This document summarizes the duties and responsibilities of doctors in cases of suspected poisoning. It describes relevant sections of the Indian Penal Code and Criminal Procedure Code, including requirements to preserve evidence and inform authorities. It also provides guidance on treating specific types of poisoning through gastric lavage, activated charcoal, and antidotes. Common poisons discussed include organophosphates, heavy metals, acids, and other toxic substances. Post-mortem findings are also outlined.
Sulphuric acid as a corrosive poison. the characteristics, fatal dose, fatal time, sign and symptoms, post-mortem appearance and medicolegal importance are discussed
Sulphuric acid as a corrosive poison. the characteristics, fatal dose, fatal time, sign and symptoms, post-mortem appearance and medicolegal importance are discussed
Toxicological aspects of Phosphorus with emphasis on its forms, uses, poisoning: types, diagnosis, treatment, autopsy features and medico-legal importance.
This presentation includes:
Other names of hydrocyanic acid
Physical appearance of cyanides
Commercial uses of cyanides
Sources of cyanides
Mode of action of cyanides
Fatal Dose and fatal period of cyanides
Acute Poisoning of cyanides
Chronic Poisoning of cyanides
Diagnosis of cyanides
Treatment of cyanides
Post-Mortem appearance
Medicolegal importance
If you like my work then thumb 👍🏻. Thanking in anticipation.
Thanatology
Types of transplants
Cause, Mechanism of Death
Manner of death
Anoxia
Signs of Death
Immediate Changes (Somatic Death)
Early Changes (Molecular Death)
Algor Mortis ......
Reference
Toxicological aspects of Phosphorus with emphasis on its forms, uses, poisoning: types, diagnosis, treatment, autopsy features and medico-legal importance.
This presentation includes:
Other names of hydrocyanic acid
Physical appearance of cyanides
Commercial uses of cyanides
Sources of cyanides
Mode of action of cyanides
Fatal Dose and fatal period of cyanides
Acute Poisoning of cyanides
Chronic Poisoning of cyanides
Diagnosis of cyanides
Treatment of cyanides
Post-Mortem appearance
Medicolegal importance
If you like my work then thumb 👍🏻. Thanking in anticipation.
Thanatology
Types of transplants
Cause, Mechanism of Death
Manner of death
Anoxia
Signs of Death
Immediate Changes (Somatic Death)
Early Changes (Molecular Death)
Algor Mortis ......
Reference
Handling of chemicals, handling of acids, ethers, calibration of burette, pipette, Heating methods, stirring methods, filtratio techniques, chemical balance and single pan balance
toxicology is a very important topic for pg entrance.....so all about it has been discussed in detail as required for pg entrance....do make use of it...
Toxicology is a very important topic and if u try reading each
poisons Separately , u will definitely forget them.
So best way to remember them more is to practice
MCQs and then read topics by correlating.
That’s what I think….
It may or may not apply for u…..
But atleast try once like this and then choose your own way .
Rasamanikya is a excellent preparation in the field of Rasashastra, it is used in various Kushtha Roga, Shwasa, Vicharchika, Bhagandara, Vatarakta, and Phiranga Roga. In this article Preparation& Comparative analytical profile for both Formulationon i.e Rasamanikya prepared by Kushmanda swarasa & Churnodhaka Shodita Haratala. The study aims to provide insights into the comparative efficacy and analytical aspects of these formulations for enhanced therapeutic outcomes.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Local Advanced Lung Cancer: Artificial Intelligence, Synergetics, Complex Sys...Oleg Kshivets
Overall life span (LS) was 1671.7±1721.6 days and cumulative 5YS reached 62.4%, 10 years – 50.4%, 20 years – 44.6%. 94 LCP lived more than 5 years without cancer (LS=2958.6±1723.6 days), 22 – more than 10 years (LS=5571±1841.8 days). 67 LCP died because of LC (LS=471.9±344 days). AT significantly improved 5YS (68% vs. 53.7%) (P=0.028 by log-rank test). Cox modeling displayed that 5YS of LCP significantly depended on: N0-N12, T3-4, blood cell circuit, cell ratio factors (ratio between cancer cells-CC and blood cells subpopulations), LC cell dynamics, recalcification time, heparin tolerance, prothrombin index, protein, AT, procedure type (P=0.000-0.031). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and N0-12 (rank=1), thrombocytes/CC (rank=2), segmented neutrophils/CC (3), eosinophils/CC (4), erythrocytes/CC (5), healthy cells/CC (6), lymphocytes/CC (7), stick neutrophils/CC (8), leucocytes/CC (9), monocytes/CC (10). Correct prediction of 5YS was 100% by neural networks computing (error=0.000; area under ROC curve=1.0).
