Congenital dislocation of the hip (CDH), also known as developmental dysplasia of the hip (DDH), is a condition where the femoral head is displaced from the acetabulum. It can occur before, during or after birth. Girls are more commonly affected than boys. Causes may include hereditary joint laxity, breech birth position, or defective acetabulum development. Treatment involves splinting or bracing in infants to encourage reduction, and may require surgery in older children if reduction does not occur. Physiotherapy focuses on maintaining reduction, improving range of motion and strengthening muscles.
Bicipital tendonitis is inflammation of long head of the biceps tendon under the bicipital groove.
In early stage, tendon becomes red and swollen, as tendonitis develops the tendon sheath can thicken.
In late stage, often become dark red in color due to inflammation.
Deformities observed with Pes cavus includes :
*clawing of the toes
*posterior hind foot deformity (described as an increased calcaneal angle)
*contracture of the plantar fascia
*cock-up deformity of the great toe
Following References were used to prepare this powerpoint presentation which makes the slides accurate and relaible for studying purpose; Therapeutic Exrercise – Carolyn Kisner
Orthopaedic Physical Assessment – Magee
Orthopaedic Medicine – L. Ombregt
Campbell’s Operative Orthopaedics
Slides includes following headings;
DEFINITION
TYPES
ORTHOPAEDIC ASSESSMENT
MEDICAL MANAGGEMENT
PHYSIOTHERAPY MANAGEMENT
SURGICAL MANAGEMENT
Bicipital tendonitis is inflammation of long head of the biceps tendon under the bicipital groove.
In early stage, tendon becomes red and swollen, as tendonitis develops the tendon sheath can thicken.
In late stage, often become dark red in color due to inflammation.
Deformities observed with Pes cavus includes :
*clawing of the toes
*posterior hind foot deformity (described as an increased calcaneal angle)
*contracture of the plantar fascia
*cock-up deformity of the great toe
Following References were used to prepare this powerpoint presentation which makes the slides accurate and relaible for studying purpose; Therapeutic Exrercise – Carolyn Kisner
Orthopaedic Physical Assessment – Magee
Orthopaedic Medicine – L. Ombregt
Campbell’s Operative Orthopaedics
Slides includes following headings;
DEFINITION
TYPES
ORTHOPAEDIC ASSESSMENT
MEDICAL MANAGGEMENT
PHYSIOTHERAPY MANAGEMENT
SURGICAL MANAGEMENT
Introduction:-
Hoffa's pad syndrome also known as Infrapatellar fat syndrome is an intrinsic disease of Hoffa's fat pad and a problem of knee joint which causes the pain at the front of knee joint so this pain is known as anterior knee pain.
Hoffa's fat pad contains pluripotent cells that can differentiate into osteoblasts and chondrocytes.
Hoffa's pad becomes inflamed or Damaged by The crushing of the pad between the femur and tibia during extension, causes inflammation of Hoffa's fat pad.
Nerve supply of fat pad is excellent( It receives branches of the femoral , common peroneal and saphenous nerves) so if it occurs any injury ,causes a sharp anterior knee pain.
The infrapatellar fat pad is a pad of adipose tissue underneath or deep to the patella tendon and the top of the fat pad attaches to the lower part of patella (knee cap).
hoffa's pad is a shock absorber ,when there is a direct force on the patella can result in pinching of the fat pad between femur and tibial plateau. The tibial plateau is the proximal tibial surface on which the femur rests.
HFP is surrounded anteriorly by the patellar tendon and the joint capsule, superiorly by the inferior pole of the patella, inferiorly by the proximal tibia and the deep infrapatellar bursa, and posteriorly by the joint synovium .
The main function of the HFP is to reduce friction between the patella, the patellar tendon, and the deep skeletal structures. In addition, it prevents pinching of the synovial membrane and it facilitates the vascularization of adjacent structures.
Causes:-
cause is usually due to single or repetitive traumatic episodes.
when you extend your knee the fat pad act as a cushion and reduces friction between outer patella facets and quadriceps tendons .
when you flex your knee ,upper part of fat pad becomes tensioned, it moves backwards in the knee.
it develops gradually over time if you repeatedly move your knee.
