What is structure of lumber disc? What is disc bulge/prolapse/herniation? What is difference between disc bulge, disc prolapse, disc herniation or disc extrusion? What is criteria to diagnose lumber disc prolapse? How lumber disc herniation is treated medically or surgically? How lumber disc herniation is treated by conservative method? How lumber disc herniation is treated through physical therapy? What is physiotherapy after various disc surgeries? What is radiological method to diagnose disc prolapse?
Bicipital tendonitis is inflammation of long head of the biceps tendon under the bicipital groove.
In early stage, tendon becomes red and swollen, as tendonitis develops the tendon sheath can thicken.
In late stage, often become dark red in color due to inflammation.
low back pain is very common in population occurring at least once a lifetime in nearly 60-80% of population.
This presentation was presented as a webinar in coordination with ypta and serving hands on 12-8-2021.
Lumbar spondylosis- Diagnosis | management | a brief medical study martinshaji
Lumbar spondylosis is a degenerative condition which affects the lower spine. In a patient with lumbar spondylosis, the spine is compressed by a narrowing of the space between the vertebrae, causing a variety of health problems ranging from back pain tone urological problems.
please comment
thank you
Bicipital tendonitis is inflammation of long head of the biceps tendon under the bicipital groove.
In early stage, tendon becomes red and swollen, as tendonitis develops the tendon sheath can thicken.
In late stage, often become dark red in color due to inflammation.
low back pain is very common in population occurring at least once a lifetime in nearly 60-80% of population.
This presentation was presented as a webinar in coordination with ypta and serving hands on 12-8-2021.
Lumbar spondylosis- Diagnosis | management | a brief medical study martinshaji
Lumbar spondylosis is a degenerative condition which affects the lower spine. In a patient with lumbar spondylosis, the spine is compressed by a narrowing of the space between the vertebrae, causing a variety of health problems ranging from back pain tone urological problems.
please comment
thank you
As part of a class presentation, we attempted to make this to briefly explain what Torticollis meas, the Types of presentation of Torticollis, and Management strategies for a Physiotherapist for Congenital Torticollis especially.
I hope this helps. :)
The pictures and information had been taken from internet, complied to make a brief presentation for the purpose of class presentation.
I do not own any content.
Supraspinatus tear - medial information martinshaji
A supraspinatus tear is a tear or rupture of the tendon of the supraspinatus muscle. The supraspinatus is part of the rotator cuff of the shoulder.
please comment
thank you ....
As part of a class presentation, we attempted to make this to briefly explain what Torticollis meas, the Types of presentation of Torticollis, and Management strategies for a Physiotherapist for Congenital Torticollis especially.
I hope this helps. :)
The pictures and information had been taken from internet, complied to make a brief presentation for the purpose of class presentation.
I do not own any content.
Supraspinatus tear - medial information martinshaji
A supraspinatus tear is a tear or rupture of the tendon of the supraspinatus muscle. The supraspinatus is part of the rotator cuff of the shoulder.
please comment
thank you ....
Herniated disk in the lower back agrasen hospital dr sandeep agrawal gondia v...Dr.Sandeep Agrawal Gondia
Back Pain
Back pain is often a common symptom of many disease conditions and the back pain may range from simple or dull pain to sudden and sharp pain. If the pain persists for few days, it is acute pain whereas if continues for more than 3 months, it is considered as chronic pain. In most cases, back pain may resolve without any treatment however if persists for more than 3 days, medical intervention is necessary.
Neck Pain
The first 7 vertebral bones on the spinal column form the cervical spine and are located in the neck region. The neck bears the weight of the head, allows significant amount of movement, and also less protected than other parts of spine. All these factors make the neck more susceptible to injury or other painful disorders. Common neck pain may occur from muscle strain or tension in everyday activities including poor posture, prolonged use of a computer and sleeping in an uncomfortable position.
Spinal Deformity Surgery
The Spine or backbone provides stability to the upper part of our body. It helps to hold the body upright. It consists of several irregularly shaped bones, called vertebrae appearing in a straight line. The spine has two gentle curves, when looked from the side and appears to be straight when viewed from the front. When these curves are exaggerated, pronounced problems can occur such as back pain, breathing difficulties and fatigue and the condition will be considered as deformity. Spine deformity can be defined as abnormality in the shape, curvature and flexibility of spine.
