Evaluation of prosthetic valve function and clinical utility.Ramachandra Barik
Many of the prosthesis-related complications can be prevented or their impact minimized through optimal prosthesis selection in the individual patient and careful medical management and follow-up after implantation.
Evaluation of prosthetic valve function and clinical utility.Ramachandra Barik
Many of the prosthesis-related complications can be prevented or their impact minimized through optimal prosthesis selection in the individual patient and careful medical management and follow-up after implantation.
A transesophageal echocardiogram, or TEE, is an alternative way to perform an echocardiogram. A specialized probe containing an ultrasound transducer at its tip is passed into the patient's esophagus. This allows image and Doppler evaluation which can be recorded. It has several advantages and some disadvantages compared with a transthoracic echocardiogram.
A transesophageal echocardiogram, or TEE, is an alternative way to perform an echocardiogram. A specialized probe containing an ultrasound transducer at its tip is passed into the patient's esophagus. This allows image and Doppler evaluation which can be recorded. It has several advantages and some disadvantages compared with a transthoracic echocardiogram.
Transesophaheal echo cardiography, the basic views. It is a diagnostic procedure to visualize the heart and have a better understanding of the structure and functions of the heart
A transesophageal echocardiogram (TEE) uses echocardiography to assess the structure and function of the heart. During the procedure, a transducer (like a microphone) sends out ultrasonic sound waves. When the transducer is placed at certain locations and angles, the ultrasonic sound waves move through the skin and other body tissues to the heart tissues, where the waves bounce or "echo" off of the heart structures. The transducer picks up the reflected waves and sends them to a computer. The computer displays the echoes as images of the heart walls and valves.
A traditional echocardiogram is done by putting the transducer on the surface of the chest. This is called a transthoracic echocardiogram. A transesophageal echocardiogram is done by inserting a probe with a transducer down the esophagus. This provides a clearer image of the heart because the sound waves do not have to pass through skin, muscle, or bone tissue. The TEE probe is much closer to the heart since the esophagus and heart are right next to each other.
Trio of Rheumatic Mitral Stenosis, Right Posterior Septal Accessory Pathway a...Ramachandra Barik
A 57-year-old male presented with recurrent palpitations. He was diagnosed with rheumatic mitral stenosis, right posterior septal accessory pathway and atrial flutter. An electrophysiological study after percutaneous balloon mitral valvotomy showed that the palpitations were due to atrial flutter with right bundle branch aberrancy. The right posterior septal pathway was a bystander because it had a higher refractory period than the atrioventricular node.
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
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Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
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Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
2. In 1987, TEE was introduced at Mayo Clinic
Rochester.
Accounts for 5-10% of all echocardiography
studies.
Semi-invasive procedure.
Skillful physician and experienced sonographer
making it extremely safe and well tolerated.
Spacious room which can accommodate a
stretcher, oxygen outlet and suction facilities, pulse
oximeter and medications. Ensure I.V. access.
3. TEE probe (modified gastroesophageal endoscopy
probe with 3-7 MHz transducer at tip) should be
examined before use.
Diameter of transducer tip in adults and pediatric
use are 9-14 mm and < 3 mm respectively.
Anterior flexion should exceed 90%, and right and
left flexion should approach 90%.
Contact the patient 12 hours, and to fast for at
least 4-6 hours before the procedure.
Patient should be accompanied due to effect of
sedation.
5. PHOTO OF A TRANSDUCER AND CLOSE UP OF THE BODY OF A
TRANSDUCER
6. Steering the imaging plane using pressure
sensitive switch.
Manipulation of anterior-posterior and right-left
flexion by control knobs.
7. TERMINOLOGY USED TO DESCRIBE THE MANIPULATION OF THE
PROBE AND TRANSDUCER DURING IMAGE ACQUISITION.
8. Informed consent, explain transient abdominal
discomfort and gagging.
Lidocaine hydrochloride spray for topical
anesthesia over pharynx and tongue, and
diazepam 2-10mg, midazolam 0.05mg/kg I.V. for
light sedation.
Infective endocarditis prophylaxis not required.
9. o Left lateral decubitus with head of bed
elevated by ~30 ͦ to avoid aspiration
(elective procedure) and supine position
(mechanically ventilated patients).
o Remove dentures
o Patient’s head to be flexed
o Imaging surface of transducer faces tongue.
Probe kept in central position to prevent entry
into piriform fossa.
11. Gentle pressure and instruction to swallow.
If resistance ,withdraw and initiate new attempt.
Bite guard always used to prevent involuntary
closure of mouth.