This is when your knee is forced to move forward from its completely straight normal position.
You may have always been able to over straighten your knee, which is called knee hyperextension or genu recurvatum .
hyperextension sports such as basketball, volleyball or high jumping may also cause inflammation of Hoffa's pad.
Hoffa's disease is more frequent in young women and the symptoms are anterior knee pain when upstairs and downstairs.
Sign and Symptoms:-
Complaints of anterior knee pain occurs when playing hyperextension sports such as basketball ,volleyball or high jumping .effusion and inflammation may be occurs and decreases the ROM of joint , stair negotiation .
Symptoms may worsen if the knee is overly straightened or bent for too long a period. Complications may include an inability to fully straighten the knee.
Diagnosis:-
Hoffa's syndrome completely diagnosed by MRI .we have requirement of an experienced orthopaedics to diagnose it.
primary Assessment have to check the Active and Passive Range Of Motion(AROM/PROM) of Hip joint and Knee joint.
Introduction:-
Hoffa's pad syndrome also known as Infrapatellar fat syndrome is an intrinsic disease of Hoffa's fat pad and a problem of knee joint which causes the pain at the front of knee joint so this pain is known as anterior knee pain.
Hoffa's fat pad contains pluripotent cells that can differentiate into osteoblasts and chondrocytes.
Hoffa's pad becomes inflamed or Damaged by The crushing of the pad between the femur and tibia during extension, causes inflammation of Hoffa's fat pad.
Nerve supply of fat pad is excellent( It receives branches of the femoral , common peroneal and saphenous nerves) so if it occurs any injury ,causes a sharp anterior knee pain.
The infrapatellar fat pad is a pad of adipose tissue underneath or deep to the patella tendon and the top of the fat pad attaches to the lower part of patella (knee cap).
hoffa's pad is a shock absorber ,when there is a direct force on the patella can result in pinching of the fat pad between femur and tibial plateau. The tibial plateau is the proximal tibial surface on which the femur rests.
HFP is surrounded anteriorly by the patellar tendon and the joint capsule, superiorly by the inferior pole of the patella, inferiorly by the proximal tibia and the deep infrapatellar bursa, and posteriorly by the joint synovium .
The main function of the HFP is to reduce friction between the patella, the patellar tendon, and the deep skeletal structures. In addition, it prevents pinching of the synovial membrane and it facilitates the vascularization of adjacent structures.
Causes:-
cause is usually due to single or repetitive traumatic episodes.
when you extend your knee the fat pad act as a cushion and reduces friction between outer patella facets and quadriceps tendons .
when you flex your knee ,upper part of fat pad becomes tensioned, it moves backwards in the knee.
it develops gradually over time if you repeatedly move your knee.
This is when your knee is forced to move forward from its completely straight normal position.
You may have always been able to over straighten your knee, which is called knee hyperextension or genu recurvatum .
hyperextension sports such as basketball, volleyball or high jumping may also cause inflammation of Hoffa's pad.
Hoffa's disease is more frequent in young women and the symptoms are anterior knee pain when upstairs and downstairs.
Sign and Symptoms:-
Complaints of anterior knee pain occurs when playing hyperextension sports such as basketball ,volleyball or high jumping .effusion and inflammation may be occurs and decreases the ROM of joint , stair negotiation .
Symptoms may worsen if the knee is overly straightened or bent for too long a period. Complications may include an inability to fully straighten the knee.
Diagnosis:-
Hoffa's syndrome completely diagnosed by MRI .we have requirement of an experienced orthopaedics to diagnose it.
primary Assessment have to check the Active and Passive Range Of Motion(AROM/PROM) of Hip joint and Knee joint.
Developmental Dysplasia Of Hip Or Displacement Of HipManisha Thakur
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Antibiotic Stewardship by Anushri Srivastava.pptxAnushriSrivastav
Stewardship is the act of taking good care of something.
Antimicrobial stewardship is a coordinated program that promotes the appropriate use of antimicrobials (including antibiotics), improves patient outcomes, reduces microbial resistance, and decreases the spread of infections caused by multidrug-resistant organisms.
WHO launched the Global Antimicrobial Resistance and Use Surveillance System (GLASS) in 2015 to fill knowledge gaps and inform strategies at all levels.
