Rheumatology
•Download as PPTX, PDF•
0 likes•51 views
This document summarizes different types of vasculitis, including: - Takayasu's disease, which mainly involves the aorta and its branches and is characterized by fever, myalgia, and weight loss in young females. - Giant cell arteritis, which occurs in the elderly and involves cranial vessels. - Polyarteritis nodosa, which is characterized by nodules along small and medium arteries. - ANCA associated vasculitis, which can involve various organ systems. The document provides details on symptoms, presentations, diagnostic criteria and treatments for each type.
Report
Share
Report
Share
Recommended
General Medicine for the Optometrist2
The document discusses several types of vasculitis, which are inflammatory diseases of blood vessels. It describes the general presentation and ocular manifestations of giant cell arteritis, polyarteritis nodosa, Wegener's granulomatosis, sarcoidosis, rheumatoid arthritis, systemic lupus erythematosus, and Sjögren's syndrome. It advises optometrists to have a high suspicion of vasculitis in elderly patients with new headaches and to urgently refer patients exhibiting ocular findings that could indicate vasculitis, such as keratitis, scleritis, or retinal problems.
Vasculitis
This document provides an overview of vasculitis, including definitions, common clinical features, pathogenic mechanisms, classification, and morphology for several specific types of vasculitis. It defines vasculitis as inflammation of blood vessels that can affect small, medium, and large vessels. Common symptoms include fever, arthralgia, myalgia, fatigue, and purpura. Pathogenic mechanisms include immune-mediated inflammation in non-infectious vasculitis and direct vascular insult by infectious pathogens in infectious vasculitis. Specific types discussed include temporal arteritis/giant cell arteritis, Takayasu arteritis, Kawasaki disease, Buerger disease, polyarteritis nodosa, and Wegener granulo
Vasculitis
This document provides an overview of vasculitis, including:
- Vasculitis refers to inflammation of blood vessel walls and can range from mild to life-threatening.
- It encompasses a large group of diseases characterized by inflammatory reactions in blood vessel walls. The causes are often unknown but can be triggered by various stimuli.
- Diagnosis involves blood tests, biopsies of affected tissues like skin and kidneys, and imaging tests. Biopsies examined under a microscope are often needed to confirm a diagnosis of vasculitis.
Vasculitis
Vasculitis refers to inflammation of blood vessels. This document discusses the pathophysiology, classification, clinical presentation, diagnosis, and treatment of various types of vasculitis. The main types include large vessel vasculitis (e.g. giant cell arteritis, Takayasu arteritis), medium vessel vasculitis (e.g. polyarteritis nodosa, Kawasaki disease), small vessel vasculitis (ANCA-associated vasculitis like granulomatosis with polyangiitis, microscopic polyangiitis, eosinophilic granulomatosis with polyangiitis), and immune complex small vessel vasculitis (e.g. IgA vasculitis, antiglomer
Vasculitis
This document discusses vasculitis, which is inflammation of blood vessels. It classifies vasculitis into large, medium, and small vessel types and discusses specific conditions like temporal arteritis, Takayasu arteritis, polyarteritis nodosa, Henoch-Schönlein purpura, Behcet's disease, Wegener's granulomatosis, microscopic polyangiitis, and Churg-Strauss syndrome. For each condition, it covers clinical manifestations, diagnosis, and treatment. Vasculitis can affect multiple organ systems and cause symptoms like fever, skin lesions, neurological issues, and kidney problems. Diagnosis involves tests like biopsy and ANCA levels while treatment consists of steroids, immunosup
Vasculitis pathology
Vasculitis is characterized by inflammation and damage to blood vessels that can lead to tissue ischemia. It can involve single or multiple organs and be primary or secondary to infections, autoimmune diseases, or malignancy. Vasculitis pathogenesis may involve immune complex formation, ANCA production, T cell activation and granuloma formation, or direct antibody-mediated damage. While immune complexes were thought to play a causal role, this has not been proven for most vasculitis syndromes. Specific types of vasculitis are associated with immune mechanisms like ANCA, T cells, or anti-endothelial cell antibodies.
Cutaneous Vasculitis
Vasculitis refers to inflammation of blood vessels which can be cutaneous or systemic. It is characterized by destruction of vessel walls by leukocytes leading to ischemia and tissue damage. It can be triggered by infections, medications, or underlying diseases. Classification systems categorize vasculitis based on vessel size. Common types include hypersensitivity vasculitis, cutaneous leukocytoclastic vasculitis, Henoch-Schonlein purpura, urticarial vasculitis, and erythema elevatum diutinum. Pathogenesis involves immune complex deposition, antineutrophil cytoplasmic antibodies, and cellular immune responses.
Systemic vasculitis 2016 shj
Different types of vasculitis have characteristic patterns of blood vessel involvement.However vasculitis is a systemic illness.The symptoms of vasculitis depend on the particular blood vessels that are involved by the inflammatory process
Recommended
General Medicine for the Optometrist2
The document discusses several types of vasculitis, which are inflammatory diseases of blood vessels. It describes the general presentation and ocular manifestations of giant cell arteritis, polyarteritis nodosa, Wegener's granulomatosis, sarcoidosis, rheumatoid arthritis, systemic lupus erythematosus, and Sjögren's syndrome. It advises optometrists to have a high suspicion of vasculitis in elderly patients with new headaches and to urgently refer patients exhibiting ocular findings that could indicate vasculitis, such as keratitis, scleritis, or retinal problems.
Vasculitis
This document provides an overview of vasculitis, including definitions, common clinical features, pathogenic mechanisms, classification, and morphology for several specific types of vasculitis. It defines vasculitis as inflammation of blood vessels that can affect small, medium, and large vessels. Common symptoms include fever, arthralgia, myalgia, fatigue, and purpura. Pathogenic mechanisms include immune-mediated inflammation in non-infectious vasculitis and direct vascular insult by infectious pathogens in infectious vasculitis. Specific types discussed include temporal arteritis/giant cell arteritis, Takayasu arteritis, Kawasaki disease, Buerger disease, polyarteritis nodosa, and Wegener granulo
Vasculitis
This document provides an overview of vasculitis, including:
- Vasculitis refers to inflammation of blood vessel walls and can range from mild to life-threatening.
- It encompasses a large group of diseases characterized by inflammatory reactions in blood vessel walls. The causes are often unknown but can be triggered by various stimuli.
- Diagnosis involves blood tests, biopsies of affected tissues like skin and kidneys, and imaging tests. Biopsies examined under a microscope are often needed to confirm a diagnosis of vasculitis.
