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DISEASES OF TONSILS AND
ADENOIDS
Dr. Junaid Shahzad
MBBS, FCPS
Senior Registrar
ENT department
ANMCH, Isra University
Islamabad Campus
Waldeyer's ring
Definition :
collection of lymphoid tissue scattered through out
the pharynx
Members of Waldeyer's ring are
1. Nasopharyngeal tonsil or the adenoids
2. Palatine tonsils or simply the tonsils
3. Lingual tonsil
4. Tubal tonsils ( fossa of rosenmuller)
• • Situated at the opening of the pharynx to the external
• environment, the tonsils and adenoid are in a position
• to provide primary defense against foreign matter.
• • produce lymphocytes
• • are active in the synthesis of immunoglobulins
• • Lymphoid tissue of Waldeyer ring is most
• immunologically active between 4 and 10 yr of age,
• with a decrease after puberty.
• • No major immunologic deficiency has been
• demonstrated after removal of either or both of the
• tonsils and adenoid.
Anatomy of tonsils
The word tonsil is derived from Latin word “Tonsilla”
meaning collection.
Types of tonsils
There are many tonsils in the body e.g.
Palatine Tonsil (faucial tonsils/Oropharyngeal tonsils)
Lingual Tonsil
Tubal Tonsil
Nasopharyngeal Tonsils (Adenoids)
Anatomy of tonsils
Location
lying in tonsillar/ Palatine fossa between Anterior &
posterior faucial pillars in the lateral wall of oropharynx.
Anterior Faucial pillar → Palato- glossus muscle
Posterior Faucial pillar → Palato- pharyngeus muscle
Blood supply of tonsil
Anatomy of tonsil
Lymphatic drainage
Jugulo-digastric (Tonsillar) nodes
Nerve supply Glossopharyngeal Nerve
Functions
a) Development of immunity in Childhood
b) Confine infection, its sampling & preventing its spread
c) Bread & butter for ENT surgeons
Acute tonsillitis :
Definition:
Acute infection of the Tonsils.
In many primary infection viral e.g adenovirus but bacteria are
Secondary invaders.
Disease of children: Age 5-6 years peak incidence
• Acute Infection
• • Symptoms :include odynophagia, dry throat,
• malaise, fever and chills, dysphagia, referred
• otalgia, headache, muscular aches, and enlarged
• cervical nodes.
• • Signs include dry tongue, erythematous enlarged
• tonsils, tonsillar or pharyngeal exudate, palatine
• petechiae, and enlargement and tenderness of
• the jugulodigastric lymph nodes
Types
• Acute catarrhal/superficial here tonsillitis is a part of
• generalized pharyngitis, mostly seen in viral infections
• • Acute follicular infection spread into the crypts with
• purulent material, presenting at the opening of crypts as
• yellow spots
• • Acute parenchymatous tonsil in uniformly enlarged
• and congested
• • Acute membranous follows stage of acute follicular
• tonsillitis where exudates coalesce to form membrane
• on the surface
Acute tonsillitis
Aetiology :
predisposing factors
A) Endogenous
local Pre- existing URTI
Ch. tonsillitis
Tonsillar remnant
General delibitating diseases
B) Exogenous
b) Over crowding
c) Poor nutrition
d) In adequate ventilation
e) Fatique and exposure to cold
Acute tonsillitis
II Exciting factors :
In many viruses are primary infective & bacteria secondary invaders where
as in others bact may be primary infective agent
Viruses :
Adenovirus, Resp syncyteal, Rhino viruses
Bacteria :
Streptococcus Haemolyticus 40%
Staph aureus, Pneumococi, H influenza
Anaerobes, Bacteriods
Pathology : Acute catarrhal
Ac follicular
Clinical features
Depends upon severity of infection & host resistance.
Local :
 Dryness in throat
 Pain in throat mild to severe
 Odynophagia
 Earache ……………… Otalgia
 Painful swelling in neck
Systemic :
 Fever mild to high accompanied with rigors
 Headache
 Pain & aches in body
Clinical features (Cont.)
Examination:
 Flushed face
 Mild Trismus
 Bad breath
 Tongue coated dirty white
 Tonsils-red, swollen, pus exudation may coalease
 J.D lymph nodes enlarged and tender
 Temp. raised
 Pulse rises in proportion to rise of temperature
(For each F◦ rise in temperature, pulse rises by
10 in number)
Investigations
1. Throat swab & culture sensitivity
2. TLC raised /DLC Neutrophils increased in number
Differential Diagnosis
Scarlet Fever
Diptheria
Vincent Angina
Thrush
Infectious Mononucleosis
Agranulocytosis
Keratosis of Tonsils
Treatment
Depends upon severity of infection.
