Cerebral palsy (CP) is a non-progressive brain injury that causes motor dysfunction. Rehabilitation aims to improve mobility and function through physiotherapy, occupational therapy, bracing, assistive devices and more. Therapies use neurofacilitation techniques like Vojta and Bobath to normalize muscle tone and facilitate normal movement. The goals are to prevent deformities and maximize a child's independence and quality of life.
Still's disease, sometimes referred to as Adult-onset Still's disease (AOSD) is a rare systemic inflammatory disease characterized by the classic triad of persistent high spiking fevers, joint pain and a distinctive salmon-colored bumpy rash.
This presentation contains detailed knowledge about Down's Syndrome its types, clinical presentation, diagnosis, medical and physio therapeutic management of the condition.
Down syndrome is a condition in which a person has an extra chromosome. Chromosomes are small “packages” of genes in the body. They determine how a baby’s body forms and functions as it grows during pregnancy and after birth. Typically, a baby is born with 46 chromosomes. Babies with Down syndrome have an extra copy of one of these chromosomes, chromosome 21. A medical term for having an extra copy of a chromosome is ‘trisomy.’ Down syndrome is also referred to as Trisomy 21. This extra copy changes how the baby’s body and brain develop, which can cause both mental and physical challenges for the baby.
Still's disease, sometimes referred to as Adult-onset Still's disease (AOSD) is a rare systemic inflammatory disease characterized by the classic triad of persistent high spiking fevers, joint pain and a distinctive salmon-colored bumpy rash.
This presentation contains detailed knowledge about Down's Syndrome its types, clinical presentation, diagnosis, medical and physio therapeutic management of the condition.
Down syndrome is a condition in which a person has an extra chromosome. Chromosomes are small “packages” of genes in the body. They determine how a baby’s body forms and functions as it grows during pregnancy and after birth. Typically, a baby is born with 46 chromosomes. Babies with Down syndrome have an extra copy of one of these chromosomes, chromosome 21. A medical term for having an extra copy of a chromosome is ‘trisomy.’ Down syndrome is also referred to as Trisomy 21. This extra copy changes how the baby’s body and brain develop, which can cause both mental and physical challenges for the baby.
CP is the most common motor disability in childhood. Cerebral means having to do with the brain. Palsy means weakness or problems with using the muscles. CP is caused by abnormal brain development or damage to the developing brain that affects a person's ability to control his or her muscles.
•Catchall term for a variety of disorders that affect a child’s ability to
•Move and
•Maintain posture and balance
•It only affects the brain’s ability to control the muscles and not muscles or nerves themselves
•The symptoms and which areas are affected will depend on the severity and location of the brain injury
•May include Intellectual Disbaility, seizures, language disorders, learning disabilities, vision and hearing problems.
Loading…
•Affects the development of the child so also known as a developmental disability
•Usually a life long disability but mild form may recover soon.
•The problem affects the child’s future ability to learn and do
•The effect depends on a number of factors like
Local Advanced Lung Cancer: Artificial Intelligence, Synergetics, Complex Sys...Oleg Kshivets
Overall life span (LS) was 1671.7±1721.6 days and cumulative 5YS reached 62.4%, 10 years – 50.4%, 20 years – 44.6%. 94 LCP lived more than 5 years without cancer (LS=2958.6±1723.6 days), 22 – more than 10 years (LS=5571±1841.8 days). 67 LCP died because of LC (LS=471.9±344 days). AT significantly improved 5YS (68% vs. 53.7%) (P=0.028 by log-rank test). Cox modeling displayed that 5YS of LCP significantly depended on: N0-N12, T3-4, blood cell circuit, cell ratio factors (ratio between cancer cells-CC and blood cells subpopulations), LC cell dynamics, recalcification time, heparin tolerance, prothrombin index, protein, AT, procedure type (P=0.000-0.031). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and N0-12 (rank=1), thrombocytes/CC (rank=2), segmented neutrophils/CC (3), eosinophils/CC (4), erythrocytes/CC (5), healthy cells/CC (6), lymphocytes/CC (7), stick neutrophils/CC (8), leucocytes/CC (9), monocytes/CC (10). Correct prediction of 5YS was 100% by neural networks computing (error=0.000; area under ROC curve=1.0).
Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
DISSERTATION on NEW DRUG DISCOVERY AND DEVELOPMENT STAGES OF DRUG DISCOVERYNEHA GUPTA
The process of drug discovery and development is a complex and multi-step endeavor aimed at bringing new pharmaceutical drugs to market. It begins with identifying and validating a biological target, such as a protein, gene, or RNA, that is associated with a disease. This step involves understanding the target's role in the disease and confirming that modulating it can have therapeutic effects. The next stage, hit identification, employs high-throughput screening (HTS) and other methods to find compounds that interact with the target. Computational techniques may also be used to identify potential hits from large compound libraries.