Spine Injections
Spine injection is a nonsurgical treatment modality recommended for treatment of chronic back pain. Injection of certain medicinal agents relieves the pain by blocking the nerve signals between specific areas of the body and the brain. The treatment approach involves injections of local anaesthetics, steroids, or narcotics into the affected soft tissues, joints, or nerve roots. It may also involve complex nerve blocks and spinal cord stimulation.
Spine Trauma
Spine trauma is damage to the spine caused from a sudden traumatic injury caused by an accidental fall or any other physical injury. Spinal injuries may occur while playing, performing normal activities, operating heavy machines, lifting heavy objects, driving automobiles, or when you suffer a fall. Injury to spine may cause various conditions including fractures, dislocation, partial misalignment (subluxation), disc compression (herniated disc), hematoma (accumulation of blood) and partial or complete tears of ligaments.
Vertebral Fractures
Vertebral compression fractures occur when the normal vertebral body of the spine is squeezed or compressed. The bone collapses when too much pressure is placed on the vertebrae, resulting in pain, limited mobility, loss of height, and spinal deformities. In severe compression fractures the vertebral body is pushed into the spinal canal which will apply pressure on the spinal cord and nerves.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
2. Learn anatomy of lumber disc bulge
Types
Find out causes of it.
Elaborate differential diagnosis (DDs) for its signs and
symptoms
Clinical tests
Radiological findings or tests
Treatment options
3. What is Lumber region?
What is disc?
What is disc bulge and disc herniation?
4.
5. It is a hydrostatic, load bearing structure between the vertebral bodies.
The intervertebral disc incorporates of:
Annulus fibrosus
Nucleus pulposus
Endplate
A thick outer ring composed of fibrous cartilage called the annulus
fibrosus,
which surrounds a central gelatinous material known as the nucleus
pulposus.
Weight is transmitted to the nucleus through the hyaline cartilage plate.
The hyaline cartilage is ideally suited
to this function because it is avascular
The fibers of the annulus can be divided into three main groups:
outermost fibers
the middle fibers
innermost fibers
The anterior fibers are strengthened by the powerful anterior
longitudinal ligament.
The posterior longitudinal ligament affords only weak reinforcement,
especially at L4-5 and L5-S1, where it is a midline, narrow, unimportant
structure attached to the annulus. The anterior and middle fibers of the
annulus are most numerous anteriorly and laterally but are deficient
posteriorly, where most of the fibers are attached to the cartilage plate.
6.
7. Disc Bulge
Extension of the disc margin beyond the
margins of the adjacent vertebral endplates. Bulge
in the disc but not a complete rupture
Protrusion / Bulging
Nucleus forced into outermost layer of annulus
fibrosus- not a
complete rupture
Extrusion/ Disc herniation
The nuclear material emerges through the
annular fibers but the posterior longitudinal
ligament remains intact. A small hole in annulus
fibrosus and fluid moves into epidural space
Sequestration or free fragment
The nuclear material emerges through the
annular fibers and the posterior longitudinal
ligament is disrupted. A portion of the nucleus
pulposus has protruded into the epidural space.
8.
9. A bulging disc is that the nucleus does not
push out of the annulus in a bulging disc.
The disc simply bulges out of the space it
normally occupies in the spine.
Considered a normal part of aging, a
bulging disc may not even cause any
symptoms.
If it bulges enough to press on spinal
nerves or narrow the spinal canal, then it
can lead to symptoms including pain,
numbness, tingling, or weakness. A
bulging disc can sometimes be a
precursor to a herniated disc.
10. AGE: 30 – 40 years
MOST COMMON LEVEL: L4-L5 (next common level is L5-S1)
MOST COMMON TYPE: Postero-lateral type
WHY DISC PROLAPSE IS MOST COMMON POSTEROLATERALLY?
Incomplete annular lamellae in this quadrant (i.e) each lamellae end with
fusion to an adjacent lamellae not completely circular.
Fibers of annulus were deficient posteriorly.
Posterior fibers are only weakly reinforced by posterior longitudinal ligament
especially L4-5 and L5-S1
11. Repetitive mechanical activities – Frequent bending, twisting, lifting, and other
similar activities without breaks and proper stretching can leave the discs damaged.
Living a sedentary lifestyle – Individuals who rarely if ever engage in physical
activity are more prone to herniated discs because the muscles that support the back
and neck weaken, which increases strain on the spine.
