Our concepts of heart disease are based on the enormous reservoir of physiologic and anatomic knowledge derived from the past 70 years' of experience in the cardiac catheterization laboratory.
As Andre Cournand remarked in his Nobel lecture of December 11, 1956, the cardiac catheter was the key in the lock.
By turning this key, Cournand and his colleagues led us into a new era in the understanding of normal and disordered cardiac function in huma
In this ppt i am going to discuss various spotters, including ECG, X-ray, fluroscopy images and there answers. These spotter now days asked in various DM cardiology exam conducted all over India, so it will help you in your DM Cardiology exam preperationn.
Our concepts of heart disease are based on the enormous reservoir of physiologic and anatomic knowledge derived from the past 70 years' of experience in the cardiac catheterization laboratory.
As Andre Cournand remarked in his Nobel lecture of December 11, 1956, the cardiac catheter was the key in the lock.
By turning this key, Cournand and his colleagues led us into a new era in the understanding of normal and disordered cardiac function in huma
In this ppt i am going to discuss various spotters, including ECG, X-ray, fluroscopy images and there answers. These spotter now days asked in various DM cardiology exam conducted all over India, so it will help you in your DM Cardiology exam preperationn.
Tachycardias are broadly categorized based upon the width of the QRS complex on the electrocardiogram (ECG). A narrow QRS complex (<120 milliseconds) reflects rapid activation of the ventricles via the normal His-Purkinje system, which in turn suggests that the arrhythmia originates above or within the His bundle (ie, a supraventricular tachycardia). The site of origin may be in the sinus node, the atria, the atrioventricular (AV) node, the His bundle, or some combination of these sites. A widened QRS (≥120 milliseconds) occurs when ventricular activation is abnormally slow. The most common reason that a QRS is widened is because the arrhythmia originates below the His bundle in the bundle branches, Purkinje fibers, or ventricular myocardium (eg, ventricular tachycardia). Alternatively, a supraventricular arrhythmia can produce a widened QRS if there are either pre-existing or rate-related abnormalities within the His-Purkinje system (eg, supraventricular tachycardia with aberrancy), or if conduction occurs over an accessory pathway. Thus, wide QRS complex tachycardias may be either supraventricular or ventricular in origin.
In a myocardial infarction transmural ischemia develops. In the first hours and days after the onset of a myocardial infarction, several changes can be observed on the ECG. First, large peaked T waves (or hyperacute T waves), then ST elevation, then negative T waves and finally pathologic Q waves develop.
preop TEE assessment of atrial septal defect is very important for making decision for device closure, properly assessed adequate rims of ASD will reduce risk of device embolization to almost nil.
differentiating between supraventicular tachycardia and ventricular tachycardia in wide complex rhythm is always confusing and management is totally different. correct diagnosis will make dramatic difference in patient management.
Speckle tracking echocardiography (STE) is an echocardiographic imaging technique that analyzes the motion of tissues in the heart by using the naturally occurring speckle pattern in the myocardium or blood when imaged by ultrasound.
Tachycardias are broadly categorized based upon the width of the QRS complex on the electrocardiogram (ECG). A narrow QRS complex (<120 milliseconds) reflects rapid activation of the ventricles via the normal His-Purkinje system, which in turn suggests that the arrhythmia originates above or within the His bundle (ie, a supraventricular tachycardia). The site of origin may be in the sinus node, the atria, the atrioventricular (AV) node, the His bundle, or some combination of these sites. A widened QRS (≥120 milliseconds) occurs when ventricular activation is abnormally slow. The most common reason that a QRS is widened is because the arrhythmia originates below the His bundle in the bundle branches, Purkinje fibers, or ventricular myocardium (eg, ventricular tachycardia). Alternatively, a supraventricular arrhythmia can produce a widened QRS if there are either pre-existing or rate-related abnormalities within the His-Purkinje system (eg, supraventricular tachycardia with aberrancy), or if conduction occurs over an accessory pathway. Thus, wide QRS complex tachycardias may be either supraventricular or ventricular in origin.
In a myocardial infarction transmural ischemia develops. In the first hours and days after the onset of a myocardial infarction, several changes can be observed on the ECG. First, large peaked T waves (or hyperacute T waves), then ST elevation, then negative T waves and finally pathologic Q waves develop.
preop TEE assessment of atrial septal defect is very important for making decision for device closure, properly assessed adequate rims of ASD will reduce risk of device embolization to almost nil.
differentiating between supraventicular tachycardia and ventricular tachycardia in wide complex rhythm is always confusing and management is totally different. correct diagnosis will make dramatic difference in patient management.
Speckle tracking echocardiography (STE) is an echocardiographic imaging technique that analyzes the motion of tissues in the heart by using the naturally occurring speckle pattern in the myocardium or blood when imaged by ultrasound.
