Congenital heart defects are abnormalities in the structure of the heart that are present at birth. They affect approximately 9 in 1000 births and can cause obstructed or abnormal blood flow. Common types include septal defects, where the walls separating the left and right sides of the heart have holes, and obstructive defects, where arteries or valves are blocked. Congenital heart defects range from mild to complex and may require medication, surgery, or both for treatment depending on severity. Their causes are usually a combination of genetic and environmental factors.
Congenital heart disease is one or more problems with the heart's structure that exist since birth. Congenital means that you're born with the defect. Congenital heart disease, also called congenital heart defect, can change the way blood flows through your heart. IF YOU LIKE GIVE YOUR LIKES AND FOLLOW THIS LINK
Congenital heart disease is one or more problems with the heart's structure that exist since birth. Congenital means that you're born with the defect. Congenital heart disease, also called congenital heart defect, can change the way blood flows through your heart. IF YOU LIKE GIVE YOUR LIKES AND FOLLOW THIS LINK
The lecture is for medical student. It is from Dr RUSINGIZA Emmanuel, MD, senior lecture at UR( UNIVERSITY OF RWANDA) .
It will help to understand heart diseases in newborn, infants and children.
Aortic insufficiency (AI), also known as aortic regurgitation (AR), is the leaking of the aortic valve of the heart that causes blood to flow in the reverse direction
Heart failure, sometimes known as congestive heart failure, occurs when your heart muscle doesn't pump blood as well as it should. Certain conditions, such as narrowed arteries in your heart (coronary artery disease) or high blood pressure, gradually leave your heart too weak or stiff to fill and pump efficiently.
commonly used for medical students, and helpful to use this ppt to study for them, and also a common man can understand very easily what is coarctation of aorta.
Kindly leave your comment if you found this helpful ;)
Some of the slides, i hide it from my real presentations for my own reference. Download to see all of them.
Austin Journal of Clinical Cardiology is an open access, peer reviewed, scholarly journal dedicated to publish articles in all areas of cardiology and angiology. The aim of the journal is to provide a forum for cardiologists, researchers, physicians, and other health professionals to find most recent advances in the areas of cardiology and cardiovascular diseases.
Austin Journal of Clinical Cardiology accepts original research articles, review articles, case reports, clinical images and rapid communication on all the aspects of cardiology and circulatory system.
Austin Journal of Clinical Cardiology strongly supports the scientific upgradation and fortification in related scientific research community by enhancing access to peer reviewed scientific literary works. Austin Publishing Group also brings universally peer reviewed journals under one roof thereby promoting knowledge sharing, mutual promotion of multidisciplinary science.
Austin Journal of Clinical Cardiology is an open access, peer reviewed, scholarly journal dedicated to publish articles in all areas of cardiology and angiology
The lecture is for medical student. It is from Dr RUSINGIZA Emmanuel, MD, senior lecture at UR( UNIVERSITY OF RWANDA) .
It will help to understand heart diseases in newborn, infants and children.
Aortic insufficiency (AI), also known as aortic regurgitation (AR), is the leaking of the aortic valve of the heart that causes blood to flow in the reverse direction
Heart failure, sometimes known as congestive heart failure, occurs when your heart muscle doesn't pump blood as well as it should. Certain conditions, such as narrowed arteries in your heart (coronary artery disease) or high blood pressure, gradually leave your heart too weak or stiff to fill and pump efficiently.
commonly used for medical students, and helpful to use this ppt to study for them, and also a common man can understand very easily what is coarctation of aorta.
Kindly leave your comment if you found this helpful ;)
Some of the slides, i hide it from my real presentations for my own reference. Download to see all of them.
Austin Journal of Clinical Cardiology is an open access, peer reviewed, scholarly journal dedicated to publish articles in all areas of cardiology and angiology. The aim of the journal is to provide a forum for cardiologists, researchers, physicians, and other health professionals to find most recent advances in the areas of cardiology and cardiovascular diseases.
Austin Journal of Clinical Cardiology accepts original research articles, review articles, case reports, clinical images and rapid communication on all the aspects of cardiology and circulatory system.
