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AN APPROACH
TO WIDE
COMPLEX
TACHYCARDIA
PRESENTOR : Dr NIHANTH REDDY V
MODERATOR : Dr KUMAR NARAYANAN
DEFINITION
Wide QRS Tachycardia is defined by a ventricular rate > 100/min and
QRS width > 120 milliseconds
CLASSIFICATION
WHY IS THERE A WIDENED
QRS ?
A widened QRS complex occurs
when ventricular contraction is
abnormally slow for one of the
following reasons :
1. The arrhythmia originates
outside of the normal
conduction system and
below the AV node -
Ventricular tachycardia
2. Abnormalities within the His
Purkinje system – SVT with
aberrancy
3. Pre excitation with an SVT
conducting antegrade over an
accessory pathway, resulting in
direct activation of ventricular
myocardium
• Atrial
fibrillation/flutter/tachycardia
with aberrant conduction or
antegrade conduction via an
accessory pathway
• Polymorphic VT
• Ventricular fibrillation
APPROACH TO WIDE COMPLEX TACHYCARDIA
• INITIAL APPROACH :
1. Assessment of hemodynamic status :
Unstable - Cardioversion irrespective of the mechanism of arrhythmia
Stable – In a stable patient, a focused clinical evaluation should include
the following :
History
Physical examination
Ancillary testing
Diagnostic maneuvers in selected patients
CLINICAL CLUES TO WIDE COMPLEX
TACHCYARDIA
VENTRICULAR TACHYCARDIA SVT
Age > 35 – 85 % Age< 35 years – 70 %
Old MI
Structural heart disease – 90 %
Signs of acute or chronic HF
Presence of ICD
Atrial arrhythmias with regular WCT
Structurally normal heart
A healed sternal incision as evidence of previous
cardiac surgery
A sequalae of peripheral artery disease or stroke
Family H/O SCD
Drugs – IC, III, LQTC Pre existing BBB/WPW
Electrolytes
DICTUM
• When the diagnosis of SVT remains uncertain, it is recommended that
the patient be treated as if the rhythm is VT until definitely proven
otherwise
• Unstable patients should be presumed to have VT and treated as such
APPROACH TO WIDE COMPLEX TACHYCARDIA
• When analyzing an ECG with a wide QRS complex, a systematic
approach is advised
• If available, comparison with a baseline ECG tracing in sinus rhythm or
atrial fibrillation is helpful
• Helpful clues on a baseline ECG may include fascicular and/or bundle
branch block, signs of prior infarction, or ventricular pre-excitation
• The following steps are recommended
1. Assessment of rate
2. Assessment of rhythm – regular or irregular ?
3. What is the QRS axis, duration ?
4. Presence and pattern of atrial activity (p wave ) should be identified
5. Relationship between atrial and ventricular activity should be
determined
6. Wide QRS morphology should be evaluated
• What is the rate ?
• Is the rhythm regular or irregular ?
 The rate of the WCT is of limited use in distinguishing VT from SVT
VT is generally regular. Slight variation in RR intervals is sometimes
seen and suggests VT as opposed to most SVTS, which are
characterized by uniformity of RR intervals
Warm up phenomenon – suggests VT
Marked irregularity of RR intervals occurs in polymorphic VT and in
AF aberrant conduction
WARM UP PHENOMENON
• If the start of tachycardia is recorded, it
is valuable to assess the initial RR
beats.
• If the RR intervals during the start of
tachycardia is irregular – it suggests VT
• This phenomenon is referred to as
warm up phenomenon and is
characteristic of VT
WHAT IS THE QRS AXIS,
DURATION AND
MORPHOLOGY ?
