Deformity: It’s the position of a limb/Joint, from which it cannot be brought back to its normal anatomical position.
Described as abnormalities of :
Length
Angulation
Rotation
Translation
Combination
Hoffa's Fracture: Diagnosis, management & New Classification System by BAGARI...Vaibhav Bagaria
Hoffa's Fracture - coronal split fracture of distal femur, its diagnosis, management strategy, a new classification and tips and tricks of management. First described Hoffa, a new classification system by Bagaria et al helps plan the surgery for these tricky fracture. The most crucial step is not to miss these fractures in ER.
Deformity: It’s the position of a limb/Joint, from which it cannot be brought back to its normal anatomical position.
Described as abnormalities of :
Length
Angulation
Rotation
Translation
Combination
Hoffa's Fracture: Diagnosis, management & New Classification System by BAGARI...Vaibhav Bagaria
Hoffa's Fracture - coronal split fracture of distal femur, its diagnosis, management strategy, a new classification and tips and tricks of management. First described Hoffa, a new classification system by Bagaria et al helps plan the surgery for these tricky fracture. The most crucial step is not to miss these fractures in ER.
Hand rehabilitation following flexor tendon injuriesAbey P Rajan
hand rehabilitation following flexor tendon injuries include introduction, clinical anatomy, tendon nutrition, tendon healing, post op. management, special cases, summary
Tensor fascia lata[tfl] muscle pedicle grafting for avn hip dr mohamed ashraf...drashraf369
slide presentation of a very promising surgical technic for a very elusive condition called avascular necrosis of femoral head.good clinical and surgical demo by dr mohamed ashraf,HOD, govt TD medical college ,alleppey,kerala, india
Hand rehabilitation following flexor tendon injuriesAbey P Rajan
hand rehabilitation following flexor tendon injuries include introduction, clinical anatomy, tendon nutrition, tendon healing, post op. management, special cases, summary
Tensor fascia lata[tfl] muscle pedicle grafting for avn hip dr mohamed ashraf...drashraf369
slide presentation of a very promising surgical technic for a very elusive condition called avascular necrosis of femoral head.good clinical and surgical demo by dr mohamed ashraf,HOD, govt TD medical college ,alleppey,kerala, india
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
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Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
9. Abbreviation Full Name Function
FPL Flexor pollicis longus Thumb flexion
PL Palmaris longus Flexion of wrist
FCU Flexor carpi ulnaris Flexion and adduction of hand
FDP Flexor digitorum profundus flexion of distal interphalangeal joints
and helps wrist flexion
FDS Flexor digitorum superficialis Flexion of proximal interphalangeal
joints and flexion of proximal
phalanges at metacarpophalangeal
joints
FCR Flexor carpi radialis Flexion and abduction of hand at
wrist
10. Abbreviation Full Name Function
APL Abductor pollicis longus Abduction of thumb
EPB Extensor pollicis brevis Extension of thumb proximal phalanx
ECRL
ECRB
Extensor carpi radialis longus/brevis Extends and abducts hand
EPL Extensor pollicis longus Extends thumb IP joint
ED
(4 tendons: II - V)
Extensor digitorum Extends digits II - V
EI Extensor indicis Extends digit II
EDM (EDC) Extensor digitorum minimi (quinti) Extends digit V
ECU Extensor carpi ulnaris Extends and abducts hand
11. Flexor Tendon Injury Zones
1: flexor digitorum profundus distal to
insertion of flexor digitorum
superficialis
2: insertion of flexor digitorum
superficialis to proximal edge of A1
pulley (“No Man’s Land”)
3: proximal edge of the A1 pulley to
distal edge of carpal tunnel
4: within the carpal tunnel
5: proximal to the carpal tunnel
12. Extensor Tendon
Injury Zones
O Extensor tendons are
divided into 8 zones
O Zones 1,3 and 5 lie
over the DIP, PIP and
MCP joints
18. ETIOLOGY
Common mechanisms of injury include
Osharp object direct laceration
(broken glass, kitchen knives or
table saws)
Ocrush injury
Oavulsions
Oburns
Oanimal or human bites
Osuicide attempts
19. Flexor tendon healing
• 2 forms:
– Intrinsic healing: occurs without direct blood
flow to the tendon
– Extrinsic healing: occurs by proliferation of
fibroblasts from the peripheral epitendon;
adhesions occur because of extrinsic healing of
the tendon and limit tendon gliding within
fibrous synovial sheaths
20. Phases of Intrinsic healing
1.Inflammatory (0-5 days) : strength of the
repair is reliant on the strength of the
suture itself
2.Fibroblastic (5-28 days) : or so-called
collagen-producing phase
3.Remodelling (>28 days)
23. • Region b/w middle aspects of middle phalanx
to finger tips
• Contains only one tendon-fdp
• Tendon laceration occurs close to its insertion
• Tendon to bone repair is required than tendon
repair
ZONE 1: ZONE OF FDP AVULSION
INJURIES
24. Leddy classification of zone I flexor tendon
injuries!!