ACCORDING TO apic.org,
Antimicrobial stewardship is a coordinated program that promotes the appropriate use of antimicrobials (including antibiotics), improves patient outcomes, reduces microbial resistance, and decreases the spread of infections caused by multidrug-resistant organisms.
ACCORDING TO pewtrusts.org,
Antibiotic stewardship refers to efforts in doctors’ offices, hospitals, long term care facilities, and other health care settings to ensure that antibiotics are used only when necessary and appropriate
According to WHO,
Antimicrobial stewardship is a systematic approach to educate and support health care professionals to follow evidence-based guidelines for prescribing and administering antimicrobials
In 1996, John McGowan and Dale Gerding first applied the term antimicrobial stewardship, where they suggested a causal association between antimicrobial agent use and resistance. They also focused on the urgency of large-scale controlled trials of antimicrobial-use regulation employing sophisticated epidemiologic methods, molecular typing, and precise resistance mechanism analysis.
Antimicrobial Stewardship(AMS) refers to the optimal selection, dosing, and duration of antimicrobial treatment resulting in the best clinical outcome with minimal side effects to the patients and minimal impact on subsequent resistance.
According to the 2019 report, in the US, more than 2.8 million antibiotic-resistant infections occur each year, and more than 35000 people die. In addition to this, it also mentioned that 223,900 cases of Clostridoides difficile occurred in 2017, of which 12800 people died. The report did not include viruses or parasites
VISION
Being proactive
Supporting optimal animal and human health
Exploring ways to reduce overall use of antimicrobials
Using the drugs that prevent and treat disease by killing microscopic organisms in a responsible way
GOAL
to prevent the generation and spread of antimicrobial resistance (AMR). Doing so will preserve the effectiveness of these drugs in animals and humans for years to come.
being to preserve human and animal health and the effectiveness of antimicrobial medications.
to implement a multidisciplinary approach in assembling a stewardship team to include an infectious disease physician, a clinical pharmacist with infectious diseases training, infection preventionist, and a close collaboration with the staff in the clinical microbiology laboratory
to prevent antimicrobial overuse, misuse and abuse.
to minimize the developme
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CRISPR-Cas9, a revolutionary gene-editing tool, holds immense potential to reshape medicine, agriculture, and our understanding of life. But like any powerful tool, it comes with ethical considerations.
Unveiling CRISPR: This naturally occurring bacterial defense system (crRNA & Cas9 protein) fights viruses. Scientists repurposed it for precise gene editing (correction, deletion, insertion) by targeting specific DNA sequences.
The Promise: CRISPR offers exciting possibilities:
Gene Therapy: Correcting genetic diseases like cystic fibrosis.
Agriculture: Engineering crops resistant to pests and harsh environments.
Research: Studying gene function to unlock new knowledge.
The Peril: Ethical concerns demand attention:
Off-target Effects: Unintended DNA edits can have unforeseen consequences.
Eugenics: Misusing CRISPR for designer babies raises social and ethical questions.
Equity: High costs could limit access to this potentially life-saving technology.
The Path Forward: Responsible development is crucial:
International Collaboration: Clear guidelines are needed for research and human trials.
Public Education: Open discussions ensure informed decisions about CRISPR.
Prioritize Safety and Ethics: Safety and ethical principles must be paramount.
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1. CONGENITAL DISLOCATION OF THE HIP
(CDH)
OR
DEVELOPMENTAL DYSPLASIA OF THE HIP
(DDH)
Presented by :
UPASANA AGARWAL
BPT 4TH YEAR
12/02/2021
2. DEFINITION :
It includes :
I. Subluxation
II. Dislocation
III. dysplasia
Girls : Boys = 6:1 i.e. girls are more affected
Spontaneous dislocation of the hip occurring before, during or shortly after birth.
