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X-RAY Chest Imaging:
 Presented by: Dr Chandni Wadhwani
Radiology Resident
SSG Hospital
Vadodara.
FOUNDER OF X-RAYS
SIR WILHEM CONRAD ROENTGEN (8th
NOV,
MRS ROENTGEN HAND
CHEST X-RAY
 BLACK OPACITIES
 WHITE OPACITIES
 GRAY OPACITIES
COMMONEST VIEW
 POSTERO-ANTERIOR VIEW( PA VIEW)
 LATERAL VIEW.
NORMAL CXR
WHAT IS TO BE SEEN ?
 1) Trachea.
 2) lung fields.
 3) cardiophrenic & costophrenic angles.
 4) hila.
 5) cardiac silhoutte.
 6) ribs.
 7) diaphragm.
 8) free gas under diaphragm is better seen on chest
PA standing view, than on abdomen standing view.
TRACHEA
 Look whether it is centrally placed, or displaced to
one side.
 Normal carinal angle is 60 degrees. It is widened in
LA enlargement & enlarged subcarinal LN.
 The left main bronchus is lower, longer & slender
than the right.
 Trachea may look displaced to one side if:
1. obliquity of the patient
2. real displacement of the trachea: pulling or pushing
due to the pathology.
TRACHEA PUSHED:
 Abnormality in the lung, on the side, opposite to
the tracheal shift.
e .g. mass, pneumothorax,
pleural effusion etc.
TRACHEA PULLED:
 Abnormality in the lung, on the same side, as
that of the tracheal shift.
e.g. collapse, fibrosis
TRACHEA IS PULLED
TRACHEA IS PUSHED
NORMAL ANATOMICAL LOBES
NORMAL ANATOMICAL LOBES
NORMAL CHEST X RAY
COSTOPHRENIC ANGLES:
 On PA view, we can seen the lateral CP angle.
 Normally it should be very sharp & clearly
lucent.
 It is blunted in the pleural effusion, pleural
thickening.
CP angles: normal & obliterated
HILA
 Normal radiographic pulmonary hila are formed
by:
* pulmonary arteries.
* upper lobe pulmonary veins.
 hilar prominence can be due to the following:
* dilated central pulmonary arteries.
* dilated upper lobe pulmonary veins.
* hilar lymphadenopathy.
NORMAL HILA
BILATERAL HILAR
LYMPHADENOPATHY
CARDIAC SILHOUTTE.
LEFT CARDIAC BORDER:
 Aortic knuckle.
 Pulmonary bay.
 Left atrial appendage.
 Left ventricle.
RIGHT CARDIAC BORDER:
 SVC.
 Right atrium.
 IVC.
AORTIC
KNUCKLE
PULMONARY
BAY
LEFT
VENTRICLE
IVC
RIGHT
ATRIUM
SVC
RIGHT ATRIUM
RIGHT VENTRICLE
LEFT ATRIUM
LEFT VENTRICLE
SVC
AORTA
ANTERIOR CARDIAC BORDER ON
LATERAL VIEW:
 Right ventricle.
POSTERIOR CARDIAC BORDER ON
LATERAL VIEW.
 Left atrium.
RIGHT
VENTRICLE
LEFT
ATRIUM
CARDIO THORACIC RATIO
x
y
a
b
 CARDIO-THORACIC RATIO
= (X + Y) / ( A + B )
 NORMAL CARDIO-THORACIC RATIO
* Adults: < 55 %.
* child: < 60 %
 If the CTR is increased, it suggests presence of
cardiomegaly / pericardial effusion.
WATER BOTTLE SIGN
EGG ON STRING HEART
SNOWMAN HEART-FIGURE OF ‘8’
BOOT SHAPED HEART
BOX SHAPED HEART
EBSTEIN’S ANOMALY
RIBS
 Identify the sternal & vertebral ends of the ribs.
 Look for any fracture of the ribs in a traumatized
patient.
 Look for the inferior rib notching at the posterior
ends, in a case of coarctation of the aorta.
 Look for the presence of the cervical ribs.
LEFT CERVICAL RIB
 BIFID RIBS ARE NORMAL VARIANTS.
