This document provides a summary of basics of electrocardiography (EKG/ECG) interpretation for emergency medicine. It begins with an overview of cardiac anatomy and the electrical conduction system. It then discusses components of the EKG including waves, intervals, rates, and rhythms. Common arrhythmias such as atrial fibrillation, supraventricular tachycardia, and ventricular tachycardia are summarized. Interpretation of EKG findings including rates, rhythms, intervals, and morphologies are covered. The document concludes with a focus on ST-elevation myocardial infarction (STEMI) including location, treatment protocols, and example EKGs.
This document discusses the interpretation of electrocardiograms (ECGs) in pediatric patients, particularly those with congenital heart disease. It covers normal variations in ECG findings with age from neonatal to adolescent periods. It then discusses ECG patterns associated with various congenital heart defects, including septal defects, obstructive lesions, cyanotic conditions and miscellaneous defects. Key findings are described for interpreting ECGs and correlating them with specific heart conditions. The document emphasizes that while not diagnostic, the ECG can provide important clues to the presence of chamber enlargement, conduction abnormalities and help classify certain congenital heart diseases.
This document discusses ECG findings in various congenital heart diseases:
1) Acyanotic CHDs like atrial septal defect (ASD) show findings of right or left ventricular hypertrophy depending on shunt direction, while ventricular septal defect (VSD) shows signs of left or bi-ventricular overload.
2) Cyanotic CHDs like transposition of the great arteries (TGA) show right axis deviation and ventricular hypertrophy initially, evolving to match pulmonary blood flow. Tricuspid atresia typically shows left axis deviation and ventricular hypertrophy.
3) Algorithms are provided to systematically analyze ECG patterns and identify the underlying CHD based on chamber
The document provides an overview of ECG interpretation including components to evaluate such as rate, rhythm, axis, wave morphology, intervals, and segments. It emphasizes considering the patient's clinical condition, obtaining prior ECGs for comparison, and getting a second opinion when needed. The normal ECG is reviewed along with abnormalities including arrhythmias, conduction defects, myocardial infarction, hypertrophy, and other cardiac and non-cardiac conditions.
The document provides information on how to interpret various electrocardiogram (EKG or ECG) rhythms and abnormalities. It defines normal sinus rhythm and various arrhythmias originating in the atria, junctional region, or ventricles. It also covers premature beats, heart blocks, ST segment changes, pathological Q waves, electrical reciprocity, and normal QRS axis definitions. The document serves as a guide for healthcare providers to understand EKG tracings and determine the underlying cardiac rhythm or injury.
The patient, a 43-year-old female, presented with palpitations, dyspnea, and chest pain after physical exertion. She has a history of rheumatism and mitral stenosis but had been stable until now. Her symptoms suggest new onset atrial fibrillation precipitated by exertion in the setting of underlying heart valve disease.
The document describes an electrocardiogram (ECG) including the leads used to record the ECG, how the leads are attached, standard measurements, the components of the cardiac cycle that are measured on ECG, and how to interpret various rhythms, intervals, waves and complexes that may appear on an ECG. It provides guidance on assessing rate, rhythm, axis, hypertrophy, ischemia/infarction, and other abnormalities and includes diagrams to demonstrate common arrhythmias and conduction abnormalities.
Inferior lead pseudo-infarct Q waves are a common finding in the Wolff-Parkinson-White (WPW) syndrome.The characteristic Q wave-T wave vector discordance results from secondary repolarization changes due to altered ventricular activation. As a corollary, the presence of T wave inversion with inferior lead Q waves and a short PR interval is strongly suggestive, but not pathognomonic of inferior ischemia.
Electrocardiography in Adult Congenital Heart DiseasesSaleh AL-Hatem
The document discusses various types of congenital heart defects seen in adults, including their typical ECG presentations. For ostium secundum atrial septal defects, unrepaired defects are commonly associated with sinus rhythm on ECG, though atrial fibrillation risk increases with age. Surgical closure may reduce but not eliminate postoperative atrial arrhythmias, especially in older patients. For ventricular septal defects, ECG findings depend on the degree of left and right ventricular overload. Large unrepaired defects can cause right and left atrial enlargement and right axis deviation on ECG. Atrioventricular canal defects are associated with first-degree atrioventricular block and left axis deviation on ECG due to
This document discusses the interpretation of electrocardiograms (ECGs) in pediatric patients, particularly those with congenital heart disease. It covers normal variations in ECG findings with age from neonatal to adolescent periods. It then discusses ECG patterns associated with various congenital heart defects, including septal defects, obstructive lesions, cyanotic conditions and miscellaneous defects. Key findings are described for interpreting ECGs and correlating them with specific heart conditions. The document emphasizes that while not diagnostic, the ECG can provide important clues to the presence of chamber enlargement, conduction abnormalities and help classify certain congenital heart diseases.
This document discusses ECG findings in various congenital heart diseases:
1) Acyanotic CHDs like atrial septal defect (ASD) show findings of right or left ventricular hypertrophy depending on shunt direction, while ventricular septal defect (VSD) shows signs of left or bi-ventricular overload.
2) Cyanotic CHDs like transposition of the great arteries (TGA) show right axis deviation and ventricular hypertrophy initially, evolving to match pulmonary blood flow. Tricuspid atresia typically shows left axis deviation and ventricular hypertrophy.
3) Algorithms are provided to systematically analyze ECG patterns and identify the underlying CHD based on chamber
The document provides an overview of ECG interpretation including components to evaluate such as rate, rhythm, axis, wave morphology, intervals, and segments. It emphasizes considering the patient's clinical condition, obtaining prior ECGs for comparison, and getting a second opinion when needed. The normal ECG is reviewed along with abnormalities including arrhythmias, conduction defects, myocardial infarction, hypertrophy, and other cardiac and non-cardiac conditions.
The document provides information on how to interpret various electrocardiogram (EKG or ECG) rhythms and abnormalities. It defines normal sinus rhythm and various arrhythmias originating in the atria, junctional region, or ventricles. It also covers premature beats, heart blocks, ST segment changes, pathological Q waves, electrical reciprocity, and normal QRS axis definitions. The document serves as a guide for healthcare providers to understand EKG tracings and determine the underlying cardiac rhythm or injury.
The patient, a 43-year-old female, presented with palpitations, dyspnea, and chest pain after physical exertion. She has a history of rheumatism and mitral stenosis but had been stable until now. Her symptoms suggest new onset atrial fibrillation precipitated by exertion in the setting of underlying heart valve disease.
The document describes an electrocardiogram (ECG) including the leads used to record the ECG, how the leads are attached, standard measurements, the components of the cardiac cycle that are measured on ECG, and how to interpret various rhythms, intervals, waves and complexes that may appear on an ECG. It provides guidance on assessing rate, rhythm, axis, hypertrophy, ischemia/infarction, and other abnormalities and includes diagrams to demonstrate common arrhythmias and conduction abnormalities.