Vasculitis
Vasculitis refers to inflammation of blood vessels. This document discusses the pathophysiology, classification, clinical presentation, diagnosis, and treatment of various types of vasculitis. The main types include large vessel vasculitis (e.g. giant cell arteritis, Takayasu arteritis), medium vessel vasculitis (e.g. polyarteritis nodosa, Kawasaki disease), small vessel vasculitis (ANCA-associated vasculitis like granulomatosis with polyangiitis, microscopic polyangiitis, eosinophilic granulomatosis with polyangiitis), and immune complex small vessel vasculitis (e.g. IgA vasculitis, antiglomer
Vasculitis
This document discusses vasculitis, which is inflammation of blood vessels. It classifies vasculitis into large, medium, and small vessel types and discusses specific conditions like temporal arteritis, Takayasu arteritis, polyarteritis nodosa, Henoch-Schönlein purpura, Behcet's disease, Wegener's granulomatosis, microscopic polyangiitis, and Churg-Strauss syndrome. For each condition, it covers clinical manifestations, diagnosis, and treatment. Vasculitis can affect multiple organ systems and cause symptoms like fever, skin lesions, neurological issues, and kidney problems. Diagnosis involves tests like biopsy and ANCA levels while treatment consists of steroids, immunosup
Vasculitis pathology
Vasculitis is characterized by inflammation and damage to blood vessels that can lead to tissue ischemia. It can involve single or multiple organs and be primary or secondary to infections, autoimmune diseases, or malignancy. Vasculitis pathogenesis may involve immune complex formation, ANCA production, T cell activation and granuloma formation, or direct antibody-mediated damage. While immune complexes were thought to play a causal role, this has not been proven for most vasculitis syndromes. Specific types of vasculitis are associated with immune mechanisms like ANCA, T cells, or anti-endothelial cell antibodies.
Cutaneous Vasculitis
Vasculitis refers to inflammation of blood vessels which can be cutaneous or systemic. It is characterized by destruction of vessel walls by leukocytes leading to ischemia and tissue damage. It can be triggered by infections, medications, or underlying diseases. Classification systems categorize vasculitis based on vessel size. Common types include hypersensitivity vasculitis, cutaneous leukocytoclastic vasculitis, Henoch-Schonlein purpura, urticarial vasculitis, and erythema elevatum diutinum. Pathogenesis involves immune complex deposition, antineutrophil cytoplasmic antibodies, and cellular immune responses.
Systemic vasculitis 2016 shj
Different types of vasculitis have characteristic patterns of blood vessel involvement.However vasculitis is a systemic illness.The symptoms of vasculitis depend on the particular blood vessels that are involved by the inflammatory process
Vasculitis undergrad: diagnosis & treatment.
1. The lecture covers the definition, pathophysiology, classification, clinical presentation, diagnosis and treatment of various types of vasculitis.
2. Vasculitis is classified according to the size of blood vessels involved, ranging from large vessel vasculitis like Takayasu arteritis and Giant Cell Arteritis, to medium vessel vasculitis like Polyarteritis nodosa, to small vessel vasculitis like Wegener's granulomatosis.
3. Key aspects of evaluation include clinical features, laboratory and serological testing, imaging, and biopsy. Management involves treatment of underlying causes, immunosuppression, and controlling disease activity and complications.
Vascul lecture
Vasculitis refers to inflammation of blood vessels. This document discusses the classification of various types of vasculitis based on vessel size, etiology, and other factors. Some of the main types covered include giant cell arteritis (temporal arteritis), Takayasu arteritis, polyarteritis nodosa, Buerger's disease, Kawasaki disease, microscopic polyangiitis, Churg-Strauss syndrome, and Wegener's granulomatosis. Key distinguishing clinical features, pathogenesis, diagnostic criteria and histopathological findings are provided for each condition.
Vasculitis
This document discusses different types of vasculitis, which are inflammatory conditions that affect blood vessels. It describes large vessel vasculitides like Takayasu arteritis and giant cell arteritis, medium vessel vasculitides like polyarteritis nodosa, and small vessel vasculitides like Wegener's granulomatosis. For each type, it provides information on clinical features, symptoms, affected organs, diagnostic imaging findings, and treatments.
IMAGING IN SARCOIDOSIS
Dr. Sandeep B S discusses the imaging findings of sarcoidosis in various organs. On thoracic CT, typical findings include bilateral hilar and right paratracheal lymphadenopathy, as well as peribronchovascular and subpleural lung nodules. Cardiac MRI can detect myocardial inflammation and scarring from sarcoidosis with high sensitivity and specificity. Hepatic and splenic sarcoidosis may appear as nodules or periportal adenopathy on abdominal imaging. Neurosarcoidosis can involve the leptomeninges, cranial nerves, dura, or spine. Bone sarcoidosis may cause lytic lesions, while musculoskeletal sarcoidosis can result
Spots with keys (2)
1. The document describes various gastrointestinal and musculoskeletal conditions seen on imaging. It includes descriptions of total colonic aganglionosis, retroperitoneal fibrosis, pectus excavatum, Reiter's syndrome, median arcuate ligament syndrome, and Haglund syndrome among others.
2. The conditions are described and key radiographic findings are highlighted, such as the displacement and tapering of ureters seen in retroperitoneal fibrosis. Common presentations, classifications, and distinguishing radiologic features are summarized for each condition.
3. Different imaging modalities are discussed, with CT and MRI findings provided where relevant to demonstrate characteristics of the various diseases and injuries.
Cutaneous vasculitis
Cutaneous involvement is very common in the different types of vasculitis. Skin lesions may be the only manifestation or may occur in the context of systemic disease
Pediatric vasculitis
This document discusses imaging findings in childhood systemic vasculitis. It describes several types of vasculitis including Takayasu arteritis, Kawasaki disease, granulomatosis with polyangiitis, microscopic polyangiitis, eosinophilic granulomatosis with polyangiitis, IgA vasculitis, Behcet's disease, and CNS involvement. Imaging plays an important role in assessing complications and organ involvement. Common findings include arterial wall thickening, aneurysms, infarctions, pulmonary nodules and hemorrhage, and nonspecific lung opacities. Differential diagnoses include infections, malignancies, and other inflammatory and non-inflammatory conditions.
Sarcoidosis imaging
This document discusses the clinical presentation and manifestations of sarcoidosis. It notes that sarcoidosis is characterized by non-caseating granulomas that can affect multiple organs, most commonly the lungs. It describes the typical involvement of the lungs, eyes, skin, joints, liver and lymph nodes. Musculoskeletal involvement and arthritis are also common. Radiographic findings include hilar lymphadenopathy and small nodules in a perilymphatic distribution. Fibrosis can develop in advanced disease. Differential diagnoses include other granulomatous diseases like tuberculosis.
Wegner's granulomatosis
This document summarizes Wegener's granulomatosis, a multisystem disease characterized by necrotizing vasculitis affecting the respiratory tract and glomerulonephritis. It involves the upper respiratory tract in 100% of cases, lower respiratory tract in 90% of cases, and kidneys in 80% of cases. Common presentations include rhinitis, sinusitis, cough, hemoptysis, and hematuria. Radiological manifestations include nodules, alveolar opacities, ground glass opacities, and consolidation. Subglottic tracheal stenosis and bronchial stenosis are also seen.
Sarcoidosis
Sarcoidosis is a systemic inflammatory disease characterized by non-caseating granulomas of unknown cause but may be due to an altered immune response in genetically susceptible individuals after exposure to environmental or infectious agents. It commonly involves the lungs causing breathing problems, eyes causing uveitis, and skin causing lesions. Diagnosis is made by biopsy showing granulomas. Treatment involves steroids and immunosuppressants to reduce symptoms from organ involvement. Prognosis is variable but may include spontaneous remission or progression to respiratory failure.