Mild cases:
.Analgesics
.Warm salt water gargles
• Chronic Infection
• • Children with chronic or cryptic tonsillitis
• often present with halitosis, chronic sore
• throats, foreign body sensation, or a history of
• expelling foul-tasting and foul-smelling cheesy
• lumps.
• • Examination can reveal tonsils of almost any
• size and often they contain copious debris
• within the crypts
Severe cases
1. Bed rest
2. Analgesics and Antipyretics
3. Plenty of liquids especially warm
4. Antibiotics
Penicillin is a drug of the choice
Augmentin and 3rd generation cephalosporin are
very effective Antibiotic may be
changed according to c/s report
Complications
Local General
 Peritonsillar abscess
 Para-pharyngeal abscess
 Retro pharyngeal abscess
 Ac. laryngeal oedema
 Ac. Suppurative cervical
lymphadenitis
 Ac. Otitis media
 Tonsillar cyst
 Tonsillolith
 Ch. Tonsillitis
 Common with strep. Haemolyticus
 Ac. Glomerulonephrits
 Sub acute bacterio- endocarditis
 Rheumatic fever
 Exacerbation of Psoriasis
 Septicemia
 Myocarditis
Prognosis
8-10 days .
If not treated → High risk of complications.
Anatomy of Adenoids
(Nasopharyngeal tonsils)
Part of Waldeyer's ring
Surface epithelium — Ciliated columnar respiratory
epithelium
Location
Situated at the junction of roof &
Postero – superior wall
of Nasopharynx
Physiology of Adenoids
Adenoids tissue is present at birth
Clinically not visible up to 1 month of age
Detectable by 4 months of age
Physiological Hypertrophy : Between the ages of
2 to 8 years
Atrophy At puberty & almost completely disappears by
the age of 20 years, sometimes it may persist for a
longer period.
Blood supply of Adenoids
Ascending palatine branch of facial artery
Ascending pharyngeal branch of external carotid artery
Pharyngeal branch of the third part of maxillary artery
Ascending cervical branch of inferior thyroid artery of
thyrocervical trunk
Lymphatic drainage of Adenoids
1) Retro-pharyngeal lymph nodes
2) Para-pharyngeal lymph nodes
Adenoids conti. • Normally, in children.
• in the posterosuperior wall
of the Nasopharynx
• just above and behind
the Uvula
• usually absent in adults.
• lacking crypts.
• Pseudostratified Columnar
Ciliated Epithelium
• Moist cushion helps to filter
impurities out of the air
Microbiology:
Haemophilus influenzae
group A beta-hemolytic
Streptococcus
Staphylococcus aureus
Moraxella catarrhalis
and Streptococcus
Pneumonia
Signs & symptoms:
1) Nasal symptoms:
Nasal obstruction, Sinusitis
2) Aural symptoms:
Sec. Otitis media,
Conductive deafness
3) Oral / pharyngeal
symptoms
Mouth breathing, cough, PND,
Change in voice
4) General symptoms
Adenoid facies, Dull minded
cor-pulmonale
Adenoid Facies:
 Chronic nasal obstruction and mouth breathing due
to enlarged adenoids leads to typical appearance of
face called
“ Adenoid Facies”.
 Face is elongated
 Dull looking face
 Open mouth
 Prominent and crowded upper teeth
 High arched palate
 Hitched up upper lip
 Pinched nose (due to disuse atrophy of alae nasi)
DIAGNOSIS
History and clinical features
Post. Rhinoscopy
Flexible Naso-endoscopy
X-ray Post nasal space for soft tissues
Treatment:
 Medical treatment:
 Surgical treatment (Adenoidectomy)
Adenoid Curette
Position of Patient during Adenoidectomy
Adenoidectomy
Definition:
Removal of Adenoids
Indications
A) Adenoid hypertrophy causing recurrent rhino- sinusitis
B) Sleep Apnoea Syndrome
C) Adenoid with secretary Otitis media
D) Adenoid hypertrophy causing repeated attacks of ASOM
leading to CSOM
E) Abnormal dental occlusion
Adenoidectomy conti.
Contraindications:
A) Acute upper respiratory infection
B) Bleeding disorders
C) Cleft palate/Short palate/ Sub mucus Cleft palate
D) During Polio epidemics
Complications of Adenoidectomy
 Hemorrhage
 Eustachian tube injury
 Otitis media
 Regrowth of residual adenoid tissue
 Rhinolalia aperta
 Velopharyngeal insufficiency
 Nasopharyngeal stenosis
 Injury to Pharyngeal musculature and vertebrae
Complications IF NOT Treated
1) Sec . Otitis Media
2) Acute Suppurative Otitis Media
3) Chronic Suppurative Otitis Media

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Diseases of tonsils and adenoids

  • 1. DISEASES OF TONSILS AND ADENOIDS Dr. Junaid Shahzad MBBS, FCPS Senior Registrar ENT department ANMCH, Isra University Islamabad Campus
  • 2. Waldeyer's ring Definition : collection of lymphoid tissue scattered through out the pharynx Members of Waldeyer's ring are 1. Nasopharyngeal tonsil or the adenoids 2. Palatine tonsils or simply the tonsils 3. Lingual tonsil 4. Tubal tonsils ( fossa of rosenmuller)
  • 3.