If nausea wait for 10 – 15 seconds and then
proceed for imaging.
12. Start with images from esophagus before gastric
views.
GE sphincter reached when probe advanced 40cm
from incisor teeth.
Descending thoracic and arch of aorta reserved for
the end of study as it causes gagging as probe is in
upper esophagus.
13. Stridor or incessant cough indicates passage into
trachea also probe would not advance beyond
30cm and image quality will be poor.
In intubated patients, introduce probe in supine
position and mandible pulled forward, if resistance
at 25 – 30 cms deflate ET tube cuff.
14. IMAGE FORMAT
No general agreement.
Right sided structures are on the left and left sided
on the right.
Apex of the imaging plane with artifact is at the top
of the screen.
15. FOUR BASIC MANEUVERS
Advancement and withdrawal : imaging views are
basal, four chamber, transgastric, and aortic views.
Rotation from side to side: useful in longitudinal
imaging planes for continuity between vertically
aligned structures, SVC and arch vessels.
16. BASAL VIEWS
Probe: midesophagus
Visualizes :Base of heart particularly AV.
Relationship Of Two Great Arteries Till Pulmonary
Bifurcation.
Proximal portions of left main and right coronary
artery.
Left atrial appendage and left pulmonary veins.
20. Horizontal plane for 4 pulmonary veins.
Right and left atrial appendages wrap around great
arteries anteriorly. Their corrugated endocardial
surface can be confused with small thrombi.
150 ͦrightward plane for dilated and tortuous
ascending aorta.
22. ATRIAL SEPTUM
Longitudinal plane at 90-120 ͦfor fossa ovalis, SVC
to RA continuity , sinus venosus ASD, foramen
ovale at superior aspect of fossa ovalis and left-
right rotation for LVOT and RVOT.
24. PULMONARY BIFURCATION
o Withdrawal of probe and at 0 ͦ.
o Pulmonary valve (thinner) and artery are superior to
aortic valve.
o Entire right and very proximal left pulmonary artery
is visualized ( for proximal pulmonary emboli).
25. FOUR CHAMBER VIEW
Middle to low esophagus.
Gentle retroflexion with withdrawal for left ventricle.
For dilated and unfolded aorta rotate by 20-30 ͦ.
Inferior septum and anterolateral wall of LV and
continuous sweep from 0-180 ͦ for LV global and
regional function.
27. Mitral valve
Continuous sweep from 0-180 ͦfor scallops of
anterior and posterior leaflets at long axis view of
aortic valve and proximal ascending aorta at 120 ͦ.
Papillary muscles and subvalvular chords
visualized ( better in transgastric view).
Four chamber view ideal for MR assessment (
number of jets, direction and severity).
28. A MID ESOPHAGEAL, FOUR-CHAMBER VIEW SUPERIMPOSED WITH
COLOR FLOW DOPPLER SHOWING MITRAL REGURGITATION. THE
VENA CONTRACTA (VC) DEPICTS THE REGURGITANT ORIFICE.
29. LVOT
o At 120-160 ͦ, opening and closing of AV and AR
assessment, also proximal ascending aorta on
slight withdrawal.
o Slight rotation to left for pulmonary valve and RVOT
and assessment of PR.
31. TRANSGASTRIC VIEWS
Slight resistance and liver indicates probe in
stomach.
Anterior flexion, leftward rotation and flexion.
Extreme anterior flexion with advancement of probe
for 5 chamber view.
32. LV
o Cross section view for intraoperative LV function
optimized by leftward rotation and leftward flexion.
o For LV apex gentle advancement with retroflexion.
o leftward rotation for LVOT and aortic valve
alignment for transaortic pressure gradient in AS.
36. MV: transducer brought close to GE junction with
horizontal imaging with anterior flexion and leftward
flexion.
Should be attempted in all patients with
myxomatous degeneration.
Papillary muscles and chords.
Coronary sinus: near GE junction and flexion knobs
in neutral position also by retroflexion in lower
esophagus.
39. TRANSGASTRIC MID-PAPILLARY SHORT AXIS VIEW. IN PANEL A THE SEPTAL AND
LATERAL WALL AREAS ARE POORLY DEFINED. IN PANEL B BOTH WALL REGIONS
ARE ADEQUATELY IMAGED BY INCREASING THE LATERAL GAIN.
40. CS located posterior to LV at AV groove draining
into RA with TV to the right and anterior.
Dilated CS indicates PLSVC which is the most
common cause which is visualized with leftward
rotation following CS.