Lecture 17 cardiovascular system
This document discusses cardiovascular pathology and includes sections on diseases of the heart, blood vessels, and lymphatic system. It describes various congenital heart defects, acquired valvular diseases such as rheumatic heart disease, infections of the heart (endocarditis), and tumors that can affect the cardiovascular system. The key topics covered are the structure and common abnormalities of the heart, acquisition and manifestations of rheumatic heart disease, characteristics of different types of endocarditis, and how tumors may cause cardiac abnormalities.
Vasculitis pathology
Vasculitis
pathology
Define and classify vasculitis.
Describe the cause, pathogenesis, morphology, and clinical presentation of various types of vasculitis.
The road to HRCT evaluation of pediatric diffuse lung diseases.part 2
This document discusses the causes, imaging findings, and classification of childhood interstitial lung disease (chILD). It lists various infectious, inflammatory, metabolic, and neoplastic causes. HRCT findings for different conditions like NEHI, surfactant dysfunction, NSIP, and LCH are described. The classification of chILD has evolved to consider clinical context and combine clinical, imaging, and pathological findings for diagnosis rather than relying on any single component. A new validated pediatric ILD classification system categorizes chILD into disorders of infancy, other categories, and unclassifiable cases.
Radiology Spotters
Progressive muscle weakness for 2 years. Giant cerebral aneurysms are greater than 25mm. Patients can present with mass effect or subarachnoid hemorrhage. On MRI, patent aneurysms appear as flow void or heterogeneous signal. Thrombosed aneurysms depend on clot age. Sturge-Weber syndrome is characterized by facial port wine stains and pial angiomas. CT detects subcortical calcification earlier than plain film. MRI shows signal changes and anatomical volume loss with age. État criblé describes diffusely widened perivascular spaces in the basal ganglia. External auditory canal atresia involves complete or incomplete bony atresia of the external auditory canal.
Sarcoidosis & orphan lung disease
This document discusses sarcoidosis, a multisystem granulomatous disorder of unknown origin. It covers the history of sarcoidosis, epidemiology, pathology, organ involvement, staging, diagnosis and treatment. Specifically, it notes that sarcoidosis was first described in 1887 and causes non-caseating granulomas. It can affect many organs, especially the lungs, and has varying presentations depending on the organs involved. Diagnosis involves ruling out other causes and identifying characteristic granulomas on biopsy.
Tutorial secondary idd aids
This document defines secondary immune deficiency diseases and discusses HIV/AIDS in particular. It covers the causes, pathogenesis, clinical presentations, diagnosis, and monitoring of secondary immune deficiencies, with a focus on HIV. Key points include: (1) Secondary immune deficiencies are acquired and common, caused by defects in antibodies, phagocytes, complement, or cell-mediated immunity; (2) HIV progresses through early, chronic, and crisis phases defined by declining CD4+ T-cell counts; (3) AIDS is diagnosed when CD4+ counts fall below 200 cells/μL and opportunistic infections or cancers develop.
Rheumatic Heart Disease
Brief overview of the clinical features, epidemiology, pathophysiology, etiology, morphology, diagnosis, complications & medical management of rheumatic heart disease
Enjoy learning!
Sarcoidosis
Sarcoidosis is a chronic inflammatory disease characterized by the formation of non-caseating granulomas in multiple organs, most commonly affecting the lungs. It has unknown causes but is thought to involve genetic and environmental factors. Historically, it was first described in the skin in 1899 and was later found to also affect the lungs and lymph nodes. It presents variably from being asymptomatic to causing respiratory symptoms or lesions in other organs. Diagnosis involves clinical features, chest imaging typically showing bilateral hilar lymphadenopathy, and biopsy demonstrating granulomas. Treatment involves observation of asymptomatic cases but corticosteroids for symptomatic or progressive disease. Prognosis is generally good with many cases resolving spontaneously but advanced lung fibrosis can occasionally occur
Clubbing summary
a compact and concise summary on clubbing.
Includes, mechanism, causes, and clinical approach to clubbing.
Wegener granulomatosis 2
This is a presentation on basic facts Wegeners Granulomatosis - History ,presentation ,Investigations and treatment.
Lecture samy- 2-4-16
1. Vasculitis is defined as inflammation of blood vessel walls and can be classified by the size of vessels involved - large, medium, or small.
2. Some examples of large vessel vasculitis include giant-cell arteritis, which commonly involves the temporal artery in older patients, and Takayasu arteritis, which usually occurs in younger patients.
3. Medium vessel vasculitis encompasses diseases like polyarteritis nodosa, characterized by necrotizing inflammation of renal arteries, and Kawasaki disease, an arteritis that can cause coronary aneurysms in children.
4. Small vessel vasculitis includes Wegener's granulomatosis, Churg-Stra
Vasculitis - Wegners, churg strauss,PAN, Temporal arteritis, Buerger's disea...
Vasculitis - Wegners, churg strauss,PAN, Temporal arteritis, Takayasu, Kawasaki : Incidence, Epidemiology, Pathogenesis, Clinical Features, Diagnosis, Investigations, Management
More Related Content
What's hot
Vasculitis undergrad: diagnosis & treatment.
1. The lecture covers the definition, pathophysiology, classification, clinical presentation, diagnosis and treatment of various types of vasculitis.
2. Vasculitis is classified according to the size of blood vessels involved, ranging from large vessel vasculitis like Takayasu arteritis and Giant Cell Arteritis, to medium vessel vasculitis like Polyarteritis nodosa, to small vessel vasculitis like Wegener's granulomatosis.
3. Key aspects of evaluation include clinical features, laboratory and serological testing, imaging, and biopsy. Management involves treatment of underlying causes, immunosuppression, and controlling disease activity and complications.
Vascul lecture
Vasculitis refers to inflammation of blood vessels. This document discusses the classification of various types of vasculitis based on vessel size, etiology, and other factors. Some of the main types covered include giant cell arteritis (temporal arteritis), Takayasu arteritis, polyarteritis nodosa, Buerger's disease, Kawasaki disease, microscopic polyangiitis, Churg-Strauss syndrome, and Wegener's granulomatosis. Key distinguishing clinical features, pathogenesis, diagnostic criteria and histopathological findings are provided for each condition.
Vasculitis
This document discusses different types of vasculitis, which are inflammatory conditions that affect blood vessels. It describes large vessel vasculitides like Takayasu arteritis and giant cell arteritis, medium vessel vasculitides like polyarteritis nodosa, and small vessel vasculitides like Wegener's granulomatosis. For each type, it provides information on clinical features, symptoms, affected organs, diagnostic imaging findings, and treatments.
IMAGING IN SARCOIDOSIS
Dr. Sandeep B S discusses the imaging findings of sarcoidosis in various organs. On thoracic CT, typical findings include bilateral hilar and right paratracheal lymphadenopathy, as well as peribronchovascular and subpleural lung nodules. Cardiac MRI can detect myocardial inflammation and scarring from sarcoidosis with high sensitivity and specificity. Hepatic and splenic sarcoidosis may appear as nodules or periportal adenopathy on abdominal imaging. Neurosarcoidosis can involve the leptomeninges, cranial nerves, dura, or spine. Bone sarcoidosis may cause lytic lesions, while musculoskeletal sarcoidosis can result
Spots with keys (2)
1. The document describes various gastrointestinal and musculoskeletal conditions seen on imaging. It includes descriptions of total colonic aganglionosis, retroperitoneal fibrosis, pectus excavatum, Reiter's syndrome, median arcuate ligament syndrome, and Haglund syndrome among others.
