Arthroscopic ACL Reconstruction By Dr Shekhar ShrivastavDelhiArthroscopy
Arthroscopic Acl Reconstruction By Dr Shekhar Shrivastav.
HOW NORMAL KNEE WORKS ?
The knee is the largest joint in the body, and one of the most easily injured. It is made up of the lower end of the thigh bone(femur), the upper end of the shin bone (tibia), and the knee cap (patella), which slides in a groove on the end of the femur. Four bands of tissue, the anterior and posterior cruciate ligaments, and the medial and lateral collateral ligaments connect the femur and the tibia and provide joint stability. The surfaces where the femur, tibia and patella touch are covered with articular cartilage, a smooth substance that cushions the bones and enables them to glide freely. Semicircular rings of tough fibrous-cartilage tissue called the lateral and medial menisci act as shock absorbers and stabilizers.
WHAT IS THE ROLE OF ACL ?
ACL along with other ligaments of the knee joint and meniscus provides stability to the knee joint.
WHAT IS LIGAMENT RECONSTRUCTION ( ACL ) ?
Ligament reconstruction involves replacing the torn ligament with a tendon (graft) from your knee and fixing the graft in place with screws. This procedure is performed with the use of the arthroscope. The anterior cruciate ligament (ACL) is the most common ligament requiring reconstruction procedures. The torn ligament is excised arthroscopically and new ligament is prepared by ligament grafts taken from your own body. Bony tunnels are prepared in femur and tibia using specialized instruments through which the new ligament is passed and fixed with special screws. This procedure requires relative rest or leave from your work or studies for about 2-3 weeks after which you will be allowed normal day to day activities.
WHEN CAN THE PATIENT BE AMBULATED AFTER SURGERY ?
The patient can walk from the same evening of the surgery. Initially the patient is advised to walk with a brace and a walking cane. Strengthening and range of motion exercises for the knee are started from the next day. The patient is discharged from the hospital 2nd or 3rd day after surgery. The patient can walk without support by 10-14 days depending on muscle strengthening. Slow Jogging and other strenuous activities are permitted after 3 months and the patient can return to active sports only 8-9 months after surgery.
Torn ACL Reconstructed ACL
For Further Queries contact your Orthopedic Surgeon at
+ 91 9971192233
Posterolateral corner injuries of knee joint Samir Dwidmuthe
Missed posterolateral corner injuries of knee joint is a common cause for failure of ACL and PCL reconstruction only next to malpositioned tunnels.
Isolated PLC injuries are uncommon, making up <2% of all acute knee ligamentous injuries. Covey JBJS 2001
Incidence of PLC injuries associated with concomitant ACL and PCL disruptions are much more common (43% to 80%). Ranawat JAAOS 2008
A recent (MRI) analysis of surgical tibialplateau fractures demonstrated an incidence of PLC injuries in 68% of cases. Gardner JOT 2005
Take home message
PLC injuries to be ruled out in every case of ACL& PCL rupture.
Neurovascular integrity to be checked in every case.
Grade I & II can be managed conservatively.
Grade III Acute- Repair.
Grade III Chronic- Anatomic PLC recon.
Beware of varus knee alignment.
Arthroscopic ACL Reconstruction By Dr Shekhar ShrivastavDelhiArthroscopy
Arthroscopic Acl Reconstruction By Dr Shekhar Shrivastav.
HOW NORMAL KNEE WORKS ?
The knee is the largest joint in the body, and one of the most easily injured. It is made up of the lower end of the thigh bone(femur), the upper end of the shin bone (tibia), and the knee cap (patella), which slides in a groove on the end of the femur. Four bands of tissue, the anterior and posterior cruciate ligaments, and the medial and lateral collateral ligaments connect the femur and the tibia and provide joint stability. The surfaces where the femur, tibia and patella touch are covered with articular cartilage, a smooth substance that cushions the bones and enables them to glide freely. Semicircular rings of tough fibrous-cartilage tissue called the lateral and medial menisci act as shock absorbers and stabilizers.
WHAT IS THE ROLE OF ACL ?
ACL along with other ligaments of the knee joint and meniscus provides stability to the knee joint.
WHAT IS LIGAMENT RECONSTRUCTION ( ACL ) ?