• QRS axis : Extreme right axis deviation
strongly favors VT
• Compared with the axis during sinus
rhythm of the old ECG, an axis shift during
the WCT of more than 40 degrees
suggests VT
• In patients with an RBBB like WCT, a QRS
axis to the left of -30 degrees suggests a
VT
• In patients with an LBBB like WCT, a QRS
axis to the right of +90 degrees suggests a
VT
• QRS duration : By definition,
the QRS duration is at least
120 milli seconds in a WCT
• In general, a wider QRS > 160
milliseconds favors VT
• In an RBBB-like WCT, a QRS
duration > 140 milliseconds
suggests VT
• In an LBBB-like WCT, a QRS
duration > 160 milliseconds
suggests VT
• CONCORDANCE : Concordance is present
when QRS complexes in all precordial leads
are monophasic with same polarity
• When present, concordance is frequently
associated with VT
• Positive concordance : V1-V6 entirely
positive with tall monophasic R waves
• Negative concordance : V1-V6 entirely
negative with deep monophasic QS
complexes – strongly suggestive of VT
• Three additional findings suggestive for a ventricular origin
tachycardia include :
1. VPC’s during SR with the same QRS configuration as during VT
2. QRS width during tachycardia less than QRS width during SR
3. A bundle branch like configuration during VT which is different from
the bundle branch block pattern during SR
• AV DISSOCIATION : When identified on ECG,
the presence of AV dissociation largely
establishes VT as the diagnosis
• AV is dissociation is characterized by atrial
activity that is independent of ventricular
activity, with ventricular rate exceeding the
atrial rate
• FUSION AND CAPTURE BEATS : Fusion and/or capture beats, when
identified on the surface ECG in a patient with WCT, are diagnostic for VT
• Fusion beats : Occur when one impulse originating from the ventricle and a
second supraventricular impulse simultaneously activate the ventricular
myocardium. The resulting QRS complex has a morphology intermediate
between that of a sinus beat and a purely ventricular complex
• Capture beats/Dressler beats : QRS complexes during a WCT that are
identical to the sinus QRS complex. It implies that the normal conduction
system has momentarily captured control of ventricular activation from the
VT focus
• Fusion and capture beats are noticed when the tachycardia rate is slower
QRS MORPHOLOGY
ECG
CRITERIA
FAVORING
VT THAN SVT
• There is no single criterion or combination of criteria that provides
complete diagnostic accuracy in evaluating a WCT
• It is therefore necessary to integrate multiple ECG findings into a
diagnostic strategy
• Of the several strategies proposed, the Brugada criteria is the most
widely used
VERECKI
ALGORITHM
WIDE QRS TACHYCARDIA WITH IRREGULAR
RHYTHM
• Atrial fibrillation or flutter with variable AV conduction
1. Bundle branch block
2. Accessory pathway
• Torsade's de pointes
THANK YOU

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Approach to Wide QRS tachycardia.pptx

  • 1. AN APPROACH TO WIDE COMPLEX TACHYCARDIA PRESENTOR : Dr NIHANTH REDDY V MODERATOR : Dr KUMAR NARAYANAN
  • 2. DEFINITION Wide QRS Tachycardia is defined by a ventricular rate > 100/min and QRS width > 120 milliseconds
  • 4. WHY IS THERE A WIDENED QRS ? A widened QRS complex occurs when ventricular contraction is abnormally slow for one of the following reasons : 1. The arrhythmia originates outside of the normal conduction system and below the AV node - Ventricular tachycardia
  • 5. 2. Abnormalities within the His Purkinje system – SVT with aberrancy
  • 6. 3. Pre excitation with an SVT conducting antegrade over an accessory pathway, resulting in direct activation of ventricular myocardium
  • 7. • Atrial fibrillation/flutter/tachycardia with aberrant conduction or antegrade conduction via an accessory pathway
  • 8. • Polymorphic VT • Ventricular fibrillation
  • 9. APPROACH TO WIDE COMPLEX TACHYCARDIA • INITIAL APPROACH : 1. Assessment of hemodynamic status : Unstable - Cardioversion irrespective of the mechanism of arrhythmia Stable – In a stable patient, a focused clinical evaluation should include the following : History Physical examination Ancillary testing Diagnostic maneuvers in selected patients
  • 10. CLINICAL CLUES TO WIDE COMPLEX TACHCYARDIA VENTRICULAR TACHYCARDIA SVT Age > 35 – 85 % Age< 35 years – 70 % Old MI Structural heart disease – 90 % Signs of acute or chronic HF Presence of ICD Atrial arrhythmias with regular WCT Structurally normal heart A healed sternal incision as evidence of previous cardiac surgery A sequalae of peripheral artery disease or stroke Family H/O SCD Drugs – IC, III, LQTC Pre existing BBB/WPW Electrolytes
  • 11. DICTUM • When the diagnosis of SVT remains uncertain, it is recommended that the patient be treated as if the rhythm is VT until definitely proven otherwise • Unstable patients should be presumed to have VT and treated as such
  • 12. APPROACH TO WIDE COMPLEX TACHYCARDIA • When analyzing an ECG with a wide QRS complex, a systematic approach is advised • If available, comparison with a baseline ECG tracing in sinus rhythm or atrial fibrillation is helpful • Helpful clues on a baseline ECG may include fascicular and/or bundle branch block, signs of prior infarction, or ventricular pre-excitation