Inferior lead pseudo-infarct Q waves are a common finding in the Wolff-Parkinson-White (WPW) syndrome.The characteristic Q wave-T wave vector discordance results from secondary repolarization changes due to altered ventricular activation. As a corollary, the presence of T wave inversion with inferior lead Q waves and a short PR interval is strongly suggestive, but not pathognomonic of inferior ischemia.
Electrocardiography in Adult Congenital Heart DiseasesSaleh AL-Hatem
The document discusses various types of congenital heart defects seen in adults, including their typical ECG presentations. For ostium secundum atrial septal defects, unrepaired defects are commonly associated with sinus rhythm on ECG, though atrial fibrillation risk increases with age. Surgical closure may reduce but not eliminate postoperative atrial arrhythmias, especially in older patients. For ventricular septal defects, ECG findings depend on the degree of left and right ventricular overload. Large unrepaired defects can cause right and left atrial enlargement and right axis deviation on ECG. Atrioventricular canal defects are associated with first-degree atrioventricular block and left axis deviation on ECG due to
This document provides an overview of ECG interpretation including:
- The anatomy of the heart's conduction system and how ECG leads are attached
- How to read an ECG strip and calculate heart rate
- Normal P, QRS, and T waves along with intervals like PR and QT
- Abnormalities that can indicate conditions like blocks, arrhythmias, and hypertrophy
- Electrolyte imbalances that can affect the ECG tracing
It concludes with examples of ECG strips and questions to test the reader's understanding.
An electrocardiogram (ECG or EKG) records the electrical signal from your heart to check for different heart conditions. Electrodes are placed on your chest to record your heart's electrical signals, which cause your heart to beat. The signals are shown as waves on an attached computer monitor or printer
An ECG provides a representation of the heart's electrical activity during each cardiac cycle. It displays distinct waveforms that can provide insight into a patient's cardiac pathophysiology. The ECG paper displays time and voltage measurements to analyze rate, regularity, P waves, PR interval, and QRS duration in order to identify normal sinus rhythm or potential arrhythmias originating from problems in the sinus node, atria, AV node, or ventricles. Arrhythmias can cause the heart to beat too slowly, too quickly, or irregularly compared to normal sinus rhythm.
This document provides an overview of AV nodal reentrant tachycardia (AVNRT) including its mechanisms, diagnosis using electrophysiology study techniques, and treatment with catheter ablation. It discusses the criteria for diagnosing dual AV nodal physiology, how AVNRT is initiated via programmed stimulation, its characteristic surface ECG patterns, and how the arrhythmia responds to different tests such as atrial and ventricular stimulation. The document emphasizes that AVNRT is the most common type of supraventricular tachycardia and is due to reentry involving slow and fast pathways in the AV node.
ECG Final Proff.Sumit Kr Ghosh Dept of Internal Medicine Medical College 88 C...Chirantan MD
This document provides an overview of electrocardiography (ECG). It discusses reading an ECG, including assessing rate, rhythm, axis, waves, intervals, and abnormalities. It covers ECG paper standards, leads, cardiac hypertrophy, coronary insufficiency, myocardial infarction, arrhythmias, conduction defects, and bundle branch blocks.
This document provides an overview of electrocardiogram (ECG) interpretation. It discusses the components of a normal ECG tracing including rate, rhythm, axis, waveforms such as P, QRS, ST segments, and T waves. It describes how to systematically evaluate an ECG and identify abnormalities. Common abnormalities are outlined such as arrhythmias, conduction blocks, myocardial infarction, hypertrophy, and electrolyte disturbances. Causes of abnormalities in various ECG components are also reviewed. The goal is to provide clinicians a methodical approach to ECG interpretation.
1. This document provides an overview of a training course on complex supraventricular tachycardia (SVT) differentiation. It discusses various SVT etiologies and electrocardiogram patterns.
2. Mechanisms of SVT discussed include atrioventricular nodal reentrant tachycardia (AVNRT), atrioventricular reentrant tachycardia (AVRT), and atrial tachycardia (AT). The document also reviews electrophysiology study findings that help differentiate the mechanisms.
3. Case examples are presented to demonstrate electrophysiology study techniques for SVT diagnosis and ablation, including ventricular overdrive pacing, ventricular extrastimuli, and induction protocols.
This document provides a template for systematically analyzing 12-lead ECGs, beginning with type and recording details, rate/rhythm/axis, P wave, PR interval, QRS complex, ST segment, T wave, and other findings. Key areas of analysis include intervals, amplitudes, morphologies, and identifying pathological patterns indicative of conditions like myocardial infarction, arrhythmias, chamber enlargement, and lethal causes of syncope. The systematic approach aids accurate ECG interpretation and diagnosis.
The document provides information about electrocardiograms (ECGs), including what an ECG is, the types of pathology that can be identified from ECGs, ECG paper specifications, the anatomy of the heart and normal ECG signal, ECG leads, determining heart rate and rhythm from ECGs, P waves, the PR interval, the QRS complex, axes determination, bundle branch blocks, ventricular hypertrophy, Q waves, the ST segment, T waves, and the QT interval. Key aspects of the ECG that can help identify conditions like myocardial infarction, pericarditis, and electrolyte abnormalities are discussed.
The document provides information about electrocardiograms (ECGs), including what an ECG is, the types of pathology that can be identified from ECGs, ECG paper specifications, heart anatomy and the normal ECG signal, ECG leads, determining heart rate from ECGs, common rhythms, P waves, the PR interval, the QRS complex, identifying left and right bundle branch block, identifying left and right ventricular hypertrophy, Q waves, the ST segment and T waves. Key details are provided about normal ECG measurements and the signs of various cardiac conditions.
This document discusses algorithms and ECG parameters for differentiating between types of narrow complex tachycardia, including atrioventricular nodal reentrant tachycardia (AVNRT) and atrioventricular reentrant tachycardia (AVRT). Key parameters discussed include the presence of pseudo waves, retrograde P wave morphology and position, and the RP interval. The Jaeggi algorithm uses these parameters to differentiate AVNRT from AVRT based on ECG analysis alone in 76% of cases. Retrograde P wave morphology varies depending on the location of the accessory pathway in cases of AVRT.
This document discusses ECG patterns in congenital heart disease. It begins by outlining the significance of ECG in diagnosing congenital heart defects. It then provides an overview of normal ECG changes in children and how they evolve over time as hemodynamics change. Next, it describes how ECG can help identify situs and ventricular position. It then discusses the characteristic ECG patterns seen in common acyanotic defects like atrial septal defects and ventricular septal defects. It also covers cyanotic defects like transposition of the great arteries. The document provides detailed information on ECG features, associated conditions, complications and evolution over time for many different congenital heart defects.