Common in the Western races
Uncommon in many Asian countries,
mainly in India
3. Hereditary – predispose to joint laxity
Hormone Induced – maternal relaxin (ligament
relaxing hormone)
Breech Delivery – 10 times more common
Defective development of Acetabulum
Swaddling of Infant
causes :
4. pathology :
BONES :
Acetabulum
Shallow acetabulum – upper part fails to
form horizontal roof
Head of femur rides up on the ilium
Bone form a pseudo-pocket
Femoral Head
Poor development
Displaced backward - upward
LIGAMENTS :
Developmental laxity
Stretched ligament (due to displacement of femoral head)
Ligamentum Teres – absent or poorly developed
MUSCLES :
Changes from subluxation or dislocation
Muscles shortening (hamstring, satorius, rectus femoris,
adductors)
5. Clinical presentations :
In Early Childhood :
1) Not very clear until the child starts to walk.
2) Asymmetry in the buttock or thigh fold.
3) Limited hip movements.
4) A click everytimes the hip is moved.
In Older Child :
1) Walking – delayed or limping(U/L) or waddling(B/L)
2) Postural Changes – scoliosis, lordosis
6. Pt assessment :
Demographic Data –
Name, Age, Gender
Chief Complains –
Parents complain about the asymmetrical folds, click during movements.
If the child has started walking, parents will complain about the peculiar gait pattern.
History Taking –
Type of delivery
Parents carrying the baby
7. Observation –
In Early Childhood :-
How the parents are carrying the baby
Asymmetric folds
Restricted abduction in hip flexion
In Older Child :-
Shortening of affected leg
Scoliosis
Lumbar lordosis
Wider perineum
Gait
Bilateral Involvement
8. Examination –
In Early Childhood :-
Barlow’s Test –
Test for subluxation of hip.
Ortolani’s Test –
Test of reduction of hip.
9. In Older Childhood :-
Galeazzi Sign –
lowering of affected side knee
Trendelenburg Test –
Stand on affected side leg
Opposite ASIS dips down
Telescopy Sign –
Test stability of hip
Positive – piston like movement is present
Gait –
Trendelenburg Gait Pattern (U/L)
Waddling Gait Pattern (B/L)
10. Investigations :
1) X – Ray :-
Below the Age of 1 Year :
• Difficult to diagnose – as ossification is not completed.
In Older Child :
• Epiphysis of the head of femur appears small
• Epiphysis – displaced laterally and upward
• Acetabulum – shallow
2) Ultrasonography :-
Position of head of the femur in newborn
11. physiotherapy management :
The treatment of CDH is age related.
Divided into parts :-
1. Up to 6 months
2. 6 months to 2 years
3. 2 years to 8 years
4. Above 8 years
WHAT ARE THE AIMS ?
to replace the femoral head in the acetabulum.
To maintain it in position until normal acetabulum development occurs.
12. Up To 6 Months :-
Splinting –
Hip Flexion and Abduction maintain
Pavlik harness
von Rosen Splint
13. 6 Months To 2 Years :-
POP Hip Spica –
With moderate medial rotation and abduction
For 3 months
Weight Traction –
Using a frame
Gallows traction
Usually for 3-4 weeks
14. 2 Years to 8 Years :-
Surgery –
Open reduction with or without osteotomy.
Chiari Pelvic Osteotomy.
Derotation Osteotomy
Plaster Immobilization continued followed by Physiotherapy
After 8 Years :-
Surgery –
No treatment is effective.
Total Hip Replacement - 2˚ OA followed by Physiotherapy
After 2 years of age, conservative management is not effective.
It requires surgical procedures.
15. Physiotherapy Management
Immobilization :-
1) Active movements to be encouraged
2) Isometrics – if possible
Gluteal muscle
Quadriceps
mobilization :-
1) Adduction limited due to immobilization - Relaxed Passive Adduction
2) Improve ROM of hip
3) Strengthening of the Glutei muscles
4) Quadruped knee standing to Kneeling
5) Improve Walking – with enough support.
17. Physiotherapy Management
mobilization :-
6) Hydrotherapy – general leg activities
5) Hippotherapy
6) After THR –
• Re-educate Gait
• Regain Muscle Strength and Movements
• Regain Patient’s Confidence
Hippo Therapy
18. references :
Essentials of Orthopedics and Applied Physiotherapy
– JAYANT JOSHI and PRAKASH KOTWAL
Tidy’s Physiotherapy – ANN THOMAS, ALISON
SKINNER and JOAN PIERCY
Essential Orthopedics – J. MAHESHWARI
Physiopedia.com