Astigmatism occurs when the cornea is not uniformly curved, causing light rays to not converge at a single focal point and resulting in blurred vision. It is most often caused by an uneven curvature of the cornea (corneal astigmatism). Symptoms include distorted or blurred vision at all distances. Astigmatism is commonly measured using a keratometer and can be corrected using spectacle lenses, contact lenses, or refractive surgery. Prescribing cylinders for spectacle correction requires determining the axis of astigmatism and cylinder power through retinoscopy techniques.
Accommodation/ Accommodation of Eye, Measurement of Accommodation of Eye (hea...Bikash Sapkota
CLICK HERE TO DOWNLOAD FULL PPT ❤❤ https://healthkura.com/measurement-of-accommodation-of-eye/ ❤❤
Dear viewers Check Out my other piece of works at ❤❤❤ https://healthkura.com ❤❤❤
Measurement of Accommodation of eye:
Amplitude, Facility,
Relative Accommodation, Fatigue, Lag,
Dynamic Retinoscopy
Presentation Layout:
-Introduction to accommodation of eye
-Mechanism
-Components
-Measurement of accommodation of eye
- Amplitude
- Facility
- Relative accommodation
- Lag
-Dynamic Retinoscopy
Accommodation
-dioptric adjustment of the crystalline lens of the eye
- to obtain clear vision for a given target of regard
-process by which the refractive power of eye is altered
- to ensure a clear retinal image
For further reading
-Clinical Procedures in Optometry by J.D. Bartlett, J.B. Eskridge, J.F. Amos
-Primary Care Optometry by Theodere Grosvenor
-Borish’s Clinical Refraction by W.J. Benjamin
-Clinical Procedures for Ocular examination by Carlson et al
-American Academy of Ophthalmology
-Optometric Clinical Practice Guideline by American Optometric Association
-Internet
Follow me to get in touch with optometric and ophthalmic updates
Accommodation/ Accommodation of Eye, Measurement of Accommodation of Eye (hea...Bikash Sapkota
CLICK HERE TO DOWNLOAD FULL PPT ❤❤ https://healthkura.com/measurement-of-accommodation-of-eye/ ❤❤
Dear viewers Check Out my other piece of works at ❤❤❤ https://healthkura.com ❤❤❤
Measurement of Accommodation of eye:
Amplitude, Facility,
Relative Accommodation, Fatigue, Lag,
Dynamic Retinoscopy
Presentation Layout:
-Introduction to accommodation of eye
-Mechanism
-Components
-Measurement of accommodation of eye
- Amplitude
- Facility
- Relative accommodation
- Lag
-Dynamic Retinoscopy
Accommodation
-dioptric adjustment of the crystalline lens of the eye
- to obtain clear vision for a given target of regard
-process by which the refractive power of eye is altered
- to ensure a clear retinal image
For further reading
-Clinical Procedures in Optometry by J.D. Bartlett, J.B. Eskridge, J.F. Amos
-Primary Care Optometry by Theodere Grosvenor
-Borish’s Clinical Refraction by W.J. Benjamin
-Clinical Procedures for Ocular examination by Carlson et al
-American Academy of Ophthalmology
-Optometric Clinical Practice Guideline by American Optometric Association
-Internet
Follow me to get in touch with optometric and ophthalmic updates
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
Follow us on: Pinterest
Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
2. Definition
Astigmatism (from the Greek “a” meaning
absence and “stigma” meaning point)
Astigmatism occurs when incident light rays
do not converge at a single focal
point.The cornea of the normal eye has a
uniform curvature, with resulting equal
refracting
power over its entire surface.
Most astigmatic corneas are normal also.
3. In some individuals, however, the cornea is not
uniform and the curvature is greater in one
meridian (plane) than another, much like a
football as a rugby ball.
6. Most astigmatism is corneal in origin.
Lenticular astigmatism is a result of uneven
curvature and differing refractive indices
within the crystalline lens.
internal or non-corneal
astigmatism was found to be -0.46X98.2° for
right eyes and -0.50X99.4° for left eyes.
