This document discusses different types of colic in horses including spasmodic, flatulent, obstructive, and specific colic. Spasmodic colic is caused by increased intestinal movement and has intermittent attacks. Flatulent colic is due to intestinal gas buildup from fermentable foods. Obstructive colic involves intestinal impaction from sand or parasites. Specific colic includes conditions like volvulus (intestinal twisting), strangulation, and invagination where intestinal segments telescope inside each other. Diagnosis involves history, exam, and tests like rectal palpation. Treatment depends on the type but may include painkillers, laxatives, fluids, massage, and in some cases surgery.
www.petsgroomingtips.com is one of the front-runners in providing complete digital information to the pet owners, which would guide theme through various process of grooming their beloved kids. A shabby puppy or kitten not only appears dirty but soon infested with disease if not treated properly. Our various tutorials and free PDF guides cover every aspect of the grooming process.
www.petsgroomingtips.com is one of the front-runners in providing complete digital information to the pet owners, which would guide theme through various process of grooming their beloved kids. A shabby puppy or kitten not only appears dirty but soon infested with disease if not treated properly. Our various tutorials and free PDF guides cover every aspect of the grooming process.
Diagnosis and Treatment of Canine Pyodermaupstatevet
Ed Jazic, DVM, DACVD
The prevalence of Canine Pyoderma is increasing very quickly and the clinical condition can present in a variety of ways. It is a common secondary manifestation of a variety of clinical conditions like allergic skin diseases, endocrinopathies, autoimmune skin diseases, and keratinization disorders. An efficient and correct diagnosis is essential as is proper therapy in the face of ever-increasing development of Canine Methicillin-Resistant Staphylococcal Pyoderma.
Mange is among the common illnesses that many dog owners are concerned about. People usually get tensed and do not know how to handle such situations. I present you this Presentation that speaks about Mange, a skin disease caused by tiny parasite mites on dogs. You can refer to this to know what Mange is all about, the types of Mange, the causes, the symptoms and a few easy ways to deal with it. Mange, if left untreated can be fatal. Therefore, timely knowledge and treatment about this disease is important. You can also add your views in the comments below.
For more information about Mange, you can refer to the following links:
http://www.vet-organics.com/types-of-mange-in-dogs/
http://pets.webmd.com/dogs/mange-dogs-canine-scabies
Diagnosis and Treatment of Canine Pyodermaupstatevet
Ed Jazic, DVM, DACVD
The prevalence of Canine Pyoderma is increasing very quickly and the clinical condition can present in a variety of ways. It is a common secondary manifestation of a variety of clinical conditions like allergic skin diseases, endocrinopathies, autoimmune skin diseases, and keratinization disorders. An efficient and correct diagnosis is essential as is proper therapy in the face of ever-increasing development of Canine Methicillin-Resistant Staphylococcal Pyoderma.
Mange is among the common illnesses that many dog owners are concerned about. People usually get tensed and do not know how to handle such situations. I present you this Presentation that speaks about Mange, a skin disease caused by tiny parasite mites on dogs. You can refer to this to know what Mange is all about, the types of Mange, the causes, the symptoms and a few easy ways to deal with it. Mange, if left untreated can be fatal. Therefore, timely knowledge and treatment about this disease is important. You can also add your views in the comments below.
For more information about Mange, you can refer to the following links:
http://www.vet-organics.com/types-of-mange-in-dogs/
http://pets.webmd.com/dogs/mange-dogs-canine-scabies
Colic Prevention: Proven tips to help reduce your horse's risksusantuller
Colic Prevention: Proven tips to help reduce your horse’s risk presented by Dr. Lydia Gray, Medical Director and Jessica Normand, Senior Director - SmartSupplements™ at SmartPak.
This is a presentation I gave on August 25, 2011 to a group of EU and UK equine veterinarians at the EBMS International Conference, at the Bartley Lodge Hotel in the New Forest, UK. I was very impressed with the discussions we had and the willingness the attendees had to implement social media in their veterinary practices. I gave them all a challenge to become dedicated users of social media in their businesses, otherwise there will be a real risk that their vet practices might not exist as they know it in the next 5-10 years. I truly believe that social media is the best opportunity we have as veterinarians to combat the numerous challenges facing our industry.
