Sinus tachycardia is characterized by a heart rate over 100 beats per minute originating from the sinus node. It can be a normal response to exercise or stress or indicate underlying conditions like heart failure. Symptoms may occur if the heart rate is very fast or the patient has heart disease. Treatment involves addressing the underlying cause and reducing stress or anxiety.
Heart arrhythmia, also known as irregular heartbeat or cardiac dysrhythmia, is a group of conditions where the heartbeat is irregular, too slow, or too fast. Arrhythmias are broken down into: Slow heartbeat: bradycardia. Fast heartbeat: tachycardia. Irregular heartbeat: flutter or fibrillation.
Wolff–Parkinson–White syndrome (WPW) is one of several disorders of the conduction system of the heart that are commonly referred to as pre-excitation syndromes. WPW is caused by the presence of an abnormal accessory electrical conduction pathway between the atria and the ventricles. Electrical signals traveling down this abnormal pathway (known as the bundle of Kent) may stimulate the ventricles to contract prematurely, resulting in a unique type of supra-ventricular tachycardia referred to as an atrio-ventricular reciprocating tachycardia.
Definition of arrhythmia - background on cardiac physiology including conduction in heart - action potential - pathogensis of arrhythmia - causes and risk factors for arrhythmia- diagnosis of arrhythmia - symptoms of tachyarrhythmias and bradyarrhythmias - investigations for arrhythmia - treatment of arrhythmia - pharmacological and other modalities of therapy for arrhythmia - managment of different types of arrhythmias
Heart arrhythmia, also known as irregular heartbeat or cardiac dysrhythmia, is a group of conditions where the heartbeat is irregular, too slow, or too fast. Arrhythmias are broken down into: Slow heartbeat: bradycardia. Fast heartbeat: tachycardia. Irregular heartbeat: flutter or fibrillation.
Wolff–Parkinson–White syndrome (WPW) is one of several disorders of the conduction system of the heart that are commonly referred to as pre-excitation syndromes. WPW is caused by the presence of an abnormal accessory electrical conduction pathway between the atria and the ventricles. Electrical signals traveling down this abnormal pathway (known as the bundle of Kent) may stimulate the ventricles to contract prematurely, resulting in a unique type of supra-ventricular tachycardia referred to as an atrio-ventricular reciprocating tachycardia.
Definition of arrhythmia - background on cardiac physiology including conduction in heart - action potential - pathogensis of arrhythmia - causes and risk factors for arrhythmia- diagnosis of arrhythmia - symptoms of tachyarrhythmias and bradyarrhythmias - investigations for arrhythmia - treatment of arrhythmia - pharmacological and other modalities of therapy for arrhythmia - managment of different types of arrhythmias
Cardiology 1.4. Palpitations - by Dr. Farjad IkramFarjad Ikram
Introduction to one of the more common symptoms of cardiac, psychiatric and metabolic disease. Palpitation is the uncomfortable awareness of heart beat and can often be the only symptom of underlying fatal arrhythmias.
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Cardiology 1.1. Chest pain - by Dr. Farjad IkramFarjad Ikram
Introduction to one of the most common symptoms that can represent a wide range of diseases, from benign to life-threatening, covering number of systems including gastrointestinal, cardiovascular, pulmonary, musculoskeletal and psychiatric. Includes a brief explanation of anti-anginal therapy.
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2. Sinus Tachycardia
• In an adult is characterized by a sinus rate of
more than 100 beats/minute
• Rate rarely exceeds 160 beats/minute except
during strenuous exercise
• Each impulse follows the normal pathway of
conduction resulting in atrial and ventricular
depolarization
3. Sinus Tachycardia
• How it happens
– Depends on the underlying cause
• May be of no clinical significance
– May be the body’s response to exercise
– May be the body’s response to high emotional state
• May also occur with hypovolemia, hemorrhage, or pain
– When the stimulus for the tachycardia is removed,
the arrhythmia spontaneously resolves
4. Sinus Tachycardia
• Causes
– Normal response to
• Exercise, pain, stress, fever, or strong emotions
– Certain cardiac conditions
• Heart failure
– Medications
• Epinephrine and atropine
– Substances
• Caffeine, nicotine, and cocaine
– Other conditions
• Anemia, respiratory distress, pulmonary embolism, sepsis,
and hyperthyroidism
5. Sinus Tachycardia
• Hard on the heart
– Not good for those with heart conditions already
– Considered a poor prognostic sign if follows MI
• Is associated with massive heart damage
– Persistent tachycardia may signal impending heart
failure or cardiogenic shock
– Consequences
• Bring on an episode of chest pain in patients with CAD
6. Sinus Tachycardia
• What to look for
– Look for a pulse rate of more than 100
beats/minute
– Rhythm is regular
8. Symptomatic Tachycardia
• If cardiac output falls and compensatory
mechanisms fail
– Will experience symptoms
• Hypotension
• Syncope
• Blurred vision
• Chest pain and palpitations
• Nervousness or anxiety
• Heart failure
– JVD
– crackles
9. Symptomatic Tachycardia
• Steps to take
– Prompt recognition is vital so treatment can be
started
– Provide the patient with a calm environment; help
to reduce fear and anxiety which can fuel the
arrhythmia
– Tachycardia is commonly the first sign of
pulmonary embolism
10. Symptomatic Tachycardia
• When to call for help and what to do until help
arrives
– Look at the patient and ask how they are doing
– Call for help if heart rate is too fast and/or
symptomatic
• Compare it their normal heart rate and rhythm
– Stay with the patient
– If the patient is not breathing and does not respond
• Call code
• ABCs/CPR
11. Sinus Tachycardia
• What to look for
– Look for a pulse rate of more than 100
beats/minute
– Rhythm is regular
12. Sinus Tachycardia
• Normal
– P wave preceding each QRS complex
– PR interval
– QRS complex
– T wave
– QT interval
13. Sinus Tachycardia
• P wave
– Normal size and shape and precedes each QRS,
but it may increase in amplitude
– As the heart rate increases, the P wave may be
superimposed on the preceding T wave and
difficult to identify
14. Sinus Tachycardia
• PR interval
– Normal indicating that the impulse is following
normal conduction pathways
• 0.12-0.20 seconds
15. Sinus Tachycardia
• QRS complex
• Normal duration representing normal
ventricular impulse conduction and recovery
– Less than 0.12 seconds
16. Sinus Tachycardia
• T wave
– Upright in lead II, confirming that normal
repolarization has taken place
17. Sinus Tachycardia
• QT interval
– Within normal limits
• 0.36 to 0.44 seconds
• QT normally shortens with tachycardia