Type I: tendon retracted into palm (fullness
in palm)
Type II: tendon trapped in the sheath at
PIP (unable to flex PIP)
Type III: tendon trapped in A4 pully
28. • From metacarpal head to middle phalanx
• Called so because initial attempts for tendon
repair here produced poor results
• FDS and FDP within one sheath
•
ZONE II-NO MANS LAND
29.
30. • B/w transverse carpal ligament and proximal
margin of tendon sheath formation
• Delayed tendon repairs are succesfull even
after several weeks of injury
ZONE III-DISTAL PALMAR CREASE
31.
32. • Lies deep to deep transverse ligament
• Tendon injuries are rare
ZONE IV-TRANSVERSE CARPAL
LIGAMENT
33.
34. • LIES PROXIMAL TO TRANSVERSE CARPAL
LIGAMENT
ZONE V-PROXIMAL
35. SIGNS & SYMPTOMS
• Unable to bend one or more finger joints
• Pain when bending finger/s
• Mild swelling over joint closest to fingertip
• Tenderness along effected finger/s on palm side of
hand
37. INSPECTION
• There is a normal arcade to hand with index
finger showing least and little finger showing
max flexion
• If affected finger shows more extension than
other digits, chance of tendon injuries are high
EXAMINATION
41. DETECTION
• History and physical Examination of wound
•
• Use of bedside ultrasonography in ER (more sensitive and specific than
physical examination)
• Wound exploration techniques or MRI.
• 3-view x-ray must be done (except most benign) to rule out foreign
bodies or bony injury.
• Radiographs to evaluate for possible fractures or dislocations (blunt
trauma cases)
42. DICTUM
• Flexor tendon repair is not a surgical
emergency. It is proved that equal or better
results can be achieved by delayed primary
repair.
• Better to repair both FDP & FDS tendons
rather than FDP alone
43. Goals of reconstruction:
•
• anatomical repair
• repair to permit active range of motion
•Pully reconstruction to minimize bow-stringing
• atraumatic surgical technique to minimize adhesions
• strict adherence to rehabilitation protocol.
44. Timing of flexor tendon repair:
Primary: repair within 24 hours (contraindicated
in case of high grade contamination i.e. human
bites, infection)
Delayed Primary: 1-10 days when the wound can
be still pulled open without incision
Early Secondary: 2-4 weeks.
Late Secondary : after 4 weeks
49. Suture Materials
• Core Non-absorbable 4/0 suture
• Different configurations
• 6/0 monofilament running epitenon suture.
• As noted by Singer MD et al. 1998, 3-0
prolene or mersilene suture may be suture
of choice
50. • WOUND EXTENDED PROXIMALLY AND DISTALLY
• PROXIMAL TENDON RETRIEVED,CORE SUTURES
ARE PLACED
• KEITH NEEDLES USED TO PASS THE SUTURES
AROUND THE DISTAL PHALANX EXITING
THROUGH NAIL PLATE DISTALLY
• REMAINING DISTAL END OF TENDON SUTURED
TO THE RE-ATTACHED PROXIMAL PORTION
ZONE 1 REPAIR
52. One method of attaching tendon to bone. A, Small area of cortex is raised with osteotome.
B, Hole is drilled through bone with Kirschner wire in drill. C, Bunnell crisscross stitch is
placed in end of tendon, and wire suture is drawn through hole in bone. D, End of tendon
is drawn against bone, and suture is tied over button.
Wilson
54. Direct repair:
if laceration is more than
1 cm from FDP insertion
Tendon advancement:
if the laceration is less
then 1 cm from insertion
55. • REPAIR BOTH TENDON LACERATIONS
• TENDON SHEATH MAY BE OPENED FOR
EXPOSURE BUT A2 AND A4 ARE PRESERVED
AS MUCH AS POSSIBLE
• FDS IS REPAIRED FIRST FOLLOWED BY FDP
ZONE II REPAIRS
56. • If both tendons are lacerated, both are
repaired, end to end with
circumferential re-enforcing sutures
• May affect lumbricalsin addition to
flexor tendons
• Damaged lumbrical is either repaired or
excised depending on severity of injury
and the location of the laceration
ZONE III REPAIRS
57. Lumbrical muscle bellies usually are
not sutured because this can
increase the tension of these
muscles and result in a “lumbrical
plus” finger (paradoxical proximal
interphalangeal extension on
attempted active finger flexion).
