Ventricular tachycardias are defined as 3 or more consecutive QRS complexes originating from the ventricles at a rate over 100 beats per minute. They can arise via reentry, automaticity, or triggered mechanisms. Sustained ventricular tachycardias last over 30 seconds, while non-sustained last less than 30 seconds. Wide complex tachycardias need to be differentiated between ventricular tachycardia and supraventricular tachycardia with aberrancy or preexcitation. Management involves determining the rate, origin, and cause of the ventricular tachycardia and using cardioversion, antiarrhythmic drugs like amiodarone, or ablation as appropriate.
Idiopathic VT refers to VT occurring in structurally normal hearts in the absence of myocardial scarring. Classification of monomorphic idiopathic VT includes outflow tract VT, fascicular VT, papillary muscle VT,annular VT, and miscellaneous (VT from the body of the RV and crux of
the heart). It is commonly seen in young patients and usually has a benign course. The 12-lead lectrocardiogram is critical in distinguishing the specific form and locations of idiopathic VT. Treatment options include medical therapy specific to the underlying mechanism of VT or catheter
ablation.
Ventricular tachycardia (VT) is a broad complex tachycardia originating from a ventricular ectopic focus. It is defined as three or more ventricular extrasystoles in succession at a rate of more than 120 beats per minute (bpm). Accelerated idioventricular rhythm refers to ventricular rhythms with rates of 100-120 bpm
Ventricular tachycardia are difficult to understand. it is classified in to two types. 1. VT in structurally normal heart, 2. VT in heart with structural diseases. I have tried to simplify the VT in structurally normal heart, which may be helpful to many students and learners.
Idiopathic VT refers to VT occurring in structurally normal hearts in the absence of myocardial scarring. Classification of monomorphic idiopathic VT includes outflow tract VT, fascicular VT, papillary muscle VT,annular VT, and miscellaneous (VT from the body of the RV and crux of
the heart). It is commonly seen in young patients and usually has a benign course. The 12-lead lectrocardiogram is critical in distinguishing the specific form and locations of idiopathic VT. Treatment options include medical therapy specific to the underlying mechanism of VT or catheter
ablation.
Ventricular tachycardia (VT) is a broad complex tachycardia originating from a ventricular ectopic focus. It is defined as three or more ventricular extrasystoles in succession at a rate of more than 120 beats per minute (bpm). Accelerated idioventricular rhythm refers to ventricular rhythms with rates of 100-120 bpm
Ventricular tachycardia are difficult to understand. it is classified in to two types. 1. VT in structurally normal heart, 2. VT in heart with structural diseases. I have tried to simplify the VT in structurally normal heart, which may be helpful to many students and learners.
Its a medical presentation describing how to approach to various cardiac arrhythmias in systematic way. Illustrated with more ECG photographs from standard sources.
Its a medical presentation describing how to approach to various cardiac arrhythmias in systematic way. Illustrated with more ECG photographs from standard sources.
June 3, 2024 Anti-Semitism Letter Sent to MIT President Kornbluth and MIT Cor...Levi Shapiro
Letter from the Congress of the United States regarding Anti-Semitism sent June 3rd to MIT President Sally Kornbluth, MIT Corp Chair, Mark Gorenberg
Dear Dr. Kornbluth and Mr. Gorenberg,
The US House of Representatives is deeply concerned by ongoing and pervasive acts of antisemitic
harassment and intimidation at the Massachusetts Institute of Technology (MIT). Failing to act decisively to ensure a safe learning environment for all students would be a grave dereliction of your responsibilities as President of MIT and Chair of the MIT Corporation.
This Congress will not stand idly by and allow an environment hostile to Jewish students to persist. The House believes that your institution is in violation of Title VI of the Civil Rights Act, and the inability or
unwillingness to rectify this violation through action requires accountability.
Postsecondary education is a unique opportunity for students to learn and have their ideas and beliefs challenged. However, universities receiving hundreds of millions of federal funds annually have denied
students that opportunity and have been hijacked to become venues for the promotion of terrorism, antisemitic harassment and intimidation, unlawful encampments, and in some cases, assaults and riots.