2. The conditions are described and key radiographic findings are highlighted, such as the displacement and tapering of ureters seen in retroperitoneal fibrosis. Common presentations, classifications, and distinguishing radiologic features are summarized for each condition.
3. Different imaging modalities are discussed, with CT and MRI findings provided where relevant to demonstrate characteristics of the various diseases and injuries.
Cutaneous vasculitis
Cutaneous involvement is very common in the different types of vasculitis. Skin lesions may be the only manifestation or may occur in the context of systemic disease
Pediatric vasculitis
This document discusses imaging findings in childhood systemic vasculitis. It describes several types of vasculitis including Takayasu arteritis, Kawasaki disease, granulomatosis with polyangiitis, microscopic polyangiitis, eosinophilic granulomatosis with polyangiitis, IgA vasculitis, Behcet's disease, and CNS involvement. Imaging plays an important role in assessing complications and organ involvement. Common findings include arterial wall thickening, aneurysms, infarctions, pulmonary nodules and hemorrhage, and nonspecific lung opacities. Differential diagnoses include infections, malignancies, and other inflammatory and non-inflammatory conditions.
Sarcoidosis imaging
This document discusses the clinical presentation and manifestations of sarcoidosis. It notes that sarcoidosis is characterized by non-caseating granulomas that can affect multiple organs, most commonly the lungs. It describes the typical involvement of the lungs, eyes, skin, joints, liver and lymph nodes. Musculoskeletal involvement and arthritis are also common. Radiographic findings include hilar lymphadenopathy and small nodules in a perilymphatic distribution. Fibrosis can develop in advanced disease. Differential diagnoses include other granulomatous diseases like tuberculosis.
Wegner's granulomatosis
This document summarizes Wegener's granulomatosis, a multisystem disease characterized by necrotizing vasculitis affecting the respiratory tract and glomerulonephritis. It involves the upper respiratory tract in 100% of cases, lower respiratory tract in 90% of cases, and kidneys in 80% of cases. Common presentations include rhinitis, sinusitis, cough, hemoptysis, and hematuria. Radiological manifestations include nodules, alveolar opacities, ground glass opacities, and consolidation. Subglottic tracheal stenosis and bronchial stenosis are also seen.
Sarcoidosis
Sarcoidosis is a systemic inflammatory disease characterized by non-caseating granulomas of unknown cause but may be due to an altered immune response in genetically susceptible individuals after exposure to environmental or infectious agents. It commonly involves the lungs causing breathing problems, eyes causing uveitis, and skin causing lesions. Diagnosis is made by biopsy showing granulomas. Treatment involves steroids and immunosuppressants to reduce symptoms from organ involvement. Prognosis is variable but may include spontaneous remission or progression to respiratory failure.
Lecture 17 cardiovascular system
This document discusses cardiovascular pathology and includes sections on diseases of the heart, blood vessels, and lymphatic system. It describes various congenital heart defects, acquired valvular diseases such as rheumatic heart disease, infections of the heart (endocarditis), and tumors that can affect the cardiovascular system. The key topics covered are the structure and common abnormalities of the heart, acquisition and manifestations of rheumatic heart disease, characteristics of different types of endocarditis, and how tumors may cause cardiac abnormalities.
Vasculitis pathology
Vasculitis
pathology
Define and classify vasculitis.
Describe the cause, pathogenesis, morphology, and clinical presentation of various types of vasculitis.
The road to HRCT evaluation of pediatric diffuse lung diseases.part 2
This document discusses the causes, imaging findings, and classification of childhood interstitial lung disease (chILD). It lists various infectious, inflammatory, metabolic, and neoplastic causes. HRCT findings for different conditions like NEHI, surfactant dysfunction, NSIP, and LCH are described. The classification of chILD has evolved to consider clinical context and combine clinical, imaging, and pathological findings for diagnosis rather than relying on any single component. A new validated pediatric ILD classification system categorizes chILD into disorders of infancy, other categories, and unclassifiable cases.
Radiology Spotters
Progressive muscle weakness for 2 years. Giant cerebral aneurysms are greater than 25mm. Patients can present with mass effect or subarachnoid hemorrhage. On MRI, patent aneurysms appear as flow void or heterogeneous signal. Thrombosed aneurysms depend on clot age. Sturge-Weber syndrome is characterized by facial port wine stains and pial angiomas. CT detects subcortical calcification earlier than plain film. MRI shows signal changes and anatomical volume loss with age. État criblé describes diffusely widened perivascular spaces in the basal ganglia. External auditory canal atresia involves complete or incomplete bony atresia of the external auditory canal.
Sarcoidosis & orphan lung disease
This document discusses sarcoidosis, a multisystem granulomatous disorder of unknown origin. It covers the history of sarcoidosis, epidemiology, pathology, organ involvement, staging, diagnosis and treatment. Specifically, it notes that sarcoidosis was first described in 1887 and causes non-caseating granulomas. It can affect many organs, especially the lungs, and has varying presentations depending on the organs involved. Diagnosis involves ruling out other causes and identifying characteristic granulomas on biopsy.
Tutorial secondary idd aids
This document defines secondary immune deficiency diseases and discusses HIV/AIDS in particular. It covers the causes, pathogenesis, clinical presentations, diagnosis, and monitoring of secondary immune deficiencies, with a focus on HIV. Key points include: (1) Secondary immune deficiencies are acquired and common, caused by defects in antibodies, phagocytes, complement, or cell-mediated immunity; (2) HIV progresses through early, chronic, and crisis phases defined by declining CD4+ T-cell counts; (3) AIDS is diagnosed when CD4+ counts fall below 200 cells/μL and opportunistic infections or cancers develop.
Rheumatic Heart Disease
Brief overview of the clinical features, epidemiology, pathophysiology, etiology, morphology, diagnosis, complications & medical management of rheumatic heart disease
Enjoy learning!
Sarcoidosis
Sarcoidosis is a chronic inflammatory disease characterized by the formation of non-caseating granulomas in multiple organs, most commonly affecting the lungs. It has unknown causes but is thought to involve genetic and environmental factors. Historically, it was first described in the skin in 1899 and was later found to also affect the lungs and lymph nodes. It presents variably from being asymptomatic to causing respiratory symptoms or lesions in other organs. Diagnosis involves clinical features, chest imaging typically showing bilateral hilar lymphadenopathy, and biopsy demonstrating granulomas. Treatment involves observation of asymptomatic cases but corticosteroids for symptomatic or progressive disease. Prognosis is generally good with many cases resolving spontaneously but advanced lung fibrosis can occasionally occur
Clubbing summary
a compact and concise summary on clubbing.
Includes, mechanism, causes, and clinical approach to clubbing.
Wegener granulomatosis 2
This is a presentation on basic facts Wegeners Granulomatosis - History ,presentation ,Investigations and treatment.
What's hot (20)
The road to HRCT evaluation of pediatric diffuse lung diseases.part 2
The road to HRCT evaluation of pediatric diffuse lung diseases.part 2
Similar to Rheumatology
Lecture samy- 2-4-16
1. Vasculitis is defined as inflammation of blood vessel walls and can be classified by the size of vessels involved - large, medium, or small.
