This document discusses the pathophysiology and evaluation of blunt ocular trauma using ultrasound techniques. It begins by explaining the different mechanisms of blunt trauma impact depending on the size of the object hitting the eye. It then outlines the different locations within the eye that can be injured, including the anterior segment, posterior segment, adnexa, and orbit. The document proceeds to describe specific injuries that can occur to structures like the cornea, iris, lens, vitreous, retina, and optic nerve. It emphasizes the importance of ultrasound, particularly ultrasonography and ultrasound biomicroscopy, in evaluating intraocular injuries and structural abnormalities in cases of blunt trauma where clinical examination is difficult or limited. Finally, it provides examples of ultrasound findings from
-IOL formula
1st generation formula : SRK, Binkhost
2nd generation formula : SRK II
3rd generation formula: Hoffer Q, Holladay 1, SRK/T
4th generation formula: Haigis, Holladay 2, Olsen
-The Hoffer Q, Holladay I, and SRK/T formula are all commonly used.
-IOL formula
1st generation formula : SRK, Binkhost
2nd generation formula : SRK II
3rd generation formula: Hoffer Q, Holladay 1, SRK/T
4th generation formula: Haigis, Holladay 2, Olsen
-The Hoffer Q, Holladay I, and SRK/T formula are all commonly used.
Ultrasonic Evaluation of Eyes With Blunt Trauma
Thesis
Submitted for partial fulfillment of the master degree in ophthalmology
By
Mohamed Ahmed El-Shafie
Resident in ophthalmology department
Mansoura University
MANAGEMENT OF MACULAR HOLE, Ophthalmology presentation, eye care in the elderly , macular hole as a consequence of trauma, Vitreoretinal surgical cases, ,
MYOPIA , basics , causes , types and treatmentssuserde6356
Myopia, also known as near-sightedness and short-sightedness, is an eye disease[5][6][7] where light from distant objects focuses in front of, instead of on, the retina.[1][2][6] As a result, distant objects appear blurry while close objects appear normal.[1] Other symptoms may include headaches and eye strain.[1][8] Severe myopia is associated with an increased risk of macular degeneration, retinal detachment, cataracts, and glaucoma.[2][9]
Myopia results from the length of the eyeball growing too long or less commonly the lens being too strong.[1][10] It is a type of refractive error.[1] Diagnosis is by the use of cycloplegics during eye examination.[11]
Tentative evidence indicates that the risk of myopia can be decreased by having young children spend more time outside.[12][13] This decrease in risk may be related to natural light exposure.[14] Myopia can be corrected with eyeglasses, contact lenses, or by refractive surgery.[1][15] Eyeglasses are the simplest and safest method of correction.[1] Contact lenses can provide a relatively wider corrected field of vision, but are associated with an increased risk of infection.[1][16] Refractive surgeries like LASIK and PRK permanently change the shape of the cornea. Surgeries like Implantable Collamer Lens (ICL) implant a lens inside the anterior chamber in front of the natural eye lens. ICL doesn't affect the cornea.[
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Acute Limb Weakness
case presentation
PBL session
3rd year
neuro ophthalmology
new mansoura university
A 54-year-old man, Mr. Stephen Smith, was brought by ambulance to the Emergency Department. He had woken up from sleep with slurring of speech and weakness of his right arm and leg. His wife was extremely distressed as Mr Smith had been perfectly well the previous night when he went to sleep. Within 20 minutes after the initial call was made Mr. Smith was admitted to the Emergency Department and was reviewed by the SpR covering the Regional Specialist Stroke Unit. Mr. Smith had been on regular antihypertensive medication (lisinopril) for 8 years. He smoked 5-8 cigarettes a day and was a social drinker consuming about 6 units of alcohol a week. He was not diabetic.
His Serum lipids were checked and was advised to reduce weight and started on a Statin (Simvastatin). There was no family history of hyperlipidaemia but his grandfather died after a Stroke. Mr. Smith had an urgent appendectomy 1 week and made an uneventful recovery. He lives with his wife in a 4-bedroom detached house.
Neurological examination showed that Mr. Smith was fully conscious and alert. He had an upper motor neuron facial palsy on the right side. He had expressive dysphasia but appeared to comprehend speech. He was just able to lift his right arm off the bed for a short period but had no grip. His right leg was weak.