Zone 3 injuries
59. Quadriga effect!!
Tendon advancement shortens the FDP
& completes the grip before the normal
fingers, if the tension on tendon graft is
set too high, and limit their flexion and
thus week grip
60. • Lacerations of flexor tendons within the
carpal canal are typically associated with
partial or complete laceration of median nerve
• Here median nerves should be repaired first
and the tendons last
ZONE IV REPAIR
61. • In this area there may be concomitant ulnar
nerve & artery damage as well as radial artery
& median nerve damage.
• Primary repair of the arteries is usually
indicated
• If wound is contaminated, arteries are
repaired and delayed repair of tendons and
nerves is planned
ZONE V REPAIR
65. Different Methods
1. Active Extention-Rubber Band Flexion Method:
e.g. Kleinert, and Brooke-Army
2. Immobilization
3. Controlled Passive Motion Methods: e.g.
Duran’s protocol
4. Strickland: Early active ROM
66. Kleinert Protocol
• Combines dorsal extension block with rubber-
band traction proximal to wrist
•
•
This passively flexes fingers, & the patient
actively extends within the limits of the splint
68. Duran protocol
• At surgery, a dorsal extension-block splint is
applied with the wrist at 20-30° of flexion, the
MCP joints at 50-60° of flexion, and the IP
joints straight
74. DONOR TENDONS FOR GRAFTING:
Palmaris Longus: Tendon of choice (fulfils
requirement of length, diam & availability)
Plantaris Tendon: Equally satisfactory &
advantage of being almost twice as long, but is
not accessible.
Others: FDS, EDC
81. Zone II injury- Middle Phalanx Level:
– Repair by interrupted suture.
– Immobilization for 5-6 weeks
– DIP joint in extension
– PIP joint left free
82.
83. Zone III injury- PIP joint level
– Most complex anatomically and physiologically
– Causes two deformities
• Boutonniere
• disruption of central tendon
Closed: splinting MCP and PIP in hyperextension for
6 weeks
Open: suture repair (figure of 8 suture)
• Swan Neck
– excessive traction of central tendon
– Closed : splinting DIP
– Open : suture repair
84.
85. Zone IV injury- shaft of proximal
phalanx level
– Repair relatively easy
– Adhesion is the
problem
86.
87. Zone V injury – MCP joint level
• Closed: splinting, 45 extension at wrist and 20
flexion at MCP
• Open: suture repair by 5.0 prolene
88.
89. Zone VI injury- Metacarpal level
Better prognosis
than in fingers
All structures, even
intertendinous
band should be
repaired.
Core type suture
possible.
Delayed suture is
possible
90.
91. Zone VII- wrist level
– Extensor tendons are under dorsal
retinaculum
– Retinaculum should be repaired or partially
preserved.
– Adhesion is the problem
– Grasping core suture should be used.
– Immobilization for 5-6 weeks.
92. EXTENSOR TENDON MANAGEMENT
3 current treatment approaches to
extensor tendon rehabilitation are
Immobilization
Early controlled passive mobilization
Early active motion
93. Position of Immobilization for Extensor
Tendon Injury
•The finger should
remain parallel to
forearm with wrist
in full extension
•PIP & DIP –
Neutral
94. IMMOBILIZATION
• Keep the tendon in a shortened position through
splinting or casting
• Tendons immobilized for 3 weeks
• In week 4, gentle active motion of the repaired
tendon is introduced
• Rehabilitation depends on zone of injury
95. IMMOBILIZATION
INJURIES IN ZONES
PROXIMAL TO MCPs
INJURIES IN ZONES
DISTAL TO MCPs
• May be immobilized for 3
weeks.
• Afterwards, finger may be
placed in removable volar splint
between exercise periods for 2
weeks
• Progressive ROM after 3 weeks
• If full flexion is not regained
rapidly, dynamic flexion may be
started after 6 weeks
• Require a longer period
of immobilization
(usually 6 weeks)
• A progressive exercise
program is initiated
• Dynamic splinting during
day and static splinting
at night to maintain
extension
96. EARLY PASSIVE MOTION
• Extensors are held in extension by dynamic,
gentle rubber band traction, and the patient is
allowed to actively flex the fingers—passively
moving repaired extensor tendons
97. EARLY ACTIVE MOTION
• Early active short arc program (developed by
Evans) allows tendon to actively move 3 days
after surgery
•
• Splinting program is complex and specific and
requires a skilled occupational therapist