Astigmatism is a refractive error where the eye focuses light unevenly due to an irregularly shaped cornea or lens. It causes blurred vision and difficulty seeing fine details. There are several types of astigmatism classified by its cause, axis, degree of refractive error in different meridians, and location of the focal points. Common symptoms include blurred vision, eye strain, headaches, and occasional tilting of the head in severe oblique astigmatism. Diagnosis involves an eye exam using a retinoscope or autorefractor to detect the different refractive errors in different meridians. Treatment options include eyeglasses, contact lenses, or refractive surgery to correct the astigmatism.
Optics of Retinoscope by Dr. Muhammad Zeeshan Hameed.pptxZeeshan Hameed
Includes
1. What is a Retinoscope?
2. Short History of Retinoscope
3. Parts of a Retinoscope
4. Detailed Optics of a Retinoscope
5. Practical Points of Retinoscopy
Optics of Retinoscope by Dr. Muhammad Zeeshan Hameed.pptxZeeshan Hameed
Includes
1. What is a Retinoscope?
2. Short History of Retinoscope
3. Parts of a Retinoscope
4. Detailed Optics of a Retinoscope
5. Practical Points of Retinoscopy
In this Presentation we learn about :-
1.What is Subjective Refraction.
2.Why we should relax the accommodation.
3.Outlines of Subjective Refraction.
4.Different Techniques or Instruments.
5.Determining Near Addition.
6.The Final Prescription.
7.References.
In this Presentation we learn about :-
1.What is Subjective Refraction.
2.Why we should relax the accommodation.
3.Outlines of Subjective Refraction.
4.Different Techniques or Instruments.
5.Determining Near Addition.
6.The Final Prescription.
7.References.
The basic concepts about refractive errors and their corrective options are explained in this lecture. It was taken at Central Park Medical College Lahore Pakistan for fourth year medical students
Topic:- Astigmatic error
This presentation only explained about Definition, types, classification of astigmatism, It did not explained about correction.
Real subjective refraction in astigmatismBipin Koirala
hope it will be beneficial for the students in eye care system . please like it and share it if you think it is beneficial for your studies. It will motivate me to upload more slides ..
When parallel rays of light enter the eye ((with accommodation relaxed) and do) and do not come to a single point focus on or near the retina.
Types of Astigmatism:
Sign & Symptoms:
Management:
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These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
4. CONTENTS OF TODAY’S LECTURE
What is Astigmatism?
Incidence
Optics
Etiology
Classification
Signs & Symptoms
Diagnosis
Treatment
5. ASTIGMATISM
Astigmatism is a refractive error of the eye in which there
is a difference in degree of refraction in different meridians
(i.e. the eye has different focal points in different planes.)
For example, the image may be clearly focused on the
retina in the horizontal (sagittal) plane, but not on the
retina in the vertical (tangential) plane.
Astigmatism causes difficulties in seeing fine detail, and in
some cases vertical lines (e.g., walls) may appear to the
patient to be leaning over.
6.
7.
8.
9. WHAT IS ASTIGMATISM?
Most astigmatic corneas have two curves, a steeper
curve and a flatter curve. This causes light to focus on
more than one point in the eye, resulting in
blurred vision.
The cornea is normally spherical, although in people with
astigmatism, it may be shaped like a rugby ball or oval
instead of tennis ball.
13. OPTICS OF ASTIGMATISM
In astigmatism, the rays of light from one sector
fall on one point & rays from another sector fall on
another point. In other words, a point focus of
light cannot be formed upon the retina.
The configuration of rays refracted through the
astigmatic surface (toric surface) is called sturms
conoid.
14.
15. ASTIGMATISM
Vertical Focal
Line
Circle of Least C
Confusion
Horizontal
Focal Line
Power Meridian A
Axis Meridian B D
Object
Source
Interval
of
Sturm
16. OPTICS OF ASTIGMATISM
Thus, there are 2 focal points separated from
each other by a focal interval, called as interval
of sturm.
The length of this focal interval is the measure
of the degree of astigmatism & the correction of
the error can only be accomplished by reducing
these two foci in to one.
18. INCIDENCE
No eye is perfectly stigmatic as almost all individuals have a
minor degree of physiological astigmatism.
About 60% cases of refractive errors have astigmatism which
needs to be corrected.
Occurs with equal frequency in males and females.
Approximate distribution according to degree of astigmatism
is:
0.25-0.5 D 50%
0.75-1.0 D 25%
1.00-4.00D 24%
>4.00 1.0%
19. INCIDENCE
The most common type is compound myopic
followed by compound hyperopic, mixed, simple
myopic & simple hyperopic.
One study reports as:
With the rule 38%
Against the rule 30%
Oblique 32%
21. ETIOLOGY
1.Corneal astigmatism:
It occurs due to abnormalities of curvature of cornea
Most common cause of astigmatism
e.g. keratoconus, pterygium, mild corneal opacities, chalazion
29. ETIOLOGY
2. Lenticular astigmatism:
It is comparatively rare.