Following hit identification, the hits are optimized to improve their efficacy, selectivity, and pharmacokinetic properties, resulting in lead compounds. These leads undergo further refinement to enhance their potency, reduce toxicity, and improve drug-like characteristics, creating drug candidates suitable for preclinical testing. In the preclinical development phase, drug candidates are tested in vitro (in cell cultures) and in vivo (in animal models) to evaluate their safety, efficacy, pharmacokinetics, and pharmacodynamics. Toxicology studies are conducted to assess potential risks.
Before clinical trials can begin, an Investigational New Drug (IND) application must be submitted to regulatory authorities. This application includes data from preclinical studies and plans for clinical trials. Clinical development involves human trials in three phases: Phase I tests the drug's safety and dosage in a small group of healthy volunteers, Phase II assesses the drug's efficacy and side effects in a larger group of patients with the target disease, and Phase III confirms the drug's efficacy and monitors adverse reactions in a large population, often compared to existing treatments.
After successful clinical trials, a New Drug Application (NDA) is submitted to regulatory authorities for approval, including all data from preclinical and clinical studies, as well as proposed labeling and manufacturing information. Regulatory authorities then review the NDA to ensure the drug is safe, effective, and of high quality, potentially requiring additional studies. Finally, after a drug is approved and marketed, it undergoes post-marketing surveillance, which includes continuous monitoring for long-term safety and effectiveness, pharmacovigilance, and reporting of any adverse effects.
Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
ADVANCED BIOPHARMACEUTICS & PHARMACOKINETICS : UNIT 5
APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMS By - AKANKSHA ASHTANKAR
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
These lecture slides, by Dr Sidra Arshad, offer a quick overview of the physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar lead (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
6. Describe the flow of current around the heart during the cardiac cycle
7. Discuss the placement and polarity of the leads of electrocardiograph
8. Describe the normal electrocardiograms recorded from the limb leads and explain the physiological basis of the different records that are obtained
9. Define mean electrical vector (axis) of the heart and give the normal range
10. Define the mean QRS vector
11. Describe the axes of leads (hexagonal reference system)
12. Comprehend the vectorial analysis of the normal ECG
13. Determine the mean electrical axis of the ventricular QRS and appreciate the mean axis deviation
14. Explain the concepts of current of injury, J point, and their significance
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
2. Cerebral palsy
CP has been defined as
Non progressive injury to the immature
brain
Leading to motor dysfunction
Lesion is not progressive, but
The clinical manifestations change over
time.
3. Progressive disorders resembling CP
Type I Arginase deficiency
Sjögren – Larsson syndrome
Lesch - Nyhan syndrome
Chiari Type I malformation
Dandy - Walker syndrome
Angelman syndrome
Nonprogressive disorders resembling CP
Mental retardation
Deprivation
Malnutrition
Non-motor handicaps (blindness)
Motor handicaps (spina bifida, myopathies)
4. Risk factors
Prenatal
Prematurity (< 36 weeks)
Low birth weight (less than 2500 g)
Maternal epilepsy
Infections (TORCH)
Bleeding in the third trimester
Severe toxemia,
Eclampsia
Multiple pregnancies
Placental insufficiency
Drug abuse and trauma
Perinatal
Prolonged and difficult labor
Premature rupture of membranes
Presentation anomalies
Vaginal bleeding at the time of labor
Bradycardia and hypoxia
Postnatal
CNS infection (encephalitis,
meningitis)
Neonatal hyperbilirubinemia
Head trauma
Seizures and Coagulopathies
6. Anatomical classification
Location Description
Hemiplegia Upper and lower extremity on one side of body
Diplegia Four extremities,
legs more affected than the arms
Quadriplegia Four extremities plus the trunk, neck and face
Triplegia Both lower extremities and one upper
extremity
Monoplegia One extremity (rare)
Double hemiplegia Four extremities, arms more affected than the
legs
7.
8.
9. Spastic CP
Increase in the physiological resistance of muscle to passive
motion.
It is part of the upper Motor neuron syndrome characterized with
Hyperreflexia
Clonus,
Extensor plantar responses and
Primitive reflexes
Spastic CP is the most common form of CP.
Approximately 70% to 80% of children with CP are spastic.