The House of Representatives will not countenance the use of federal funds to indoctrinate students into hateful, antisemitic, anti-American supporters of terrorism. Investigations into campus antisemitism by the Committee on Education and the Workforce and the Committee on Ways and Means have been expanded into a Congress-wide probe across all relevant jurisdictions to address this national crisis. The undersigned Committees will conduct oversight into the use of federal funds at MIT and its learning environment under authorities granted to each Committee.
• The Committee on Education and the Workforce has been investigating your institution since December 7, 2023. The Committee has broad jurisdiction over postsecondary education, including its compliance with Title VI of the Civil Rights Act, campus safety concerns over disruptions to the learning environment, and the awarding of federal student aid under the Higher Education Act.
• The Committee on Oversight and Accountability is investigating the sources of funding and other support flowing to groups espousing pro-Hamas propaganda and engaged in antisemitic harassment and intimidation of students. The Committee on Oversight and Accountability is the principal oversight committee of the US House of Representatives and has broad authority to investigate “any matter” at “any time” under House Rule X.
• The Committee on Ways and Means has been investigating several universities since November 15, 2023, when the Committee held a hearing entitled From Ivory Towers to Dark Corners: Investigating the Nexus Between Antisemitism, Tax-Exempt Universities, and Terror Financing. The Committee followed the hearing with letters to those institutions on January 10, 202
Read| The latest issue of The Challenger is here! We are thrilled to announce that our school paper has qualified for the NATIONAL SCHOOLS PRESS CONFERENCE (NSPC) 2024. Thank you for your unwavering support and trust. Dive into the stories that made us stand out!
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Antifertility, Toxicity studies as per OECD guidelines
The French Revolution, which began in 1789, was a period of radical social and political upheaval in France. It marked the decline of absolute monarchies, the rise of secular and democratic republics, and the eventual rise of Napoleon Bonaparte. This revolutionary period is crucial in understanding the transition from feudalism to modernity in Europe.
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Synthetic Fiber Construction in lab .pptxPavel ( NSTU)
Synthetic fiber production is a fascinating and complex field that blends chemistry, engineering, and environmental science. By understanding these aspects, students can gain a comprehensive view of synthetic fiber production, its impact on society and the environment, and the potential for future innovations. Synthetic fibers play a crucial role in modern society, impacting various aspects of daily life, industry, and the environment. ynthetic fibers are integral to modern life, offering a range of benefits from cost-effectiveness and versatility to innovative applications and performance characteristics. While they pose environmental challenges, ongoing research and development aim to create more sustainable and eco-friendly alternatives. Understanding the importance of synthetic fibers helps in appreciating their role in the economy, industry, and daily life, while also emphasizing the need for sustainable practices and innovation.
2. V. TACHYCARDIA
• 3 or More consecutive QRS complexes of ventricular
origin at a rate of > 100 beats / min
• VTs arise distal to bifurcation of HIS bundle
• THREE MECHANISMs
– RE-ERNTRY (90 % VT with IHD)
– AUTOMATICITY ( escape rhythms, idiopathic VTs )
– TRIGGERED ( digitalis , catecholaminergic )
3. Some TERMS
• Sustained
– For More than 30 seconds
• Non Sustained
– For Less than 30 seconds
• Frequent PVCs
– > 5 / minute
– > 30 / hour
• Occasional PVCs
5. Wide Complex Tachycardias
• Any WCT in a patient with IHD is VT
• If you don’t know the reason, its VT
• Horizontal entry of patient in ER, is VT
6. Wide Complex Tachycardias
• Regular
– VT
– Abberant ventricular conduction
– Preexisting LBBB or RBBB
– Preexisting Nonspecific Intra ventricular conduction defects
– Anterograde conduction with pre excitation ( WPW with antedromic AVRT )
– Anterograde conduction over atriofascicular or nodo ventricular connection
