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• Testicular torsion refers to the torsion of the
spermatic cord structures and subsequent
loss of the blood supply to the ipsilateral
testicle.
• This is a urological emergency; early
diagnosis and treatment are vital to saving
the testicle and preserving future fertility.
• Testicular torsion is primarily a disease of
adolescents and neonates. It is the most
common cause of testicular loss in these
age groups.
INTRODUCTION
Normal anatomy
• The tunica vaginalis does not
completely surround the testis and
epididymis, which are attached to the
posterior scrotal wall.
Anatomy review
• Bell-clapper anomaly.
The tunica vaginalis completely surrounds
the testis, epididymis, and part of the
spermatic cord, predisposing to torsion.
Anatomy review
TESTICULAR ARTERIAL ANATOMY
Testicular artery
– Branch off aorta
– Major intra-testicular blood supply
• Cremaster and deferential artery
– Extra-testicular
Anatomy review
• Colour Doppler should reveal bilaterally symmetric and relatively
uniform flow through both testes and epididymides.
NORMAL ULTRASOUND AND DOPPLER FINDINGS
• Spectral Doppler tracings of testicular arterial inflow demonstrate
relatively low resistance
NORMAL ULTRASOUND AND DOPPLER FINDINGS
• The cremasteric and deferential arteries which have relatively
high resistance to flow.
• The normal testicular artery resistive indices in adults range from
46% to 78%, with a mean of 64%.
NORMAL ULTRASOUND AND DOPPLER FINDINGS
ULTRASOUND: SPECTRAL
DOPPLER
Extratesticular blood flow-
High resistance, low flow
Intratesticular blood flow-
Low resistance, high flow
• Torsion occurs as the testicle rotates between 90° and 180°,
compromising blood flow to and from the testicle.
• Complete torsion usually occurs when the testicle twists 360° or more;
incomplete or partial torsion occurs with lesser degrees of rotation.
The degree of torsion may extend to 720°.
• The twisting of the testicle causes venous occlusion and engorgement
as well as arterial ischemia and infarction of the testicle.
• The degree of torsion the testicle endures may play a role in the
viability of the testicle over time.
• In addition to the extent of torsion, the duration of torsion prominently
influences the rates of both immediate salvage and late testicular
atrophy. Testicular salvage is most likely if the duration of torsion is less
than 6-8 hours.
• If 24 hours or more elapse, testicular necrosis develops in most
patients.
PATHOPHYSIOLOGY
Intravaginal torsion
Is the more common type, occurring
most frequently at puberty. It results
from anomalous suspension of the testis
by a long stalk of spermatic cord,
resulting in complete investment of the
testis and epididymis by the tunica
vaginalis.
• This anomaly has been likened to a
bell-clapper
TWO TYPES OF TESTICULAR
TORSION
Extravaginal torsion
• Most often occurs in newborns
without the “bell clapper”
deformity.
• It is thought to result from a poor or
absent attachment of the testis to
the scrotal wall, allowing rotation of
the testis, epididymis, and tunica
vaginalis as a unit and causing
torsion of the cord at the level of
the external ring
TWO TYPES OF TESTICULAR
TORSION
Testicle rotates on spermatic
cord