It may be:
Curvatural….lenticonus
Positional…..congenital tilting & traumatic subluxation of lens
Index……….developing cataract/nuclear sclerosis/diabetic cat.
34. Severity of Astigmatism
The severity of astigmatism can be classified as
follows:
Mild Astigmatism < 1.00 diopter
Moderate Astigmatism 1.00 to 2.00 diopters
Severe Astigmatism 2.00 to 3.00 diopters
Extreme Astigmatism > 3.00 diopters
37. CLASSIFICATION
1. Astigmatism - Based on asymmetry of structure
• Corneal astigmatism - astigmatism due to an irregularly shaped
cornea
• Lenticular astigmatism - astigmatism due to an irregularly shaped
lens
38. CLASSIFICATION
2. Astigmatism - Based on axis of the principal meridians
a. Regular astigmatism
Against-the-rule astigmatism
With-the-rule astigmatism
b. Oblique astigmatism
c. Bioblique astigmatism
d. Irregular astigmatism
39. a. Regular astigmatism:
The astigmatism is said to be regular if there is different
refraction by the eye in two meridia at right angles to each
other.
Can be corrected with spectacles.
Normally, horizontal curvature of cornea is flatter than
vertical & this is attributed to the pressure of lids on the
corneal surface. This is physiological. So, vertical cornea
should be more curve than horizontal.
On this basis, it has two types:
With the rule (WTR) & against the rule (ATR)
40.
41. With-the-rule (direct astigmatism):
Principle meridia are at right angle to each other.
Vertical curve is more than horizontal.
Concave cylinder is prescribed in horizontal axis
(180) and convex are prescribed in vertical axis
(90).
Normally the vertical meridian is rendered 0.25 D
more convex than horizontal by the pressure of
fleshy upper eyelid.
42. Against-the-rule (indirect astigmatism):
Principle meridia are at right angle to each other.
Horizontal curve is more than Vertical.
Convex cylinder is prescribed in horizontal axis
(180) and concave are prescribed in vertical axis
(90).
Usually associated with old age.
43. b. Oblique astigmatism:
A type of astigmatism in which principle meridia are
not horizontal or vertical but are at right angle to
each other (45 & 135).
Usually symmetrical in both the eyes (cylinder
required at 30 in both the eyes)
Or complementary (cylinder required at 30 in one
eye & 150 in other eye)
44.
45. c. Bioblique astigmatism:
In this type of astigmatism, the two principle
meridia are not at right angle to each other.
e.g. one may be at 30 & other at 100.
46. d. Irregular astigmatism:
o It is characterized by an irregular change of
refractive power in different meridia.
o There are multiple meridia which admit no
geometrical analysis.
o Cannot be corrected by spectacles.
o It occurs due to corneal scars, during maturation
of cataract, etc.
47. CLASSIFICATION
3. Astigmatism - Based on focus of the principal
meridians
Simple astigmatism
Simple hyperopic astigmatism
Simple myopic astigmatism
Compound astigmatism
Compound hyperopic astigmatism
Compound myopic astigmatism
Mixed astigmatism
48. SIMPLE ASTIGMATISM
In simple astigmatism, one of the foci falls on retina &
other focus falls in front or behind retina.
This leads to one meridian being emmetropic & other being
myopic (one focus on the retina & other focus falls in front of retina) or
hyperopic (one focus on retina & other focus behind retina), so called
as simple myopic astigmatism & simple hyperopic
astigmatism respectively.
It can be with-the-rule or against-the-rule.
-2 D cyl at 90 is example of simple myopic astigmatism.
+2 D cyl at 90 is example of simple hyperopic astigmatism.
51. COMPOUND ASTIGMATISM
• Neither of the two foci fall on the retina.
• The condition is known as compound hyperopic
if both foci are at back of retina.
• The condition is known as compound myopic if
both foci are at front of retina.
• It can be with-the-rule or against-the-rule.
• -3 DS with -2DC at 90 is example of compound
myopic astigmatism.
• +3 DS with +2DC at 90 is example of compound
hyperopic astigmatism.
54. MIXED ASTIGMATISM
In mixed astigmatism, one of the two foci
lies at back while other at front of the retina.
It can be with-the-rule or against-the-rule.
-3 DS with +8DC at 90 is an example of
mixed astigmatism.
57. NOTE:
If cyl power is less than spherical power, then it is
not mixed but compound astigmatism.
For example, -3DS with +1DC at 180 sounds as if it
is mixed astigmatism, but actually is compound
astigmatism, as cyl is less than sphere.
58. RESIDUAL ASTIGMATISM
The largest element of the total astigmatism is due to
anterior corneal surface.
While the other components like:
Posterior corneal surface
Lens
Refractive indices
constitute the residual astigmatism.
RESIDUAL ASTIGMATISM= TOTAL – CORNEAL ASTIGMATISM
60. SIGNS & SYMPTOMS
Type of the symptoms produced, depends upon the type
of astigmatism:
1. Blurring of vision:
Transient blurring of vision in low astigmatism.
Relieved by closing/rubbing the eyes.
Circles elongate into ovals.
