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ECG ConferenceECG Conference
Henry Tran, MDHenry Tran, MD
January 13, 2016January 13, 2016
Goals
 Review basic principles of EKG
 RHYTHM: Identify sinus vs atrial arrhythmias
 Recognize patterns of STEMI
 Recognize patterns of ischemia
 Distinguish Wide Complex Tachycardia: Ventricular
Tachycardia vs. Aberrant Conduction
Conduction System
ECG
Positioning Precordial Leads
12 Lead ECG
Axis
ECG Interpretation Methodology
 #1 Rhythm: what rhythm governs the atria
 Sinus or Not sinus
 AV Block
 Bundle Branch Conduction
 #2 Axis:
 Look for LAFB or Right Axis
 #3 Waveform Analysis
 #4 The Big Picture
Pt #1 Bruce Stage 2
P Waves: ? Sinus or Ectopic
SINUS P WAVE IS UPRIGHT IN LEAD I and LEAD II
Pt #1 Resting ECG prior to regular exercise stress test
Pt #1 Bruce Stage 1, 2:45 min
Pt 2
Pt 3
Typical Atrial Flutter
ECG Interpretation Methodology
 #1 Rhythm: what rhythm governs the atria
 Sinus or Not sinus
 AV Block
 Bundle Branch Conduction
 #2 Axis:
 Look for LAFB or Right Axis
 #3 Waveform Analysis
 #4 The Big Picture
Mobitz I Wenckebach
• Although it is possible to syncopize due
to Mobitz I, it is generally considered
benign and a normal variant
• If you see it at night during sleep, not an
emergency
Mobitz 2
Where’s the block?
Mobitz I
Mobitz II
Pt 3
Don’t miss this…
Conduction in AVRT
Orthodromic Antidromic
Vi / Vt
Bundle Branch Blocks
LAFP
•Axis >45o
•qR in I, AVL
LPFP
•Very rare
• >100o
V TACH!!!
Axis Deviation: Causes
NORMAL
Northwest LEFT
RIGHT
Lead
I
Lead aVF
•normal finding in children
and tall thin adults
•RVH
•COPD
•ASD/VSD
•PE
•Anterolateral MI
•left posterior hemiblock (>125 degree)
•left anterior hemiblock (>-45o
)
•Q waves of inferior MI
•WPW-Right Pathway
LVH DOES NOT CAUSE LEFT AXIS
Pt 6
WPW
Pt 7
Pt 8: 43 yo female complains of chest tightness
55 yo male POD #1 s/p laminectomy, remains
intubated
Don’t Miss This…
Don’t Miss These…
27 yo asian male complains of malignant syncope
NO TERMINAL S WAVE AS IN RBBB
Brugada Syndrome
• Channelopathy: Na+ SCN5A, L-Type calcium
• Increase epicardial dispersion of repolarization in the RV
44 yo male with schizophrenia and HCV complains of chest pain:
83 yo male hx of afib on coumadin brought to ER due to poor PO intake:
Accelerated junctional rhythms
33 yo male with no PMHX complains of chest pain x 3 hrs. Do you give kayexalate?
25 yo female complains of palpitations and dizziness. What imaging modality should
be ordered next?
Digoxin Toxicity LBBBLVH
GUESS THE REPOLARIZATION ABNORMALITY!
Summary
 What’s the rhythm?????
Thanks!

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Principles of ECG Interpretation

Editor's Notes

  1. Atrial Flutter
  2. Atrial Flutter
  3. Mobitz 1
  4. Mobitz II
  5. Antidromic AVRT Axis is normal, VI/VT is >1
  6. Ventricular Tachycardia
  7. Sinus with APCs, nl axis, LBBB
  8. WPW with LBBB, PVC is more narrow than sinus conducted beats!
  9. Left Anterior Fascicular block Note poor R wave progression,
  10. Incomplete RBBB, RV strain pattern Sinus tach PE!
  11. Inferior STEMI
  12. Brugada Type I “Coved” Pattern
  13. USE FLECAINIDE CHALLENGE (NA+ 1C)
  14. Prolonged QT
  15. Dig toxicity: Not short QT, scooped ST, afib, ventricular ectopy
  16. The increase in intracellular sodium causes an augmentation of the exchange of intracellular sodium for extracellular calcium. This shift can cause an increase in permeability to potassium; an increase in the outward current during the plateau phase, with a reduction in the duration of the action potential; and a short QT interval
  17. Hyperkalemia
  18. Hypercalcemia: 1) Digoxin toxicity 2) Hypercalcemia 3) Congenital Short QT Short QT < 300
  19. Hyperacute T wave with ST depession ->>STEMI
  20. Arvd
  21. Dig Toxicity:scooped” ST segment depression, most prominent in the inferolateral precordial leads and usually absent in the rightward leads; flattened T waves; increased U wave; and shortening of th QT interval