In esophageal views PLSVC is sandwiched
between LAA and LSPV.
41. AORTIC VIEWS
Descending thoracic aorta: in horizontal plane with
transducer rotated leftward and posterior followed
by slow withdrawal from diaphragm to aortic arch
with slight rotational adjustment.
Dilated and tortuous aorta adjust imaging plane by
0-90 ͦ.
42. THE VARIOUS HORIZONTAL AND LONGITUDINAL VIEWS OF THE
AORTA THAT CAN BE OBTAINED WITH TEE
43. Aortic Arch: longitudinal plane at 90 ͦ.
Withdraw slightly for arch vessels( all 3 visualized in
one-third patients). Brachiocephalic artery being
most difficult due to rightward and anterior location
and interposing trachea.
Proximal pulmonary artery seen in suspected PTE.
44. DOPPLER EXAMINATION
No additional information over TTE.
EXCEPT:
LAA FLOW: imaged from midesophagus aortic
valve short axis view( 30 to 60 ͦ) or midesophagus
two chamber view(80 to 100 ͦ).
In AF regular atrial contraction wave is absent.
In atrial flutter velocity waves are regular and
greater due to slower atrial rate.
45. Normal LAA velocity: contraction (60±14cm/s),filling
(52±13cm/s) and early diastolic filling(20±11cm/s).
In AF with LAA velocity <20cm/s more likely have LA
thrombus and 2.6 fold greater risk of ischemic stroke
compared to patient’s with velocity > 20cm/s.
Lower LAA velocity also seen in stroke patients in sinus
rhythm.
LAA velocity may predict successful cardioversion of AF
and maintenance of sinus rhythm at 1 year.
49. INDICATIONS FOR TEE
Nondiagnostic TTE
Evaluation of native valve disease
Evaluation of prosthetic valves
Evaluation of suspected and definite IE
Evaluation of suspected cardioembolic event(35%)
Evaluation of cardiac tumors and masses
Evaluation of a atrial septal abnormality
Evaluation of acute aortic syndrome or aortic
disease
50. LEFT: TRANSESOPHAGEAL ECHOCARDIOGRAPHIC VIEW OF A PATIENT
WITH SEVERE MITRAL REGURGITATION DUE TO A FLAIL POSTERIOR
LEAFLET. THE ARROW POINTS TO THE PORTION OF THE POSTERIOR
LEAFLET THAT IS UNSUPPORTED AND MOVES INTO THE LEFT ATRIUM
DURING SYSTOLE. RIGHT: COLOR-FLOW IMAGING DEMONSTRATING A
LARGE MOSAIC JET OF MITRAL REGURGITATION DURING SYSTOLE.
51. TRANSESOPHAGEAL STILL-FRAME ECHOCARDIOGRAPHIC VIEW OF A PATIENT WITH A
DILATED AORTA, AORTIC DISSECTION, AND SEVERE AORTIC REGURGITATION. THE
ARROW POINTS TO THE INTIMAL FLAP THAT IS SEEN IN THE DILATED ASCENDING
AORTA. LEFT: THE LONG-AXIS APEX-DOWN VIEW OF THE BLACK-AND-WHITE TWO-
DIMENSIONAL IMAGE IN DIASTOLE. RIGHT: COLOR-FLOW IMAGING THAT
DEMONSTRATES A LARGE MOSAIC JET OF AORTIC REGURGITATION
52. A 3D ZOOM MODE IMAGE ROTATED TO DISPLAY THE SURGEONS
VIEW FROM THE LEFT ATRIUM ONTO THE MITRAL VALVE, WITH A
P2 PROLAPSE AND RUPTURED CHORDAE TENDINAE (RC)
53. Atrial fibrillation ( TEE-guided strategy for “early”
cardioversion) (34%)
Evaluation of naïve and surgically corrected
congenital heart disease
Detection of coronary artery anomalies and
coronary artery disease
Evaluation of postoperative cardiac tamponade and
pericardial disease
Evaluation of the critically ill patient
Intraoperative monitoring
Guidance of interventional procedures
54. TRANSESOPHAGEAL STILL-FRAME ECHOCARDIOGRAPHIC
IMAGES OF A PATIENT WITH A LEFT ATRIAL MYXOMA. THERE IS A
LARGE ECHO-DENSE MASS IN THE LEFT ATRIUM, ATTACHED TO
THE ATRIAL SEPTUM. THE MASS MOVES ACROSS THE MITRAL
VALVE IN DIASTOLE
55. CONTRAINDICATIONS TO TEE
ABSOLUTE
Uncooperative patient
Severe respiratory depression
Tenuous cardiorespiratory status
Esophageal obstruction (stricture, mass)
Esophagectomy or esophagogastrectomy
Tracheoesophageal fistula
Perforated viscus
Active upper gi bleed
56. RELATIVE
Esophageal diverticulum
Esophageal varices
Previous esophageal surgery
History of dysphagia
Recent upper gi bleed
Severe cervical arthritis with restricted mobility
Atlantoaxial joint disease with restricted mobility
Severe Coagulopathy
57. PROCEDURAL COMPLICATIONS WITH TEE
MAJOR
o Death
o Esophageal rupture/perforation
o Upper gi bleed
o Laryngospasm or bronchospasm
o Congestive heart failure or pulmonary edema
o Sustained ventricular tachycardia
58. MINOR
o Excessive retching or vomiting
o Sore throat and Hoarseness
o Minor pharyngeal or lip bleeding
o Nonsustained or sustained supraventricular
tachycardia/atrial fibrillation, NSVT and Bradycardia
or heart block
o Transient hypotension or hypertension and Angina
o Transient hypoxia
o Tracheal intubation
o Dental injury
59. 1.9% failed esophageal intubation during TEE.
