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12 Lead ECG
Interpretation: Color
Coding for MI’s
Zagarzusem.E MD
1
Objectives
 review the ECG waveform and intervals
 Define myocardial ischemia, injury and infarction
 Identify the 5 major infarct areas on the 12 lead
 Name occluded arteries common to the area
 Differentiate ECG changes reflecting ischemia,
injury and infarction
 Identify cardiac enzymes associated with ACS
2
3
4
ECG graph paper
The 12-Lead view
 Each limb lead I, II, III, AVR, AVL, AVF records
from a different angle
 All six limb leads intersect and visualize a frontal
plane
 The six chest leads (precordial) V1, V2, V3, V4,
V5, V6 view the body in the horizontal plane to the
AV node
 The 12 lead ECG forms a camera view from 12
angles
5
Views from Augmented and Limb Leads-
Frontal
6
Precordial lead snapshots
 Think of each
precordial lead as a
horizontal view of
the heart at the AV
node
 With the limb leads
and the precordial
leads you have a
snapshot of heart
portions
7
Unipolar and Bipolar
 Limb leads I, II, III are bipolar and have a negative
and positive pole
 Electrical potential differences are measured between
the poles
 AVR, AVL and AVF are unipolar
 No negative lead
 The heart is the negative pole
 Electrical potential difference is measured betweeen the
lead and the heart
 Chest leads are unipolar
 The heart also is the negative pole
2004 Anna Story8
Lead Placement is Important
 Each positive electrode
acts as a camera
looking at the heart
 Ten leads attached for
twelve lead diagnostics.
The monitor combines 2
leads.
 Mnemonic for limb
leads
 White on right
 Smoke(black) over
fire(red)
 Snow(white) on
grass(green) 2004 Anna Story 9
Precordial Leads
10
Arrangement of Leads on the EKG
Anatomic Groups
(Septum)
Anatomic Groups
(Anterior Wall)
Anatomic Groups
(Lateral Wall)
Anatomic Groups
(Inferior Wall)
Anatomic Groups
(Summary)
The ECG Tracing: Waves
 P- wave
 Marks the beginning of the cardiac cycle and
measures the electrical impulse that causes atrial
depolarization and mechanical contraction
 QRS- Complex
 Measures the impulse that causes ventricular
depolarization
 Q-wave- may or may not be evident on the ECG
 R-wave- first upward deflection following P wave
 S-wave- the first downward deflection following the R-
wave
 T- wave
 Marks ventricular repolarization that ends the
cardiac cycle 18
Intervals and Segments
 P-R interval-
 Time interval for impulse to go from the SA to the AV node
 normal 0.12-0.20 secs
 QRS Interval
 Time interval for impulse to go from AV node to stimulate Purkinjie
fibers
 Less than 0.12 secs
 QT Interval
 Time interval from beginning of depolarization to the end of
repolarization
 Should not exceed ½ the length of the R-R
 ST segment
 end of the S to the beginning of the T
19
The ECG Tracing
20
MI Definition
 A result of occlusion of arterial flow to the myocardium.
 Ischemia, injury and necrosis is result
 Occlusion occurs via spasm, blood clot or stenosis
21
ECG Changes : Ischemia
 T-wave inversion ( flipped T)
 ST segment depression
 T wave flattening
 Biphasic T-waves
22
Baseline
Inversion-tongoroh
Flattening-havtgairah
Depressed-dooshloh
ECG Changes: Injury
 ST segment elevation of greater than 1mm in at least 2
contiguous leads
 Heightenedөндөрсөх or peaked T waves
 Directly related to portions of myocardium rendered
electrically inactive
23
Baseline
ECG Changes: Infarct
 Significant Q-wave where none previously existed
 Why?
 Impulse traveling away from the positive lead
 Necrotic tissue is electrically dead
 No Q-wave in Subendocardial infarcts
 Why?
 Not full thickness dead tissue
 But will see a ST depression
 Often a precursor to full thickness MI
 Criteria
 Depth of Q wave should be 25% the height of the R wave
 Width of Q wave is 0.04 secs
 Diminished height of the R wave
24
Evolving MI and Hallmarks of AMI
25
1 year
•Q wave
•ST Elevation
•T wave inversion
Dissecting the 12 Lead ECG
 Horizontal marks time
 Vertical marks amplitude
 6 limb leads
 6 precordial leads
 Positioning measures 12 perspectives or views of
the heart
 The 12 perspectives are arranged in vertical
columns
 Limb leads are I, II, III, AVR, AVL, AVF
 Precordial leads are V1, V2, V3, V4, V5, V6
26
A Normal 12 Lead ECG
27
A Normal 12 Lead ECG
28
Color Coding ECG’s Anterior
 Yellow indicates V1, V2,
V3, V4
 Anterior infarct with ST elevation
 Left Anterior Descending Artery
(LAD)
 V1 and V2 may also indicate septal
involvement which extends from
front to the back of the heart along
the septum
 Left bundle branch block
 Right bundle branch block
 2nd Degree Type2
 Complete Heart Block
29
Anterior MI
30
Color Coding ECG- Inferior
 Blue indicates leads II, III,
AVF
 Inferior Infarct with ST
elevations
 Right Coronary Artery (RCA)
 1st degree Heart Block
 2nd degree Type 1, 2
 3rd degree Block
 N/V common, Brady
31
Inferior MI
32
As an aside….