2. Some examples of large vessel vasculitis include giant-cell arteritis, which commonly involves the temporal artery in older patients, and Takayasu arteritis, which usually occurs in younger patients.
3. Medium vessel vasculitis encompasses diseases like polyarteritis nodosa, characterized by necrotizing inflammation of renal arteries, and Kawasaki disease, an arteritis that can cause coronary aneurysms in children.
4. Small vessel vasculitis includes Wegener's granulomatosis, Churg-Stra
Vasculitis - Wegners, churg strauss,PAN, Temporal arteritis, Buerger's disea...
Vasculitis - Wegners, churg strauss,PAN, Temporal arteritis, Takayasu, Kawasaki : Incidence, Epidemiology, Pathogenesis, Clinical Features, Diagnosis, Investigations, Management
neurologic presentations of systemic vasculitis
- Temporal arteritis (TA), also known as giant cell arteritis, is an inflammatory disease that affects medium and large arteries, especially the cranial arteries. It is more common in adults over 50 years of age.
- Left untreated, TA can cause permanent vision loss. Treatment involves high-dose corticosteroids to suppress inflammation. Prednisone is gradually tapered over months or years to prevent relapses.
- Takayasu arteritis is a large vessel vasculitis that predominantly affects women aged 10-30. It causes stenosis and occlusion of the aorta and its main branches. Treatment consists of corticosteroids along with monitoring for vascular complications like strokes.
CVS Pathology 6 Vasculitis 2019 sufia husain
This document summarizes several types of vasculitis, including giant cell arteritis, polyarteritis nodosa, Wegener's granulomatosis, cutaneous hypersensitivity vasculitis, and thromboangiitis obliterans. Giant cell arteritis primarily affects large arteries in the head and is characterized by headache, jaw pain, and vision loss. Polyarteritis nodosa typically involves medium and small arteries throughout the body, especially the kidneys. Wegener's granulomatosis is defined by lung and kidney involvement and the presence of C-ANCA antibodies. Cutaneous hypersensitivity vasculitis mainly causes skin purpura. Thromboangiitis obliterans, or Buerger's disease,
Vaskulitis/ Giant cell arteritis/ temporalis arteritis
This document discusses several types of vasculitis:
1. Giant cell arteritis affects large arteries in the head and is characterized by granulomatous inflammation and headache along the temporal artery.
2. Polyarteritis nodosa involves medium and small arteries throughout the body and causes tissue ischemia. It is associated with hepatitis B/C.
3. Wegener's granulomatosis is defined by necrotizing granulomas of the respiratory tract, necrotizing vasculitis of small vessels, and renal disease. It is linked to C-ANCA antibodies.
4. Leukocytoclastic vasculitis mainly involves small skin blood vessels and is characterized by skin purpura
Endocarditis
Infective endocarditis is a condition where the inner lining of the heart (endocardium) becomes inflamed due to a bacterial or fungal infection. It often involves the heart valves and can cause growths called vegetations that can break off and travel to other organs. It is classified based on the causative organism (bacteria, fungus) and whether the person has a normal or abnormal heart/valves. Common symptoms include fever, heart murmur, skin lesions, and potential complications involving the brain, lungs, kidneys, and spleen. Diagnosis involves blood cultures, echocardiography, and the modified Duke criteria.
Rheumatic heart disease
Rheumatic heart disease is caused by rheumatic fever, which occurs after a streptococcal throat infection and causes inflammation of the heart valves. Genetic factors influence the risk of developing rheumatic heart disease. Acute rheumatic fever causes inflammatory lesions called Aschoff bodies in heart tissue that contain characteristic giant cells. Long-term rheumatic heart disease results in thickened and scarred heart valves that may become narrowed or insufficient.
Rheumatic heart disease agp
Rheumatic fever and rheumatic heart disease are conditions that can occur after a streptococcal throat infection. Rheumatic fever is an inflammatory disease that affects the heart, joints, skin, and blood vessels. It is characterized by lesions called Aschoff bodies and rheumatic granulomas. Long term, it can lead to rheumatic heart disease where the heart valves are damaged. The disease process involves an immune response to streptococcal antigens that causes inflammation and tissue damage. Symptoms vary by the organs involved but can include fever, arthritis, heart valve abnormalities, and skin rashes.
Vasculitis
This document discusses vasculitis, which is inflammation of blood vessels. It defines vasculitis and describes the different types including large vessel, medium vessel, and small vessel vasculitis. Specific conditions are discussed such as giant cell arteritis, granulomatosis with polyangiitis, Churg-Strauss syndrome, Behcet's disease, thromboangiitis obliterans, and infectious vasculitis. The pathology, clinical features, morphology, and treatment of some of these conditions are summarized. Images are also included showing histological features.
Infective endocarditis@ghanem@
A lecture on common problem infective endocarditis prepared by IslamGhanem Ahmed Ghanem assistant lecturer of cardiology Zagazig university 2013
Approach to a patient with vasculitis and its
Vasculitis refers to inflammation of blood vessels. It can be primary or secondary to other conditions like drugs, infections, collagen disorders or malignancies. It involves various sizes of blood vessels from large arteries to small vessels. Common symptoms include fever, fatigue, rashes, arthritis, and organ involvement depending on the vessel type. Diagnosis is based on clinical features, lab tests like ANCA, and biopsy. Treatment involves immunosuppression. Prognosis depends on type and organ involvement.
D. Fadhil Vasculitis-7 (Muhadharaty) (1).pptx
Vasculitides are a group of rare diseases characterized by inflammation and necrosis of blood vessels. They are classified based on the size of affected vessels into large, medium, and small vessel vasculitis. Some of the main types discussed in the document include giant cell arteritis/polymyalgia rheumatica, Takayasu's arteritis, polyarteritis nodosa, Churg-Strauss syndrome, Wegener's granulomatosis, and Henoch Schonlein purpura. The document provides details on clinical features, investigations, treatments, and prognosis for each type of vasculitis.
APPROACH TO VASCULITIS..........pptx
This document provides an overview of different types of vasculitis. It discusses the pathogenesis of vasculitis including immune complex formation and ANCA-mediated mechanisms. It then describes the clinical presentations and characteristics of various vasculitides that involve different vessel sizes such as giant cell arteritis, Takayasu's arteritis, polyarteritis nodosa, Kawasaki disease, ANCA-associated vasculitis (granulomatosis with polyangiitis, microscopic polyangiitis, Churg-Strauss syndrome), cutaneous small vessel vasculitis, and IgA vasculitis. It provides details on the patterns of organ involvement, diagnostic criteria and clinical course for each type of vasculitis.
Rheumatic heart disease
This document provides information about rheumatic heart disease. It begins with definitions, noting it is a chronic condition resulting from rheumatic fever which involves scarring and deformity of the heart valves. Incidence is higher among those from poor socioeconomic backgrounds living in crowded, damp places. The disease is caused by group A beta-hemolytic streptococcus infection leading to rheumatic fever. This causes inflammation of the heart valves and formation of vegetations, resulting in valvular damage and potential heart failure. Clinical manifestations and treatments are also summarized.
Vasculitis.pptx
This document discusses the classification of vasculitis. It begins by defining vasculitis as a heterogeneous group of conditions where inflammation of blood vessels occurs independently or secondary to other diseases. It then describes how vasculitis typically presents depending on vessel size, such as limb claudication for large vessel vasculitis or palpable purpura for small vessel vasculitis. Specific types of vasculitis are then outlined according to vessel size and other characteristics, such as the diagnostic criteria for giant cell arteritis and Takayasu arteritis as examples of large vessel vasculitis.