Complications occur in 3.5% patients ,
predominantly minor.
0.2-0.5% suffer major complication.
< 0.01% TEE-related mortality.
Recent review of 17 studies encompassing 42,355
patients identified only 4 TEE procedure deaths.
Esophageal perforation and major upper GI
bleeding in 0.01% and 0.03% of TEE studies
respectively.
Dental injuries in 0.03% patients.
60. Minor sore throat may be present for 24 hrs after TEE.
0.1% have persistent odynophagia requiring further
investigation and Laryngospasm in <0.02%.
Topical anesthetic agents can cause acute toxic
methemoglobinemia, manifesting clinically in 0.115% of
TEE studies.
Agents oxidizes hb and prevents oxygen delivery to
tissues -cyanosis, lethargy, tachycardia, dyspnea, and
death.
Methylene blue is indicated if methemoglobin levels
>30%, or patient has cyanosis, CNS depression, or
cardiorespiratory compromise.
Intraoperative TEE complication rate 2.4% (excluding
failed intubations).
Editor's Notes
Short-axis view of ascending aorta and main pulmonary artery (MPA), with bifurcation and origin of right pulmonary artery (RPA), from the upper transoesophageal window.
Transverse view of upper left atrium. Colour Doppler display of inflow from right upper pulmonary vein (RUPV, arrow). SVC, superior vena cava; AoA, ascedending aorta.
Long-axis view of the ascending aorta. (A) Proximal ascending aorta. (B) In the same patient, after retraction of the probe and adjustment of the plane orientation, a long portion of the dilated ascending aorta is seen. RPA, right pulmonary artery.
Left and right atrium and atrial septum in longitudinal (sagittal) view. Note orifice of superior (SVC) and inferior vena cava (IVC) and right atrial appendage (RAA).
Low transoesophageal view of right ventricle (RV), right atrium (RA), and tricuspid valve. This is a transoesophageal four-chamber view modified by slight counterclockwise shaft rotation.
Transoesophageal long-axis view of the left ventricle. Aoa, ascending aorta.
Transgastric short-axis view of the left (LV) and right ventricle (RV).
Transgastric two-chamber view. The apex is to the left, and the left atrium to the right in the image. (A) Cross-section showing the cavity of the left ventricle. (B) Slightly modified view intersecting both papillary muscles and chordal subvalvular apparatus. AW, anterior wall; IW, inferior wall; AL, anterolateral papillary muscle; PM, posteromedial papillary muscle.
Short-axis view of the open mitral valve from the transgastric position. AL, anterolateral; PM, posteromedial commissure. A1–A3 and P1–P3 denominate the respective leaflet scallops.
(A) Left atrial appendage. (B) Pulsed wave Doppler recording of emptying (upward) and filling (downward) velocities in atrial fibrillation. The velocities are quite high (>25 cm/s), indicating relatively low risk of thrombus generation. LUPV, left upper pulmonary vein. (C) Example of left atrial appendage with marked pectinate muscles (arrow). There is no thrombus.
Left: left upper pulmonary vein (LUPV) imaged in an approximately longitudinal view. Right: pulsed Doppler recording of normal pulmonary venous inflow from the left upper pulmonary vein. Positive (upward) velocities are directed into the left atrium. S, systolic wave; D, diastolic wave; R, reverse wave.