How to read ECG systematically with practice strips Khaled AlKhodari
This lecture simplifies the steps of reading ECG systematically. It starts with a simple heart anatomy and the logical steps that should be followed to perfect ECG reading without missing any abnormality. Finally, there are some practice ECG strips that include but not only MI, STEMI, Wellens syndrome, Pulmonary embolism, LVH, arrhythmias... and others
Ventricular tachycardia are difficult to understand. it is classified in to two types. 1. VT in structurally normal heart, 2. VT in heart with structural diseases. I have tried to simplify the VT in structurally normal heart, which may be helpful to many students and learners.
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The Roman Empire, a vast and enduring power, stands as one of history's most remarkable civilizations, leaving an indelible imprint on the world. It emerged from the Roman Republic, transitioning into an imperial powerhouse under the leadership of Augustus Caesar in 27 BCE. This transformation marked the beginning of an era defined by unprecedented territorial expansion, architectural marvels, and profound cultural influence.
The empire's roots lie in the city of Rome, founded, according to legend, by Romulus in 753 BCE. Over centuries, Rome evolved from a small settlement to a formidable republic, characterized by a complex political system with elected officials and checks on power. However, internal strife, class conflicts, and military ambitions paved the way for the end of the Republic. Julius Caesar’s dictatorship and subsequent assassination in 44 BCE created a power vacuum, leading to a civil war. Octavian, later Augustus, emerged victorious, heralding the Roman Empire’s birth.
Under Augustus, the empire experienced the Pax Romana, a 200-year period of relative peace and stability. Augustus reformed the military, established efficient administrative systems, and initiated grand construction projects. The empire's borders expanded, encompassing territories from Britain to Egypt and from Spain to the Euphrates. Roman legions, renowned for their discipline and engineering prowess, secured and maintained these vast territories, building roads, fortifications, and cities that facilitated control and integration.
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Roman architecture and engineering achievements were monumental. They perfected the arch, vault, and dome, constructing enduring structures like the Colosseum, Pantheon, and aqueducts. These engineering marvels not only showcased Roman ingenuity but also served practical purposes, from public entertainment to water supply.
2024.06.01 Introducing a competency framework for languag learning materials ...Sandy Millin
http://sandymillin.wordpress.com/iateflwebinar2024
Published classroom materials form the basis of syllabuses, drive teacher professional development, and have a potentially huge influence on learners, teachers and education systems. All teachers also create their own materials, whether a few sentences on a blackboard, a highly-structured fully-realised online course, or anything in between. Despite this, the knowledge and skills needed to create effective language learning materials are rarely part of teacher training, and are mostly learnt by trial and error.
Knowledge and skills frameworks, generally called competency frameworks, for ELT teachers, trainers and managers have existed for a few years now. However, until I created one for my MA dissertation, there wasn’t one drawing together what we need to know and do to be able to effectively produce language learning materials.
This webinar will introduce you to my framework, highlighting the key competencies I identified from my research. It will also show how anybody involved in language teaching (any language, not just English!), teacher training, managing schools or developing language learning materials can benefit from using the framework.
Operation “Blue Star” is the only event in the history of Independent India where the state went into war with its own people. Even after about 40 years it is not clear if it was culmination of states anger over people of the region, a political game of power or start of dictatorial chapter in the democratic setup.
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This Gasta posits a strategic approach to integrating AI into HEIs to prepare staff, students and the curriculum for an evolving world and workplace. We will highlight the advantages of working with these technologies beyond the realm of teaching, learning and assessment by considering prompt engineering skills, industry impact, curriculum changes, and the need for staff upskilling. In contrast, not engaging strategically with Generative AI poses risks, including falling behind peers, missed opportunities and failing to ensure our graduates remain employable. The rapid evolution of AI technologies necessitates a proactive and strategic approach if we are to remain relevant.
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2. Overview
• Case scenario
• ECG based diagnosis
• Introduction
• Wide complex tachycardia and diagnostic algorithm
• VT vs SVT
• VT
– Classification
– Etiology
– Localization
• ECG examples
3. Case
• 40 Y old male, Taxi driver
• Non smoker, occasional tobacco
• K/C/O Bronchial asthma, No other co-morbidities
• C/O
– Retro-sternal burning sensation
– Palpitation
• Past history: Episodic palpitations from last 10 yrs
• Holter study: Sinus bradycardia with occasional VPCs (May’2011)
• Family history: NAD
On examination
• PR 256/ min
• BP 90/60 mm Hg
• SE:
– CVS: S1/S2 + , no murmur, no added sounds
– R/S: bilateral equal vesicular BS
6. • CBC: 15/12050/141000
• RFT: 17.63/1.24
• Na/K: 146/5.7
• Trop T : 0.032
• Echo: Normal chamber dimensions, Normal LV systolic function,
LVEF 55%, RA, RV normal, RVOT-PLAX- 16, PSAX- 21
• Cardiac MRI: Normal biventricular size and function, focal non
specific enhancement at inferior RV insertion point, no e/o
ischemic/fibrotic changes
Diagnosis
Hemodynamically unstable sustained monomorphic VT ? Idiopathic
7. Introduction
• Most common cause of WCT(80%)
• Relatively organized tachyarrhythmia with discrete QRS
• Major cause of
– Morbidity and mortality in pt with structural heart disease
– Sudden cardiac death (60% cases) on holter monitoring
• Diagnosis on presentation…….. still a challenge
• Reentry is most common mechanism
• Recurrence is common in less than year
• ICD implantation is absolute indication in presence of LVEF< 30%
All WCT is VT until proven otherwise
András Vereckei et al, current Cardiology Reviews, 2014, 1
10. Brugada algorithm for
D/D of WCTs
Brugada P et al,A new approach to the differential diagnosis of a regular tachycardia with a wide QRS
complex. Circulation 1991; 83: 1649-59.