Venous occlusion, edema

Arterial ischemia

Infarction
• Severe unilateral scrotal pain
• Previous episodes, spontaneous resolution
• Related to activity, trauma, during sleep
• Nausea, vomiting, abdominal pain, fever
HISTORY
• Prenatal torsion, firm, hard, scrotal
mass, which does not transilluminate
in an otherwise asymptomatic
newborn male. The scrotal skin
characteristically fixes to the
necrotic gonad.
• Older patient, swollen, tender, high-
riding testis with abnormal transverse
lie and loss of the cremasteric reflex
PHYSICAL EXAMINATION
• CLINICAL SUSPICION
• Nuclear scintigraphy
– Radiation, limited availability
• Ultrasound
– Altered echotexture (B-mode)
– Vascular flow (Color / Spectral / Power Doppler)
• Infrared scrotal Spectroscopy
DIAGNOSIS
• Sensitivity 86%, specificity 100% experienced provider using color /
power doppler1
• Gray-scale findings on ultrasound depend on how much time has
passed since the torsion occurred.
• The gray-scale findings of acute and subacute torsion are not
specific and may be seen in testicular infarction caused by
epididymitis, epididymo-orchitis, and traumatic testicular rupture or
infarction.
ULTRASOUND FOR TESTICULAR TORSION
• Early stages, scrotal contents may have a normal sonographic
appearance.
• After 4 to 6 hours, the testis becomes swollen and hypoechoic,
• After 24 hours, the testis becomes heterogeneous as a result of
hemorrhage, infarction, necrosis, and vascular congestion
• The epididymal head appears enlarged and may have
decreased echogenicity or may become heterogeneous.
• The spermatic cord immediately cranial to the testis and
epididymis is twisted, causing a characteristic torsion knot or
“whirlpool pattern” of concentric layers
ULTRASOUND FOR TESTICULAR TORSION
• A reactive hydrocele and scrotal skin thickening are often seen
with torsion.
Large, echogenic or complex extratesticular masses caused by
hemorrhage in the tunica vaginalis or epididymis may be seen in
patients with undiagnosed torsion.
ULTRASOUND FOR TESTICULAR TORSION
ULTRASOUND – B-MODE
– Early ischemia: enlargement, no Δ echogenicity
– Hemorrhage: hyperechoic areas in an
infarcted testis, heterogenous
• Late ischemia/infarct:
hypoechoic
• Color/power Doppler sonography is the most useful and
most rapid technique to establish the diagnosis of
testicular torsion and to help distinguish torsion from
epididymo-orchitis
• Blood flow is absent in the affected testicle or
significantly less than in the normal, contralateral testicle.
COLOR/POWER DOPPLER SONOGRAPHY
ULTRASOUND: COLOR DOPPLER
• Early Torsion
– No flow, echogenicity similar
• Late Torsion
– Heterogenous echotexture
– Increased extra testicular
blood flow
Meticulous scanning of the testicular parenchyma with
the use of low-flow detection Doppler settings
(low pulse repetition frequency, low wall filter, high
Doppler gain)
is important because testicular vessels are small and
have low flow velocities, especially in prepubertal
boys.
DIFFERENTIAL DIAGNOSIS OF ACUTE SCROTUM
• Epididymitis
• Scrotal abscess
• Torsion of epididymal appendage
• Intratesticular hematoma
• Is a common cause of acute scrotal pain and may mimic
testicular torsion clinically.
• Patients are rarely referred for imaging because the pain is
usually not severe, and the twisted appendage may be evident
clinically as the “blue dot” sign.
• The sonographic appearance of the twisted testicular
appendage has been described as an avascular hypoechoic
mass adjacent to a normally perfused testis and surrounded by
an area of increased color Doppler perfusion.
• However, the twisted appendage may appear as an echogenic
extratesticular mass situated between the head of the
epididymis and the upper pole of the testis.
TORSION OF THE TESTICULAR APPENDAGE
• Technetium-99m pertechnetate is the agent of choice.
• Immediate radionuclide angiograms are obtained, with
subsequent static images as well.
• In the healthy patient, images show symmetric flow to the
testes, and delayed images show uniformly symmetric
activity.
NUCLEAR IMAGING
• Static images demonstrate a photopenic area in the
involved testis.
• In the subacute and late phases of torsion (missed torsion),
there is often increased flow to the affected hemiscrotum
via the pudendal artery with a photopenic testis and a rim
of surrounding increased activity on static images. This has
been called a rim, doughnut, or bull's-eye sign.
NUCLEAR IMAGING
• Near-infrared spectroscopy (NIRS) is an emerging tool to assess
testicular torsion.
• It can measure oxygen saturation 3-4 cm deep in the skin, is rapid
(lasting 20 seconds), and is noninvasive.
• Aydogdu et al performed a small prospective study evaluating 16
adult patients with testicular torsion and found NIRS to be 100%
sensitive and specific for torsion when compared with the
contralateral testis.
NEAR-INFRARED SPECTROSCOPY
TREATMENT
• Definitive treatment: surgical
detorsion and orchioplexy
• Manual detorsion: medial to
lateral; “opening a book”
– May need to rotate 2-3 times for
complete detorsion
Roberts: Clinical Procedures in Emergency Medicine
Testicular Torsion