Traumatic injury to lumbar discs- commonly occurs when lifting while bent at
the waist, rather than lifting with the legs while the back is straight. Sports/
Automobile injury
Obesity – Spinal degeneration can be quickened as a result of the burden of
supporting excess body fat.
Practicing poor posture – Improper spinal alignment while sitting, standing, or
lying down strains the back and neck. Sitting and bending forwards, lifting heavy
weight bending back.
Tobacco abuse – The chemicals commonly found in cigarettes can interfere with
the disc’s ability to absorb nutrients, which results in the weakening of the disc.
12. Patient presents with LOW BACK
PAIN with or without buttock
involvement.
AGGRAVATING FACTORS Pain
will aggravate on bending, stooping,
lifting heavy weight.
RELIEVING FACTORS: Pain
relieved on lying or rest. No position of
comfort in case of high lumbar root
lesions.
MORNING STIFFNES
Patient presents with lower leg pain more
dominated than low back pain. follows a
dermatomal pattern
Weakness And Paresthesia (pins and needles
feeling)
Numbness
Difficulty / painful walking in case of herniated
disc causing stenosis
Difficulty/ painful stepping down stairs in case of
herniated disc causing stenosis
AGGRAVATING FACTORS Pain will aggravate on
bending, stooping, lifting heavy weight, coughing,
sneezing, deep breathing or laughing. But in case of
stenosis aggravate on extension.
RELIEVING FACTORS Pain relieved on extension but
pain relieved on flexion posture if stenosis.
MORNING STIFFNES
BULGING HERNIATION
13. AROM
• Flexion- Painful and restricted
• Lateral bending to same side- Painful and restricted
• If stenosis, extension painful and restricted
PROM (carefully)
Tenderness +ve on involved vertebra or Over The Spinous Process
Paraspinal muscle spasm- Central Furrow sign
Gait- Antalgic/ limping gait
Loss of lumber lordosis
Scoliosis in Lumbar spine/ Sciatic scoliosis
Loss of Normal Lumbar Lordosis
Perform clinical tests
Dermatome and myotome examination
14. 1) SLR Test/ LASEGUE’S TEST
2) BRAGGARD’S SIGN
3) BOWSTRING SIGN
4) Contralateral SLR Positive/ Crossed Straight Leg Raise Test (Crossed
Lasègue test)/ CONTRALATERAL STRAIGHT LEG RAISING TEST
(FRAJERSZTAGN TEST)
5) NAFFZIGER’S TEST
6) Tripod Test/Flip Sign
7) Valsalva maneuver
8) Slump test
9) FEMORAL NERVE Traction TEST (REVERSE SLR TEST)
10) Prone knee bend (PKBI)
16. Perform SLR, move out of painful
range and add dorsiflexion
Stretching of the sciatic nerve will
cause intense pain
17.
18. Indicates a large central disc
herniation or sequestration
Contralateral side pain reproduction
on 40 to 60 degree hip flexion in
knee extended position
19. Here pressure applied on the
jugular vein for 10 seconds, the
patient face flush. Now patient
asked to cough which produce
pain in back indicate test is
positive.
Indication of thecal sac / spinal
theca compression
20. Shooting pain in entire leg in case of
nerve root involvement but muscular
tension in case of hamstring
contracture.
21. Pt. seated, asked to take a breath,
hold breath, and bear doen as if
evacuating stool.
Pain reproduce.
Indicate thecal sac compression
22. Hands behind back, slump
back of pt. ,flex head, exert
over pressure to head in
flexion, extend involved knee
with added pressure in
dorsiflexion
Neurological signs or
shooting pain reproduction
23. Patient side lying, extent hip
15 degree flex knee
Test for L2 to L4 nerve root
compression test
Pain in groin or hip that radiate
down anterior medial thigh
indicate L3 nerve lesion
Pain extending to midtibia
indicates L4 nerve root
problem
24. Pt. prone, flex knee to 90 degree pain,
maintain position for 45 to 60 seconds.
Pain provoke.
When taking heel to buttock indicate SI
or lumber pain.
Could indicate tight rectus femoris.
Maintaining position with careful
history and its modification lead to find
femoral nerve compression
25.
26.
27.
28.
29. MYOTOMES
L2: Hip Flexion
L3: Knee extension
L4: Ankle Dorsiflexion
L5: Great toe extension
S1: Ankle plantar flexion, eversion, hip extension
S2: Knee flexion
30. The diagnosis of disc rupture is dependent on
demonstration of root impairment as reflected by
signs of motor weakness, changes in sensory
appreciation or reflex activity.