• Irregular
– A Fib with abberant conduction/ BBB/ IVCD
– A fib with venricular pre excitation
– Polymorphic VT ( catecholaminergic )
– Torsade de Pointes
7. Differentiating points
VT
History of MI
AV dissociation ( Pathognomic )
Capture beats
Fusion beats
Extreme Axis
Very Broad complexes ( > 140 ms )
No response to Vagal manouvers or adenosine
8. Fusion beat• Marriot et al Criteria
1. Contour and Duration of QRS are intermediate
2. PR of Fusion < PR of supraventricular complex
3. PS (PJ) of Fusion > PR of supraventricular complex
4. Vector of Fusion complex is always different from
supraventricular
• EXCEPTIONs
– BBB/ IVCD/ SVT with variable Av conduction times
11. How to Assess a WCT
• Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
• Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
12. BRUGADA (1991)
• Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
SVT WITH ABBERANCY
RS100PM
Brugada’s sign
13. V1,2,6 Based Criteria
used by BRUGADA
• Wellens Criteria for VT-RBBB (1978)
– V1
• Monophasic R
• Biphasic qR, QR, RS
– V6
• rS , QS , qR
• Kindwall Criteria for VT-LBBB(1988)
– V1 & V2
• R > 30ms
– V1 & V2 Start of q to nadir of s > 60ms
– Notch on Downslop of S in V1 / V2
– Any Q in V6
• Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
14. V1,2,6 Based Criteria
used by BRUGADA
• Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
LBBB
• Delayed onset of intrinsicoid deflection in leads I, V5, V6
• Broad monophasic R waves in leads I, V5, V6 that are
usually notched or slurred
• rS or QS complex in right precordial leads
RBBB
• rsR’ or rSR
• Delayed onset of intrinsicoid deflection (beginning
of QRS to peak of R wave > 0.05 seconds) in V1
and V2
• Wide slurred S wave in leads I, V5, and V6
15. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
QRSP
17. Wide Complex Tachycardias
• Any WCT in a patient with IHD is VT
• If you don’t know the reason, its VT
• Horizontal entry of patient in ER, is VT
18. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
STEP 1
STEP 2
Tip shows the origin
19. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
20. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
21. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
23. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
Lead iii ulti
24. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
25. • General Management
– Unstable
• DCC
– Synchronous for VT with pulse; Asynchronus for pulseless VT
– Stable
• Medical initiallly
• Search reversible cause and correct
– Ischemia
– Electrolyte imbalance
– Bradycardia
– Hypotension
– drugs
• Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
26. • Acute medical therapy
– Amiodarone ( agent of choice )
– Lidocaine ( ischemic origin )
– Procainamide
– B blockers ( for ACS, idiopathic RVOT )
– Calcium channel blockers ( RVOT, fascicular and digitalis )
– MgSO4 ( Torsade de pointes )
– Sodium Bicarbonate ( Acidosis ; TCA )
• RFA ( Curative for 90% of idiopathic VT )
• Surgical ablation ( for scar VT; with aneurysmectomy )
• ATP ( anti tachycardia pacing )
• Alcohol injection in coronary branch for the area
• Prevention
– B blockers ( primary )
– ICD ( primary and secondary )
• Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
27. Amiodarone
• Intravenous infusion
– Initial 5 mg/ kg over 20-120 minutes with ecg
monitoring; subsequent infusion given if necessary
according to response upto max. 1.2 g in 24 hours
• Pulseless VT or V fib. ( ACLS )
– Pre filled syringe or 300 mg diluted in 20 ml glucose 5
% after adrenaline if refractory to defibrillation.
Additional dose of 150 mg can be given by IV injection
if nececcary followed by infusion of 900 mg over 24
hours
Reference; BNF 67
28. Amiodaroone
• 5mg / kg bolus = 300 mg for 60 kg person
• INFUSION
– 1 mg / min for 6 hours
– ½ mg /min for 18 hours
24 hours
2 injections ( 300 mg ) in 100 ml burrette at 20 microdrops / min for 6 hours
then
At 10 microdrops for 18 hours
29. Amiodarone
• Oral
– Start: LOAD 800-1600mg PO qd x 1-3 wk until
response
• (for loading dose more than 1000mg / day divided w/
meals bid – tid)
– Maintenence
• 200-600 mg PO qd
• Divided bid-tid if GI intolerence
30. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
31. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT
32. • Rate
• QRS duration
• Differentiate from SVT with Abberancy
• Differentiate from pre-exicitation
• Focus of VT
• Cause of VT
• Manage VT