Infective endocarditis
Infective endocarditis is a microbial infection of the heart valves or intracardiac devices. It can be caused by various bacteria, fungi, and viruses. Risk factors include structural heart disease, intravenous drug use, and invasive procedures. Common symptoms are fever, chills, weight loss, and new heart murmurs. Diagnosis involves blood cultures and the modified Duke criteria. Treatment involves antibiotics over extended periods along with surgery for severe cases or device removal.
Takayasu Arteritis
Takayasu arteritis is a chronic inflammatory disease that causes stenosis, occlusion, dilation or aneurysm of the aorta and its branches. It most commonly affects adolescent girls and young women. Symptoms include limb claudication, decreased brachial pulse, hypertension, bruits, and vascular ischemia. Diagnosis is based on meeting criteria such as age of onset under 40, decreased pulse, blood pressure difference between arms, and angiographic evidence of vascular involvement. Treatment involves glucocorticoids which can control inflammation and symptoms, though relapses may occur. Additional immunosuppressants may be needed, and management of hypertension is important, especially during pregnancy which carries higher risks but fertility is not affected.
Acute rheumatic fever
Rheumatic fever is an inflammatory disease that affects the heart, joints, skin and nervous system following a streptococcal throat infection. It can cause permanent heart valve damage. Symptoms include fever, arthritis and heart inflammation. Treatment focuses on relieving symptoms, eliminating streptococcal infection, and preventing cardiac damage through bed rest and medications like salicylates or corticosteroids. Long-term antibiotic prophylaxis is needed to prevent recurrences, especially for those who develop heart valve problems.
Vasculitis
Vasculitis refers to inflammation of blood vessels. There are many types classified by the size of vessels involved and pathogenic mechanisms. Noninfectious vasculitis can be immune complex-mediated, associated with ANCAs, anti-endothelial cell antibodies, or autoreactive T cells. Infectious vasculitis involves direct invasion of vessels by pathogens. Common noninfectious vasculitides include giant cell arteritis, Takayasu arteritis, polyarteritis nodosa, Kawasaki disease, microscopic polyangiitis, Wegener's granulomatosis, Churg-Strauss syndrome, and Buerger's disease. Clinical manifestations depend on the organs involved. Treatment involves immunos
Systemic vasculitides - Wegener's Granulomatosis, Microscopic Polyangitis, Ch...
Systemic vasculitides - Wegener's Granulomatosis, Microscopic Polyangitis, Churg Strauss Syndrome, PAN, Giant cell arteritis, Polymyalgia rheumatica
Similar to Rheumatology (20)
Vasculitis - Wegners, churg strauss,PAN, Temporal arteritis, Buerger's disea...
Vasculitis - Wegners, churg strauss,PAN, Temporal arteritis, Buerger's disea...
Vaskulitis/ Giant cell arteritis/ temporalis arteritis
Vaskulitis/ Giant cell arteritis/ temporalis arteritis
Systemic vasculitides - Wegener's Granulomatosis, Microscopic Polyangitis, Ch...
Systemic vasculitides - Wegener's Granulomatosis, Microscopic Polyangitis, Ch...
More from Tess Jose
Anesthesia implication in Dextrocardia and situs inversus
This document presents a case report of a 45-year-old man with dextrocardia and situs inversus who presented with a right inguinal hernia. Key points discussed include:
1. The patient's history of dextrocardia diagnosed in childhood and current presentation of a irreducible right inguinal hernia.
2. Anesthesia management considerations for the laparoscopic hernia repair including placement of monitors in reverse positions and intubation occurring on the left side.
3. The intraoperative and postoperative periods were uneventful with pain management including a right-sided TAP block.
4. Challenges of situs inversus include proper
Neuromuscular monitoring
This document discusses neuromuscular transmission monitoring during anesthesia using muscle relaxants. It describes how neuromuscular block depth is monitored using a nerve stimulator to deliver electrical stimuli and evaluate the evoked muscular response. Different patterns of nerve stimulation are used, including single twitch, train-of-four, tetanic and double-burst stimulation to assess the degree of block. Factors like stimulus current, frequency and waveform are discussed in monitoring neuromuscular transmission.
Inhalational agent.pptx
1. Several gases were discovered to have anesthetic properties throughout the 19th century, including nitrous oxide, diethyl ether, and chloroform.
2. Between 1920-1940, newer inhaled anesthetics like ethylene, cyclopropane, and divinyl ether were introduced with faster induction and recovery times but also serious drawbacks like flammability and toxicity.
3. Modern inhaled anesthetics developed through fluorination have greater stability and lesser toxicity compared to earlier agents. Their uptake and distribution involves transfer between the anesthesia machine, breathing circuit, alveoli, and pulmonary blood based on partial pressures and solubility properties.
Patient positioning and associated risk
This document discusses various patient positions used during surgery and their associated risks. It describes positions such as supine, lateral, prone, lithotomy, and variations like Trendelenburg. For each position, it details anatomical support, physiological impacts, and potential complications. Positioning-related nerve injuries, pressure points, vascular complications, and retinal ischemia are addressed. Proper padding, stabilization, and frequent reassessment are emphasized to prevent issues from sustained pressures.
Local anaesthetics.pptx
Local anaesthetics are weak bases that are prepared as water soluble salts for commercial use. They consist of an aromatic part linked to a tertiary amine via an intermediate chain containing an ester or amide linkage. Local anaesthetics act by blocking sodium channels, and their potency, onset, and duration of action depend on properties like lipid solubility, pKa, and protein binding. Adverse effects include both central nervous system and cardiovascular toxicity.
Opioids drugs
This document provides information on opioids including their classification, mechanism of action, receptors, and effects. It discusses specific opioids like morphine, fentanyl, and sufentanil. Morphine is naturally occurring and has a duration of action of 3-4 hours. Fentanyl is 75-125 times more potent than morphine. Sufentanil has an analgesic potency 5-10 times that of fentanyl. All three opioids can cause respiratory depression, the most serious adverse effect. Their use requires monitoring to prevent this.
Epidural Anaesthesia.pptx
This document provides an overview of epidural anesthesia. It discusses the anatomy of the spine and structures in the epidural space, including the dura mater, spinal cord, nerve roots, fat, and blood vessels. It describes how epidural blocks are performed in different regions of the spine and the physiology of how local anesthetics spread within the epidural space to produce analgesia. Indications, complications, and the history of epidural anesthesia are also summarized.
Techniques of mask ventilation
This document discusses techniques of mask ventilation. It begins by introducing mask ventilation as a noninvasive airway management technique used for short anesthetics or as a bridge to a more definitive airway. It then describes the types of face masks, including open masks for oxygen delivery and closed masks for ventilation. It discusses the parts of masks including the connector and harness. The document outlines techniques for mask application, including one-handed and two-handed techniques. It describes signs of inadequate mask ventilation and complications of mask ventilation such as skin allergy, nerve injury, gastric inflation, and eye or skin injuries. Throughout, it provides details on positioning, types of masks, application techniques, and complications to summarize techniques of mask ventilation.