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Testicular Torsion

  • 1.
  • 2. • Testicular torsion refers to the torsion of the spermatic cord structures and subsequent loss of the blood supply to the ipsilateral testicle. • This is a urological emergency; early diagnosis and treatment are vital to saving the testicle and preserving future fertility. • Testicular torsion is primarily a disease of adolescents and neonates. It is the most common cause of testicular loss in these age groups. INTRODUCTION
  • 3. Normal anatomy • The tunica vaginalis does not completely surround the testis and epididymis, which are attached to the posterior scrotal wall. Anatomy review
  • 4. • Bell-clapper anomaly. The tunica vaginalis completely surrounds the testis, epididymis, and part of the spermatic cord, predisposing to torsion. Anatomy review
  • 5. TESTICULAR ARTERIAL ANATOMY Testicular artery – Branch off aorta – Major intra-testicular blood supply • Cremaster and deferential artery – Extra-testicular Anatomy review
  • 6. • Colour Doppler should reveal bilaterally symmetric and relatively uniform flow through both testes and epididymides. NORMAL ULTRASOUND AND DOPPLER FINDINGS
  • 7. • Spectral Doppler tracings of testicular arterial inflow demonstrate relatively low resistance NORMAL ULTRASOUND AND DOPPLER FINDINGS
  • 8. • The cremasteric and deferential arteries which have relatively high resistance to flow. • The normal testicular artery resistive indices in adults range from 46% to 78%, with a mean of 64%. NORMAL ULTRASOUND AND DOPPLER FINDINGS
  • 9. ULTRASOUND: SPECTRAL DOPPLER Extratesticular blood flow- High resistance, low flow Intratesticular blood flow- Low resistance, high flow
  • 10. • Torsion occurs as the testicle rotates between 90° and 180°, compromising blood flow to and from the testicle. • Complete torsion usually occurs when the testicle twists 360° or more; incomplete or partial torsion occurs with lesser degrees of rotation. The degree of torsion may extend to 720°. • The twisting of the testicle causes venous occlusion and engorgement as well as arterial ischemia and infarction of the testicle. • The degree of torsion the testicle endures may play a role in the viability of the testicle over time. • In addition to the extent of torsion, the duration of torsion prominently influences the rates of both immediate salvage and late testicular atrophy. Testicular salvage is most likely if the duration of torsion is less than 6-8 hours. • If 24 hours or more elapse, testicular necrosis develops in most patients. PATHOPHYSIOLOGY
  • 11. Intravaginal torsion Is the more common type, occurring most frequently at puberty. It results from anomalous suspension of the testis by a long stalk of spermatic cord, resulting in complete investment of the testis and epididymis by the tunica vaginalis. • This anomaly has been likened to a bell-clapper TWO TYPES OF TESTICULAR TORSION
  • 12. Extravaginal torsion • Most often occurs in newborns without the “bell clapper” deformity. • It is thought to result from a poor or absent attachment of the testis to the scrotal wall, allowing rotation of the testis, epididymis, and tunica vaginalis as a unit and causing torsion of the cord at the level of the external ring TWO TYPES OF TESTICULAR TORSION
  • 13. Testicle rotates on spermatic cord  Venous occlusion, edema  Arterial ischemia  Infarction
  • 14. • Severe unilateral scrotal pain • Previous episodes, spontaneous resolution • Related to activity, trauma, during sleep • Nausea, vomiting, abdominal pain, fever HISTORY
  • 15. • Prenatal torsion, firm, hard, scrotal mass, which does not transilluminate in an otherwise asymptomatic newborn male. The scrotal skin characteristically fixes to the necrotic gonad. • Older patient, swollen, tender, high- riding testis with abnormal transverse lie and loss of the cremasteric reflex PHYSICAL EXAMINATION
  • 16. • CLINICAL SUSPICION • Nuclear scintigraphy – Radiation, limited availability • Ultrasound – Altered echotexture (B-mode) – Vascular flow (Color / Spectral / Power Doppler) • Infrared scrotal Spectroscopy DIAGNOSIS
  • 17. • Sensitivity 86%, specificity 100% experienced provider using color / power doppler1 • Gray-scale findings on ultrasound depend on how much time has passed since the torsion occurred. • The gray-scale findings of acute and subacute torsion are not specific and may be seen in testicular infarction caused by epididymitis, epididymo-orchitis, and traumatic testicular rupture or infarction. ULTRASOUND FOR TESTICULAR TORSION
  • 18. • Early stages, scrotal contents may have a normal sonographic appearance. • After 4 to 6 hours, the testis becomes swollen and hypoechoic, • After 24 hours, the testis becomes heterogeneous as a result of hemorrhage, infarction, necrosis, and vascular congestion • The epididymal head appears enlarged and may have decreased echogenicity or may become heterogeneous. • The spermatic cord immediately cranial to the testis and epididymis is twisted, causing a characteristic torsion knot or “whirlpool pattern” of concentric layers ULTRASOUND FOR TESTICULAR TORSION
  • 19. • A reactive hydrocele and scrotal skin thickening are often seen with torsion. Large, echogenic or complex extratesticular masses caused by hemorrhage in the tunica vaginalis or epididymis may be seen in patients with undiagnosed torsion. ULTRASOUND FOR TESTICULAR TORSION
  • 20. ULTRASOUND – B-MODE – Early ischemia: enlargement, no Δ echogenicity – Hemorrhage: hyperechoic areas in an infarcted testis, heterogenous • Late ischemia/infarct: hypoechoic
  • 21. • Color/power Doppler sonography is the most useful and most rapid technique to establish the diagnosis of testicular torsion and to help distinguish torsion from epididymo-orchitis • Blood flow is absent in the affected testicle or significantly less than in the normal, contralateral testicle. COLOR/POWER DOPPLER SONOGRAPHY
  • 22. ULTRASOUND: COLOR DOPPLER • Early Torsion – No flow, echogenicity similar • Late Torsion – Heterogenous echotexture – Increased extra testicular blood flow
  • 23.
  • 24. Meticulous scanning of the testicular parenchyma with the use of low-flow detection Doppler settings (low pulse repetition frequency, low wall filter, high Doppler gain) is important because testicular vessels are small and have low flow velocities, especially in prepubertal boys.
  • 25. DIFFERENTIAL DIAGNOSIS OF ACUTE SCROTUM • Epididymitis • Scrotal abscess • Torsion of epididymal appendage • Intratesticular hematoma
  • 26. • Is a common cause of acute scrotal pain and may mimic testicular torsion clinically. • Patients are rarely referred for imaging because the pain is usually not severe, and the twisted appendage may be evident clinically as the “blue dot” sign. • The sonographic appearance of the twisted testicular appendage has been described as an avascular hypoechoic mass adjacent to a normally perfused testis and surrounded by an area of increased color Doppler perfusion. • However, the twisted appendage may appear as an echogenic extratesticular mass situated between the head of the epididymis and the upper pole of the testis. TORSION OF THE TESTICULAR APPENDAGE
  • 27.
  • 28. • Technetium-99m pertechnetate is the agent of choice. • Immediate radionuclide angiograms are obtained, with subsequent static images as well. • In the healthy patient, images show symmetric flow to the testes, and delayed images show uniformly symmetric activity. NUCLEAR IMAGING
  • 29. • Static images demonstrate a photopenic area in the involved testis. • In the subacute and late phases of torsion (missed torsion), there is often increased flow to the affected hemiscrotum via the pudendal artery with a photopenic testis and a rim of surrounding increased activity on static images. This has been called a rim, doughnut, or bull's-eye sign. NUCLEAR IMAGING
  • 30.
  • 31.
  • 32. • Near-infrared spectroscopy (NIRS) is an emerging tool to assess testicular torsion. • It can measure oxygen saturation 3-4 cm deep in the skin, is rapid (lasting 20 seconds), and is noninvasive. • Aydogdu et al performed a small prospective study evaluating 16 adult patients with testicular torsion and found NIRS to be 100% sensitive and specific for torsion when compared with the contralateral testis. NEAR-INFRARED SPECTROSCOPY
  • 33. TREATMENT • Definitive treatment: surgical detorsion and orchioplexy • Manual detorsion: medial to lateral; “opening a book” – May need to rotate 2-3 times for complete detorsion Roberts: Clinical Procedures in Emergency Medicine