This is a presentation I gave at the 2015 AAEP Focus Meeting in August 2015.
My goal in the presentation was to highlight various technologies that can help make a veterinarians life easier. This could mean helping with productivity, interacting with clients and other members of the veterinary health team, treating a patient, educating clients, or making the use of social media much easier.
If you click the "Notes" button on the right under the screen you can see the notes I included for each slide.
Let me know if there is a technology that you use that could benefit other veterinarians.
Thanks
Mike Pownall
Equine Dentistry: Equine Teeth Cleaning with Equine Dentist Mark BurnellDeborah Davis
Cleaning your horse's teeth can have an impact on his health and well-being. Learn the importance of equine teeth cleaning in this presentation from Horseland.com.au with equine dentist Mark Burnell.
Ten things vets wished owners knew #horsehealthHorse SA
#horsesa #horse #horsecare #horsehealth
Dr Kendra McLeod, Equine Primary Opinion, University of Adelaide Equine Health & Performance Centre presented this talk at the Horse SA Northern Horse Forum, April 2017
http://www.adelaide.edu.au/vetsci/centres/ehpc/
Hosted by Horse SA https//www.horsesa.asn.au a non-profit organisation working with and for horse owners on common topics. Why not join, sponsor or donate today https://www.horsesa.asn.au/join-sponsor-or-donate or visit our website for more events, webinars or download free resources.
Note: Horse SA has hosted this presentation only. It is up to individuals to seek their own advice. It is also the responsibility of individuals to familiarise themselves with laws relating to the provision of animal services which may fall under Acts or Regulations associated with veterinary services, animal welfare and related areas, which vary between Australian states, and in jurisdictions within different countries.
Colic: Diagnosis, Treatment and Prevention (Carr)Gwyn Shelle
Colic, a symptom feared by horse owners, can seemingly appear without warning. There are many situations that can preclude a horse to colic, ranging from parasite infestation, dehydration to grain overload. Colic symptoms can appear mild to traumatic, but the actual severity and appropriate treatment options may be hard to determine until the veterinarian examines the horse. Dr. Elizabeth Carr will give a basic review of colic, discussing when the horse owner should worry, treatment options your veterinarian can use, and management practices to prevent certain kinds of colic.
Dentistry - How It Can Improve Your Driving HorseEqdent
A power point presentation put together for the James River Driving Club March 2012 meeting. Equine dentistry is an important aspect of keeping your carriage driving horse going comfortably.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
263778731218 Abortion Clinic /Pills In Harare ,sisternakatoto
263778731218 Abortion Clinic /Pills In Harare ,ABORTION WOMEN’S CLINIC +27730423979 IN women clinic we believe that every woman should be able to make choices in her pregnancy. Our job is to provide compassionate care, safety,affordable and confidential services. That’s why we have won the trust from all generations of women all over the world. we use non surgical method(Abortion pills) to terminate…Dr.LISA +27730423979women Clinic is committed to providing the highest quality of obstetrical and gynecological care to women of all ages. Our dedicated staff aim to treat each patient and her health concerns with compassion and respect.Our dedicated group ABORTION WOMEN’S CLINIC +27730423979 IN women clinic we believe that every woman should be able to make choices in her pregnancy. Our job is to provide compassionate care, safety,affordable and confidential services. That’s why we have won the trust from all generations of women all over the world. we use non surgical method(Abortion pills) to terminate…Dr.LISA +27730423979women Clinic is committed to providing the highest quality of obstetrical and gynecological care to women of all ages. Our dedicated staff aim to treat each patient and her health concerns with compassion and respect.Our dedicated group of receptionists, nurses, and physicians have worked together as a teamof receptionists, nurses, and physicians have worked together as a team wwww.lisywomensclinic.co.za/
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
6. II-Clinical classification of colic
1- Spasmodic colic
2- Flatulent colic
3- Obstructive colic
4- Specific colic due to outside
obstruction of bowel :
e.g. Volvulus; invagination; torsion;
strangulated hernia; scrotal,
diaphragmatic & umbilical hernia.
7. 1-Spasmodic colic
Definition :
It is a severe attacks of abdominal
pain caused by increase peristaltic
movement of the intestine,
characterized by:
intermittent fits of colic, rapid course
(short duration) and favorable
prognosis.