Austin Journal of Clinical Cardiology strongly supports the scientific upgradation and fortification in related scientific research community by enhancing access to peer reviewed scientific literary works. Austin Publishing Group also brings universally peer reviewed journals under one roof thereby promoting knowledge sharing, mutual promotion of multidisciplinary science.
Austin Journal of Clinical Cardiology is an open access, peer reviewed, scholarly journal dedicated to publish articles in all areas of cardiology and angiology
TGA is a complex congenital heart disease.Understanding the anatomy,physiology,surgery and anaesthetic management is very important for patient's better outcome.This ppt explains all these points in detail.
Congenital Adr Hyperplasia (CAH) can appear at any age from birth to puberty where it can lead to ambiguous genitalia. It is due to absolute or relative deficiency of 17 Hydroxylase or 21 Hydroxylase enzyme.
A congenital heart defect is a problem with the structure of the heart. It is present at birth. Congenital heart defects are the most common type of birth defect. The defects can involve the walls of the heart, the valves of the heart, and the arteries and veins near the heart. They can disrupt the normal flow of blood through the heart. The blood flow can slow down, go in the wrong direction or to the wrong place, or be blocked completely.
Doctors use a physical exam and special heart tests to diagnose congenital heart defects. They often find severe defects during pregnancy or soon after birth. Signs and symptoms of severe defects in newborns include
Rapid breathing
Cyanosis - a bluish tint to the skin, lips, and fingernails
Fatigue
Poor blood circulation
Many congenital heart defects cause few or no signs and symptoms. They are often not diagnosed until children are older.
Many children with congenital heart defects don't need treatment, but others do. Treatment can include medicines, catheter procedures, surgery, and heart transplants. The treatment depends on the type of the defect, how severe it is, and a child's age, size, and general health.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
2. Congenital heart defect
Congenital heart defect (CHD) or congenital heart
anomaly is a defect in the structure of the heart and
great vessels that is present at birth. Many types of
heart defects exist, most of which either obstruct blood
flow in the heart or vessels near it, or cause blood to
flow through the heart in an abnormal pattern. Other
defects, such as long QT syndrome, affect the heart's
rhythm. Heart defects are among the most common
birth defects and are the leading cause of birth defect-related
deaths. Approximately 9 people in 1000 are born
with a congenital heart defect. Many defects do not
need treatment, but some complex congenital heart
defects require medication or surgery.
3. Causes
The cause of congenital heart disease may be
either genetic or environmental, but is usually
a combination of both.
4. Classification
A number of classification systems exist for
congenital heart defects. In 2000 the
International Congenital Heart Surgery
Nomenclature was developed to provide a
generic classification system.
5. Hypoplasia
Hypoplasia can affect the heart, typically resulting in the
underdevelopment of the right ventricle or the left ventricle. This
causes only one side of the heart to be capable of pumping blood
to the body and lungs effectively. Hypoplasia of the heart is rare
but is the most serious form of CHD. It is called hypoplastic left
heart syndrome when it affects the left side of the heart and
hypoplastic right heart syndrome when it affects the right side of
the heart. In both conditions, the presence of a patent ductus
arteriosus (and, when hypoplasia affects the right side of the heart,
a patent foramen ovale) is vital to the infant's ability to survive until
emergency heart surgery can be performed, since without these
pathways blood cannot circulate to the body (or lungs, depending
on which side of the heart is defective). Hypoplasia of the heart is
generally a cyanotic heart defect
6. Obstruction defects
Obstruction defects occur when heart valves,
arteries, or veins are abnormally narrow or
blocked. Common defects include pulmonic
stenosis, aortic stenosis, and coarctation of the
aorta, with other types such as bicuspid aortic
valve stenosis and subaortic stenosis being
comparatively rare. Any narrowing or blockage
can cause heart enlargement or hypertension.
7. Septal defects
The septum is a wall of tissue which separates the
left heart from the right heart. Defects in the
interatrial septum or the interventricular septum
allow blood to flow from the right side of the heart to
the left, reducing the heart's efficiency.Ventricular
septal defects are collectively the most common type
of CHD, although approximately 30% of adults have
a type of atrial septal defect called probe patent
foramen ovale.