32. Lumbar strain, spasm (70%)
Facet joint pain
Degenerative processes of disc and facets, usually age-related(10%)
Spinal stenosis
Osteoporotic compression fracture
Spondylolisthesis
Traumatic fracture
Congenital disease
Cauda equina
Inflammatory/metabolic causes: Diabetes, Ankylosing spondylitis, Paget’s disease,
Arachnoiditis, Sarcoidosis
Intraspinal synovial cysts
Severe kyphosis, Severe scoliosis, Internal disk disruption
Non mechanical- systemic cause
33.
34. Majority of disc prolapse respond well to conservative therapy. Resolution of
first disc prolapse takes place approximately 75% of patients over a period of
3 months ( 12 weeks) surgery should not be recommended for at least 6
weeks of treatment.
BED REST In very acute condition patient must be kept on bed rest.
Adequate analgesic relive the pain and this helps the muscle spasm to
subside. Patient should not be kept in bed rest for not more than 2 to 3days.
35.
36. Modalities
Cold or hot pack
TENS
Ultrasound therapy
Hydrotherapy
Lumbar Traction
Manual treatment
37.
38. Pain control
Ambulation and resumption of exercise
Education maintaining healthy weight
restoration of functional deficit
Restoration of neurological deficits associated with symptomatic disc
herniation.
39. Acute/ protection phase- 0 to 4 weeks
Subacute/ Controlled motion phase- 4 to 12 weeks
Chronic/ Return to function phase- >12 weeks (6
months in some cases)
40. Educate patient- encourage to engage in activities
Pain control – modalities, soft tissue mobilization, traction, mobilization, rest
for 2 days if needed to settle nerve root irritation
Lumber traction for relieving paresthesia
Educate good posture - add braces or lumber support if needed
Initiate neuromuscular activation and control of stabilizing muscles-
Core strengthening – drawing in maneuver , bridging ,
Start extension bias protocol (McKenzie protocol)
Teach safe performance of ADLs- add adjacent muscle strengthening
44. Educate patient – engage in all activities in safe mechanics, home exercise
program , ergonomics adaptation of work
Progress control of stabilizing muscles- Lifting one leg in crawling position
Lifting crossed arms and legs in crawling position Lunges
Flexibility exercises (eg, yoga and stretching)
Proprioception/coordination/balance (medicine ball and wobble/tilt board)
strengthening exercises
Trunk curls
SLR
aerobic activity (eg, walking, cycling)
45.
46.
47. Educate patient – engage in all activities in safe mechanics, home exercise program ,
ergonomics adaptation of work - progression
Progress control of stabilizing muscles- Lifting one leg in crawling position Lifting
crossed arms and legs in crawling position, Lunges progress
Flexibility exercises (eg, yoga and stretching)
Proprioception/coordination/balance (challange balance progress)
strengthening exercises- progression
aerobic activity (eg, walking, cycling)
(McKenzie approach progression)
motor control exercises MCEs
Endurance , agility, strength
Lumber Traction
48.
49.
50. Post Surgical Rehab - In case of surgery, program start regularly 4-6 weeks
post-surgery.
Patient education about the rehabilitation program they will follow the
next few weeks. Rehabilitation programs that start four to six weeks post-
surgery with exercises versus no treatment found that exercise programs
are more effective.
The patients are instructed and accompanied in daily activities such as:
coming out of bed, going to the bathroom and clothing
Patients have to pay attention on the ergonomics of the back.
Home exercise programs.
51. Duration of rehabilitation program: 4 weeks
Frequency: every day
Duration of one session: approximately 60 minutes
Treatment: dynamic lumbar stabilization exercises + home exercises
Exercises: Prior to the DLS training session patients are provided with instruction or
technique to ensure and protect a neutral spine position. During the first 15 minutes of
each session stretching of back extensors, hip flexors, hamstrings and Achilles tendon
should be performed.
(DLS consists of: Quadratus exercises Abdominal strengthening Bridging with ball
Straightening of external abdominal oblique muscle Lifting one leg in crawling position
Lifting crossed arms and legs in crawling position Lunges)
Home Exercises - should be added to the treatment. These should be performed every
day. 5 repetitions during the first week up to 10-15 reps in the following weeks