Ligament reconstruction involves replacing the torn ligament with a tendon (graft) from your knee and fixing the graft in place with screws. This procedure is performed with the use of the arthroscope. The anterior cruciate ligament (ACL) is the most common ligament requiring reconstruction procedures. The torn ligament is excised arthroscopically and new ligament is prepared by ligament grafts taken from your own body. Bony tunnels are prepared in femur and tibia using specialized instruments through which the new ligament is passed and fixed with special screws. This procedure requires relative rest or leave from your work or studies for about 2-3 weeks after which you will be allowed normal day to day activities.
WHEN CAN THE PATIENT BE AMBULATED AFTER SURGERY ?
The patient can walk from the same evening of the surgery. Initially the patient is advised to walk with a brace and a walking cane. Strengthening and range of motion exercises for the knee are started from the next day. The patient is discharged from the hospital 2nd or 3rd day after surgery. The patient can walk without support by 10-14 days depending on muscle strengthening. Slow Jogging and other strenuous activities are permitted after 3 months and the patient can return to active sports only 8-9 months after surgery.
Torn ACL Reconstructed ACL
For Further Queries contact your Orthopedic Surgeon at
+ 91 9971192233
Posterolateral corner injuries of knee joint Samir Dwidmuthe
Missed posterolateral corner injuries of knee joint is a common cause for failure of ACL and PCL reconstruction only next to malpositioned tunnels.
Isolated PLC injuries are uncommon, making up <2% of all acute knee ligamentous injuries. Covey JBJS 2001
Incidence of PLC injuries associated with concomitant ACL and PCL disruptions are much more common (43% to 80%). Ranawat JAAOS 2008
A recent (MRI) analysis of surgical tibialplateau fractures demonstrated an incidence of PLC injuries in 68% of cases. Gardner JOT 2005
Take home message
PLC injuries to be ruled out in every case of ACL& PCL rupture.
Neurovascular integrity to be checked in every case.
Grade I & II can be managed conservatively.
Grade III Acute- Repair.
Grade III Chronic- Anatomic PLC recon.
Beware of varus knee alignment.
Osseous anatomy, Types of approaches(Position,landmarks,Incision,Superficial and Deep surgical dissection) , structures at risk, Extensile approaches with diagrams and eponymous .
Detailed history and its evaluation , examination of spine in general and local with special tests in cervical , thoracic outlet syndrome , lumbar spine and SI joint with diagrams, neurological examination both sensory and motor.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
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Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
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The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
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Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
2. BRIEF INTRODUCTION
• Defined as Fracture occurring at or proximal to
surgical neck
• 80 % of all humeral #
• 7% of all #..
• Pt > 65 yrs – Second most common fracture of
the upper extremity
• 65% of # occur in Pt’s > 60 yrs
• F:M – 3:1
• Incidence increases with age.
3. MECHANISM
• High energy injuries in normal bone resulted in
marked communition of the surgical neck area with
extension into proximal humerus shaft.
• FOOSH with shoulder in flexion , abduction &
internal rotation, the humerus head is forced into
valgus, hinging around the inferomedial aspect of
the stronger calcar bone.
• Patient falls directly on the shoulder, the deforming
forces on the humeral head will create a varus
deformity.
4. • Most # are non displaced, good prognosis –
nonsurgical
• Risk factors: Poor quality bone ,impaired vision &
balance, medical co morbidities, decreased muscle
tone.
• Young Pts – High energy trauma
• Severe soft tissue disruption always require surgical
intervention
• Seizures & electric shock – indirect causes
5.
6. ANATOMY
• Muscle insertions on these
segments and the magnitude
and directionof the forces
causing injury, determine the
pattern of fracture lines
,displacement and angulation.
• Greater tuberosity has three
regions into which the
supraspinatus, infraspinatus,
and teres minor insert
• Subscapularis tendon
lesser tuberosity, which is
separated from the greater
tuberosity by the bicipital
groove.
7. VASCULARITY
• The axillary artery is known as “tethered
trifurcation” at the level of the surgical
neck.
• Tethered Trifurcation - In the third part of
axillary artery where the vessel is
tethered to the humerus by the anterior
and posterior humeral circumflex
branches
• ACHA is the main arterial supply , in
complex fractures PCHA was preserved
in 85% of cases and only 20% of ACHA.
• If the medial calcar of the humerus is
spared by the fracture, the vessel is
spared.
12. DEFORMING FORCES OF PHF
• The greater tuberosity is
pulled posteromedially by
the effect of the supra-
and infraspinatus
tendons.
• The lesser tuberosity is
pulled antero medially by
the subscapularis
tendon.
• The shaft segment is
pulled anteromedially by
the pectoralis major
tendon.