 Right sided EKG
 Ever heard of it?
 Ever done one?
 Think about it…..
 For your cases that are clearly inferior MI’s
 Obtain a dextrocardiogram whenever ST segment elevation is
noted in Inferior leads
33
Right Sided EKG????
 RVI occurs around 40% in
inferior MI’s
 Significance
 Larger area of infarct
 Both ventricles
 Different treatment
 Right leads “look” directly at
Right Ventricle and can
show ST elevations in leads
II. III. AVF, V4R , V5R and
V6R
 Occlusion in RCA and
proximal enough to involve
the RV
34
The single most accurate
tool used in measuring RVI.
90% sensitive and specific
Clinical Triad of RVI
 Hypotension
 Jugular vein
distention
 Dry lung sounds
35
Color Coding ECG- Lateral
 Red indicates leads
I, AVL, V5, V6
 Lateral Infarct with
ST elevations
 Left Circumflex
Artery
 Rarely by itself
 Usually in combo
36
Lateral MI
37
Color Coding ECG- Posterior
 Green indicates leads V1,
V2
 Posterior Infarct with ST
Depressions and/ tall R wave
 RCA and/or LCX Artery
 Understand Reciprocal changes
 The posterior aspect of the
heart is viewed as a mirror
image and therefore
depressions versus elevations
indicate MI
 Rarely by itself usually in
combo
38
Posterior MI
39
Color Coding ECG- SubEndo
 No color for
SubEndocardial infarcts
since they are not
transmural
 Look for diffuse or
localized changes and
non – Q wave
abnormalities
 T-wave inversions
 ST segment depression
40
SubEndo MI
41
More than one color shows abnormality
 A combination of infarcts such as:
Anterolateral yellow and red
Inferoposterior blue and green
Anteroseptal yellow and green
42
Putting it ALL together
43
44
Practice 1
45
Anterior MI with lateral
involvement
ST elevations V2, V3, V4
ST elevations II, AVL, V5
Click for
answer
Practice 2
46
Anteroseptal MI
ST elevations V1, V2, V3, V4
Click for
answer
Practice 3
47
Click for
answer
Inferior MI
ST elevation 2,3 AVF
Practice 4
48
Click for
answer
Inferior lateral MI
ST elevations 2, 3, AVF
ST elevations V5
Practice 5
49
•Acute inferior MI
•Lateral ischemia
Click for
answer
Additional Practice Strips
50
Additional Practice Strips
51
Additional Practice Strips
52
Additional Practice Strips
53
Additional Practice Strips
54
Additional Practice Strips
55
Additional Practice Strips
56
Additional Practice Strips
57
Additional Practice Strips
58
Additional Practice Strips
59
Additional Practice Strips
60
Additional Practice Strips
61
Additional Practice Strips
62
Additional Practice Strips
63
Additional Practice Strips
64
Additional Practice Strips
65
Cardiac Enzymes Indicating Infarct
 Normals
 CPK- 10-155u/liter
 begin rise 3-6 hours and peaks 12-24 with return to norm 3-5
days
 CPK-MB < than 5% IU/liter
 LDH 85-200 IU/liter
 Begin rise 12 hours, peaks 36-72 and normal around 10 days
 LDH 1- 18.1% - 29% of total
 LDH 2- 27.4% to 37.5% of total
66
Cardiac Enzymes Indicating Infarct
 Troponins- Now the
Gold Standard!