  • 4. • • Situated at the opening of the pharynx to the external • environment, the tonsils and adenoid are in a position • to provide primary defense against foreign matter. • • produce lymphocytes • • are active in the synthesis of immunoglobulins • • Lymphoid tissue of Waldeyer ring is most • immunologically active between 4 and 10 yr of age, • with a decrease after puberty. • • No major immunologic deficiency has been • demonstrated after removal of either or both of the • tonsils and adenoid.
  • 5. Anatomy of tonsils The word tonsil is derived from Latin word “Tonsilla” meaning collection. Types of tonsils There are many tonsils in the body e.g. Palatine Tonsil (faucial tonsils/Oropharyngeal tonsils) Lingual Tonsil Tubal Tonsil Nasopharyngeal Tonsils (Adenoids)
  • 6.
  • 7.
  • 8. Anatomy of tonsils Location lying in tonsillar/ Palatine fossa between Anterior & posterior faucial pillars in the lateral wall of oropharynx. Anterior Faucial pillar → Palato- glossus muscle Posterior Faucial pillar → Palato- pharyngeus muscle
  • 10. Anatomy of tonsil Lymphatic drainage Jugulo-digastric (Tonsillar) nodes Nerve supply Glossopharyngeal Nerve Functions a) Development of immunity in Childhood b) Confine infection, its sampling & preventing its spread c) Bread & butter for ENT surgeons
  • 11. Acute tonsillitis : Definition: Acute infection of the Tonsils. In many primary infection viral e.g adenovirus but bacteria are Secondary invaders. Disease of children: Age 5-6 years peak incidence
  • 12. • Acute Infection • • Symptoms :include odynophagia, dry throat, • malaise, fever and chills, dysphagia, referred • otalgia, headache, muscular aches, and enlarged • cervical nodes. • • Signs include dry tongue, erythematous enlarged • tonsils, tonsillar or pharyngeal exudate, palatine • petechiae, and enlargement and tenderness of • the jugulodigastric lymph nodes
  • 13. Types • Acute catarrhal/superficial here tonsillitis is a part of • generalized pharyngitis, mostly seen in viral infections • • Acute follicular infection spread into the crypts with • purulent material, presenting at the opening of crypts as • yellow spots • • Acute parenchymatous tonsil in uniformly enlarged • and congested • • Acute membranous follows stage of acute follicular • tonsillitis where exudates coalesce to form membrane • on the surface
  • 14.
  • 15.
  • 16. Acute tonsillitis Aetiology : predisposing factors A) Endogenous local Pre- existing URTI Ch. tonsillitis Tonsillar remnant General delibitating diseases B) Exogenous b) Over crowding c) Poor nutrition d) In adequate ventilation e) Fatique and exposure to cold
  • 17. Acute tonsillitis II Exciting factors : In many viruses are primary infective & bacteria secondary invaders where as in others bact may be primary infective agent Viruses : Adenovirus, Resp syncyteal, Rhino viruses Bacteria : Streptococcus Haemolyticus 40% Staph aureus, Pneumococi, H influenza Anaerobes, Bacteriods Pathology : Acute catarrhal Ac follicular
  • 18. Clinical features Depends upon severity of infection & host resistance. Local :  Dryness in throat  Pain in throat mild to severe  Odynophagia  Earache ……………… Otalgia  Painful swelling in neck Systemic :  Fever mild to high accompanied with rigors  Headache  Pain & aches in body
  • 19. Clinical features (Cont.) Examination:  Flushed face  Mild Trismus  Bad breath  Tongue coated dirty white  Tonsils-red, swollen, pus exudation may coalease  J.D lymph nodes enlarged and tender  Temp. raised  Pulse rises in proportion to rise of temperature (For each F◦ rise in temperature, pulse rises by 10 in number)
  • 20. Investigations 1. Throat swab & culture sensitivity 2. TLC raised /DLC Neutrophils increased in number
  • 21. Differential Diagnosis Scarlet Fever Diptheria Vincent Angina Thrush Infectious Mononucleosis Agranulocytosis Keratosis of Tonsils
  • 22. Treatment Depends upon severity of infection. Mild cases: .Analgesics .Warm salt water gargles
  • 23.