This document provides an overview of ECG interpretation and arrhythmia recognition. The course objectives are to recognize normal sinus rhythm, the 13 most common rhythm disturbances, and acute myocardial infarction on ECG. The learning modules cover ECG basics, rhythm analysis, normal sinus rhythm, common arrhythmias, and 12-lead interpretation. Various arrhythmias are described that can arise from problems in the sinus node, atrial cells, AV node, or ventricular cells.
This document provides an overview of systematically analyzing an ECG. It discusses the normal components of an ECG including rate, rhythm, axis, P wave, PR interval, QRS complex, QT interval, ST segment, T wave, and other waves. It then examines each component in more detail, providing examples of normal and abnormal findings. Key aspects that could indicate conditions like myocardial infarction, arrhythmias, conduction abnormalities, and structural heart disease are emphasized. The document concludes with sample ECG rhythm and segment interpretation questions.
This document provides a guide to ECGs. It begins by outlining the objectives which are to cover electrical conduction in the heart, lead placement, ECG settings, components, waves, complexes, and abnormalities. It then discusses the cardiac conduction system, ECG waves and components, 12-lead ECG placement, components of the ECG including rate, rhythm, axis, P wave, PR interval, QRS complex, QT interval, ST segment, T wave, and other waves. It provides examples of normal ECG characteristics and discusses how to systematically analyze an ECG by examining rate, rhythm, axis, P wave, PR interval, QRS complex, QT interval and QTc, ST segment, and T wave
This document provides an overview of common cardiac rhythms seen on electrocardiograms (ECGs) including normal sinus rhythm, sinus tachycardia, sinus bradycardia, premature atrial contractions, atrial flutter, atrial fibrillation, paroxysmal atrial tachycardia, premature junctional contractions, junctional escape rhythm, junctional tachycardia, supraventricular tachycardia, premature ventricular complexes, and ventricular tachycardia. For each rhythm, the typical heart rate, rhythm, P wave characteristics, PR interval, QRS duration, and examples are described. Causes of each rhythm are also listed.
Bundle branch block, left ventricular hypertrophy, ventricular rhythms, and early repolarization can all produce ECG changes that mimic acute coronary syndrome. These imitators make it difficult to identify ST elevation or depression that may indicate ACS. However, new or presumed new bundle branch block accompanied by suggestive clinical symptoms is still cause for thrombolysis. The presence of an imitator does not rule out an underlying acute coronary syndrome.
This document provides information on cardiac arrhythmias, including:
- The normal cardiac conduction system and blood flow through the heart.
- Definitions and characteristics of various normal and abnormal sinus rhythms, atrial rhythms, junctional rhythms, and ventricular rhythms based on heart rate, rhythm, P wave presence and morphology, PR interval, and QRS width.
- Key arrhythmias summarized include normal sinus rhythm, sinus bradycardia, sinus tachycardia, premature atrial contractions, atrial fibrillation, atrial flutter, premature junctional contractions, junctional tachycardia, premature ventricular contractions, ventricular tachycardia, and ventricular flutter.
1) STEMI equivalents refer to patients with acutely occluded coronary arteries who do not present with classical ECG changes but have worse outcomes. Common equivalents include de Winter ST/T waves, Wellens' syndromes, ST elevation in aVR, new LBBB, isolated posterior MIs, and upright T waves in V1.
2) Wellens' syndromes present with progressive T wave inversions in leads V2-V3 and little cardiac marker elevation, indicating critical proximal LAD stenosis.
3) ST elevation in aVR with widespread ST depression indicates high-risk left main or three-vessel coronary disease requiring emergent angiography.
The document provides criteria for diagnosing ST-elevation myocardial infarction (STEMI) on electrocardiogram (ECG). It lists cut-off values for ST elevation in different leads used to identify STEMI based on the patient's age, sex and lead location. It cautions that baseline ECG abnormalities like left bundle branch block (LBBB) could obscure interpretation and provides examples of STEMI in different heart locations identified by affected leads on ECG.
This document provides an overview of ECG interpretation including:
- The anatomy of the heart's conduction system and how ECG leads are attached
- How to read an ECG strip and calculate heart rate
- Normal P, QRS, and T waves along with intervals like PR and QT
- Abnormalities that can indicate conditions like blocks, arrhythmias, and hypertrophy
- Electrolyte imbalances that can affect the ECG tracing
It concludes with examples of ECG strips and questions to test the reader's understanding.
An electrocardiogram (ECG or EKG) records the electrical signal from your heart to check for different heart conditions. Electrodes are placed on your chest to record your heart's electrical signals, which cause your heart to beat. The signals are shown as waves on an attached computer monitor or printer
An ECG provides a representation of the heart's electrical activity during each cardiac cycle. It displays distinct waveforms that can provide insight into a patient's cardiac pathophysiology. The ECG paper displays time and voltage measurements to analyze rate, regularity, P waves, PR interval, and QRS duration in order to identify normal sinus rhythm or potential arrhythmias originating from problems in the sinus node, atria, AV node, or ventricles. Arrhythmias can cause the heart to beat too slowly, too quickly, or irregularly compared to normal sinus rhythm.
This document provides an overview of AV nodal reentrant tachycardia (AVNRT) including its mechanisms, diagnosis using electrophysiology study techniques, and treatment with catheter ablation. It discusses the criteria for diagnosing dual AV nodal physiology, how AVNRT is initiated via programmed stimulation, its characteristic surface ECG patterns, and how the arrhythmia responds to different tests such as atrial and ventricular stimulation. The document emphasizes that AVNRT is the most common type of supraventricular tachycardia and is due to reentry involving slow and fast pathways in the AV node.
ECG Final Proff.Sumit Kr Ghosh Dept of Internal Medicine Medical College 88 C...Chirantan MD
This document provides an overview of electrocardiography (ECG). It discusses reading an ECG, including assessing rate, rhythm, axis, waves, intervals, and abnormalities. It covers ECG paper standards, leads, cardiac hypertrophy, coronary insufficiency, myocardial infarction, arrhythmias, conduction defects, and bundle branch blocks.
This document provides an overview of electrocardiogram (ECG) interpretation. It discusses the components of a normal ECG tracing including rate, rhythm, axis, waveforms such as P, QRS, ST segments, and T waves. It describes how to systematically evaluate an ECG and identify abnormalities. Common abnormalities are outlined such as arrhythmias, conduction blocks, myocardial infarction, hypertrophy, and electrolyte disturbances. Causes of abnormalities in various ECG components are also reviewed. The goal is to provide clinicians a methodical approach to ECG interpretation.
1. This document provides an overview of a training course on complex supraventricular tachycardia (SVT) differentiation. It discusses various SVT etiologies and electrocardiogram patterns.