8. Etiology
(1) Drinking cold water when horse is hot
and sweat after work.
(2) Exposure of the body to external cold or
wetness.
9. Symptoms
(1) It occurs suddenly in the form of
fits of colic, each fit lasts from 5-15
minutes (short attacks).
during attack the animal kicks at the
abdomen, lies down on the ground
and rolls with violence .
10. but often rise again after rolling.
(2)There is slight increase in body
temperature, respiration, pulse &
congested mucosa.
(3) An increases in the peristaltic sound.
(4) Defecation occurs at short intervals and
the feces may be semisolid or fluid.
11. (5) Frequent passage of small quantity of
urine.
(6) Local or patchy sweating on back,
gluteal region, brisket & hindleg region.
(7) Horse may looks at the flank region
towards the site of colic.
(8) After the attach, the horse look normal
till second fit.
13. Treatment
(1) Give intestinal sedative and
antispasmodic drugs
R/ Atropine sulfate 4-6 ml 1%IM or IV.
R/ Anlagen 10 ml/100KgBW injected IV
(2) Rectal enema using warm water
and soft soap to stimulates and
regulates peristaltic movement of the
intestine and consequently relief pain.
15. Etiology
(1) Feeding on large quantities of highly
fermentable green feed such as
barseem.
(2) Ingestion of spoiled or moldy food or
grains, which has tendency to swell.
(3) Sudden change in the ration.
(4) Atony of the bowel.
(5) Obstruction of the bowel by sands.
16. Symptoms
(1) Sudden attacks of
continuous abdominal pain.
(2) Abdominal distension
(especially in the right flank).
(3) Cecal percussion gives
tympanic sound
17. (6) Congested mucosa, increase of
pulse & respiration, with moderate
to severe dyspnea.
(5) Affected horse may roll and
paw violently and lies down very
carefully.
(7) Small amount of feces may be
voided.
19. Treatment:
(1) Apply massage externally in flank
regions and internally through the
rectum using the hand.
(2) Rectal enema
(3) Purgatives:
Oral liquid paraffin 2-3 liters by
stomach tube
20. (5) Non steroidal anti-
nflammatory drugs:
Finadin
(6) Antifermentive such as
formaline solution, 10-20 ml
dissolved in 2 liters of water
given per os.
22. 3-Obstructive colic
(Colic due to impaction of the intestine)
It occurs when the large intestine
remains impacted with undigested food
material causing partial obstruction,
colic, depression & anorexia.
27. Etiology
(1) Overfeeding of grains or coarse food
rich in cellulose and bran for long period.
(2) Ingesting food materials, which contain
large amounts of mud or sand.
(3) Sluggish intestinal peristalsis
especially in old debilitated & or draught
horses.
(4) Greedy feeding & defective teeth.
28. (5) Obstruction of the intestine or natural
opening by large foreign bodies or parasite.
(6) Inadequate water intake or green food.
(7) Enterolith, fiber balls, hairball.
(8) Encephalitic (equine rectal paralysis).
29. Colic may be due to
(1) Impaction of the small intestine.
(2) Impaction of the colon.
30. [1] Impaction of the small
intestine
Causes:
It occurs due to accumulation of sand
in the small intestine (sandy colic) or
large number of parasites (Ascaris).
31. Symptoms
(1) The symptoms varies according to the
location of impaction:
when the duodenum is affected, the
symptoms occurs after feeding within few
hours.
when the ileum is affected symptoms
appear after several hours.
(2) Restlessness and beats the ground
with the forelimbs.
.
32. (3) In severe attacks the animal lies on the
ground and rolls
(4) During urination the animal throw the
hindlegs backward and outward and urine
comes out at intervals.
33. [2] Impaction of the colon
It is due to
accumulation of
undigested
materials in the
colon.
The large colon is
the most common
seat of impaction in
horse.
34. Symptoms
(1) Subacute colic which occurs slowly,
started with dullness and abdominal
discomfort, the animal looks at the flank
and kicking its belly.
(2) Constipation, Feces are passed in
small amounts, hard in consistency and
covered with thick & sticky mucous.
(3) Intestinal sound are absent or much
decreased in intensity.
35. 4) Moderate increase in pulse but the
temperature & respiration are normal.