8. Cyanotic defects
Cyanotic heart defects are called such because
they result in cyanosis, a bluish-grey
discoloration of the skin due to a lack of
oxygen in the body. Such defects include
persistent truncus arteriosus, total anomalous
pulmonary venous connection, tetralogy of
Fallot, transposition of the great vessels, and
tricuspid atresia.
10. 11. Mitral stenosis
12. Pulmonary atresia
13.Pulmonary stenosis
14.Transposition of the great vessels
15. dextro-Transposition of the great arteries (d-
TGA)
16. levo-Transposition of the great arteries (l-TGA)
17.Tricuspid atresia
18.Persistent truncus arteriosus
19.Ventricular septal defect (VSD)
20.Wolff-Parkinson-White syndrome (WPW)
11. Aortic valve stenosis
Aortic valve stenosis (AS) is a disease of the
heart valves in which the opening of the
aortic valve is narrowed. The aortic valve is
the valve located between the left ventricle of
the heart and the aorta, the largest artery in
the body, which carries the entire output of
blood to the systemic circulation.
12.
13. Atrial septal defect
Atrial septal defect (ASD) is a form of a congenital heart
defect that lets blood flow between the normally
separated two upper chambers, the atria of the heart.
The atria are separated by a dividing wall, the interatrial
septum. If this septum is defective or absent, then
oxygen-rich blood can flow directly from the left side of
the heart to mix with the oxygen-poor blood in the right
side of the heart, or vice versa. This can lead to lower-than-
normal oxygen levels in the arterial blood that
supplies the brain, organs, and tissues.
14.
15. Atrioventricular septal defect
Atrioventricular septal defect (AVSD) or
atrioventricular canal defect (AVCD), previously
known as "common atrioventricular canal"
(CAVC) or "endocardial cushion defect", is
characterized by a deficiency of the
atrioventricular septum of the heart. It is caused
by an abnormal or inadequate fusion of the
superior and inferior endocardial cushions with
the mid portion of the atrial septum and the
muscular portion of the ventricular septum.
16.
17. Bicuspid aortic valve
A bicuspid aortic valve (BAV) is most commonly a
congenital condition of the aortic valve where two of the
aortic valvular leaflets fuse during development resulting
in a valve that is bicuspid instead of the normal tricuspid
configuration. Normally the only cardiac valve that is
bicuspid is the mitral valve (bicuspid valve) which is
situated between the left atrium and left ventricle.
Cardiac valves play a crucial role in ensuring the
unidirectional flow of blood from the atrium to the
ventricles, or the ventricle to the aorta or pulmonary
trunk.
18.
19. Dextrocardia
Dextrocardia is a congenital defect in which the
apex of the heart is situated on the right side of
the body. There are two main types of
dextrocardia: dextrocardia of embryonic arrest
(also known as isolated dextrocardia[citation
needed]) and dextrocardia situs inversus.
20.
21. Double inlet left ventricle
A double inlet left ventricle (DILV) or
"single ventricle", is a congenital heart
defect appearing in 5 in 100,000 newborns,
where both the left atrium and the right
atrium feed into the left ventricle. The right
ventricle is hypoplastic or doesn't exist.
22.
23. Double outlet right ventricle
Double outlet right ventricle (DORV) is a form of
congenital heart disease where both of the great
arteries connect (in whole or in part) to the right
ventricle (RV). In some cases it is found that this
occurs on the left side of the heart rather than the
right side.
24.
25. Ebstein's anomaly
Ebstein's anomaly is a congenital heart
defect in which the septal and posterior
leaflets of the tricuspid valve are
displaced towards the apex of the right
ventricle of the heart.
26.
27. Hypoplastic left heart syndrome
Hypoplastic left heart syndrome (HLHS) is
a rare congenital heart defect in which the
left ventricle of the heart is severely
underdeveloped.
28.