13. IMAGING AND OTHER DIAGNOSTIC
STUDIES
• Radiographs :- consist of three views
• AP- Perpendicular to the plane of scapula(Grashey view)
• Neer View (Scapula Y view)
• Axillary view
• CT scan
• MRI
• Angiography
• USG
• Dual energy X–ray absorptiometry (DEXA).
18. NON-OPERATIVE TREATMENT OF
PROXIMAL HUMERAL FRACTURES
• The majority of proximal humeral fractures are
nondisplaced or minimally displaced and nonoperative
treatment is indicated.
• Fracture stability can be assessed both
radiographically and clinically.
• Radiographically, stable fractures exhibit impaction or
interdigitation between bone fragments
• Clinically, fracture stability may be assessed by
palpating the proximal humerus just distal to the
acromion with one hand, while rotating the arm at the
elbow with the other. If the proximal humerus is felt to
move as a unit with the distal segment, the fracture is
19. INDICATIONS FOR NON OPERATIVE
TREATMENT
• Surgical neck fractures with contact and no gross
instability.
• Greater and lesser tuberosity fractures.
-Displacement < 1cm
-Overlap with the head < 20%
• Varus posteromedial fractures.
-Varus < 45 degrees.
-Minor tuberosity displacement.
• Valgus fractures.
-Head not pointing superior or lateral.
-Minor tuberosity displacement.
• Elderly patients with contraindications for anaesthesia.
20. RELATIVE CONTRAINDICATIONS
• Open fracture
• Associated vascular injury
• Polytrauma
• Associated disruption of suspensory complex
shoulder
• Pathologic fractures , Head dislocation ,
Excessive tuberosity displacement , excessive
varus or valgus displacement in frail , elderly
patients.
21. • In GT fractures displacement must be less than 1 cm for
non operative, if GT heals with displacement of > 1 cm lead
to impingement in ext. rotation, elevation or both.
• If LT heals with displacement of > 1 cm lead to
impingement in internal rotation.
• Varus posteromedial fractures have a better outcome than
valgus impacted fractures with non operative
management.
• Valgus fractures having worst prognosis when treated non
operatively , marked valgus displacements leads to higher
chances of disruption of the vascularity of the humeral
head and more displacement of tuberosities.
22. NON OPERATIVE TREATMENT
PROTOCOL
• Immobilization of the arm to the chest using a simple
collar and cuff sling.
• Gilchrist or Velpeau type shoulder immobilizer used.
• For fractures involving GT fragment, it is better to
immobilise in some external rotation because the
posterosuperior rotator cuff is under more tension in
int. rotation.
• For fractures of surgical neck, to counteract the pull of
pectoralis major on the proximal aspect of diaphysis ,
considerate to put a pillow or small bump in the axilla.
23.
24. • Surgical neck fractures with potential for instability
should be evaluated with radiographs on a weekly
basis for 1st
4 weeks.
• Rest of the fractures obtain radiographs at week 3,6
& 12 after the injury.
• Early sling immobilization for 7 – 10 days.
• Active finger , wrist , elbow movements.
• First 2-3 weeks , passive ROM exercises are best
tolerated in supine position.
• At 6 weeks, active assisted ROM exercises are
added.
• Strengthening exercises are started after 3 months.
25. OUTCOMES
• 60 % of the shoulders can be expected to be
completely pain free.
• 10% complain of moderate to severe pain.
• Complete recovery only seen in 46%.
• Motion is usually limited, between 90 – 120 degrees
of elevation.
• Unsatisfactory results-
Valgus impacted fractures > Varus
posteromedial fractures > Isolated fractures of GT.
26. OPERATIVE TREATMENT
Indications :
• Fracture dislocations.
• Head depression fractures involving more than
20% - 25% of the articular surface.
• Unstable surgical neck fractures with tuberosity
displacement over 1cm.
• Severe valgus/varus displacement of humeral head.
• Open fractures , fractures associated with
disruption of suspensory complex of shoulder.
• Fractures associated with vascular injuries and
pathologic fractures.
27. Surgical options :
• Open Reduction and Internal Fixation
• Intramedullary Nailing
• Closed Reduction and Internal fixation with
Percutaneous pins and screws
• Tension Band Fixation
• Hemiarthroplasty
• Reverse Total Shoulder Arthroplasty
28. Open reduction and internal fixation
DELTOPECTORAL APPROACH
• Most common surgical exposure used for both internal
fixation and arthroplasty worldwide.