2. Mechanisms of SVT discussed include atrioventricular nodal reentrant tachycardia (AVNRT), atrioventricular reentrant tachycardia (AVRT), and atrial tachycardia (AT). The document also reviews electrophysiology study findings that help differentiate the mechanisms.
3. Case examples are presented to demonstrate electrophysiology study techniques for SVT diagnosis and ablation, including ventricular overdrive pacing, ventricular extrastimuli, and induction protocols.
This document provides a template for systematically analyzing 12-lead ECGs, beginning with type and recording details, rate/rhythm/axis, P wave, PR interval, QRS complex, ST segment, T wave, and other findings. Key areas of analysis include intervals, amplitudes, morphologies, and identifying pathological patterns indicative of conditions like myocardial infarction, arrhythmias, chamber enlargement, and lethal causes of syncope. The systematic approach aids accurate ECG interpretation and diagnosis.
The document provides information about electrocardiograms (ECGs), including what an ECG is, the types of pathology that can be identified from ECGs, ECG paper specifications, the anatomy of the heart and normal ECG signal, ECG leads, determining heart rate and rhythm from ECGs, P waves, the PR interval, the QRS complex, axes determination, bundle branch blocks, ventricular hypertrophy, Q waves, the ST segment, T waves, and the QT interval. Key aspects of the ECG that can help identify conditions like myocardial infarction, pericarditis, and electrolyte abnormalities are discussed.
The document provides information about electrocardiograms (ECGs), including what an ECG is, the types of pathology that can be identified from ECGs, ECG paper specifications, heart anatomy and the normal ECG signal, ECG leads, determining heart rate from ECGs, common rhythms, P waves, the PR interval, the QRS complex, identifying left and right bundle branch block, identifying left and right ventricular hypertrophy, Q waves, the ST segment and T waves. Key details are provided about normal ECG measurements and the signs of various cardiac conditions.
This document discusses algorithms and ECG parameters for differentiating between types of narrow complex tachycardia, including atrioventricular nodal reentrant tachycardia (AVNRT) and atrioventricular reentrant tachycardia (AVRT). Key parameters discussed include the presence of pseudo waves, retrograde P wave morphology and position, and the RP interval. The Jaeggi algorithm uses these parameters to differentiate AVNRT from AVRT based on ECG analysis alone in 76% of cases. Retrograde P wave morphology varies depending on the location of the accessory pathway in cases of AVRT.
This document discusses ECG patterns in congenital heart disease. It begins by outlining the significance of ECG in diagnosing congenital heart defects. It then provides an overview of normal ECG changes in children and how they evolve over time as hemodynamics change. Next, it describes how ECG can help identify situs and ventricular position. It then discusses the characteristic ECG patterns seen in common acyanotic defects like atrial septal defects and ventricular septal defects. It also covers cyanotic defects like transposition of the great arteries. The document provides detailed information on ECG features, associated conditions, complications and evolution over time for many different congenital heart defects.
This document provides an overview of ECG interpretation and arrhythmia recognition. The course objectives are to recognize normal sinus rhythm, the 13 most common rhythm disturbances, and acute myocardial infarction on ECG. The learning modules cover ECG basics, rhythm analysis, normal sinus rhythm, common arrhythmias, and 12-lead interpretation. Various arrhythmias are described that can arise from problems in the sinus node, atrial cells, AV node, or ventricular cells.
This document provides an overview of systematically analyzing an ECG. It discusses the normal components of an ECG including rate, rhythm, axis, P wave, PR interval, QRS complex, QT interval, ST segment, T wave, and other waves. It then examines each component in more detail, providing examples of normal and abnormal findings. Key aspects that could indicate conditions like myocardial infarction, arrhythmias, conduction abnormalities, and structural heart disease are emphasized. The document concludes with sample ECG rhythm and segment interpretation questions.
This document provides a guide to ECGs. It begins by outlining the objectives which are to cover electrical conduction in the heart, lead placement, ECG settings, components, waves, complexes, and abnormalities. It then discusses the cardiac conduction system, ECG waves and components, 12-lead ECG placement, components of the ECG including rate, rhythm, axis, P wave, PR interval, QRS complex, QT interval, ST segment, T wave, and other waves. It provides examples of normal ECG characteristics and discusses how to systematically analyze an ECG by examining rate, rhythm, axis, P wave, PR interval, QRS complex, QT interval and QTc, ST segment, and T wave
This document provides an overview of common cardiac rhythms seen on electrocardiograms (ECGs) including normal sinus rhythm, sinus tachycardia, sinus bradycardia, premature atrial contractions, atrial flutter, atrial fibrillation, paroxysmal atrial tachycardia, premature junctional contractions, junctional escape rhythm, junctional tachycardia, supraventricular tachycardia, premature ventricular complexes, and ventricular tachycardia. For each rhythm, the typical heart rate, rhythm, P wave characteristics, PR interval, QRS duration, and examples are described. Causes of each rhythm are also listed.
Bundle branch block, left ventricular hypertrophy, ventricular rhythms, and early repolarization can all produce ECG changes that mimic acute coronary syndrome. These imitators make it difficult to identify ST elevation or depression that may indicate ACS. However, new or presumed new bundle branch block accompanied by suggestive clinical symptoms is still cause for thrombolysis. The presence of an imitator does not rule out an underlying acute coronary syndrome.
This document provides information on cardiac arrhythmias, including:
- The normal cardiac conduction system and blood flow through the heart.
- Definitions and characteristics of various normal and abnormal sinus rhythms, atrial rhythms, junctional rhythms, and ventricular rhythms based on heart rate, rhythm, P wave presence and morphology, PR interval, and QRS width.
- Key arrhythmias summarized include normal sinus rhythm, sinus bradycardia, sinus tachycardia, premature atrial contractions, atrial fibrillation, atrial flutter, premature junctional contractions, junctional tachycardia, premature ventricular contractions, ventricular tachycardia, and ventricular flutter.
1) STEMI equivalents refer to patients with acutely occluded coronary arteries who do not present with classical ECG changes but have worse outcomes. Common equivalents include de Winter ST/T waves, Wellens' syndromes, ST elevation in aVR, new LBBB, isolated posterior MIs, and upright T waves in V1.
2) Wellens' syndromes present with progressive T wave inversions in leads V2-V3 and little cardiac marker elevation, indicating critical proximal LAD stenosis.
3) ST elevation in aVR with widespread ST depression indicates high-risk left main or three-vessel coronary disease requiring emergent angiography.
The document provides criteria for diagnosing ST-elevation myocardial infarction (STEMI) on electrocardiogram (ECG). It lists cut-off values for ST elevation in different leads used to identify STEMI based on the patient's age, sex and lead location. It cautions that baseline ECG abnormalities like left bundle branch block (LBBB) could obscure interpretation and provides examples of STEMI in different heart locations identified by affected leads on ECG.