A point of light appears tailed off.
A line appears as a succession of strokes fused into a
blurred image.
61.
62.
63.
64. SIGNS & SYMPTOMS
2. Asthenopic symptoms:
More marked in patients with low astigmatism
(more accommodative effort)
Severe in hyperopic astigmatism
(more accommodative effort)
Tiredness of eyes
Headaches (from mild frontal ache to explosions of
pain)
Nervous disturbances:
Dizziness
Fatigue
Irritability
65. SIGNS & SYMPTOMS
3. Tilting of the head:
Some patients with high oblique astigmatism, may
hold the head tilted to one side to reduce image
distortion.
Some children may even develop scoliosis.
(The condition of side-to-side spinal curves is called scoliosis. On an X-
ray, the spine of an individual with scoliosis looks more like an "S" or a
"C" than a straight line.)
66. SIGNS & SYMPTOMS
4. Half closure of the lids:
Seen in patients with high astigmatism.
This is to make a sort of stenopaeic slit & cutting
out the rays from one meridian..
This also causes Asthenopic symptoms.
67. SIGNS & SYMPTOMS
5. Reading material is held too close:
Reading material is held too close to the eyes by
the patient to achieve blur but large image just
like a myope.
68. SIGNS & SYMPTOMS
6. Burning & itching:
May be seen in patients with low astigmatism
B/c of rubbing the eyes
Falling of eye lashes
Hyperemia
Styes& chalazia
70. DIAGNOSIS
VA with and without correction monocularly
Pinhole VA
Retinoscopy
Keratometry
Keratoscopy with placido’s disc
Computerised corneal topography/videograph
Subjective verification:
Jackson cross cylinder
Astigmatic fan & block
Trial & error technique (axis then power)
Maddox V
Stenopaeic slit
78. ASTIGMATISM TEST
Close one eye and then the other one , if you do not see all
the lined squares, in the same black color , if you do see
.
one or more squares grey, you then have an astigmatism.
84. The typical spiral pattern of keratoconus progression. In
color-coded topographic images, red represents steeper
corneal curvature, and the spectrum of yellow, green,
and blue represents progressively flatter curvatures.
87. SPECTACLES
Astigmatism is corrected optically with a cylindrical
lens.
A combination of a spherical lens and a cylindrical
lens (spherocylindrical lens) is used to correct a
spherical error with an astigmatic error.
Cyl has power (curvature) in one meridian and no
power in the other meridian.
The axis of the cylinder is lined up with the axis of
astigmatism to correct the astigmatic power
difference.
93. CONTACT LENSES
Various types of contact lenses are used:
Soft
Hard
Rigid gas permeable
Hybrid (hard center & soft periphery, used in
keratoconus)
Depending upon the degree of astigmatism:
Spherical
Toric
Bitoric
94.
95. Eugene Kalt, MD, first to propose the use of a contact
lens for keratoconus.
99. REFERENCES
Theory and practice of optics and refraction by
A K Khurana
Duke Elder's Practice of refraction (Tenth
edition)
Clinical Optics by Elkington, Frank and
Greaney (Third edition)
Text book of ophthalmology (Volume : 1) by
Jaypee publishers
and many websites.
Editor's Notes
Astigmatism This diagram represents the imaging of an astigmatic system (convex sphero-cylindrical lens or an astigmatic eye). Below the optical representation are the cross-sections that would be found if a screen was placed perpendicular to the optical axis at the positions shown. The line foci, ellipses and their orientations are shown, along with the circle of least confusion.
The figure shows the imaging of a positive spherical lens, of radius of curvature r s . The principal meridia are seen at 90 and 180 . The principal meridia of a lens are always positioned at right angles to each other, whatever the orientation of the lens in front of the eye. The power of a spherical lens is related to the radius of curvature of the lens surfaces and the material from which the lens is made. At each surface the power can be calculated as follows: F = (n’-n) / r where n’ is the refractive index of the media that the incident light is entering into and n is the refractive index of the media that the lens is coming from. Often the lens is in air, so the power equation at the front surface would read: F 1 =(n’ - 1) / r 1 where F 1 is the power of the front surface, n’ is the refractive index of the lens material and r 1 is the radius of curvature of the front surface. The power equation at the back surface of the lens would read as follows: F 2 = (1-n’) / r 2 where F 2 is the power of the back surface of the lens and r 2 is its radius of curvature.
The figure shows a cylindrical lens. Light incident normal to the lens surface will pass through undeviated. Light incident towards the edge of the lens will meet a curved surface and will cone to a point focus along the optic axis. If light incident on the upper portion of the lens is considered, it can be seen that rays grazing the edge of the lens will also be refracted to a single point focus, but the point focus will not be coincident with the point focus on the optic axis. This is due to there being no condensing power in the vertical plane of the lens. If any series of rays between those illustrated were traced, a series of point foci would be seen to fall between the two extremes drawn forming a line image for the point object.
The dioptric separation of the line foci is known as the Interval of Sturm and this distance should be equivalent to any axial astigmatism that the cylindrical lens is to be used to correct.