amblyopia
• Strabismus
• Nystagmus
3. Visual acuity
• Preverbal children
– Motor or sensory response to a threshold stimulus
– Known size at a known distance
• Pre-literate and verbal Children
– Smallest target of known size at known distance
correctly verbally identified by a child
4. Visual Milestones
Age Visual Behavior
0-1 month Fixate &Turns eye and head to look at the light source /
Looks high contrast object (mother’s hairline) + Horizontal Tracking
At 1 month, fixation is central, steady and maintained
3 month Vertical and circular tracking
Interested in mobiles
Lip-reading / responsive smile
6 months Reaches towards, and later grasps the hanging objects
Observes toys falling and rolling away
10 Looks for hidden toys
Interested in pictures
1 year Recognize people
2 years Picture Matching
3 years Letter matching of single letters
5 years Snellen chart by matching or naming
5. Gross Estimate of VA
Age Snellen’s Equivalent
Birth 6/120
4 months 6/60
6 months 6/36
1 year 6/18
2 years 6/6
7. Resolution Acuity
• Preferential Looking
– Child prefers to look at the grating
• Striped grating
• Uniform filed
– Forced choice preferential looking (FCPL)
– 2 months to 1 year or pt with mental disabilities
– Visual threshold=75% correct inferences
• Drawbacks of PFL
– Difficult to hold a child’s attention for long
– Reliability is dependent on many trials
– Near test
10. VEP
• Measures electrical activity directly from the
scalp through surface electrodes
– Amplitude and timing of wave of electrical activity
– Go on presenting finer and finer stimuli
• Until zero response amplitude
• Abnormal VEP?
– Problem with the visual
information reaching cortex
11.
12. Age-wise VA assessment
• Infants
– PFL/OKN/VEP
– Cat ford Drum
• 1-2 years
– STYCAR
• For retards too
– Boeck Candy test
– Worth’s Ivory Ball test
• 2-3 years
– Miniature Toy test
– Cardiff Acuity test
– Coin test
– Lea symbols
• 3-5 years
– Allen’s picture cards
– Sheridan’s HOTVX letter test
– Lippman’s HOTV Test
• 5-6 years
– Snellen
13. Screening
• Age ranges and visual/ocular pathology to be
detected by screening:
Pre-term Perinatal/infantile Pre-school School age
ROP Congenital cataract
Glaucoma
Anterior segment
disorder
Amblyogenic
factors
-anisometropia
-strabismus
-high refractive
error
-Ptosis
-Media opacity
Refractive error
14. Fixation Reflex
• CSM
– Monocular central fixation (C)
• No eccentric fixation
– Hold steady Fixation (S)
– Maintain fixation (M)
• With the viewing eye
• Even when the fellow eye is uncovered
• F + F
– Fixate and Follow the light stimuli
– Monocular viewing conditions
15. Pupillary Response
• Birth- 3 years (or upto when VA can’t be
measured)
• Pupil reactions to the changes of light stimulus
– Direct
– Consensual
• Not equivalent to visual ability but indicates
– Intact neurologic visual pathways
16. Red Light Reflex
• Bruckner Test
• 0-3 years
• Ophthalmoscope
– Position the light to illuminate BE
• Reflex
– Symmetrical
– Equal intensity
– Healthy red color
17. Corneal Light Reflex
• Hirschberg’s test
• 2 months- 8 years
• Strabismus (D/Dx: pseudostrabismus)
• Corneal reflection of Penlight
– Positioned symmetrically on both pupils
18. Cover test
• To know if eye deviates when fusion is
interrupted
– Observing the fixating eye when other eye is
covered (Cover test)
– Observing the eye being uncovered for movement
when the occluder is shifted to the other eye
(alternate cover)
– Observing the eye being uncovered for movement
when the occluder is removed (cover uncover)
19. Stereo-acuity
• 3-8 years
• Needs polarized glass
• Random dot E or stereo butterfly
• To check problems with stereo-acuity and
depth perception
20. Color vision
• At 6 years (first grader)
• Others: optional
• To check color deficiency
– Ishihara Pseudo-isochromatic Plates,
– Color Vision Testing Made Easy
– Good-Lite Book of Color Plates.
• Fluorescent desk lamp - if enough natural
daylight is not available.
