Volkmann’s Contracture
KIMBERLY WALSH
PT INTERN
SPANISH TOWN HOSPITAL
Brief Background
Definition
Clinically Relevant Anatomy
Epidemiology
Etiology
Pathophysiology
Clinical Manifestations
Dx
Differential Dx
Mx
Medical
Surgical
Physiotherapy
Prognosis
Brief Background
 Richard von Volkmann in 1881 was the first
to describe ischaemic muscle paralysis and
contracture.
 His name was later associated with the
contracture later in 1889 by Hildebrand to
describe the end result of any untreated/
inadequately treated compartment
syndrome
Definition
Volkmann’s Contracture (a.k.a Volkmann’s
Ischaemic Contracture) is a deformity of the
hand, fingers and wrist cause by injury to the
muscles of the forearm.
Definition cont’d
It is a permanent shortening of forearm
muscles which occur following injury and
results in a claw like deformity of the hand,
fingers and wrist
Clinically Relevant Anatomy
 The relevant anatomy of Volkmann’s
contracture includes the superficial and
deep flexors of the forearm.
Superficial Flexors of the Forearm
Pronator Teres
 Flexor Capri Ulnaris
 Palmaris Longus
 Flexor Digitorium
Superficialis
Deep Flexors of the Forearm
Pronator Quadratus
Flexor Pollicis Longus
Flexor Digitorium Profundus
Epidemiology
Overall, Volkmann’s contractures are rare.
The condition occurs most commonly in
children and follows injuries particularly to the
elbow
Etiology
The incidence of Volkmann’s Contracture is low.
Factors that can lead to the condition;
Neglected compartment syndrome
Trauma
Fractures
Tight immobilizers
Bleeding disorders
Animal bites
Burns
Excessive exercise
Pathophysiology
Volkmann’s Contracture is usually seen in children with
displaced supracondylar fractures of the humerus or
forearm.
It results from severe injury to the deep tissues and
muscles of the volar compartment secondary to
increased compartmental pressures
Pathophysiology cont’d
Volkmann’s contracture is the end
result of prolonged ischaemia of
muscles and nerves in the forearm.
The muscles undergoes necrosis,
fibrosis and contracture
Pathophysiology (cont’d)
There are three classification of Volkmann’s
Contracture:
Mild
Moderate
Severe
Clinical Manifestations
 The clinical presentation
includes the five “Ps” which are;
 Pain
 Pulselessness
 Pallor
 Paresthesia
 Paralysis
Clinical Manifestations cont’d
Special findings include;
Pain with passive stretching of the
flexors
Elbow flexion
Forearm pronation
Finger flexion
MCPJ in extension
The wrist is in palmar flexion
Clinical Manifestations cont’d
Palpation of the affected region creates pain.
Also, upon palpation, firmness of the tissues can be
felt.
Swelling
Decreased sensation
Diagnosis
X- rays of the humerus, elbow and
forearm are useful for assessing the
amount of displacement of supracondylar
fractures
Non- displaced fractures rarely cause
Volkmann’s contracture
Diagnosis cont’d
Manometry : measures
intracompartmental pressure
Angiogram: blood vessel rupture
MRI: fibrosis of the muscles
Differential Dx
Pseudo – Volkmann’s Contracture
Entrapment of the flexor digitorium profundus
secondary to fractures of radius or ulna occurs 2 days
– 16 years after close reduction of shafts of radius
and ulna
Differential Diagnosis cont’d
The forearm muscles are of normal length
It can be prevented by routine check of PROM
after closed reduction of fractures
Management
Medical Management
Initial treatment for Volkmann's contracture consist of
removing occlusive dressings or removal of cast
Analgesics provide symptomatic relief in chronic cases.
Surgical Management
 Fasciotomy: This is done to prevent
progression to Volkmann’s Contracture
Moderate stages may need tenolysis, neurolysis
and extensor transfer procedures.
Severe stages may need debridement of damaged
muscles.
Physical Therapy Management
In the mild stages, physical therapy alone can
be use to treat the contracture
The aim is to improve joint motion and muscle
force
Physical Therapy Management (cont’d)
Heating Modalities
Passive stretching
Muscle re- education using
electrical stimulation (can be
done with functional activities)
Strengthening of the remaining
muscle groups
Static Progressive Splinting
Post operative PT mx (moderate stage sx)
includes;
Immobilization in a splint or cast approx. 4
weeks with PROM exercises during that time.
Tendon/ Neural glides can be done.
 Isometric exercises begin at 4 weeks and a thermoplastic
splint may be worn full – time
 At week six, there is a transition to wearing night splint only.
Prognosis
The outcome will depend upon the stage at which
treatment is instituted.
The earlier it is undertaken, the better the outcome
likely will be.
The outlook is grave in the severe types.
References
 Farzad, M., Hussein,S. Layeghi, F. Non- Surgical Treatment of
Established Forearm’s Volkmann Contracture in Child: A Case
Report. Iranian Rehabilitation Journal,. Vol (8). (2010).
 Reurings, J.,Verhofstad, M. The Volkmann Ischemic
Contracture of the Forearm is Preventable. European Journal
of Trauma and Emergency Surgery.October 2007.
 Singh, A. Volkmann Ischemic Contracture Presentation and
Treatment. Bone and
Spine.https://boneandspine.com/Volkmann-ischemic-
contracture/
 Volkmann’s
Contracture.https://www.physiopedia.com/Volkmann%27s_Contr
acture
 Stevanovic, M., Sharpe, F. Compartment Syndrome and
Volkmann Ischemic Contracture.
THE END