Cirrhosis
This document discusses alcoholic liver disease and cirrhosis. It covers the pathology and symptoms of alcoholic hepatitis and cirrhosis, including jaundice, hepatomegaly, portal hypertension, and fibrosis. It also mentions common lab abnormalities seen in alcoholic liver disease such as anemia, increased bilirubin, AST>ALT. Potential treatments discussed include pentoxyfilline, steroids, and acamprosate to aid abstinence from alcohol. The Lille score is mentioned as a method to assess treatment response to steroids in alcoholic hepatitis using 6 variables.
Viral hepatitis
This document summarizes different types of viral hepatitis. It discusses the transmission routes and at-risk groups for Hepatitis A, B, C, D, E, and G. It also provides recommendations for immunization against Hepatitis B and post-exposure prophylaxis. Additionally, it briefly describes the pathological findings seen in acute viral hepatitis, including panlobular infiltration, hepatic cell necrosis, and ballooning of cells.
Antisocial personality disorder
Antisocial personality disorder is characterized by a disregard for social norms and the rights of others since age 15, as demonstrated by behaviors such as failure to conform to social norms, deceitfulness, impulsivity, irritability and aggression, recklessness, irresponsibility, and lack of remorse. The disorder typically follows a chronic unremitting course, with symptoms peaking in late adolescence. Common comorbid conditions include substance abuse disorders and depressive disorders. Treatment may involve psychotherapy, though patients often respond better in peer settings rather than isolation. Pharmacotherapy can also be used to treat comorbid conditions like anxiety, rage, and depression.
Breaking bad news
This document discusses how to effectively deliver bad news to patients. It notes that bad news includes diagnoses of serious illnesses, discussing prognoses, do-not-resuscitate orders, death pronouncements, and organ donation talks. Doctors are worried about damaging their reputation, facing questions from relatives, and dealing with legal and compensation issues when delivering bad news. The document recommends following the Kübler-Ross model of how people process bad news, which involves denial, anger, bargaining, depression, and acceptance. It emphasizes that breaking bad news requires a tactful approach and is a sensitive task that benefits from practice and guidelines.
urinary tract infection
Lower urinary tract infections (UTIs) include cystitis, urethritis, and prostatitis. Symptoms include fever, suprapubic pain, dysuria, urgency, and frequency. UTIs are classified as uncomplicated or complicated based on underlying conditions. Diagnosis involves urine analysis and culture. Treatment for uncomplicated UTIs includes antibiotics like nitrofurantoin or TMP-SMX for 5 days. Complicated UTIs require culture-guided antibiotics. Acute pyelonephritis requires IV antibiotics for 7-10 days with fluid resuscitation. Prostatitis causes urinary symptoms and pelvic pain, treated with quinolones for 28 days. Epididymo-
Ryles tube insertion and its care
Ryles tube insertion and its care
This document discusses the history, indications, types, equipment, and procedure for inserting a Ryles tube. It notes that enteral feeding has been practiced for thousands of years using enemas. There are short tubes like Levin tubes that are inserted through the nose into the stomach for decompression and washing. Longer tubes can reach the duodenum or jejunum. The procedure involves lubricating the tube, inserting it through the nose while the patient swallows, checking placement is in the stomach, and monitoring for complications. Common complications include clogged tubes, aspiration, and electrolyte imbalances.
Supraglottic airway devices
The document discusses different types of supraglottic airway devices. It describes laryngeal mask airways (LMAs) like the Classic LMA, ProSeal LMA, LMA Supreme, and Intubating LMA. It also discusses pharyngeal sealers like the Laryngeal Tube and Combitube. Cuffless preshaped sealers like the i-gel and SLIPA are also outlined. Key features of different devices are highlighted, along with their indications, proper usage, advantages, and complications. Supraglottic airways provide an alternative to endotracheal intubation and help with ventilation in difficult airway scenarios.
E fast
The document discusses the eFAST (extended focused assessment with sonography in trauma) protocol, which was developed in the mid-1990s and expanded in 2004 to include pneumothorax detection. It describes the 7 areas to scan in an eFAST exam, including the right and left upper quadrants, subxiphoid/subcostal, and suprapubic and intercostal areas. Signs of a pneumothorax on ultrasound include absent lung sliding, irregularly spaced A-lines, and no B-lines. The eFAST protocol plays an important role in diagnosing blunt abdominal injuries as physical exams and CT scans are not always possible or optimal in trauma settings.
Ca stomach
This document summarizes carcinoma of the stomach, including its anatomy, blood supply, classification, risk factors, common sites, spread, and surgical management. It notes that stomach cancer can be classified as intestinal, diffuse, or unclassified based on histological features. Risk factors include conditions like HNPCC or pernicious anemia. Surgical options include partial or total gastrectomy depending on the location and extent of the cancer, often with removal of nearby organs.
Peripheral vestibular disorders
The document discusses peripheral vestibular disorders. It describes the anatomy of the peripheral vestibular system including the vestibular end organs containing the semicircular canals and otoliths. Common peripheral vestibular disorders are then outlined, including Meniere's disease, BPPV, vestibular neuronitis, and labyrinthitis. Vestibular neuronitis and labyrinthitis are discussed in more detail regarding their causes, symptoms, and treatment options. Other causes of peripheral vestibular disorders mentioned include vestibulotoxic drugs, head trauma, perilymph fistula, syphilis, and acoustic neuroma.
Developmental dysplasia of hip
This document discusses congenital dislocation of the hip. It defines developmental dysplasia of the hip as partial or complete displacement of the femoral head from the acetabulum present since birth. Risk factors include being female, a first born, having a family history, or experiencing faulty intrauterine positioning such as breech presentation. Diagnosis involves physical exam findings like leg length discrepancy and a positive Ortolani or Barlow maneuver. Treatment depends on age and may involve closed or open reduction along with hip spica casting or surgical procedures like femoral and pelvic osteotomies.
Cranial nerve iv and vi
The trochlear nerve emerges from the dorsal aspect of the brainstem and is the only cranial nerve that decussates. It innervates the superior oblique muscle. The superior oblique muscle functions to depress, abduct, and intort the eye. Damage to the trochlear nerve results in limitations in depression, especially in adduction, which can cause diplopia that is worse on downward gaze. Compensatory head positioning such as tilting the head contralaterally may help alleviate diplopia.
More from Tess Jose (20)
Anesthesia implication in Dextrocardia and situs inversus
Anesthesia implication in Dextrocardia and situs inversus
Recently uploaded
Nano-gold for Cancer Therapy chemistry investigatory project
chemistry investigatory project
The development of nanogold-based cancer therapy could revolutionize oncology by providing a more targeted, less invasive treatment option. This project contributes to the growing body of research aimed at harnessing nanotechnology for medical applications, paving the way for future clinical trials and potential commercial applications.
Cancer remains one of the leading causes of death worldwide, prompting the need for innovative treatment methods. Nanotechnology offers promising new approaches, including the use of gold nanoparticles (nanogold) for targeted cancer therapy. Nanogold particles possess unique physical and chemical properties that make them suitable for drug delivery, imaging, and photothermal therapy.