29. Hypoplastic right heart syndrome
Hypoplastic right
heart syndrome is a
congenital heart
defect in which the
right atrium and right
ventricle are
underdeveloped.
30. Mitral valve stenosis
Mitral stenosis is a valvular heart disease
characterized by the narrowing of the orifice of
the mitral valve of the heart.
31. Pulmonary atresia
Pulmonary atresia is a congenital malformation
of the pulmonary valve in which the valve orifice
fails to develop. The valve is completely closed
thereby obstructing the outflow of blood from
the heart to the lungs. The pulmonary valve is
located on the right side of the heart between
the right ventricle and pulmonary artery.
32.
33. Pulmonic stenosis
Pulmonic stenosis, also known as pulmonary
stenosis, is a dynamic or fixed obstruction of
flow from the right ventricle of the heart to the
pulmonary artery. It is usually first diagnosed in
childhood. Pulmonic stenosis is usually due to
isolated valvular obstruction (Pulmonary valve
stenosis), but may be due to subvalvular or
supravalvular obstruction. It may occur in
association with more complicated congenital
heart disorders.
34.
35. dextro-Transposition of the great arteries
Dextro-Transposition of the great arteries
(d-Transposition of the great arteries,
dextro-TGA, or d-TGA), sometimes also
referred to as complete transposition of the
great arteries, is a birth defect in the large
arteries of the heart. The primary arteries
(the aorta and the pulmonary artery) are
transposed.
36.
37. levo-Transposition of the great arteries
levo-Transposition of the great arteries (L-Transposition
of the great arteries, levo-TGA, or l-TGA), also
commonly referred to as congenitally corrected
transposition of the great arteries (CC-TGA), is an
acyanotic congenital heart defect (CHD) in which the
primary arteries (the aorta and the pulmonary artery) are
transposed, with the aorta anterior and to the left of the
pulmonary artery; the morphological left and right
ventricles are also transposed.
38.
39. Total Anomalous Pulmonary Venous
Return (TAPVR)
The pulmonary veins are the four blood vessels (two
on each side) that return oxygen-rich blood from the
lungs to the left atrium of the heart.
Total anomalous pulmonary venous return (TAPVR)
is a rare congenital malformation in which all four
pulmonary veins do not connect normally to the left
atrium. Instead the four pulmonary veins drain
abnormally to the right atrium by way of an abnormal
(anomalous) connection.
40.
41. Tricuspid atresia
Tricuspid atresia is a form of congenital heart
disease whereby there is a complete absence
of the tricuspid valve. Therefore, there is an
absence of right atrioventricular connection.
This leads to a hypoplastic (undersized) or
absent right ventricle. This defect is contracted
during prenatal development, when the heart
does not finish developing. It causes the heart
to be unable to properly oxygenate the rest of
the blood in the body.
42.
43. Persistent truncus arteriosus
Persistent truncus arteriosus (or patent truncus
arteriosus), also known as Common arterial
trunk, is a rare form of congenital heart disease
that presents at birth. In this condition, the
embryological structure known as the truncus
arteriosus fails to properly divide into the
pulmonary trunk and aorta. This results in one
arterial trunk arising from the heart and
providing mixed blood to the coronary arteries,
pulmonary arteries, and systemic circulation.
44.
45. Ventricular septal defect
A ventricular septal defect (VSD) is a defect
in the ventricular septum, the wall dividing
the left and right ventricles of the heart.
The ventricular septum consists of an
inferior muscular and superior membranous
portion and is extensively innervated with
conducting cardiomyocytes.
46.
47. Wolff–Parkinson–White syndrome
Wolff–Parkinson–White syndrome (WPW) is one of
several disorders of the conduction system of the heart
that are commonly referred to as pre-excitation
syndromes. WPW is caused by the presence of an
abnormal accessory electrical conduction pathway
between the atria and the ventricles. Electrical signals
traveling down this abnormal pathway (known as the
bundle of Kent) may stimulate the ventricles to contract
prematurely, resulting in a unique type of supraventricular
tachycardia referred to as an atrioventricular reciprocating
tachycardia.