11. Adam M. May. Journal of the American Heart Association. Wide Complex Tachycardia Differentiation: A
Reappraisal of the State‐of‐the‐Art, 2020 Volume: 9, Issue: 11
12. Classification of VT
• On basis of
– Clinical presentation
– ECG (Duration and morphological) presentation
– Disease entity
ACC/AHA/ESC 2006 guidelines
23. VT in patients with CAD
• Typically based on a reentrant mechanism
Principles for noninvasive localization of the VT circuit’s exit
1.Post-MI VTs almost always arise in the left ventricle or IVS.
•Knowledge of the location of the prior infarct facilitates the localization
process
•VTs associated with inferior MI arise from the inferobasal septum or free
wall
•VTs associated with anterior MI arise from the antero-apical or infero-
apical septum or free wall.
2. VTs arising from the IVS have narrower QRS durations compared to free
wall VTs.
3.LBBB VTs almost always localize to the septum, (or within 1 cm of the
septum) while RBBB VTs can arise anywhere in the left ventricle, posing a
greater challenge for localization.
4.The presence of positive or negative concordance in the precordial leads
strongly suggests a basal or apical exit site, respectively.
5.A superior axis in general points to an IVT exit location (thus many inferior
MI VTs have a superior axis) or apical locations (in anterior MI), while an
inferior axis generally implies an antero-basal location.
Masry et al Thoracic key 2018
24. Schematic representation of anatomical areas in right anterior
oblique (RAO) and left anterior oblique (LAO) views. LAO: left
anterior oblique; RAO: right anterior oblique.
Algorithm for identification of exit sites of VT.
Condori-Leandro HI, et al. Journal of Geriatric Cardiology (2019) 16: 362 377
25.
26. VT in absence of Structural heart disease
• ~ 10% of all patients present with VT
• An absence of structural heart disease is usually suggested if an ECG
(except in Brugada syndrome and LQTS]), echo, and coronary
arteriogram collectively are normal.
• Types of VT in absence of structural heart disease:
– RV outflow tract (RVOT) VT
– LV outflow tract (LVOT) VT
– Idiopathic LV tachycardia (ILVT)
– Idiopathic propranolol-sensitive (automatic) VT (IPVT)
– Catecholaminergic polymorphic VT (CPVT),
– Brugada syndrome, and LQTS.
• Idiopathic VT -RVOT VT, LVOT VT, ILVT, and IPVT
Komandoor Srivathsan et al, Indian Pacing and Electrophysiology Journal (ISSN 0972-6292), 5(2): 106-121 (2005)
27. Diagnostic scheme for monomorphic ventricular tachycardia in structurally normal
hearts (collectively normal electrocardiogram, echo, and coronary angiogram)
Komandoor Srivathsan et al, Indian Pacing and Electrophysiology Journal (ISSN 0972-6292), 5(2): 106-121 (2005)
28. Proposed diagnostic scheme for polymorphic VT or ventricular fibrillation in
structurally normal hearts (collectively normal electrocardiogram [except in
Brugada syndrome and LQTS], echocardiogram, and coronary angiogram).
Komandoor Srivathsan et al, Indian Pacing and Electrophysiology Journal (ISSN 0972-6292), 5(2): 106-121 (2005)
31. LVOT VT ECG
ECG illustrating LVOT VT. The S wave in LI and R-wave transition in V1
suggest LVOT VT. In addition, an R:S amplitude ratio of 30% and an R:QRS
duration ratio of 50% are seen. Presence of an S wave in leads V5 and V6
suggests an infravalvular origin of the tachycardia.
32. Leandro et al, Journal of Geriatric Cardiology (2019) 16: 362 377
34. Epicardial VT
•A pseudo delta wave ≥ 34 ms
(measured from the earliest
ventricular activation to the earliest
fast deflection in any precordial lead)
•Intrinsicoid deflection V2 ≥ 85 ms
(defined as the interval measured from
the earliest ventricular activation to
the peak of QRS in V2)
•Shortest RS complex ≥ 121 ms
(defined as the interval measured from
the earliest ventricular activation to
the nadir of the first S wave in any
precordial lead)
Leandro et al, Journal of Geriatric Cardiology (2019) 16: 362 377
35. Brugada type ECG
RBBB with ST elevation in V1 to V3
ECG of a patient with Brugada syndrome. The right bundle branch block pattern with coved ST
segment elevation (J-point elevation) is more than 2 mm, particularly in lead V2.