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
Integrating Ayurveda into Parkinson’s Management: A Holistic ApproachAyurveda ForAll
Explore the benefits of combining Ayurveda with conventional Parkinson's treatments. Learn how a holistic approach can manage symptoms, enhance well-being, and balance body energies. Discover the steps to safely integrate Ayurvedic practices into your Parkinson’s care plan, including expert guidance on diet, herbal remedies, and lifestyle modifications.
2. Duties of a doctor in suspected
poisoning is described under
S.39 CrPC
S.175 CrPC
S.193 IPC
S.176 IPC
S.201 IPC
S.202 IPC
3. 201 –(punishment IPC no. if not
done)preserve articles , food, excreta ,
stomach wash , bottles , capsules , paper
packets should be collected and preserved
39 – if a private practitioner is convinced that it
is a homicidal poisoning he should inform
police officer or magistrate
4. If 39 not done doctor is punishable under 176
If he is sure that it is suicidal poisoning no
need to inform police since 309 is not included
in the section
But under 175 if asked by police officer Dr
should give all information
If conceals liable to get punished under 202
7. Gastric lavage
Within 3 hrs
For salycylates,phenothiazenes,antihistaminics,TCA, -- lavage
can be done upto 12 – 18 hours
Ewald’s / Baos tube
First 250 ml then 500 ml
CI – corrosive poisoning except for phenol
Complications –
1. Laryngeal spasm
2. Aspiration pneumonitis
3. Perforation of the stomach
4. Sinus Bradycardia
27. Organophosphate poisoning
Mixed in a solvent called Aromax – kerosine
like smell in the body cavity,stomach contents ,
vomitus , froth, etc
Inactivation of Che becomes irriversible after 24 –
36 hrs
Sign & symptoms – when activity drops to 30% of
normal activity
Death – paralysis of the respiratory muscles
28. Choline esterase test
5 ml of heparinised blood
average normal values
77 – 142 in red cells
41 – 140 in plasma
Diagnosis can be confirmed by giving 2 mg of
atropine in normal signs of atropinisation and
in poisoned releives the symptoms
31. Post Mortem appearance
Signs of Asphyxia
Congestion – face,all internal organs
Cyanosis of lips,fingers , nose
Blood stained froth--Respiratory path ,
mouth and nose
Stomach content with smell of
kerosene
Organophosphorous can be detected in
putrified bodies
32. Oxims
DAM – Di Acetyl Monoxime
Praldioxime Iodide
Pralidoxime chloride
37. Treatment of Sulphuric acid
poisoning
Avoid gastric lavage
250 ml of water / milk / milk of magnesia /
lime water – within 30min
Demulcents
Prednisolone 60 mg / day - to prevent
esophageal stricture and shock
If stricture develops – 4cm diameter Hg filled
bougie should be passed daily
Give nothing by mouth
Topica l paste of Magnesium oxide / sodium
bicarbonate
38.