Artificial Intelligence Symposium (THAIS)
Artificial Intelligence Symposium (THAIS). Parc Taulí. Sabadell
biomechanics of running. Dr.dhwani.pptx
The biomechanics of running involves the study of the mechanical principles underlying running movements. It includes the analysis of the running gait cycle, which consists of the stance phase (foot contact to push-off) and the swing phase (foot lift-off to next contact). Key aspects include kinematics (joint angles and movements, stride length and frequency) and kinetics (forces involved in running, including ground reaction and muscle forces). Understanding these factors helps in improving running performance, optimizing technique, and preventing injuries.
5 Effective Homeopathic Medicines for Irregular Periods
Discover the benefits of homeopathic medicine for irregular periods with our guide on 5 common remedies. Learn how these natural treatments can help regulate menstrual cycles and improve overall menstrual health.
Visit Us: https://drdeepikashomeopathy.com/service/irregular-periods-treatment/
NAVIGATING THE HORIZONS OF TIME LAPSE EMBRYO MONITORING.pdf
Time-lapse embryo monitoring is an advanced imaging technique used in IVF to continuously observe embryo development. It captures high-resolution images at regular intervals, allowing embryologists to select the most viable embryos for transfer based on detailed growth patterns. This technology enhances embryo selection, potentially increasing pregnancy success rates.
NARCOTICS- POLICY AND PROCEDURES FOR ITS USE
This document outlines policies and procedures for handling narcotic and controlled drugs in NABH accredited hospitals.
Histololgy of Female Reproductive System.pptx
Dive into an in-depth exploration of the histological structure of female reproductive system with this comprehensive lecture. Presented by Dr. Ayesha Irfan, Assistant Professor of Anatomy, this presentation covers the Gross anatomy and functional histology of the female reproductive organs. Ideal for students, educators, and anyone interested in medical science, this lecture provides clear explanations, detailed diagrams, and valuable insights into female reproductive system. Enhance your knowledge and understanding of this essential aspect of human biology.
June 2024 Oncology Cartoons By Dr Kanhu Charan Patro
une 2024 Oncology Cartoons By Dr Kanhu Charan Patro
SENSORY NEEDS B.SC. NURSING SEMESTER II.
Sensation is the neurological process by which we become aware of surrounding environment.
Patellar Instability: Diagnosis Management
Pictorial and detailed description of patellar instability with sign and symptoms and how to diagnose , what investigations you should go with and how to approach with treatment options . I have presented this slide in my 2nd year junior residency in orthopedics at LLRM medical college Meerut and got good reviews for it
After getting it read you will definitely understand the topic.
Pollen and Fungal allergy: aeroallergy.pdf
Pollen and Fungal allergy: aeroallergy.pdfChulalongkorn Allergy and Clinical Immunology Research Group
Pollen and Fungal allergy
Presented by Chaloemchai Chumsaengchotsakul, MD.
June 14, 2024Acute Gout Care & Urate Lowering Therapy .pdf
In this document , the management of acute gout attacks and a description of urate lowering therapy is mentioned.
Foundation of Yoga, YCB Level-3, Unit-1
Unit -1 of Yoga certification board, level 3, all topics covered. An exam conducted by ministry of Ayush for yoga enthusiastic students.
Pharmacology of 5-hydroxytryptamine and Antagonist
5-hydroxytryptamine or 5-HT or Serotonin is a neurotransmitter that serves a range of roles in the human body. It is sometimes referred to as the happy chemical since it promotes overall well-being and happiness.
It is mostly found in the brain, intestines, and blood platelets.
5-HT is utilised to transport messages between nerve cells, is known to be involved in smooth muscle contraction, and adds to overall well-being and pleasure, among other benefits. 5-HT regulates the body's sleep-wake cycles and internal clock by acting as a precursor to melatonin.
It is hypothesised to regulate hunger, emotions, motor, cognitive, and autonomic processes.
Cervical Disc Arthroplasty ORSI 2024.pptx
Indication and installation of a mobile cervical disc prosthesis. Benefits of the PRODISC C VIVO mobile disc prosthesis (Centinel Spine)
Recently uploaded (20)
Tele Optometry (kunj'sppt) / Basics of tele optometry.
Tele Optometry (kunj'sppt) / Basics of tele optometry.
Nano-gold for Cancer Therapy chemistry investigatory project
Nano-gold for Cancer Therapy chemistry investigatory project
5 Effective Homeopathic Medicines for Irregular Periods
5 Effective Homeopathic Medicines for Irregular Periods
NAVIGATING THE HORIZONS OF TIME LAPSE EMBRYO MONITORING.pdf
NAVIGATING THE HORIZONS OF TIME LAPSE EMBRYO MONITORING.pdf
June 2024 Oncology Cartoons By Dr Kanhu Charan Patro
June 2024 Oncology Cartoons By Dr Kanhu Charan Patro
Pharmacology of 5-hydroxytryptamine and Antagonist
Pharmacology of 5-hydroxytryptamine and Antagonist
Rheumatology
- 2. Inflammation of blood vessel and its complication.
- 3. Large vessel Takayasu Giant cell arthritis Medium vessel Polyarteritis nodosa kawasaki Small vessel Anca associated Immune complex
- 4. Pulseless disease, young female arteritis, or Harbitiz-Raedu syndrome F>>M ;<40years Takayasu’s disease is a chronic vasculitis mainly involving the aorta and its main branches such as the brachiocephalic, carotid, subclavian, vertebral and renal arteries as well as the coronary and pulmonary arteries.
- 5. : The early symptoms are non-specific. These include fever, myalgia, arthralgia ,weightloss,anorexia. tenderness of carotid artery subclavian, vertebral and renal arteries as well as the coronary and pulmonary arteries
- 6. This form of systemic connective tissue disease occurs in the elderly, in the sixth and seventh decades. It is characterized by panarteritis with giant cell infiltration, predominantly affecting the cranial vessels. The etiology is not established.
- 7. Both sexes are equally affected Onset is with severe,unilateral or bilateral headache in the temporal or less commonly in the occipital regions( visual loss), associated with vomiting. Diagnostic feature is the palpably thickened, tender and non pulsatile temporal arteries. Loss of vision due to occlusion of the ophthalmic artery is a dreaded complication. Rarely patients present as facial neuralgia with jaw claudication on chewing, deafness, necrosis of the tongue, or an associated widespread necrotizing arteritis of small vessels which presents as peripheral neuropathy.
- 8. ESR often exceeds 100 mm Temporal artery biopsy Arterial doppler
- 10. Characterised by severe pain and stiffness in the neck, back, shoulder, upper arms and thighs, particularly worse in the morning
- 11. Polyarteritis nodosa (PAN) is characterized by the formation of multiple nodules along blood vessels, mainly affecting the smaller arteries, arterioles, and adjacent veins. Capillaries are spared. Pathologically the lesion consists of polymorphonuclear infiltration, necrosis and aneurysmal dilatation of the arterial wall
- 12. Idiopathic Hepatitis B
- 13. Though all ages may be affected, adults suffer more frequently. Males predominate. The onset is variable. The onset is with general symptoms like fever, muscle pains, arthralgia, rash and anorexia.
- 14. Skin manifestation Inflammatory nodules ranging in size from 5- 10 mm may be palpable over the superficial arteries. These nodules are tender. Livido reticularis
- 16. Rheumatoid Arthritis is a chronic inflammatory autoimmune disease that involves the joints and may cause systemic manifestations.