  • 24. • Chronic Infection • • Children with chronic or cryptic tonsillitis • often present with halitosis, chronic sore • throats, foreign body sensation, or a history of • expelling foul-tasting and foul-smelling cheesy • lumps. • • Examination can reveal tonsils of almost any • size and often they contain copious debris • within the crypts
  • 25. Severe cases 1. Bed rest 2. Analgesics and Antipyretics 3. Plenty of liquids especially warm 4. Antibiotics Penicillin is a drug of the choice Augmentin and 3rd generation cephalosporin are very effective Antibiotic may be changed according to c/s report
  • 26. Complications Local General  Peritonsillar abscess  Para-pharyngeal abscess  Retro pharyngeal abscess  Ac. laryngeal oedema  Ac. Suppurative cervical lymphadenitis  Ac. Otitis media  Tonsillar cyst  Tonsillolith  Ch. Tonsillitis  Common with strep. Haemolyticus  Ac. Glomerulonephrits  Sub acute bacterio- endocarditis  Rheumatic fever  Exacerbation of Psoriasis  Septicemia  Myocarditis
  • 27. Prognosis 8-10 days . If not treated → High risk of complications.
  • 28. Anatomy of Adenoids (Nasopharyngeal tonsils) Part of Waldeyer's ring Surface epithelium — Ciliated columnar respiratory epithelium Location Situated at the junction of roof & Postero – superior wall of Nasopharynx
  • 29. Physiology of Adenoids Adenoids tissue is present at birth Clinically not visible up to 1 month of age Detectable by 4 months of age Physiological Hypertrophy : Between the ages of 2 to 8 years Atrophy At puberty & almost completely disappears by the age of 20 years, sometimes it may persist for a longer period.
  • 30. Blood supply of Adenoids Ascending palatine branch of facial artery Ascending pharyngeal branch of external carotid artery Pharyngeal branch of the third part of maxillary artery Ascending cervical branch of inferior thyroid artery of thyrocervical trunk
  • 31. Lymphatic drainage of Adenoids 1) Retro-pharyngeal lymph nodes 2) Para-pharyngeal lymph nodes
  • 32.
  • 33. Adenoids conti. • Normally, in children. • in the posterosuperior wall of the Nasopharynx • just above and behind the Uvula • usually absent in adults. • lacking crypts. • Pseudostratified Columnar Ciliated Epithelium • Moist cushion helps to filter impurities out of the air
  • 34. Microbiology: Haemophilus influenzae group A beta-hemolytic Streptococcus Staphylococcus aureus Moraxella catarrhalis and Streptococcus Pneumonia
  • 35. Signs & symptoms: 1) Nasal symptoms: Nasal obstruction, Sinusitis 2) Aural symptoms: Sec. Otitis media, Conductive deafness 3) Oral / pharyngeal symptoms Mouth breathing, cough, PND, Change in voice 4) General symptoms Adenoid facies, Dull minded cor-pulmonale
  • 36. Adenoid Facies:  Chronic nasal obstruction and mouth breathing due to enlarged adenoids leads to typical appearance of face called “ Adenoid Facies”.  Face is elongated  Dull looking face  Open mouth  Prominent and crowded upper teeth  High arched palate  Hitched up upper lip  Pinched nose (due to disuse atrophy of alae nasi)
  • 37. DIAGNOSIS History and clinical features Post. Rhinoscopy Flexible Naso-endoscopy X-ray Post nasal space for soft tissues
  • 38.
  • 39. Treatment:  Medical treatment:  Surgical treatment (Adenoidectomy)
  • 41. Position of Patient during Adenoidectomy
  • 42. Adenoidectomy Definition: Removal of Adenoids Indications A) Adenoid hypertrophy causing recurrent rhino- sinusitis B) Sleep Apnoea Syndrome C) Adenoid with secretary Otitis media D) Adenoid hypertrophy causing repeated attacks of ASOM leading to CSOM E) Abnormal dental occlusion
  • 43. Adenoidectomy conti. Contraindications: A) Acute upper respiratory infection B) Bleeding disorders C) Cleft palate/Short palate/ Sub mucus Cleft palate D) During Polio epidemics
  • 44. Complications of Adenoidectomy  Hemorrhage  Eustachian tube injury  Otitis media  Regrowth of residual adenoid tissue  Rhinolalia aperta  Velopharyngeal insufficiency  Nasopharyngeal stenosis  Injury to Pharyngeal musculature and vertebrae
  • 45. Complications IF NOT Treated 1) Sec . Otitis Media 2) Acute Suppurative Otitis Media 3) Chronic Suppurative Otitis Media