1. Skin incision is placed lateral to the tip of coracoid i.e,
junction between the medial 3rd
and lateral 2/3rd
of distance
between tip of coracoid and acromion.
2. Identify deltopectoral interval and cephalic vein proximally
and mobilizing the cephalic vein medially or laterally allows
for improved exposure .
29. 3. Arm is then placed in abduction to relax the deltoid.
4. Anterior distal deltoid insertion is gently elevated off the
lateral side of the humerus , just enough to allow the
correct lateral plate placement.
5. Next , clavipectoral fascia is opened , careful soft tissue
management is required to avoid devascularization of the
fractured fragments.
6. Identification of the long head of the biceps on the
anterior aspect of the proximal shaft will facilitate
understanding of fracture pattern and aid with fracture
reduction and plate placement.
7. Proximity to the ascending branch of ACHA, dissection
around and behind long head of biceps tendon avoided
whenever possible.
30. NOTE:
-Biceps tendon may be injured with the fracture
then biceps tenodesis commonly performed.
- Biceps tendon intact may make fracture reduction
more difficult , especially in subacute injuries with
substantial humerus shortening , then also tenodesis done.
31.
32. SUPERIOR DELTOID SPLITTING APPROACH
• This approach to the fracture by longitudinal incision
starting from anterolateral corner of acromion and
splitting the deltoid (Ant. and Middle raphe).
• This approach benefits better access to the greater
tuberosity and lateral humeral shaft plate fixation and
lesser disruption of the fracture vascularity.
• This approach caries a higher risk of iatrogenic axillary
nerve injury ( If deltoid split more than 5 cm ).
• This approach not useful for antero inferior fracture
dislocations.
33.
34.
35. Internal fixation using Locking plates
• Orif using periarticular precontoured locking plates.
• High rates of post operative displacement and varus
collapse mainly in elderly with 3 & 4 part fractures.
• Most frequent complication :
Intra-articular screw penetration.
1. Primary screw penetration ( Intra operative insertion of
excessively long screws )
2. Secondary screw penetration ( fracture collapse with
loss of reduction leading to late screw penetration )
37. • Structural bone grafting and cementing techniques have
demonstrated in increase in the strength of fixation and
decrease the rates of loss of reduction ,collapse and screw
penetration.
• The use of fibular strut grafting has been described to
improve stability of varus-impacted fractures in which the
medial calcar may not be reliably reconstructed.
• Goal being to create a buttress at the inferior aspect of
the anatomic neck to prevent delayed varus collapse
• Split of the head in only two segments and fracture dislocations
in the very young patients considered for plate fixation.
• In fracture dislocations, head is reduced first and provisional
fixation is done.
39. Placement of plate and fixation
• The plate should be placed low enough, so that its superior
border is at least 5mm below the superior edge of the
greater tuberosity.
• But high enough so that lower locking screws will support
the calcar region.
• In osteoporotic patients- typically augment with cement at
least 4 screws, especially at superior aspect of the head
and at calcar region. About 5CC of cement is introduced
through each screw.
• Once the plate and screws have been placed
transtendinous sutures are tied onto the plate to provide
additional fixation
43. Internal fixation : Intramedullary Nailing
• Biomechanical advantages in osteoporotic bone
• It allows stabilization with minimum surgical invasion
• Indications-
• Displaced two part surgical neck fractures
• Pathological fractures
• Contraindications-
• Varus four-part fractures with lateral displacement
of the humeral head
• Head-splitting fractures
44. • Most modern implants , using now a days are “Third
Generation Proximal Humeral IM nails”.
• The most usual entry point is just lateral to the articular
surface of the humeral head and just medial to greater
tuberosity.
• In lateral view entry point should be approximately
1 to 1.5 cm posterior to anterior margin of GT.
• IM nailing violates the rotator cuff , and thus may lead to
post operative shoulder pain.
45.
46. Closed Reduction and internal fixation
with Percutaneous pins and screws
• It has theoretical advantage of minimizing soft tissue
trauma, thereby promoting healing and reducing the
risk of AVN of the humeral head.
• It also has the advantage of decreased scarring in the
• scapulohumeral interface and subsequent easier
rehabilitation.
• Indications-
• Fracture without significant communition in pt with
good quality bone.
• Pt should be willing to comply with postop care
plan.
47. • Contra indications
• Comminution of calcar ,greater tuberosity and
osteopenia are absolute contraindications.