39. PM change
Stomach – soft , spongy , black mass ,
when readily disintegrates when
touched
40. Treatment of vitriolage
Wash with plenty of water
Soap of Sodium or Potassium carbonate
Thick paste of magnesium oxide / carbonate
is applied
Eyes – irrigated with Dilute Sodium
bicarbonate solution
Later a few drops of olive oil or castor oil is
applied to the eyes
42. Sign and symptoms
Greater abdominal distension due to gas
formation
Tissues are stained yellow
In esophagu and stomach …the corrosion of
the mucus membrane may not be associated
with yellow colour –Brown or Brown black due
to acid hemetin
43. HCl
Corrosion is less severe
Stomach contains brownish fluid
The folds of the whole stomach mucosa are
brownish
Perforation is rare
Acute inflammation and edema of the
respiratory passage are common
45. Treatment of Oxalic Acid poisoning
Stomach wash – Calcium lactate or gluconate
Antidote – preparation of Calcium
Lime water , Calcium gluconate ,Calcium
chloride
Parathyroid extracts bowel may be evacuated
by enema or castor oil
46. Carbolic acid
Carboluria – further oxidation of
hydroquinone and pyrocatechol in the
urine cause green colouration
Chronic Poisoning – Phenolic
Marasmus-Anorexia , headache ,wt
loss ,headache ,darkurine , and
oochronosis
Oochronosis – pigmentation of skin
and sclera
47. Post Mortem findings in carbolic acid poisoning
i) Esophagus - Mucosa is tough, corrugated,
arranged
in longitudinal folds.
ii) Stomach - Mucosal folds are swollen covered
by opaque,
coagulated mucous membrane which is
thickened and leathery. Partial separation of
necrotic mucosa.
iii) Duodenum, - Similar to stomach
upper SI
iv) Liver, Spleen - Whitish, hardened patch where
stomach has
been in contact.
v) Kidneys - Haemorrhagic nephritis
vi) Brain - Congested, edematous
vii) Blood - Dark, semifluid, only partially coagulated.
48. Formic acid
Action – corrosive actioo on GI mucosa
Causes hemolysis leading to acute renal
failure
ATP synthesis is diminished
T/T –Milk –Folinic acid 1mg /Kg 4th
hourly
51. Metallic arsenic is not poisonous as it is
not absorbed from the alimentary canal
Arsenic trioxide / white Arsenic
MOA – binds and inhibits Pyruvate
oxidase which is a mitochondrial enz
Affects vascular endothelium –
increased permiabilityesp in the
intestinal canal
Irritation of the mucucs membrane
Depression of the NS
Interfers with glycolysis
53. Fulminent tyoe
Massive doses – 3 -5 gm Arsenic death in 1 –
3hrs – due to shock & peripheral vascular
failure
54. Gastro enteric type
Common form of acute poisoning
Mostly half an hour after ingestion
Sweetish metallic taste
Constriction of the throat and difficulty in
swallowing
Burning and colicky pain in the esophagus,
stomach and bowel
Purging
Stools – frequently and involuntary first
Dark coloured stinking and bloody
resembles rice water stools
Garlicky odour in breath and feces may be
noted
Death is usually due to circulatory failure
55. Narcotic Form
Giddiness
Formication and tenderness of the muscle
Delerium coma and death
Arseniurated hydrogen direct poison to Hb
hemolysis ,hemoglobinuria , renal failure
Death is almost instantaneous
56. Treatment of Arsenic Poisoning
Emetics should not be used
Alkalis should not be used
Wash with large amount of warm water /
milk
Ferric oxide
BAL
DMPS
DMSA
Penicillamine
Demulscents
Glucose saline with Sodium bicarbonate
Hemodyalisis and exchange transfusion
59. Mercury
Mercuric
Egg white
Ca – EDTA should not be used as it is
nephrotoxic with mercury
PM – if the patient survives forfew days
..LI shows necrosis due to the reexcretion
of mercury into the large bowel
60. Chronic poisoning – Hydrargyrism
PRESENCE OF TISSUE DEPOSITS OF Cu
-CHALCOSIS
65. Preservation for viscera in case of
suspected poisoning
Stomach and its contents
Upper part of small intestine and its contents
30 m
Liver 200 300 g
Kidney – half of each
Blood 30 ml
Urine 30 ml