Spastic CP is anatomically distributed into
Hemiplegia
Diplegia
Quadriplegia
10. Early signs of CP
Signs suggestive of CP in an infant
Abnormal behaviour
Excessive docility or irritability
Poor eye contact
Poor sleep
Oromotor problems
Frequent vomiting
Poor sucking
Tongue retraction
Persistent bite Grimacing
Poor mobility
Poor head control
Hand preference before 2 years of age
Abnormal tone
11. Early differential diagnosis in
developmental disability
Cerebral palsy Mental retardation
Risk factors Often present Mostly absent
Complaints Irritable, sleepless
baby
Easy baby
Milestones Delayed Delayed
Muscle tone Increased Normal or reduced
Primitive reflexes Persist Normal disappearance
Postural reflexes Delayed appearance Normal or Delayed
appearance
Focal signs Appear Absent
12. Neuromotor problems in CP
Difficulty with flexing and extending
the body against gravity
Sitting
Functional ambulation
13. Impairments
Primary impairments (due to the brain lesion)
Muscle tone (spasticity, dystonia)
Balance
Strength
Selectivity
Sensation
Secondary impairments
Contractures (equinus, adduction)
Deformities (scoliosis)
Tertiary impairments
Adaptive mechanisms (knee hyperextension in stance)
14. Contractures and deformity
Common sites for
contracture
Upper extremity
Pronator
Wrist and finger flexor
Thumb adductor
Lower extremity
Hip adductor-flexor
Knee flexor
Ankle plantar flexor
Common sites for
deformity
Spine
Scoliosis and kyphosis
Hip
Subluxation, Dislocation
Femur & tibia
Internal or external torsion
Foot
Equinus, valgus and
varus.
15. Associated problems in CP
Seizures
Visual impairments
Intellectual impairment
Learning disabilities
Hearing problems
Communication problems
Oromotor dysfunction
Gastrointestinal problems and nutrition
Teeth problems
Respiratory dysfunction
Bladder and bowel problems
Social and emotional disturbances
16. Epileptic seizures
Seizures affect about 30 to 50% of CP patients
They are most Common in the
Quadriplegics and
Hemiplegics,
Patients with mental retardation
Postnatally acquired CP.
Seizures most resistant to drug therapy occur in hemiplegics.
Seizure frequency increases in the preschool period.
Electroencephalograms are necessary for the diagnosis of
seizure disorder
17. Visual impairments seen in
CP
Pathology Clinical finding
Damage to the visual cortex Cortical blindness
Damage to the optic nerve Blindness
Loss of oculomotor control Loss of binocular vision
Refraction problems Myopia
18. Nutritional and oromotor issues
Teeth problems
Dentin Primary or hyperbilirubinemia
Malocclusion Spasticity
Tooth decay Feeding, swallowing
problems
Gingival hyperplasia Antiepileptic drug use
Causes of inadequate food intake
Difficulty chewing and swallowing
Hyperactive gag reflex
Spasticity of oropharyngeal muscles
Loss of selective control of oropharyngeal muscles
Gastroesophageal reflux
20. Psychosocial problems
Extremely stressful for the family and the child when he
grows up.
Stress leads from denial to anger, guilt and depression.
Coping with the emotional burden of disability is easier if the
family has strong relationships, financial security, and
supportive members of the community.
The child and the family need to find ways to connect to
each other.
A healthy relationship between the mother and the child
forms the basis of future happiness.
Prevention or appropriate treatment of associated
problems Improves the quality of life of the child and the
family.
21. Rehabilitation
Rehabilitation is the name given to all
diagnostic and therapeutic procedures
Which aim to develop maximum
physical social and vocational function
in a diseased or injured person
22. Goals of rehabilitationGoals Objectives of rehabilitation
Improve mobility Teach the child to use his remaining potential
Teach the child functional movement
Gain muscle strength
Prevent deformity Decrease spasticity
Improve joint alignment
Educate the parents To set reasonable expectations
Do the exercises at home
Teach daily living
skills
Have the child participate in daily living activities
Social integration Provide community and social support
24. Rehabilitation planning
Example: Independent standing
State the necessary time period
Plan the methods to achieve this goal
Evaluate the end state.
Revise the treatment program
25. Therapy program
Age group Program
Infant Stimulating
Advanced postural equilibrium
Balance reactions for head and trunk
control
Toddler & pre-schooler Stretching the spastic muscles
Strengthening the weak ones
Promoting mobility
Adolescent Improving cardiovascular status
26. Principles of therapy
methods
Support the development of multiple systems such as
Cognitive
Visual
Sensory
Musculoskeletal
Involve play activities to ensure compliance
Enhance social integration
Involve the family
Have fun
27. Gross Motor Function Classification
System
Class Goal of treatment
1- Walks independently, speed,
balance & coordination reduced.
Diminish energy expenditure
Decrease level of support
Improve appearance
2- Walks without assistive devices but
limitations present.
Diminish energy expenditure
Decrease level of support
Improve appearance
3- Walks with assistive devices Improve gait
Improve standing
4- Transported or uses powered
mobility
Decrease pain
Improve sitting & standing
5- Severely limited, dependent on
wheelchair.