7. 1.2 Epidemiology - prevalence
Astigmatism (more than 0.5 diopters) is a
commonly encountered refractive error,
accounting for about 13 per cent of the refractive
errors of the human eye.
Human infants exhibit both high prevalence and
high degrees of astigmatism, largely corneal in
origin
In adult Americans, the prevalence of
astigmatism has been reported to be 20% higher
among men than women but was not associated
with number of years of formal education
8. astigmatism types, classification
Ocular astigmatism can occur as a result of
unequal curvature along the two principal
meridian of the anterior cornea (known as
corneal astigmatism)
9. unequal curvatures of the front and back
surfaces of the crystalline lens,
decentration or tilting of the lens or unequal
refractive indices across the crystalline lens
(known as internal or non-corneal
astigmatism)
10. Corneal astigmatism is often classified
according to the axis of astigmatism as being
either with-the-rule (WTR),
oblique or against-the-rule (ATR)
.
11. The principal meridians-the meridians of
maximum and
minimum corneal curvature-are usually at
right angles to each other in astigmatism
and are
usually (but not necessarily) in the
vertical and horizontal planes.
12. Astigmatism can be described as regular or
irregular.
In regular astigmatism, there are two principal
meridians separated by 90 degrees; the best
spectacle-corrected visual acuity (BSCVA) is at
least 20/20 and, in the case of corneal
astigmatism, corneal topography displays a
symmetrical bow—tie pattern.
13. In regular astigmatism, the refractive power
varies successively from one meridian to the
next, and each meridian has a uniform
curvature at every point across the entrance
pupil.
14. Types of regular astigmatism
Astigmatic eyes:
Simple myopic astigmatism: one meridian focuses light
in front of the retina, the other on
the retina;
Simple hyperopic astigmatism: one meridian focuses
light on the retina, the other
theoretically behind the retina;
Compound myopic astigmatism: both meridians focus
light in front of the retina;
Compound hyperopic astigmatism: both meridians focus
light theoretically behind the
retina;
Mixed astigmatism: one meridian focuses light in front of
the retina, the other behind the
retina.
15. In irregular astigmatism, which is less common, the
corneal “rugby ball” would appear out
of its customary shape and/or bumpy.
Irregular astigmatism can be regularly irregular or
irregularly irregular
In regularly irregular astigmatism, two principal
meridians exist but are either asymmetrical or not
90 degrees apart and is typified by either unequal
slopes of the hemi meridians along a single meridian
(the “asymmetric bow-tie”) or hemimeridians of
equal slope but not aligned with
each other (the “angled bow-tie” or nonorthogonal
astigmatism).
16.
17. Irregularly irregular astigmatism does not have
identifiable prime meridians. In irregular astigmatism,
which can be clinically significant in
conditions such as keratoconus and other corneal
ectasias; corneal basement membrane and
stromal dystrophies; corneal scarring; and post-surgical
corneas (e.g., following penetrating keratoplasty, radial
keratotomy, and complicated refractive surgery), the
magnitude and the
axis of astigmatism vary from point to point across the
entrance pupil
18. Symptoms
Distortion or blurring of images at all distances is
one of the most common astigmatism
symptoms.This may happen vertically,
horizontally, or diagonally.
There can be
indistinctness of objects, circles become elongated
into ovals and a point of light begins to
tail off. Symptoms of eye strain such as headaches ,
photophobia, and fatigue are also among the most
common astigmatism symptoms.
Reading small print is difficult with astigmatism.
19.
20. Keratometric
Performed with a device called keratometer
or ophthalmometer, keratometry is the
measurement of a patient’s corneal
curvature.As such, it provides an objective,
quantitative measurement of corneal
astigmatism, measuring the curvature in each
meridian as well as the axis.
21. Non-surgical treatment of astigmatism
Retinoscopy of Regular Astigmatism
Most eyes have some regular astigmatism. In such cases,
light is refracted differently by the 2 principal astigmatic
meridians.
Sweeping the retinoscope back and forth measures the
power along only a single axis.
Moving the retinoscope from side to side (with the streak
oriented at 90°) measures the optical power in the 180°
meridian. Power in this meridian is provided by a cylinder
at the 90° axis.