 Rises after 3-6 hours
 Negative Troponin
within 6 hours of
onset of S&S rules
out the MI
 Peaks at about 20
hours
 May be raised for 14
days
67
Cardiac Enzymes Indicating Infarct
 Troponin T
 84% sensitivity for MI 8 hours after onset of symptoms
 22% for unstable angina
 Advantages
 Highly sensitive for detecting myocardial ischemia
 Levels may help to stratify risks
 Disadvantages
 Less specific than Troponin I
 Increased in angina
 Increased in chronic renal failure
68
Cardiac Enzymes Indicating Infarct
 Troponin I
 90% sensitivity for MI 8 hours after onset of S&S and
95% specificity
 Level greater than 1.2 suggest MI
 Negative rules out MI
 Obtain two negative troponin values 4 hours apart
 Normally exceedingly low
Even a small elevation indicates
myocardial damage
69
THANKS FOR ATTENTION
References
 Хүний физиологи-Г.Дашзэвэг
 Дотор өвчний оношзүй-Б.Гомбосүрэн
 www.slideshare.net
 www.wikipedia.org
71

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Зүрхний цахилгаан бичлэг

  • 1. 12 Lead ECG Interpretation: Color Coding for MI’s Zagarzusem.E MD 1
  • 2. Objectives  review the ECG waveform and intervals  Define myocardial ischemia, injury and infarction  Identify the 5 major infarct areas on the 12 lead  Name occluded arteries common to the area  Differentiate ECG changes reflecting ischemia, injury and infarction  Identify cardiac enzymes associated with ACS 2
  • 3. 3
  • 5. The 12-Lead view  Each limb lead I, II, III, AVR, AVL, AVF records from a different angle  All six limb leads intersect and visualize a frontal plane  The six chest leads (precordial) V1, V2, V3, V4, V5, V6 view the body in the horizontal plane to the AV node  The 12 lead ECG forms a camera view from 12 angles 5
  • 6. Views from Augmented and Limb Leads- Frontal 6
  • 7. Precordial lead snapshots  Think of each precordial lead as a horizontal view of the heart at the AV node  With the limb leads and the precordial leads you have a snapshot of heart portions 7
  • 8. Unipolar and Bipolar  Limb leads I, II, III are bipolar and have a negative and positive pole  Electrical potential differences are measured between the poles  AVR, AVL and AVF are unipolar  No negative lead  The heart is the negative pole  Electrical potential difference is measured betweeen the lead and the heart  Chest leads are unipolar  The heart also is the negative pole 2004 Anna Story8
  • 9. Lead Placement is Important  Each positive electrode acts as a camera looking at the heart  Ten leads attached for twelve lead diagnostics. The monitor combines 2 leads.  Mnemonic for limb leads  White on right  Smoke(black) over fire(red)  Snow(white) on grass(green) 2004 Anna Story 9
  • 11. Arrangement of Leads on the EKG
  • 17.
  • 18. The ECG Tracing: Waves  P- wave  Marks the beginning of the cardiac cycle and measures the electrical impulse that causes atrial depolarization and mechanical contraction  QRS- Complex  Measures the impulse that causes ventricular depolarization  Q-wave- may or may not be evident on the ECG  R-wave- first upward deflection following P wave  S-wave- the first downward deflection following the R- wave  T- wave  Marks ventricular repolarization that ends the cardiac cycle 18
  • 19. Intervals and Segments  P-R interval-  Time interval for impulse to go from the SA to the AV node  normal 0.12-0.20 secs  QRS Interval  Time interval for impulse to go from AV node to stimulate Purkinjie fibers  Less than 0.12 secs  QT Interval  Time interval from beginning of depolarization to the end of repolarization  Should not exceed ½ the length of the R-R  ST segment  end of the S to the beginning of the T 19
  • 21. MI Definition  A result of occlusion of arterial flow to the myocardium.  Ischemia, injury and necrosis is result  Occlusion occurs via spasm, blood clot or stenosis 21
  • 22. ECG Changes : Ischemia  T-wave inversion ( flipped T)  ST segment depression  T wave flattening  Biphasic T-waves 22 Baseline Inversion-tongoroh Flattening-havtgairah Depressed-dooshloh
  • 23. ECG Changes: Injury  ST segment elevation of greater than 1mm in at least 2 contiguous leads  Heightenedөндөрсөх or peaked T waves  Directly related to portions of myocardium rendered electrically inactive 23 Baseline
  • 24. ECG Changes: Infarct  Significant Q-wave where none previously existed  Why?  Impulse traveling away from the positive lead  Necrotic tissue is electrically dead  No Q-wave in Subendocardial infarcts  Why?  Not full thickness dead tissue  But will see a ST depression  Often a precursor to full thickness MI  Criteria  Depth of Q wave should be 25% the height of the R wave  Width of Q wave is 0.04 secs  Diminished height of the R wave 24
