Simple ECG lecture about sinus arrest, sinoatrial exit block, AV block and escape rhythms. Slideshow was made with an audience of medical professionals in mind.
Biatrial enlargement is diagnosed when criteria for both right and left atrial enlargement are present on the same ECG.
The diagnosis of biatrial enlargement requires criteria for LAE and RAE to be met in either lead II, lead V1 or a combination of leads.
Case-1:
A 23 years old medical student presented with occasional palpitation, shortness of breath and chest discomfort. He had the following ECG.
A 53 years old gentleman presented with palpitations for last 5 hours. He is smoker, diabetic, dyslipidemic and hypertensive. He had exertional chest discomfort for last 5 years and did coronary angiogram 3 years back and CAG revealed TVD and advised for revascularization. But he refused and was irregular in medication and reluctant for life style modification. He came to emergency department with this ECG.
ECG in Emergency Department - Advances in ACS ECGDr.Mahmoud Abbas
ECG in Emergency Department -Advances in ACS ECG. Lecture presented by Dr Hesham Ibrahim at the Egyptian Critical Care Summit , the leading educational event and medical exhibition in Egypt.
Biatrial enlargement is diagnosed when criteria for both right and left atrial enlargement are present on the same ECG.
The diagnosis of biatrial enlargement requires criteria for LAE and RAE to be met in either lead II, lead V1 or a combination of leads.
Case-1:
A 23 years old medical student presented with occasional palpitation, shortness of breath and chest discomfort. He had the following ECG.
A 53 years old gentleman presented with palpitations for last 5 hours. He is smoker, diabetic, dyslipidemic and hypertensive. He had exertional chest discomfort for last 5 years and did coronary angiogram 3 years back and CAG revealed TVD and advised for revascularization. But he refused and was irregular in medication and reluctant for life style modification. He came to emergency department with this ECG.
ECG in Emergency Department - Advances in ACS ECGDr.Mahmoud Abbas
ECG in Emergency Department -Advances in ACS ECG. Lecture presented by Dr Hesham Ibrahim at the Egyptian Critical Care Summit , the leading educational event and medical exhibition in Egypt.
A lecture on degenerative complete heart block. Given at the PHC-Pintig Puso Foundation 14th Post Graduate Course (in Cooperation with Department of Adult Cardiology of Philippine Heart Center).
Atrioventricular blocks are related to delay in conduction of the AV node..
Their recognition is primarily by ECG, anatomical correlation is by EP study.
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Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
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2 Case Reports of Gastric Ultrasound
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
The POPPY STUDY (Preconception to post-partum cardiovascular function in prim...
ECG Lecture: Sinus arrest, sinoatrial exit block, AV block and escape rhythms
1. Sinus arrest, Sinoatrial exit block,
AV blocks, Escape Rhythms
Dr. Michael-Joseph F. Agbayani, MD FPCP FPCC
@HeartRhythmMD
2. Images used are mine or under a creative commons
license or public domain.
3.
4.
5. SA node: 60 to 100
SA node: 60 to 100
bpm
bpm
AV Junction: 40 to
AV Junction: 40 to
60 bpm
60 bpm
Ventricle: <40 bpm
Ventricle: <40 bpm
6.
7.
8. Sinoatrial Exit Block
• Sinus node still firing
• Impulse fails to conduct beyond SA node
• Degrees
– First degree SA exit block
– Second degree SA exit block (Type 1 and type 2)
– Third degree SA exit block
9. First degree sinoatrial block
• First degree
– Delay in conduction of sinus impulse
– Not evident in a surface ECG
10. Second degree sinoatrial block
• Intermittent failure of the sinus impulse to
exit the node
• Type 1
– Progressive delay in sinus impulse resulting in
non-conducted impulse
– Group beating (Wenckebach periodicity)
12. Second-degree sinoatrial block
• Type 2
– Failed conduction of sinus impulse without
progressive prolongation of sinoatrial conduction
time
– Pause is a multiple of baseline P-P interval
13.
14. Third-degree sinoatrial block
• None of the sinus node impulses are able to
exit the node
• Absence of p waves
• Indistinguishable from sinus arrest
15. Sinus Pause / Sinus Arrest
• Sinus node doesn’t fire
• P-P interval of the pause not a multiple of the
baseline P-P interval
• Usually said to be abnormal if > 3 seconds
16.
17.
18. First degree AV block
•
•
•
•
PR interval is > 200 ms and is constant
Each p wave is followed by QRS complex
Marked first degree is >300 ms
May be symptomatic
19.
20. Second Degree AV Block
•
•
•
•
One non-conducted p wave
Mobitz Type 1 (Wenckebach Block)
Mobitz Type 2
2:1 AV block
21. Mobitz Type 1 (Wenckebach)
• Gradual prolongation of PR interval until nonconducted p wave
• RP-PR reciprocity
• Lengthening of the PR interval at progressively
shorter decrements
• Progressively shorter R-R intervals and group beating
• Shortening back to baseline PR interval after the
blocked cycle
22.