Editor's Notes

  1. The right and left testicular arteries originate from the aorta just below the renal arteries. They course through the deep inguinal ring to enter the spermatic cord, accompanied by the cremasteric and deferential arteries, which supply the soft tissues of the scrotum , epididymis and vas deferens. The testicular artery penetrates the tunica albuginea along the posterior aspect of the testis and gives off capsular branches which course through the tunic a vasculosa. The se capsular branches then give rise to the centripetal arteries which carry blood from the capsular surface , centrally towards the mediastinum along the septula (Figs 13-5 and 13-6). Branches of the centripetal arteries then course backward towards the capsular surface; these are known as recurrent rami.,
  2. Transverse colour Doppler ultrasound image of both testicles using a split screen format. Note the symmetry of echotexture and the uniformity and symmetry of colour flow.
  3. Spectral Doppler waveform of a normal left testicular artery. The resistive index is normal (approx. 60%).
  4. Intrastesticular supply- TESTICULAR a off aorta, low resistance with high flow Extratesticular supply- DEFERENTIAL and CREMASTERIC a, high resistance with low flow Flow during diastole
  5. Looking at echotexture Straddle view to see portions of each in same image. Enlarged to engorged blood
  6. Epidid: enlarge hyperemic epydidymis Abscess: comples fluid collection Appendix: mass next to epididymal head with absence of flow Hematoma has surrounding blood flow.