Better positioning
Decrease pain
Improve hygiene
29. Neurofacilitation techniques
Sensory input to the CNS produces reflex motor output.
Various neurofacilitation techniques are based on this
basic principle.
All of the techniques aim to normalize muscle tone
To establish advanced postural reactions and to
facilitate normal movement patterns.
30. Vojta method of therapy
18 points in the body –
crawling and reflex rolling.
placing the child in particular positions and stimulation of the key
points in the body would enhance CNS development
In this way the child is presumed to learn normal movement
patterns in place of abnormal motion.
Applied by the primary caregiver at home at least 4-5 times daily
and stopped after a year if there is no improvement
31. Bobath neurodevelopmental
therapy
This is the most commonly used therapy method in CP worldwide.
It aims to
Normalize muscle tone
Inhibit abnormal primitive reflexes
Stimulate normal movement.
It uses the idea of reflex inhibitory positions to decrease spasticity
and stimulation to promote the development of advanced postural
reactions.
32. It is believed that through positioning and stimulation a
sense of normal movement will develop.
To teach the mother how to position the child at home
during feeding and other activities.
The baby is held in the antispastic position to prevent
contracture formation.
33. Occupational therapy
OT aims to improve hand and upper extremity function in the child
through play and purposeful activity .
There are defined systematic treatment methods for occupational
therapy.
Sensory integration therapy aims to enhance the child’s ability to
organize and integrate sensory information.
In response to sensory feedback, CNS perception and
execution functions may improve and the motor planning
capacity of the child may increase.
.
34. Constraint induced movement therapy
Where the normal hand is constrained
Paralytic hand is forced to function
Useful in children with hemiplegia.
Begin therapy toward one year of age when the child can
feed himself using a spoon and play with toys.
Teach the child age appropriate self care activities such as
dressing, bathing and brushing teeth.
Encourage the child to help with part of these activities even if
he is unable to perform them independently
35. play
Always include play activities in the rehabilitation
program.
Play improves mental capacity and provides
psychological satisfaction.
Organized play can address specific gross and fine
motor problems
This increases the child’s compliance with therapy.
Riding a toy horse may improve
Weight shift over the pelvis
Swinging may improve sensation of movement.
36. Advantages of swimming
Normalizes muscle tone
Decreases rate of contracture
Strengthens muscles
Improves cardiovascular fitness
Improves walking
Horseback riding Improves
Head control
Trunk balance
Normalize muscle tone
Positive emotions
Self esteem
37. Recreational programs
Arts and crafts
Music
Dancing
Wheelchair dancing
Drama,
Camping
Fishing
Scuba diving
38. OTHERS
Speech therapy
Hearing aids and implants
Drugs
Antispasmodics
Baclofen Diazepam
Clonidine Thizanidine
Dantroline Botulinum injections
Neoromuscular blockers
Proton pump inhibitors for GERD
Analgesics for pain
Respiratory physiotherapy
39. Goals of brace
Increase function
Prevent deformity
Keep joint in a functional position
Stabilize the trunk and extremities
Facilitate selective motor control
Decrease spasticity
Protect extremity from injury in the
postoperative phase
40. Braces in CP
Ankle foot orthoses
Knee-ankle foot orthoses
Hip abduction orthoses
Thoracolumbosacral orthoses
Supramalleolar orthoses
Foot orthoses
Hand splints
41. Functions of the AFO
Main function Keep the foot in a
plantigrade
position
Stance phase Stable base of support
Swing phase Prevent drop foot
At night Prevent contracture
42. Spinal braces
To slow the progression of deformity
To delay surgery
To allow skeletal growth
To assist sitting balance
To protect the surgical site from excessive loading
after surgery
44. Benefits of Standers
Support erect posture
Enable weight bearing
Stretch muscles to prevent contractures
Decrease muscle tone
Improve head and trunk control
45. Goals of orthopaedic surgery
Goal Operate to achieve
Walking potential Functional
ambulation
Good hip and knee
extension Stable hips
Plantigrade stable feet
No walking
potential
Sitting balance Straight spine
Horizontal pelvis
Stable hips
46. Ortho surgeries in CP
Types of CP Surgical procedures most often performed
Quadriplegic Hip adductor flexor release
Osteotomy Spine fusion
Diplegic Hamstring-gastrocnemius lengthening
Hip adductor-flexor lengthening
Derotation osteotomy
Rectus femoris transfer
Hemiplegic Gastrocnemius lengthening
Split tibialis anterior & posterior transfer
Tibialis posterior lengthening
47. Message
Enhance Familial bonding and support
Therapy along with peers
No aggressive therapy
Don’t give false hopes
Include sports and recreational activities
Successful rehabilitation includes
Prevention of additional problems
Reduction of disability
Community integration.
Rehabilitation is successful if
Child is happy
Parents are well adjusted