  • 25. Evolving MI and Hallmarks of AMI 25 1 year •Q wave •ST Elevation •T wave inversion
  • 26. Dissecting the 12 Lead ECG  Horizontal marks time  Vertical marks amplitude  6 limb leads  6 precordial leads  Positioning measures 12 perspectives or views of the heart  The 12 perspectives are arranged in vertical columns  Limb leads are I, II, III, AVR, AVL, AVF  Precordial leads are V1, V2, V3, V4, V5, V6 26
  • 27. A Normal 12 Lead ECG 27
  • 28. A Normal 12 Lead ECG 28
  • 29. Color Coding ECG’s Anterior  Yellow indicates V1, V2, V3, V4  Anterior infarct with ST elevation  Left Anterior Descending Artery (LAD)  V1 and V2 may also indicate septal involvement which extends from front to the back of the heart along the septum  Left bundle branch block  Right bundle branch block  2nd Degree Type2  Complete Heart Block 29
  • 31. Color Coding ECG- Inferior  Blue indicates leads II, III, AVF  Inferior Infarct with ST elevations  Right Coronary Artery (RCA)  1st degree Heart Block  2nd degree Type 1, 2  3rd degree Block  N/V common, Brady 31
  • 33. As an aside….  Right sided EKG  Ever heard of it?  Ever done one?  Think about it…..  For your cases that are clearly inferior MI’s  Obtain a dextrocardiogram whenever ST segment elevation is noted in Inferior leads 33
  • 34. Right Sided EKG????  RVI occurs around 40% in inferior MI’s  Significance  Larger area of infarct  Both ventricles  Different treatment  Right leads “look” directly at Right Ventricle and can show ST elevations in leads II. III. AVF, V4R , V5R and V6R  Occlusion in RCA and proximal enough to involve the RV 34 The single most accurate tool used in measuring RVI. 90% sensitive and specific
  • 35. Clinical Triad of RVI  Hypotension  Jugular vein distention  Dry lung sounds 35
  • 36. Color Coding ECG- Lateral  Red indicates leads I, AVL, V5, V6  Lateral Infarct with ST elevations  Left Circumflex Artery  Rarely by itself  Usually in combo 36
  • 38. Color Coding ECG- Posterior  Green indicates leads V1, V2  Posterior Infarct with ST Depressions and/ tall R wave  RCA and/or LCX Artery  Understand Reciprocal changes  The posterior aspect of the heart is viewed as a mirror image and therefore depressions versus elevations indicate MI  Rarely by itself usually in combo 38
  • 40. Color Coding ECG- SubEndo  No color for SubEndocardial infarcts since they are not transmural  Look for diffuse or localized changes and non – Q wave abnormalities  T-wave inversions  ST segment depression 40
  • 42. More than one color shows abnormality  A combination of infarcts such as: Anterolateral yellow and red Inferoposterior blue and green Anteroseptal yellow and green 42
  • 43. Putting it ALL together 43
  • 44. 44
  • 45. Practice 1 45 Anterior MI with lateral involvement ST elevations V2, V3, V4 ST elevations II, AVL, V5 Click for answer
  • 46. Practice 2 46 Anteroseptal MI ST elevations V1, V2, V3, V4 Click for answer
  • 47. Practice 3 47 Click for answer Inferior MI ST elevation 2,3 AVF
  • 48. Practice 4 48 Click for answer Inferior lateral MI ST elevations 2, 3, AVF ST elevations V5
  • 49. Practice 5 49 •Acute inferior MI •Lateral ischemia Click for answer
  • 66. Cardiac Enzymes Indicating Infarct  Normals  CPK- 10-155u/liter  begin rise 3-6 hours and peaks 12-24 with return to norm 3-5 days  CPK-MB < than 5% IU/liter  LDH 85-200 IU/liter  Begin rise 12 hours, peaks 36-72 and normal around 10 days  LDH 1- 18.1% - 29% of total  LDH 2- 27.4% to 37.5% of total 66
  • 67. Cardiac Enzymes Indicating Infarct  Troponins- Now the Gold Standard!  Rises after 3-6 hours  Negative Troponin within 6 hours of onset of S&S rules out the MI  Peaks at about 20 hours  May be raised for 14 days 67
  • 68. Cardiac Enzymes Indicating Infarct  Troponin T  84% sensitivity for MI 8 hours after onset of symptoms  22% for unstable angina  Advantages  Highly sensitive for detecting myocardial ischemia  Levels may help to stratify risks  Disadvantages  Less specific than Troponin I  Increased in angina  Increased in chronic renal failure 68
  • 69. Cardiac Enzymes Indicating Infarct  Troponin I  90% sensitivity for MI 8 hours after onset of S&S and 95% specificity  Level greater than 1.2 suggest MI  Negative rules out MI  Obtain two negative troponin values 4 hours apart  Normally exceedingly low Even a small elevation indicates myocardial damage 69
  • 71. References  Хүний физиологи-Г.Дашзэвэг  Дотор өвчний оношзүй-Б.Гомбосүрэн  www.slideshare.net  www.wikipedia.org 71

Editor's Notes

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