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Dr. Abraham
 Identify
 Classify
 Rectify
 Location
 SA nodal artery – RCA>LCx
 S & PS
 Asymptomatic – ECG changes
 Symptomatic – tachy
brady
tachy – brady
 Overdrive suppression
 SA disease – brady
pause
arrest
exit block
chronotropic incompetence
 <85% predicted heart rate
 <100
 <2 SD of expected
 Block sympathetics – 0.2mg/kg propranolol
 Block para – 0.04mg/kg atropine
 Intrinsic heart rate –
117.2-(0.53*age)
 Type I second-degree SA block results from
progressive prolongation of SA node
conduction with intermittent failure of the
impulses originating in the sinus node to
conduct to the surrounding atrial tissue.
Second-degree SA block appears on the ECG
as an intermittent absence of P waves
“WENKEBACH”
 no change in SA node conduction before the
pause
 Complete or third-degree SA block results in
no P waves on the ECG
 ECG – holter
loop study
event monitors
 Chronotropic incompetence
 IHR
 Invasive testing – SNRT
SACT
 Exclude extrinsic causes
 Short term – pharmacotherapy – atropine
theophylline
isoproterenol
 Long term – pacemaker – SA disease
Carotid
hypersensitivity
 OF KOCH
 AV nodal , septal perforators
 S & PS
 HIS and further down – minimal autonomic
innervation
 1ST DEGREE
Mobitz type I “wenkebach”
 2ND DEGREE
Mobitz type II
 3RD DEGREE
DEFINITE DIAGNOSIS – “HIS” ELECTROGRAM
 Exculde functional causes
Transcutaneous
Temporary
 Pacing Transjugular
Permanent
 1 – A, V, D, S, O
 2 – A, V, D, S, O
 3 – I, T, D, O
 4 – R
 5 – P, S, D, O
 Of the procedure
 Twiddler syndrome
 Pacemaker syndrome – fatigue
neck pulsation
palpitation
dizziness
raised JVP
cannon waves
CCF
 Class I - those for which there is evidence or
consensus of opinion that therapy is useful
and effective
 Class II - there is conflicting evidence or a
divergence of opinion about the efficacy of a
procedure or treatment
 II A - weight of evidence or opinion favors
treatment
 II B - efficacy is less well established by the
evidence or opinion of experts
 Class III - evidence or weight of opinion
indicates that the therapy is not efficacious
or useful and may be harmful
Bradyarrhythmias

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Bradyarrhythmias

  • 2.
  • 3.
  • 4.
  • 5.
  • 7.  Location  SA nodal artery – RCA>LCx  S & PS
  • 8.  Asymptomatic – ECG changes  Symptomatic – tachy brady tachy – brady  Overdrive suppression  SA disease – brady pause arrest exit block chronotropic incompetence
  • 9.
  • 10.
  • 11.
  • 12.
  • 13.  <85% predicted heart rate  <100  <2 SD of expected
  • 14.  Block sympathetics – 0.2mg/kg propranolol  Block para – 0.04mg/kg atropine  Intrinsic heart rate – 117.2-(0.53*age)
  • 15.
  • 16.  Type I second-degree SA block results from progressive prolongation of SA node conduction with intermittent failure of the impulses originating in the sinus node to conduct to the surrounding atrial tissue. Second-degree SA block appears on the ECG as an intermittent absence of P waves “WENKEBACH”
  • 17.
  • 18.  no change in SA node conduction before the pause
  • 19.
  • 20.  Complete or third-degree SA block results in no P waves on the ECG
  • 21.
  • 22.  ECG – holter loop study event monitors  Chronotropic incompetence  IHR  Invasive testing – SNRT SACT
  • 23.  Exclude extrinsic causes  Short term – pharmacotherapy – atropine theophylline isoproterenol  Long term – pacemaker – SA disease Carotid hypersensitivity
  • 24.  OF KOCH  AV nodal , septal perforators  S & PS  HIS and further down – minimal autonomic innervation
  • 25.
  • 26.
  • 27.
  • 28.  1ST DEGREE Mobitz type I “wenkebach”  2ND DEGREE Mobitz type II  3RD DEGREE
  • 29.
  • 30.
  • 31.
  • 32.
  • 33. DEFINITE DIAGNOSIS – “HIS” ELECTROGRAM
  • 34.
  • 35.
  • 36.
  • 37.  Exculde functional causes Transcutaneous Temporary  Pacing Transjugular Permanent
  • 38.
  • 39.
  • 40.  1 – A, V, D, S, O  2 – A, V, D, S, O  3 – I, T, D, O  4 – R  5 – P, S, D, O
  • 41.  Of the procedure  Twiddler syndrome  Pacemaker syndrome – fatigue neck pulsation palpitation dizziness raised JVP cannon waves CCF
  • 42.  Class I - those for which there is evidence or consensus of opinion that therapy is useful and effective  Class II - there is conflicting evidence or a divergence of opinion about the efficacy of a procedure or treatment  II A - weight of evidence or opinion favors treatment  II B - efficacy is less well established by the evidence or opinion of experts  Class III - evidence or weight of opinion indicates that the therapy is not efficacious or useful and may be harmful