1) STEMI equivalents refer to patients with acutely occluded coronary arteries who do not present with classical ECG changes but have worse outcomes. Common equivalents include de Winter ST/T waves, Wellens' syndromes, ST elevation in aVR, new LBBB, isolated posterior MIs, and upright T waves in V1.
2) Wellens' syndromes present with progressive T wave inversions in leads V2-V3 and little cardiac marker elevation, indicating critical proximal LAD stenosis.
3) ST elevation in aVR with widespread ST depression indicates high-risk left main or three-vessel coronary disease requiring emergent angiography.
Non-invasive imaging plays an important role in the management of cardiovascular diseases. Different imaging modalities have advantages and limitations. Echocardiography is useful for assessing cardiac structure and function but limited for coronary artery disease evaluation. Nuclear imaging can evaluate perfusion and function but not coronary anatomy directly. CT and MRI can assess coronary anatomy in addition to function but CT involves radiation. The appropriate choice of imaging modality depends on the clinical question and no single test can replace all others for evaluating cardiovascular diseases. Integrating complementary information from different tests provides the most comprehensive assessment.
ECG in Emergency Department - Advances in ACS ECGDr.Mahmoud Abbas
ECG in Emergency Department -Advances in ACS ECG. Lecture presented by Dr Hesham Ibrahim at the Egyptian Critical Care Summit , the leading educational event and medical exhibition in Egypt.
This document discusses the stages of pulmonary edema seen on chest x-rays and associated wedge pressures. Stage I shows early signs like deer antler sign and Kerley B lines with pressures of 12-18 mmHg. Stage II shows interstitial edema on x-ray with pressures of 19-25 mmHg. Stage III is alveolar edema appearing as bat wing shadowing on x-ray associated with pressures over 25 mmHg.
The document provides an overview of the pathology of lung diseases as seen on chest x-rays, including signs, appearances and common causes of conditions such as consolidation, pleural effusion, atelectasis, pneumothorax, lung masses, fibrosis and infections like tuberculosis. Differential diagnoses are also provided for various lung abnormalities seen on x-rays.
The document discusses electrocardiograms (ECGs) in the context of acute coronary syndrome. It begins by describing the normal conduction system and the 12 standard ECG leads. It then explains how ECGs are recorded and the positioning of limb and precordial leads. The document discusses ST segments, T waves, and how to evaluate for ST elevations. It defines acute coronary syndrome and describes the classifications of ST-elevation MI, non-ST-elevation MI, and unstable angina based on ECG and cardiac enzyme findings. Specific ECG patterns for lateral, inferior, septal, and posterior wall MIs are also shown.
This document summarizes diagnostic radiology techniques for imaging the cardiovascular system. It describes normal appearances and abnormalities seen on x-rays, CT, MRI, echocardiography and nuclear medicine imaging. Key sections outline normal cardiac anatomy and sizes seen on x-ray, as well as abnormalities such as heart enlargement, pulmonary blood flow changes, aortic abnormalities like aneurysms and dissections. Imaging methods for evaluating these conditions are also mentioned.
This document discusses chest x-ray interpretation and provides guidance on evaluating x-rays. It explains that tissue density determines how an x-ray beam penetrates, with denser tissues appearing whiter and less dense tissues appearing blacker. It also outlines different chest x-ray views and factors to consider like patient orientation, age, gender, and rotation. Abnormalities are described as appearing too white, too black, too large, or in the wrong place. The document stresses a systematic approach of identifying, localizing, describing lesions, and providing differential diagnoses.
- The document discusses the anatomy and physiology of the heart's conduction system and how it generates the normal cardiac rhythm. It describes the roles of the sinoatrial node, atrioventricular node, Bundle of His, and Purkinje fibers in conducting electrical impulses through the heart.
- Various types of cardiac arrhythmias are defined based on disruptions to the heart's normal conduction system. These include premature beats, rhythms originating from the atria, AV junction, or ventricles. Characteristics like P wave presence/morphology, rate, and regularity are used to identify arrhythmias.
- A 12-lead electrocardiogram (EKG or ECG) is used to
This document provides an overview of ECG interpretation, including conduction pathways, a systematic method of interpretation, and common abnormalities seen in critical care. It discusses supraventricular and ventricular arrhythmias, bundle branch blocks, heart block, and life-threatening arrhythmias such as ventricular tachycardia, ventricular fibrillation, and asystole. It also covers the basics of 12-lead ECG interpretation including lead placement and axis.
1. The document discusses rhythm identification and interpretation of electrocardiograms (EKGs). It covers the basics of rate, rhythm, axis, hypertrophy, and infarction.
2. Key rhythms discussed include normal sinus rhythm, wandering pacemaker, multifocal atrial tachycardia, and atrial fibrillation. Characteristics of each rhythm like rate, P wave morphology, and regularity are examined.
3. The document provides an overview of the PQRST waveform and reviews concepts like axis, intervals, and rhythm identification. Common arrhythmias are categorized including irregular rhythms, escape beats/rhythms, premature beats, tachyarrhythmias, and heart blocks.
This document is a module on ECG rhythm interpretation focusing on supraventricular and ventricular arrhythmias. It provides learning objectives and outlines several modules to be covered, including ECG basics, normal sinus rhythm, common arrhythmias, diagnosing myocardial infarction, and advanced 12-lead interpretation. Specific arrhythmias covered are sinus rhythms, premature beats, supraventricular arrhythmias like atrial fibrillation, atrial flutter, and paroxysmal supraventricular tachycardia. Ventricular arrhythmias of ventricular tachycardia and ventricular fibrillation are also discussed. Examples are provided of different arrhythmias and their characteristics.
This document provides an overview of ECG interpretation presented by Dr. Raghoba. It outlines the normal ECG components and measurements. It then describes various arrhythmias including tachycardias like sinus tachycardia, atrial fibrillation and ventricular tachycardia. Bradycardias like sinus bradycardia and heart block are also reviewed. Common ECG findings for myocardial infarction, bundle branch blocks, ectopics and structural heart disease are presented. Electrolyte disturbances and other miscellaneous ECG patterns are also discussed.
The document discusses the conduction system of the heart and components of an electrocardiogram (ECG). It provides descriptions of normal sinus rhythm and several types of heart block, including first, second (Mobitz I and II), and third-degree heart block. Characteristics of the rhythm, rate, P wave, PR interval, and QRS complex are described for each type of block.
This document provides an overview of how to read an electrocardiogram (ECG). It describes the basic anatomy and electrical conduction system of the heart and how the ECG machine records and displays the heart's electrical activity. It then outlines a systematic approach for interpreting an ECG, including evaluating the rhythm, rate, axes, voltages, waves, segments, intervals, and any signs of ischemia, injury, or arrhythmia. Localization of abnormalities is also addressed. Examples are provided throughout to illustrate various normal and pathological ECG patterns.