 Crowding of ribs will occur in volume loss of the
lung.
 Increased distance between the ribs will occur in
emphysema.
DIAPHRAGM
 The right hemi-diaphragm is higher than the left
hemi-diaphragm by
< 2.5 cm, or <2 intercostal spaces.
 The diaphragm may be flattened in emphysema,
massive pleural effusion, large pneumothorax,
phrenic nerve palsy.
 The diaphragm may be abnormally elevated by;
* phrenic nerve palsy.
* collapse of the lung.
* massive ascitis.
* large abdominal pathology.
* subphrenic abscess.
* diaphragmatic hernia.
Free gas under diaphragm:
 best seen in the chest film; & not in the x-ray
abdomen standing.
Causes :
 Peptic perforation of stomach or duodenum.
 Enteric perforation, due to trauma, typhoid
ulcer.
 Colonic perforation due to trauma, UC,
malignancy.
 After procedures: tubal ligation, HSG,
laparoscopy.
 Chiladity syndrome: colonic interposition
between the diaphragm & liver or stomach.
Free gas vs. chiladity
TYPES
 Congenital diaphragmatic herniation can be
classified into two basic types on location
 Bochdalek hernia
 commoner on the left : 75-90%
 posterior
 large and associated with poorer outome
 presents earlier
 Morgagni hernia
 less common
 anterior
 presents later
MORGAGNI HERNIA
BOCHDALEK HERNIA
Basic rules for localizing
a chest lesion
 SILHOUETTE SIGN
 When two isodense structures are in
anatomical contiguity with each other, their
interface is obscured.
THE LESION IS SILHOUTTING WITH
THE
RIGHT CARDIAC BORDER
RESPIRATORY SYSTEM
 1) CONSOLIDATION.
 2) COLLAPSE.
 3) PLEURAL EFFUSION.
 4) D/D of opaque hemithorax.
 5) MASS & SPN.
 6) PNEUMOTHORAX.
 7) HYDROPNEUMOTHORAX.
 8) TUBERCULOSIS.
 9) EMPHYSEMA.
CONSOLIDATION
 Inhomogenous radiopacity, with air
bronchogram within it.
 Borders may be sharply defined, if the
consolidation is confined to the lobar or
segmental boundaries.
 Presence of the Airbronchogram suggests that:
* the lesion is intra-parenchymal.
* the bronchus, supplying that lobe
or segment is patent.
COLLAPSE
 1) HOMOGENOUS RADIOPACITY.
 2) CROWDING OF THE BRONCHO-
VASCULAR MARKINGS.
 3) DISPLACEMENT OF THE HILA,
FISSURES, DIAPHRAGM,
MEDIASTINUM.
 4) COMPENSATORY OVER-INFLATION OF
THE OPPOSITE LUNG OR THE OTHER
LOBES.
COLLAPSE OF RUL.
TRACHEAL SHIFT IN
COLLAPSE
PLEURAL EFFUSION
 1) homogenous radiopacity obliterating
the CP angle, & diaphragm.
 2) concave upper border.
 3) no broncho-vascular markings.
 How to detect a very small quantity of pleural
effusion ?
* 15 min Lateral decubitus film.
D/D of opaque hemithorax
 Mediastinum shifted to opposite side:
* massive pleural effusion
* diaphragmatic hernia.
 Mediastinum shifted to same side:
* massive collapse of the lung.
* massive fibrosis
* post pneumonectomy.
 Mediastinum central:
* massive consolidation
Opaque hemithorax with
trachea pulled towards the
same side
Opaque hemithorax with the
mediastinum pushed to opp.
side
Opaque hemithorax with the
mediastinum in center.
MASS
 PLEURAL / PARENCHYMAL / MEDIASTINAL ?
Pleural mass
PERIPHERAL PARENCHYMAL MASS:
SPN
PNEUMOTHORAX.
 Radiolucency in the CP angle,
 No broncho-vascular markings.
 Razor-sharp medial border.
 There may be associated changes, as under:
* collapse of the underlying lung.
* mediastinal displacement.
* diaphragmatic displacement.
HYDROPNEUMOTHORAX
 Air fluid level.