• Inability to reduce Fracture Fragments
• Fracture Dislocation
• Non Compliant patients
48. To avoid injury to the axillary nerve, lateral
pins should enter the humeral cortex at a
point at least twice the distance from the
upper aspect of the head to the inferior
head margin with the wire angulated
approximately 45 degrees to the cortical
surface. The end point for the greater
tuberosity pin should be >2 cm from the
inferior most margin of the humeral head.
49. Tension Band Fixation (Osteosuture)
• It is most frequently used as an adjunct to plates and
screw fixation, IM nailing, and arthroplasty.
• The main goal of tension band fixation is the
neutralization of tension forces generated by the
rotator cuff at the level of the tuberosities, and
bending at the level of the surgical neck.
• The main advantage of tension band fixation is the
minimal amount of hardware that is required. Thus
avoiding the risks associated with hardware, which
include pain, neurovascular compromise, migration,
failure, and the need for removal.
53. Hemiarthroplasty
• Also known as humeral head replacement
Indications-
• Four-part fractures,
• Three-part fractures in older patients with
osteoporotic bone,
• Fracture-dislocations
• Comminuted head-splitting fractures
• Head depression fractures involving more than 40%
of the articular surface
54. Favours hemiarthroplasty –
-Intact tuberosity / large tuberosity fragments
-Younger , Male
Contraindications-
• Active infection of the shoulder joint and/or the
surrounding soft tissue
55. Postoperative Care
• Passive range-of-motion exercises are started on the
first postoperative day. They are limited to neutral
rotation and 90 degrees of forward elevation.
• Patients are followed up clinically and radiographically
at 2 weeks, 6 weeks, and 3 months.
• Active-assisted range-of-motion exercises are started
at 6 weeks and strengthening exercises at 3 months
56.
57. Reverse Total Shoulder Arthroplasty
• By placing a hemisphere onto the glenoid surface and a
concave tray onto the humeral stem, reverse shoulder
arthroplasty allows for rotation to occur at the glenohumeral
joint through activation of the deltoid, without the need for a
functional rotator cuff/tuberosity unit.
Indications
• Complex acute proximal humeral fractures
• Proximal humerus malunion or nonunion where the normal
anatomy of the tuberosities cannot be reliably restored
• Glenohumeral joint arthritis with advanced rotator cuff
• pathology
• Massive irreparable rotator cuff tears with painful
pseudoparesis
59. The ideal candidate for reverse total shoulder arthroplasty in a patient with a
complex proximal humerus fracture is a low demand elderly patient with pre-
existing rotator cuff pathology and glenoid pathology.
60. COMPLICATIONS
• Avascular necrosis of humeral head and/or tuberosity
• Non-union- The normal time for clinical union of a
proximal humeral fracture is typically 4 to 8 weeks.
Nonunion is said to be present if a fracture site is still
mobile 16 weeks post injury.
• Malunion
• Post-traumatic Shoulder stiffness
• Post traumatic arthritis
• Infection
• Iatrogenic-such as inadequate reduction, incorrectly
positioned implants, screw penetration into the joint, loss
of fixation, tuberosity disruption, and nerve injury.
• Heterotopic bone formation
61. Risk of Avascular Necrosis
Four-part fractures and fracture dislocations are
considered to have the highest risk for humeral head
necrosis.
Hertel’s Criteria
• Metaphyseal extension of the humeral head < 8
mm
• Medial hinge disruption of >2 mm, and
• Fracture through anatomical neck
• The combination above above factors had 97%
positive predictive value for humeral head ischemia
62. Metaphyseal extension of the humeral
head of >9 mm
Metaphyseal extension of the humeral
head of <8 mm.
64. Humeral Head Osteonecrosis
• Due to primary and secondary causes.
Primary- Due to fracture pattern.
Secondary- Due to consequence of operative
management ( Ecxessive fracture manipulation and
stripping of soft tissues).
• Predisposing factors : Medical comorbidities , Drug
treatment , smoking , Alcohol abuse and malnutrition.
65. • Radiological changes : Patchy humeral head sclerosis, to
complete humeral head resorption and collapse.
• In some individuals, osteonecrosis is not associated with
severe symptoms and no treatment is required other than
periodic follow up evaluations.
TREATMENT :
Patients who have early radiological changes , Core
Decompression may done occasionally.
but most patients have advanced collapse and enough
symptoms then conversion to shoulder arthroplasty.
patients with isolated humeral head AVN and no
other fracture related issues , Hemiarthroplasty is the
procedure of choice.