23.
24. Mobitz Type 2
• No change in PR before non-conducted P
wave
• Usually associated with His-Purkinje disease
25.
26.
27.
28. 2:1 AV block
• Second degree AV block
• Every other beat is conducted
• PR interval in conducted beats is usually
constant
29. 2:1 AV block
• Classifying into Mobitz Type 1 or type 2 is
discouraged
• Clues to level of block
– Narrow vs wide complex
– Long or short PRs in conducted beats
– PR and RP relationship
– Presence of Mobitz Type 1 block
– Response to Atropine or exercise
32. High Grade AV Block
• At least 2 consecutive non-conducted P waves
(3:1 AV Block)
• Also called “advanced second-degree heart
block”
• Look at the PR interval of conducted beats:
should be constant
34. 3rd Degree or Complete Heart Block
• None of the P waves are conducted
• P waves and QRS complexes occur
independently of each other
• R-R intervals are usually regular (junctional or
ventricular escape rhythm)
35. SA node: 60 to 100
SA node: 60 to 100
bpm
bpm
AV Junction: 40 to
AV Junction: 40 to
60 bpm
60 bpm
Ventricle: <40 bpm
Ventricle: <40 bpm
36.
37. • R-R intervals are usually regular (escape
rhythm)
• “PR” intervals are not constant / no pattern
• Escape rhythm can be junctional or ventricular
38. Escape Rhythms
• Atrial escape rhythm (<60 bpm)
– P wave morphology abnormal
• Junctional rhythm (40 to 60 bpm)
– Narrow QRS
– Retrograde P waves shortly before or after QRS, if
any
• Ventricular rhythm (<40 bpm)
– Wide, “bizarre” QRS complexes
39.
40. SA node: 60 to 100
SA node: 60 to 100
bpm
bpm
AV Junction: 40 to
AV Junction: 40 to
60 bpm
60 bpm
Ventricle: <40 bpm
Ventricle: <40 bpm
41.
42.
43.
44. Images
•
•
•
•
•
•
•
•
•
•
•
Heart image with conduction system: Public Domain image from Gray’s Anatomy
QRS complex: Public Domain image from Wikipedia user
http://en.wikipedia.org/wiki/User:Agateller
Road Block sign: http://en.wikipedia.org/wiki/File:Road_block.jpg
Traffic: http://en.wikipedia.org/wiki/File:Traffic_jam_on_Phu_Nhuan_district.JPG
Sinoartial exit block: http://commons.wikimedia.org/wiki/User:Jer5150
First degree AV block: http://www.flickr.com/photos/popfossa/
Second Degree AV block Mobitz Type 1:
http://commons.wikimedia.org/wiki/User:Jer5150
Mobitz type 2 and 2:1: http://commons.wikimedia.org/wiki/User:Jer5150
Complete heart block: http://commons.wikimedia.org/wiki/User:Jer5150
Complete heart block strip: http://en.wikipedia.org/wiki/User:MoodyGroove
Junctional rhythm: http://www.flickr.com/photos/nottinghamvets/
45. Images
•
•
•
•
•
•
•
•
Sinus pause: http://commons.wikimedia.org/wiki/User:Stevenfruitsmaak
Mobitz Type 1 with escape: Michael Rosengarten BEng, MD.McGill
(ecgpedia.org)
SCT with Pause: Michael Rosengarten BEng, MD.McGill (ecgpedia.org)
Mobitz type 1 and type 2 strips: Munther Homoud, M.D.
(http://ocw.tufts.edu/)
Mobitz Type II: http://lifeinthefastlane.com/author/edward-burns/
High Grade AV block: http://lifeinthefastlane.com/author/edward-burns/
Ventricular Escape rhythm: http://lifeinthefastlane.com/author/edwardburns/
Fixed ratio blocks: http://lifeinthefastlane.com/author/edward-burns/
46. SA exit block, Sinus Arrest, AV Blocks and Escape Rhythms
ECG EXERCISE
47.
48.
49.
50.
51.
52.
53.
54.
55.
56. Images
•
•
•
•
•
•
•
•
•
First Degree AV block: http://www.flickr.com/photos/popfossa/
4:1 AV block: http://www.flickr.com/photos/popfossa/
Mobitz type 1: http://www.flickr.com/photos/popfossa/
First Degree AV block: http://www.flickr.com/photos/popfossa/
Type II Sinoatrial exit block: http://lifeinthefastlane.com/author/edward-burns/
Mobitz type 1: http://lifeinthefastlane.com/author/edward-burns/
Complete Heart Block; http://lifeinthefastlane.com/author/edward-burns/
Sinus arrest: http://lifeinthefastlane.com/author/edward-burns/
High grade Av block and Mobitz Type 2:
http://lifeinthefastlane.com/author/edward-burns/
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