The document provides guidance on how to analyze an electrocardiogram (ECG). It discusses how to calculate heart rate from the ECG tracing and determine rhythm. It describes how to identify abnormalities in regular and irregular rhythms. It also outlines how to analyze the P wave, P-R interval, QRS complex, ST segment, T waves, and QT interval. The document notes the usefulness of ECG for evaluating conditions like myocardial ischemia, arrhythmias, conduction defects, and chamber hypertrophy.
1) The ECG trace represents the electrical activity of the heart over one cardiac cycle and is made up of three distinct waves: the P wave from atrial depolarization, the QRS complex from ventricular depolarization, and the T wave from ventricular repolarization.
2) When interpreting an ECG, the clinician should check patient details, calibration, rate, rhythm, and abnormalities in each lead to identify any arrhythmias, conduction defects, or signs of myocardial infarction.
3) Common rhythms include normal sinus rhythm, sinus bradycardia/tachycardia, atrial fibrillation, ventricular tachycardia, and various types of heart block. ST segment elevation in certain leads
The ECG represents the electrical activity of the heart. It can provide insight into cardiac pathophysiology by analyzing the distinctive waveforms of each cardiac event. The ECG can identify arrhythmias, ischemia, infarction, pericarditis, chamber hypertrophy, and electrolyte disturbances. The standard 12-lead ECG consists of 3 limb leads, 3 augmented limb leads, and 6 precordial leads, which provide different views of the heart. Analysis of the P wave, PR interval, QRS complex, ST segment, T wave, and QT interval can reveal normal sinus rhythm or abnormalities that require further investigation.
This document provides an overview of cardiac conduction systems, electrocardiograms, arrhythmias, and how to interpret ECG readings. It defines common arrhythmias like sinus tachycardia, atrial fibrillation, ventricular tachycardia and discusses how to identify them using a 6-step approach examining rate, rhythm, P-waves, P-R interval, QRS complex and the relationship between P-waves and QRS. Examples are given of normal sinus rhythm and various arrhythmias with their characteristic ECG patterns. Special situations and normal variants are also addressed.
The 11-step method provides a systematic approach to reading EKGs:
1. Gather data such as heart rate, intervals, and axis.
2. Diagnose rhythm, conduction blocks, enlargement, and infarction by applying specific criteria.
3. Potential diagnoses are identified through disturbances of rhythm, conduction, hypertrophy, and ischemia. The relationship between P waves and QRS complexes helps determine block types.
The 11-step method provides a systematic approach to reading EKGs:
1. Gather data such as heart rate, intervals, and axis.
2. Diagnose rhythm, conduction blocks, enlargement, and infarction by applying specific criteria.
3. Potential diagnoses are identified through disturbances of rhythm, conduction, hypertrophy, and ischemia. The four questions framework is used to characterize rhythms.
1) Bradyarrhythmias include sinus bradycardia, junctional rhythm, sinoatrial block, and atrioventricular block which can be first, second, or third degree.
2) Tachyarrhythmias include ventricular tachycardia, ventricular fibrillation, atrial fibrillation, AV nodal reentrant tachycardia, and atrial tachycardia. Narrow complex tachycardias require analyzing the P waves and PR and RP intervals to determine the mechanism.
3) Different arrhythmias are caused by abnormalities in impulse conduction through the heart or abnormal automaticity in the sinoatrial node or elsewhere. Diagnosis requires interpreting the rhythm on
Heart arrhythmia, also known as irregular heartbeat or cardiac dysrhythmia, is a group of conditions where the heartbeat is irregular, too slow, or too fast. Arrhythmias are broken down into: Slow heartbeat: bradycardia. Fast heartbeat: tachycardia. Irregular heartbeat: flutter or fibrillation.
This ECG shows three different rhythms: atrial flutter, a sinus beat, and atrial fibrillation. Atrial flutter has a sawtooth pattern best seen in certain leads and an atrial rate of 250-350 bpm with a regular ventricular rate half the atrial rate. Atrial fibrillation has irregular undulations representing disorganized atrial activation without contraction. This ECG also shows third-degree heart block with independent atrial and ventricular rhythms, indicated by an atrial rate of 88 bpm and ventricular rate of 50 bpm.
1. The document discusses electrocardiographic (ECG) interpretation including determining cardiac rate and rhythm, identifying conduction disturbances, myocardial ischemia or infarction, and other abnormalities.
2. It provides details on properly placing ECG leads and determining the cardiac axis. Common rhythms, conduction blocks, hypertrophy, and other ECG findings are explained.
3. A mnemonic device, RRAHIM, is presented to guide the systematic interpretation of an ECG, covering rate, rhythm, axis, hypertrophy, ischemia/infarction, and other findings.
Lec 14 basic ecg interpretation for mohsEhealthMoHS
This document provides an overview of basic ECG interpretation. It begins by describing the spatial orientation of the 12 lead ECG and how it relates to different areas of the heart. It then discusses normal cardiac conduction, including the roles of the sinoatrial node, atrioventricular node, bundle branches, and Purkinje fibers. Key intervals like the PR and QT intervals are also explained. Common rhythms are then summarized, focusing on identifying features like rate, regularity, and relationship between P waves and QRS complexes to determine if a rhythm is normal or abnormal.
The document provides an overview of electrocardiography (ECG) basics including lead positions, ECG paper and timing, standardization, the normal ECG waves including P, PR, QRS, ST segments, T waves, and QT interval, and abnormalities. Key findings of right and left ventricular hypertrophy, atrial enlargement, bundle branch blocks, myocardial infarction, and various degrees of atrioventricular block are also summarized.
Hurricane Maria caused widespread devastation in Puerto Rico in September 2017. Luis E Rios Jr's Disaster Medical Assistance Team (DMAT) deployed to Puerto Rico to support response efforts. Over the course of their deployment, the DMAT set up field medical stations, supported hospitals that lacked power and supplies, and provided medical care to displaced populations. Their work highlighted the extensive damage to Puerto Rico's infrastructure and healthcare system from the back-to-back hurricanes. Upon completing their mission, the DMAT team faced challenges with logistics of demobilizing from the remote island location.
The document discusses several major causes of childhood injuries including motor vehicle crashes, falls, burns, bicycling accidents, and firearm injuries. It reviews research demonstrating the effectiveness of interventions like car seats, bicycle helmets, safe firearm storage, and smoke detectors at preventing injuries. The goal of injury prevention is to understand how and why injuries occur so that targeted strategies can be developed and implemented to reduce injuries in children.