Volksmann contracture

Editor's Notes

  • #5 He described a contracture of the muscles of the wrist and fingers which followed tight bandaging of the arm in the Rx of # of the elbow. He believed that it was essentially due to ischemic of the muscles
  • #8 A fracture of the supracondylar ridge causes a deficit in circulation of the biracial artery
  • #12 Origin: medial epicondyle of humerus and coronoid process of Ulnar Insertion: middle of lateral surface of radius Nerve supply: Median nerve Artery: Ulnar artery Action: pronates forearm
  • #13 Origin: medial epicondyle Insertion: Pisiform, hook of hamate and the base of the 5th metacarpal Nerve Supply: Ulnar nerve Artery: Ulnar artery Action: Flexes and addicts the wrist
  • #14 Origin: medial epicondyle of the humerus Insertion: Palmar aponeurosis and the flexor retinaculum of the hand Nerve supply: median nerve Action: flexes the wrist Artery: Ulnar artery
  • #15 Origin: medial epicondyle , Ulnar collateral ligament and coronoid process of Ulna Insertion: Middle phalanges of digits 2-5 Nerve supply: median nerve Artery: Ulnar artery Action: Flexors of the fingers, primarily the PIP joints
  • #17 Origin: Anterior surface of the Ulna Insertion: Anterior surface of the radius Nerve supply : median nerve ( anterior I. Nerve Artery: Ant. I artery Action : pronate the forearm
  • #18 Origin : Anterior surface of radius and adjacent interosseous membrane Insertion: base of distal phalanx of the thumb Nerve supply: median nerve ( ant interosseous nerve) C8 and T1 Artery: Anterior interosseous artery Action: Flexes phalanges of the 1st thumb
  • #19 Origin: Anterior and medial surfaces of ulna, interosseous membrane and deep fascia of the forearm Insertion: Base of the distal phalanx of digits 2-5 Nerve supply: medial part : Ulnar nerve (C8 & T1) Lateral part: anterior interosseous branch of median nerve (C8- T1) Artery: Ulnar and anterior interosseous arteries Action: Flexes distal phalanges and assists with flexion of the hand.
  • #25 Mild – deep flexor compartment of the hand esp Flexor digitorium profundus Nerve involvement may be absent or mild.
  • #26 Degeneration of the muscles FDP and FPL and partial degeneration of the flexor superficialis Neurological impairment is always presents. Intrinsic Ms present
  • #27 Degeneration of all flexor muscles of fingers and wrist and can extend to the wrist extensions.
  • #28 Pain – earliest sign Pallor unhealthy pale appearance
  • #29 elbow flexion forearm pronation wrist flexion thumb adduction metacarpophalangeal joint extension finger flexion.
  • #30 Swelling; due to swelling pulsations cannot be felt in the arm mainly in the distal part
  • #32 Manometry: Insertion of the needle within the compartment(s) The normal is between 0-8 MMHG (millimeters of mercury) Angiogram: esp in the case of supracondylar fractures
  • #37 Fasciotomy: Release of the constricted compartment Incision into the skin Emergency! 0-8mmhg normal Greater than 30 mmhg sx
  • #38 Tendon slide – correct tightness of the flexor, Neurolysis –of the median and ulna nerve “free up” the nerves. Extension transfer procedures- when finger flexors are very weak or absent there can be transfer of the extensor carpi radials Debridement- removal of damaged tissues
  • #40 Within Pain limits
  • #42 Strengthening of remaining Ms groups: grip strengthening Wrist flexor – isometric excs
  • #43 Static progressive splinting. For extending elbow, wrist and plus position of the fingers Increase ROM. Once ROM is improving then there is re-splinting Correcting the deformity
  • #44 Neurolysis : freeing up the nerves. Tendon slides
  • #46 Thermoplastic splint. Lighter, it can be removed for cleaning, easily molded around the body Night splint to maintain wrist and finger extension