  • 13. • The following steps are recommended 1. Assessment of rate 2. Assessment of rhythm – regular or irregular ? 3. What is the QRS axis, duration ? 4. Presence and pattern of atrial activity (p wave ) should be identified 5. Relationship between atrial and ventricular activity should be determined 6. Wide QRS morphology should be evaluated
  • 14. • What is the rate ? • Is the rhythm regular or irregular ?  The rate of the WCT is of limited use in distinguishing VT from SVT VT is generally regular. Slight variation in RR intervals is sometimes seen and suggests VT as opposed to most SVTS, which are characterized by uniformity of RR intervals Warm up phenomenon – suggests VT Marked irregularity of RR intervals occurs in polymorphic VT and in AF aberrant conduction
  • 15. WARM UP PHENOMENON • If the start of tachycardia is recorded, it is valuable to assess the initial RR beats. • If the RR intervals during the start of tachycardia is irregular – it suggests VT • This phenomenon is referred to as warm up phenomenon and is characteristic of VT
  • 16. WHAT IS THE QRS AXIS, DURATION AND MORPHOLOGY ? • QRS axis : Extreme right axis deviation strongly favors VT • Compared with the axis during sinus rhythm of the old ECG, an axis shift during the WCT of more than 40 degrees suggests VT • In patients with an RBBB like WCT, a QRS axis to the left of -30 degrees suggests a VT • In patients with an LBBB like WCT, a QRS axis to the right of +90 degrees suggests a VT
  • 17. • QRS duration : By definition, the QRS duration is at least 120 milli seconds in a WCT • In general, a wider QRS > 160 milliseconds favors VT • In an RBBB-like WCT, a QRS duration > 140 milliseconds suggests VT • In an LBBB-like WCT, a QRS duration > 160 milliseconds suggests VT
  • 18. • CONCORDANCE : Concordance is present when QRS complexes in all precordial leads are monophasic with same polarity • When present, concordance is frequently associated with VT • Positive concordance : V1-V6 entirely positive with tall monophasic R waves • Negative concordance : V1-V6 entirely negative with deep monophasic QS complexes – strongly suggestive of VT
  • 19. • Three additional findings suggestive for a ventricular origin tachycardia include : 1. VPC’s during SR with the same QRS configuration as during VT 2. QRS width during tachycardia less than QRS width during SR 3. A bundle branch like configuration during VT which is different from the bundle branch block pattern during SR
  • 20. • AV DISSOCIATION : When identified on ECG, the presence of AV dissociation largely establishes VT as the diagnosis • AV is dissociation is characterized by atrial activity that is independent of ventricular activity, with ventricular rate exceeding the atrial rate
  • 21. • FUSION AND CAPTURE BEATS : Fusion and/or capture beats, when identified on the surface ECG in a patient with WCT, are diagnostic for VT • Fusion beats : Occur when one impulse originating from the ventricle and a second supraventricular impulse simultaneously activate the ventricular myocardium. The resulting QRS complex has a morphology intermediate between that of a sinus beat and a purely ventricular complex • Capture beats/Dressler beats : QRS complexes during a WCT that are identical to the sinus QRS complex. It implies that the normal conduction system has momentarily captured control of ventricular activation from the VT focus • Fusion and capture beats are noticed when the tachycardia rate is slower
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  • 26. • There is no single criterion or combination of criteria that provides complete diagnostic accuracy in evaluating a WCT • It is therefore necessary to integrate multiple ECG findings into a diagnostic strategy • Of the several strategies proposed, the Brugada criteria is the most widely used
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  • 29. WIDE QRS TACHYCARDIA WITH IRREGULAR RHYTHM • Atrial fibrillation or flutter with variable AV conduction 1. Bundle branch block 2. Accessory pathway • Torsade's de pointes
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Editor's Notes

  1. Classic monomorphic VT with uniform QRS complexes Indeterminate axis Very broad QRS (~200 ms) Notching near the nadir of the S wave in lead III = Josephson’s sign
  2. Av dissociation Capture beats Avr positive
  3. The rationale behind the vi/vt criterion is that during WCT due to SVT the initial activation of the septum (occurring either left-to-right or right-to-left) should be invariably rapid over the normal His-Purkinje system and the intraventicular conduction delay causing the wide QRS complex occurs in the mid to terminal part of the QRS, thus the vi/vt >1 during SVT. During WCT due to VT, however, an initial slower muscle-to-muscle spread of activation occurs until the impulse reaches the His-Purkinje system, after which the rest of the ventricular muscle is more rapidly activated, thus, the vi/vt ≤1 during VT.