The document discusses the origins and progression of the opioid epidemic, beginning with increased opioid prescribing in the 1990s based on limited research. This led to widespread prescription drug abuse and a subsequent shift to heroin use. Heroin was then contaminated by increasingly potent synthetic opioids like fentanyl and its analogs, dramatically increasing overdose deaths. While naloxone programs aimed to reduce these deaths, issues with take-home naloxone use may undermine its effectiveness and encourage further risky opioid use.
This document provides an overview of treating sepsis in 8 steps:
1) Rapidly assess for critical instability and signs of organ dysfunction
2) Administer empiric fluids and perform ultrasound exams to identify potential infection sources
3) Administer early, broad-spectrum antibiotics within 1 hour
4) Identify and control infection sources
5) Understand appropriate vasoactive medications
6) Recognize patients that deteriorate rapidly
7) Use lactate levels to guide resuscitation but recognize limitations
8) Optimize hemodynamics prior to intubation for shock patients
Two case studies are then presented and managed using the stepwise approach.
This document discusses managing hypertensive emergencies in the emergency department. It defines hypertension and hypertensive urgency versus emergency. For hypertensive urgency, when BP is markedly elevated but there are no symptoms, treatment is usually not required in the ED and patients can be referred for outpatient follow up. For hypertensive emergency, when there are progressive symptoms of end organ damage, treatment in the ED is warranted to lower BP by about 25% aiming to avoid hypotension. Goals of treatment and commonly used oral and IV antihypertensive agents are reviewed.
The ECMO program at UF Health has treated 28 patients referred since starting in July 2017, with 7 patients placed on ECMO support. The program has accomplished several milestones like treating two patients simultaneously and cannulating patients back-to-back. Survival rates for patients treated with ECMO are over 80%. The program aims to progress through three phases, currently rescuing patients at UF Health and from the Jacksonville area who need advanced support. A recent case described a 21-year-old trauma patient who required multiple surgeries and ultimately ECMO support, showing improvement over his ICU course before being successfully decannulated.
This document discusses post-trauma resuscitation debriefing. It defines debriefing as a facilitated discussion of actions and thought processes during a trauma resuscitation to encourage reflection and improve future performance. Debriefing should identify the underlying rationales behind behaviors and occur immediately after the event with the entire multi-professional trauma team. The Plus-Delta-Discuss method is recommended, focusing on what went well, what could be improved, and discussing keys to success and barriers. Debriefing has been shown to improve performance in trauma resuscitations when done focused, succinctly and without blame.
This document provides information about an emergency and trauma care symposium discussing managing chaos in pediatric resuscitations. The symposium features two speakers - Dr. Todd Wylie and Dr. Robert C. Luten - who will discuss identifying factors that contribute to chaos in pediatric resuscitations, describing reliable means for initial pediatric patient assessment, and identifying tools and resources to reduce complexity. The document includes objectives, background on the two patient populations of adults and pediatrics seen in emergency departments, epidemiological data on pediatric emergency medical services, and an overview of contributors to chaos in pediatric resuscitations.
The document discusses prediction rules for identifying pediatric patients at low risk for intra-abdominal injuries after blunt abdominal trauma. It summarizes the PECARN prediction rule, which identifies variables from history and physical exam that predict whether a child needs intervention. The rule achieved high sensitivity and specificity. The document also discusses the UF-Jax algorithm for managing pediatric blunt abdominal trauma, which incorporates physical exam findings, labs, ultrasound, and CT imaging to determine need for observation, admission, or intervention. The goal is identifying injuries requiring treatment while avoiding unnecessary radiation exposure.
These case studies describe situations where victims of human trafficking presented for medical care but were not recognized. In the first case, "Jill" was brought to the emergency department by her trafficker after suffering complications from an attempted abortion. Due to a busy emergency department and missed signs of abuse, she was not identified as a trafficking victim. The second case involved an 18-year-old female who was actually a 15-year-old trafficking victim. These cases highlight how traffickers coach victims and manipulate healthcare encounters to avoid detection. Emergency departments frequently encounter trafficking victims but often fail to recognize them.
This document discusses three cases presenting with metabolic emergencies:
1) A woman with altered mental status was diagnosed with thyroid storm based on clinical manifestations and lab results. Treatment focused on reducing thyroid hormone levels and controlling symptoms.
2) A woman with abdominal pain and abnormal vital signs was found to have diabetic ketoacidosis based on high blood glucose and acidotic lab results. Treatment centered around rehydration, correcting acidosis, and identifying the precipitating cause.
3) An intubated trauma patient developed hypotension, which was diagnosed as adrenal insufficiency/crisis based on lab results and abrupt cessation of glucocorticoids during hospitalization. Treatment involved starting glucocortico
Geiger- Interdisplinary approach to wound managementUFJaxEMS
An interdisciplinary approach to wound management is discussed, focusing on providing quality care and positive patient outcomes. Key points covered include the physiology of wound healing, assessing wounds and barriers to healing, and guidelines for treating pressure injuries, venous insufficiency, arterial disease, and diabetic foot ulcers. Referrals to specialists are recommended when needed to properly diagnose and manage complex chronic wounds.
This document discusses trauma during pregnancy, including:
1) Trauma affects 1 in 12 pregnancies and is a leading cause of nonobstetric death, though 90% of injuries are minor.
2) Special considerations in the assessment and management of pregnant trauma patients include changes in cardiovascular, respiratory, gastrointestinal/genitourinary systems as well as injury patterns.
3) The goals in treating pregnant trauma patients are to first stabilize the mother to save the fetus, as maternal demise will lead to fetal demise in most cases. A multidisciplinary approach between trauma and obstetrics teams is important.
This document discusses the implementation of an EMT mid-line team at UF Health Jacksonville to reduce central line-associated bloodstream infections (CLABSIs). It describes how EMTs in the emergency department were trained to place ultrasound-guided peripheral IV lines, which led to an 80% reduction in central line placements. The success of this program then prompted the creation of an EMT mid-line team to place mid-lines in intensive care unit patients to further reduce CLABSI rates across the hospital. Key aspects of implementing this team included developing an EMT training program, ensuring continuity of ultrasound equipment, tracking placement success rates, and changing policies to allow peripheral mid-lines to remain in place for up to 29 days.
This document discusses the approach to managing difficult airways in the pre-hospital setting. It begins by defining normal, difficult, and failed airways. It then reviews indications for intubation and outlines an algorithm for assessing and managing difficult airways. Key factors that can predict a difficult airway are described. Techniques for managing difficult bag-mask ventilation, laryngoscopy, endotracheal intubation, and criricothyrotomy are outlined. Evidence on outcomes of airway management strategies in trauma and cardiac arrest patients is presented. The document concludes with a quiz question about the most important airway device.