21. Age vs Refractive Error
General Trends
Premature Infant High myopia ( ± ROP)
Newborn (full term) baby Hyperopia ( 1-2 D)
Toddler (preschool) Less hyperopia / low Myopia
(towards Emmetopia)
School Children Emmetropia or school myopia
Adolescent Myopia
22. Refraction
• Cycloplegic Refraction:
– Greater range of accommodation
– Fluctuating fixation
– Pseudomyopia
– Strabismus
– High hyperopia / latent Hyperopia
– Inconsistent results
• Near Retinoscopy
23. Prescription
• Should not interfere with the
emmetropization
• Only if beyond normal range for that age
24. Hyperopia upto 1 year
• Isohyperopia
– Error ≥ 2D & deviation present
• Prescribe the full cyclopegic correction
• Mandate frequent F/u if < 2 D
– Prescribe partial (2/3rd) if ≥ 5D
• Anisohyperopia
– < 2.5 D (No Rx if no deviation)
– ≥ 2.5
• Partial for no deviation
• Full Rx if esodeviation
25. Myopia upto 1 year
• Isomyopia < -5 D
– No Rx (Regular monitoring)
• Isomypia ≥ 5 D
– Rx with 1-2 D less
• Aniso-mypia ≥ 2.5 D
– Prescribe
26. Hyperopia in preschoolers
(upto 6 years)
• Isohyperopia
– Deviation present? Full Rx if error ≥ 1.5 D
– No deviation? Partial Rx (2/3rd) if ≥ 2.5 D
• Anisohyperopia
– < 1.5 ( No Rx, R/w 3 months)
– ≥1.5 D (partial for no deviation)
• Full for esodeviation
27. Myopia in Preschoolers
• Isomyopia < 3
– No Rx
• Isomyopia ≥ 3
– Prescribe but undercorrection
• Aniso-myopia ≥ 2D
– Prescribe
28. School Age
• Asymptomatic Isometropia ≥ 1.5 Prescribe
– Near full correction (emmetropization ended)
• Hyperopic anisometropia ≥ 1D
– Full correction
• Myopia?
– Optimum correction is warranted
29. Aphakic Correction
• Challenges
– High plus
– Astigmatism
– No accommodation
• Correction
– Overcorrect by 2-3 D in first few months
• Near consideration
– Decrease the overcorrection to 1-2D ≈1 year
• Intermediate distance consideration
– Bifocals in school age
• N+D consideration
Lawrence formula for aphakic correction,
P = +11 + half of pre-op error
30. Amblyopia
• Visual impairment without obvious organic
ocular pathology
– observer sees nothing and the patient very little
(Albrecht von Graeffe)
• Critical period
– Visual development permanently disrupted if
deprived of stimulation
– Age of neural plasticity
• Weeks of deprivation betn 3-6 months
• Months of deprivation below 8 years
32. Amblyopia
• Most common cause
– Anisometropia (>1.5D)
• Anisohyperopia is more amblyogenic than anisomyopia
• Astigmatism
– Can’t be overcome by accommodation
• >1.5D is amblyogenic
• >1 D (>10o from90/180)
– Full cylinder correction recommended
• Myopia ≥ 3D
• Hyperopia ≥ 3.5
33. Strabismus
• Constancy
– Constant or intermittent
• Laterality
– Unilateral, alternate
• Horizontal deviation
– Exo or eso
• Vertical deviation
– Hypo or Hyper
• Comitancy
– Comitant or incomitant
• Cause
– Primary or secondary
• Age of onset
– Infantile or acquired
34. Esodeviation
1. Comitant Eso
A. Accommodative
• Refractive (normal AC/A)
• Non-refractive (High AC/A)
• Hypo-accommodative
• Partially accommodative
B. Non-accomodative
• Infantile (birth to 6 months)
• Acquired
– Basic
– Non accommodative convergence excess
– Esotropia in Myopia
– Acute esotropia
– Divergence insuffieciency
– Cyclic esotropia
C. Microtropia
D. Nystagmus Blockage Syndrome
35. 2. Incomitant Eso
A. Paralytic
B. Non paralytic or non-paretic
i. A and V pattern
ii. Retraction Syndromes
iii. Mechanical restrictive esodeviation
– Congenital fibrosis
– Acquired-restrictive (trauma, myopathy, surgery, etc)
3. Secondary Eso
A. Sensory
B. Consecutive
38. Congenital Manifest Nystagmus
• First 3 months of life
• Cause
– Cataract
– Congenital glaucoma
– Aniridia
– Achromatopsia
– Down’s syndrome
– High Myopia
– Optic Nerve Hypoplasia
– Leber’s amaurosis
– OCA
39. Latent Vs Manifest-latent
• Latent
– Evoked by occluding one eye
– Absence when both eyes are open
• Manifest latent
– Latent nystagmus that gets evident at some gazes
– Is lesser in amplitude than the latent one
• Ocular Nystagmus
– Congenital (or with in 1st week ) ocular defects
• Spasmus Nutans
– 1st year of life and disappears 1-2 years
– High Frequency (7Hz), small amplitude
– Head nodding
40. • Sensory Defect Nystagmus
– Cause: inadequate image formation at fovea
– Anterior visual pathway disease
– Always B/L and horizontal
– Pendular and oscillates with equal velocity in both
direction
• Motor Defect
– Cause: efferent mechanism disruption
– Centers or pathways for oculomotor pathways
– Null Point: VA improved due to dampening of
Nystagmus