(5) Rectal palpation revealed balloon
shape colon impacted with fecal mass.
(6) Dehydration, thirst, anorexia occur
with constant effort to urinate.
37. 1-Case history
A-How sever has the pain been?
B-When did the horse last defecate?
And what was the character of the
feces?
C-Has the horse shown specific
behaviour such as playing with water?
D-Could the horse have gaine access
highly fermentable food?
38. 2- Examination of pulse:
The heart rate and the character of
pulse are important in determining the
degree of severity of colic.
Rates greeter than 80 bpm should be
considered the result of sever lesion or
disease
39. 3- Examination of M.M
In simple dehydration the oral mm is
slight bluish but in sever dehydration it
becomes brick red or cyanosed. In
sever cases it becoms pale blue-grey
color.
40. 4- Auscultaion of
abdomen
Site: the caudal edge of the last ribe
from the paralumber fossa to ventral
abdomen on both right and left side:
Deacrease or absence of intestinal
sounds in case of clonic impaction
NB:
Gas/fluid interface sounds cause by the
movments of the horse should not be
mistaken
41.
42. 5-Rectal examination
In lare colon impaction is characterized
by an enlarged, firm, filled viscous
located on the pelvic floor
43. 6-Abdomenocentesis
Analysis of peritoneal fluid reflects the
changes. More than 500 white blood
cells/Ul. increase Of protein and RBcs
in colonic impaction & strangulation.
44. 7-Laboratory ex
PCV and Total plasma protine are used
to detrmine the hydration state of the
animal.
Electrolyte and acid base balance.
50. (2) Laxative:
per os or direct into the cecum by long
needle as liquid paraffin or mineral oil
or lin seed oil (2-3 liters).
51. (3)Fluid therapy:
R/Saline (0.09%Na Cl) 5-6LI/V
(4) Rectal enema :
using warm water and soft soap or lubricant.
According to the the degree of dehydration
(5) Parasympathetic drug:
R/Neo-stigmine 1 amp/70 Kg BW
:مهمة ملحوظة
وعمل البرافين زيت شربة إعطاء من ساعة بنصف الدواء هذا يعطى
ساعة نصف لمدة للحصان المشى رياضة عمل ثم الشرجية الحقنة
رقم4و5اإلتاث من للحوامل اليعطى
52. Meconium impaction in
foals
Meconium is the first fecal material
produced in the uterus by a newborn
foal.
It is Composed of cellular debris,
amniotic fluid and intestinal secretions.
53. It is normally passes within 30 minutes to
two hours after birth.
It is dark brown and forms small balls,
but in meconium impaction it becoms dry
and hard.
54. Causes
lack of oxygen at birth may affect
intestinal motility.
Dehydration may also effect the
passage of fecal material.
A narrower pelvis in male foal may
predispose it to impaction.
55. Abdominal pain
manifested by tail
swishing and
restlessness
Reduced attempts
to nurse
recumbency and
violent rolling
Clinical finding
60. [1] Embolic colic
(Verminous aneurism)
It is disorder in the intestine due to
the presence of larvae of strongylus
vulgaris in the anterior mesenteric
artery of the horse, causing
aneurisms, emboli and thrombi of the
mesenteric artery and its branches.
61. Clinical symptoms
It is characterized by intermittent
and attacks of colic which
occurs suddenly during work as
in spasmodic colic, beside that
the feces are bloody ¶sitic
eggs are present in fecal
examination
65. CausesCauses
(1) Severe attack of colic.
(2) violent movements, or sudden fall of
the animal.
(2) Injections of large dose of
parasympathetic drugs
(3) Heavy infestation with parasite
(Ascaris) cause irregularity in peristaltic
movement of the intestine .
66. SymptomsSymptoms
(1) Peracute abdominal pain, .
(2) Profuse sweating due to fatigue & pain.
(3) Initially temperature is elevated,
terminally becomes subnormal.
(6) Clean rectum by rectal palpation
(7) Short course, death within 48 hours.
69. [[33] Intestinal strangulation] Intestinal strangulation
It occurs when a lope of the intestine
passes through a natural or artificial
opening in the peritoneum as in case of
inguinal hernia in stallion. Also in case
of pedunculated tumour which cause
strangulation.
Symptoms: as torsion.
Treatment: Surgical.