This document provides goals and objectives for a training on evaluating and treating stroke patients in the pre-hospital setting. It reviews relevant neuroanatomy, stroke pathophysiology, risk factors, clinical assessment tools like NIHSS and LAMS scoring. It discusses distinguishing between ischemic and hemorrhagic stroke as well as common stroke mimics. The training emphasizes the importance of the pre-hospital evaluation and communicating findings to the hospital to expedite diagnosis and treatment. A case study demonstrates applying these clinical skills to assess a patient and consider differential diagnoses.
This document discusses common complications and emergencies that may occur in transplant patients presenting to the emergency department. It notes that infection is the most common reason for admission within the first year, with a variety of potential infectious etiologies outlined. It also discusses rejection, the effects of immunosuppressive medications, and graft-versus-host disease. Specific complications that may arise include urinary tract infections, renal artery thrombosis, biliary issues, and dysrhythmias. Treatment approaches for various infections, rejection, and complications are provided.
This document discusses advances in ECMO and REBOA treatment.
ECMO provides lung and heart support to allow time for treatment when time is the treatment. It is used for conditions like ARDS, lung trauma, heart attacks, and as a bridge to transplant. Outcomes have improved at ECMO centers.
REBOA is the placement of a balloon in the aorta to control hemorrhage from the abdomen or pelvis. It is indicated for trauma with suspected bleeding below the diaphragm and can control bleeding until surgery.
Update on drugs of abuse trends in FloridaUFJaxEMS
This document discusses trends in drugs of abuse in Florida. It provides case studies and discusses the clinical effects and management of various substances including opioids, synthetic cannabinoids, fentanyl, marijuana, synthetic cathinones like flakka, MDMA, and excited delirium syndrome. Key points are the rise in heroin and fentanyl deaths, increasing potency of marijuana products, constant production of new synthetic drugs, and importance of controlling agitation in excited delirium cases.
The document discusses an ED and trauma symposium presentation on sepsis management. The presentation covers updated sepsis definitions, principles of early severe sepsis management, tips for sepsis care, common pitfalls to avoid, and implications of recent literature. Key points include early recognition and treatment of sepsis, initial fluid resuscitation, early antibiotics, source control, hemodynamic support and lactate monitoring to guide resuscitation and improve outcomes.
A Strategic Approach: GenAI in EducationPeter Windle
Artificial Intelligence (AI) technologies such as Generative AI, Image Generators and Large Language Models have had a dramatic impact on teaching, learning and assessment over the past 18 months. The most immediate threat AI posed was to Academic Integrity with Higher Education Institutes (HEIs) focusing their efforts on combating the use of GenAI in assessment. Guidelines were developed for staff and students, policies put in place too. Innovative educators have forged paths in the use of Generative AI for teaching, learning and assessments leading to pockets of transformation springing up across HEIs, often with little or no top-down guidance, support or direction.
This Gasta posits a strategic approach to integrating AI into HEIs to prepare staff, students and the curriculum for an evolving world and workplace. We will highlight the advantages of working with these technologies beyond the realm of teaching, learning and assessment by considering prompt engineering skills, industry impact, curriculum changes, and the need for staff upskilling. In contrast, not engaging strategically with Generative AI poses risks, including falling behind peers, missed opportunities and failing to ensure our graduates remain employable. The rapid evolution of AI technologies necessitates a proactive and strategic approach if we are to remain relevant.
A workshop hosted by the South African Journal of Science aimed at postgraduate students and early career researchers with little or no experience in writing and publishing journal articles.
A review of the growth of the Israel Genealogy Research Association Database Collection for the last 12 months. Our collection is now passed the 3 million mark and still growing. See which archives have contributed the most. See the different types of records we have, and which years have had records added. You can also see what we have for the future.
Main Java[All of the Base Concepts}.docxadhitya5119
This is part 1 of my Java Learning Journey. This Contains Custom methods, classes, constructors, packages, multithreading , try- catch block, finally block and more.
How to Fix the Import Error in the Odoo 17Celine George
An import error occurs when a program fails to import a module or library, disrupting its execution. In languages like Python, this issue arises when the specified module cannot be found or accessed, hindering the program's functionality. Resolving import errors is crucial for maintaining smooth software operation and uninterrupted development processes.
it describes the bony anatomy including the femoral head , acetabulum, labrum . also discusses the capsule , ligaments . muscle that act on the hip joint and the range of motion are outlined. factors affecting hip joint stability and weight transmission through the joint are summarized.
How to Add Chatter in the odoo 17 ERP ModuleCeline George
In Odoo, the chatter is like a chat tool that helps you work together on records. You can leave notes and track things, making it easier to talk with your team and partners. Inside chatter, all communication history, activity, and changes will be displayed.
How to Build a Module in Odoo 17 Using the Scaffold MethodCeline George
Odoo provides an option for creating a module by using a single line command. By using this command the user can make a whole structure of a module. It is very easy for a beginner to make a module. There is no need to make each file manually. This slide will show how to create a module using the scaffold method.
Thinking of getting a dog? Be aware that breeds like Pit Bulls, Rottweilers, and German Shepherds can be loyal and dangerous. Proper training and socialization are crucial to preventing aggressive behaviors. Ensure safety by understanding their needs and always supervising interactions. Stay safe, and enjoy your furry friends!
Executive Directors Chat Leveraging AI for Diversity, Equity, and InclusionTechSoup
Let’s explore the intersection of technology and equity in the final session of our DEI series. Discover how AI tools, like ChatGPT, can be used to support and enhance your nonprofit's DEI initiatives. Participants will gain insights into practical AI applications and get tips for leveraging technology to advance their DEI goals.
147. Which of the following are proven to
improve survival from STEMI?
• Lights and Sirens
• Aspirin
• Morphine
• Oxygen
• Cardiac Catheterization
• ALS unit
• Helicopter transport
• Nitroglycerin
197. Benign Early Repolarization
Widespread concave STE
Most prominent V2-V5
STE usually < 2mm
Notching at J-point
Usually prominent in V4
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203. Brugada Type I
STE > 2mm in >1 of V1-V3
Followed by negative T
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Editor's Notes
Anatomical Leads
1st degree: prolonged PR > 200 ms
2nd degree:
Mobitz Type I: Wenckebach = progressively increasing PR interval until non-conducting p wave
Mobitz Type II = Intermittent non-conducting p waves without progressive prolongation of PR interval
2nd degree:
Mobitz Type I: Wenckebach = progressively increasing PR interval until non-conducting p wave
Mobitz Type II = Intermittent non-conducting p waves without progressive prolongation of PR interval
2nd degree:
Mobitz Type I: Wenckebach = progressively increasing PR interval until non-conducting p wave
Mobitz Type II = Intermittent non-conducting p waves without progressive prolongation of PR interval
3rd degree: Complete heart block – complete dissociation between P and QRS