22. Finding the cylinder axis
Before the powers in each of the principal
meridians can be determined, the axes of the
meridians must be determined. Four
characteristics of the streak reflex aid in this
determination:
1) Break. A break is observed when the streak is
not oriented parallel to 1 of the principal
meridians.The reflex streak in the pupil is not
aligned with the streak projected on the iris
and surface of the eye, and the line appears
broken.
23. The break disappears (ie, the line
appears continuous) when the projected
streak
is rotated to the correct axis.
24. Width.The width of the reflex in the pupil
varies as it is rotated around the correct
axis.The reflex appears narrowest when the
streak, or intercept, aligns with the axis.
25.
26. Intensity.The intensity of the line is brighter
when the streak is on the correct axis.
Skew. Skew (oblique motion of the streak
reflex) may be used to refine the axis
in small cylinders. If the retinoscope streak is
off-axis, it moves in a slightly different
direction from that of the pupillary reflex.
27. The reflex and streak move in the same direction
when the streak is aligned with 1 of the
principal meridians.
28. When the streak is aligned at the correct axis,
the sleeve may be lowered (Copeland
instrument)
or raised (Welch Allyn instrument) to narrow
the streak, allowing the axis to be
determined more easily
29. The axis can be confirmed through a
technique known as straddling, which is
performed with the estimated correcting
cylinder in place
The retinoscope streak is turned 45° off-axis
in both directions, and if the axis is correct,
the width of the reflex should be equal in
both off-axis positions.
30. If the axis is not correct, the
widths are unequal in these 2 positions.
The axis of the correcting plus-cylinder
should be moved toward the narrower reflex
and the straddling repeated until the widths
are equal.
This technique is often more accurate than
subjective cross-cylinder axis refinement.
31.
32. Finding the cylinder power
After the 2 principal meridians are identified,
the previously explained spherical techniques
are applied to each axis:
With 2 spheres. Neutralize 1 axis with a
spherical lens; then neutralize the axis 90°
away..
33. The difference between these readings is the
cylinder power.
For example, if the 90° axis is neutralized
with a+ 1.50 sphere and the 180° axis is
neutralized with a +2.25 sphere, the gross
retinoscopy is+ 1.50 +0.75 x 180
34. The examiner's working distance (ie, + 1.50) is
subtracted from the sphere to obtain the final
refractive correction: plano +0.75 x 180.
With a sphere and cylinder. Neutralize 1 axis with
a spherical lens.To enable the
use of with reflexes, neutralize the less plus axis
first.Then, with this spherical
lens in place, neutralize the axis 90° away by
adding a plus cylindrical lens.The
spherocylindrical gross retinoscopy is read
directly from the trial lens apparatus.
35. Common Guidelines for Prescribing
Cylinders for Spectacle Correction
1. In children, give the full astigmatic correction.
2. In adults, try the full astigmatic correction first.
Give warning and encouragement.
If problems are anticipated, try a walking-around
trial with trial frames before
prescribing.
3.To minimize distortion, use minus cylinder
lenses and minimize vertex distances.
4. Spatial distortion from astigmatic spectacle
corrections is a binocular phenomenon.
Occlude 1 eye to verify that this is indeed the
cause of the patient's complaints.
36. 5. If necessary, reduce distortion still further by
rotating the cylinder axis toward
180° or 90° (or toward the old axis) and/or by
reducing the cylinder power. Balance
the resulting blur with the remaining distortion,
using careful adjustment of
cylinder power and sphere. Residual astigmatism
at any position of the cylinder
axis may be minimized with the Jackson cross-
cylinder test for cylinder power.
Adjust the sphere using the spherical equivalent
concept as a guide, but rely on a
final subjective check to obtain best visual acuity.
37. 6. If distortion cannot be reduced sufficiently
by altering the astigmatic spectacle
correction, consider contact lenses (which
cause no appreciable distortion) or iseikonic
corrections.
38. Refrence
American academy clinical optics
Duke-elder practise of refraction
Elkington
Google images