Reflexes on the right side were exaggerated and his right plantar was extensor. He responded to touch and pin prick equally on both sides. He either had visual inattention or a visual field defect on the right side. He had no papilloedema His blood pressure was 164/96, pulse 84 per min, regular. Other systems were entirely normal.
ECG and all routine blood tests were performed. An emergency CT scan was requested and even though the scan was normal the SpR ruled out emergency thrombolysis in this instance. Mr. Smith was admitted to the Acute Stroke Unit and was seen by the Stroke Consultant. Mr. Smith remained fully conscious and alert but had some difficulty in swallowing. Hence an intravenous infusion was commenced and an alternate strategy was adopted for providing his nutritional requirements. A carotid Doppler scan was requested.
After 3 weeks, Mr. Smith seemed to make good progress and the MDT meeting recorded a consistent improvement in his Barthel Index. The Stroke Team met him with his wife and discussed arrangements for discharge home. His wife was keen to know what support measures were available to them when Mr. Smith returned home. She also wanted to know about the risk of a future stroke and how this could be cut down.
ILOs:-
1- Consider the differential diagnosis of speech.
2- Discuss the risk factors for stroke and primary prevention of stroke.
3- Discuss the overall management of a patient with an acute stroke.
4- Complications of stroke
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Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
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Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
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(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
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Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
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1. UBM and US of Eye
Mohamed ELShafie
Assistant lecturer of ophthalmology
Kafr ELShiekh university
2. Pathophysiology of Blunt Ocular Trauma
If a large object hits the eye,
most of the impact is usually
taken by the orbital margin.
If a small object hits the eye, the
eye itself may take most of the
impact.
3. There are four main mechanisms:
1- Coup (injury at the same point)
2- Contrecoup (injury at the opposite point)
3- Equatorial expansion
4- Global repositioning
Mechanism of Blunt Ocular Trauma
16. Orientations of the B-scan Probe
• Axial:
Lesion in relation to lens &optic nerve .
•Transverse:
Lateral extent, 6 clock hours .
•Longitudinal:
AP extent,1 clock hour.
17.
18. Ultrasound biomicrscopy
UBM uses high frequency
ultrasound (50-100MHZ) to
produce images of the eye with
high resolution (50 um) with
reduced depth of penetration
(5mm).
19. Structural abnormalities
Guide to treatment
Follow up after treatment
A new method for gonioscopy and
quantitative angle measurement
20. Orientations of the UBM probe
• Transverse section
Lateral extent
• Radial section
21. Examination Technique of UBM:
• Patient is lying down in supine position
• Monitor is at comfortable height
• Hand controller is in accessible position.
• Eye cup of suitable size separate the two lids, filled with saline solution.
24. Male patient of 45 years old was exposed to
blunt trauma 2 years ago .. Clinical
examination show traumatic cataract
B-scan US show rupture of posterior
capsule which cant be detected by clinical
examination
25. A case with Vit. Hge that couldn't be detected
clinically due to corneal oedema
26. A case with RD
Retinal break could be localized only by US
27. A case with PVD
Mobility of PVD is more than RD.
PVD becomes more prominent in higher gain settings
33. 25 years old man exposed to blunt trauma ..
Clinically slit lamp showed corneal oedema,
which mask visaulization of the anterior
segment
UBM examination showed subluxated lens
with vitreous prolapsed in AC.
34. A case with iridodialysis
separation of the iris root from its attachment to the ciliary body
I can apply the probe in different techniques:in transverse I apply brope tangential to limbus..in long probe is perpendicular to limbus,mark to ward limbus…in axial brope through cornea
The probe was placed over the limbus in radial fashion
with anesthetic drops. The...
Fresh hemorrhage dots or lines Old hemorrhage dots gets brighter
bright continuous, folded mem. Of high spike with insertion into the disc and ora serrata.
Mobility of PVD is more than RD. PVD becomes more prominent in higher gain settings
Adherence of posterior hyaloid to peripheral retinal tear
Retinal step sign from an edematous retina to bare sclera. Vitreous hemorrhage
separation of the iris root from its attachment to the ciliary body
tear is in the ciliary body itself, between the circular and the longitudinal fibers