 Razor sharp border of the collapsed lung.
HYDROPNEUMOTHORAX
EMPHYSEMA
 Hyperlucent lung field.
 Widening of the rib space.
 Flattening of the diaphragm.
 More than 7 visible anterior ends of the ribs.
 Tear drop shaped heart.
EMPHYSEMA
LUNG ABSESS
 Air- fluid level.
 Thick wall.
 Surrounding consolidation.
Lung abscess
D/D of miliary opacities
 Miliary opacities are < 2 mm sized, multiple,
opacities of uniform sizes & shapes
homogenously distributed in both the lung
fields.
D/D of miliary opacities are as under:
 Miliary tuberculosis.
 Pnemoconiosis
 Histoplasmosis.
 Miliary metastasis in thyroid, pancreas,breast
malignancies.
MILLIARY MOTTLING.
Primary pulmonary
tuberculosis Occurs in children, who are infected with the
tuberculous bacilli for the first time, or who have
been given the BCG vaccine.
 Charaterized by GHOHN’S COMPLEX, which is
made up of :
* Ghohn’s focus: an area of
consolidation.
* typically unilateral hilar
lymphadenopathy, on the same side
as that of the consolidation.
POST PRIMARY TUBERCULOSIS
 Defined as the development of the disease in a
patient, who had previous
subclinical / clinical infection; or who had been
vaccinated with the BCG vaccine.
KEY FEATURES OF POST-PRIMARY TB:
 Predominant involvement of the apices of the lungs
by:
* consolidation.
* cavitation.
* fibrosis.
* volume-loss.
 NO HILAR LYMPHADENOPATHY.
 Pleural involvement in the form of effusion or
empyema.
 Bronchiectasis ( if present ) is typically in the upper
lobes.
POST PRIMARY TB
TRY YOUR KNOWLEDGE!!
Miliary mottling
PNEUMOPERITONEUM-Free gas under
diaphragm
CONSOLIDATION-Air bronchogram
COLLAPSE OF LINGULAR LOBE
EMPHYSEMATOUS LUNG
CANON BALL Pulmonary metastasis
Chest XRAY made easy-for UG/PG

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Chest XRAY made easy-for UG/PG

Editor's Notes

  1. THYMIC SHADOW CARDIO-THORACIC RATIO FOR NEONATES / INFANTS : LESS THAN 0.6 ASD VS VSD : LEFT ATRIAL ENLARGEMENT ONLY IN VSD.
  2. To sustain life, a communication (eg: a patent foramen ovale, atrial septal defect, ventricular septal defect, or a combination of these) must be present between the systemic and the pulmonary circulation, in addition to systemic collateral arteries. It is isolated in 90% of those affected and rarely is associated with a syndrome or an extracardiac malformation.
  3. THIS CHEST RADIOGRAPH SHOWS CONVEX RADIOOPACITIES IN THE MEDIASTINUM IN THE SUPRACARDIAC REGION GIVING FIGURE OF 8 OR SNOWMAN APPEARANCE WHICH IS CHARACTERISTIC OF SUPRACARDIAC TYPE OF TOTAL ANOMALOUS PULMONARY VENOUS RETURN.
  4. and are best differentiated according to the site at which the anomalous pulmonary veins terminate. Typically, four anomalous pulmonary veins (two from each lung) converge directly behind the left atrium and form a common vein, known as the vertical vein, that passes anterior to the left pulmonary artery and the left main bronchus to join the innominate vein
  5. RIGHT SIDED AORTIC ARCH CONCAVITY OF MAIN PULMONARY ARTERY UPTURNED CARDIAC APEX DUE TO RVH; WITH OLIGEMIA CHARACTERISTIC PRESENTATION OF TOF
  6. Right ventricular hypertrophy typically develops in long-standing untreated disease.
  7. YOUNG HYPERTENSIVE MALE: DIFFERENTIAL UPPER AND LOWER LIMB BLOOD PRESSURES. HIGH IN PRE-STENOTIC VESSELS; LOWER IN POST STENOTIC VESSELS.
  8. INFANT SHOW MASSIVE CARDIOMEGALY WITH DECREASED PULMONARY FLOW.