Dr. Teo Zue Hiong
Content
   Hydrocele
   Hematocele
   Spermatocele & eppidydymal cyst
   Varicocele
   Testicular tumor
   Testic torsion
   Epidydymo-orchitis

Undescended testis
hydrocele
 Excessive collection of fluid within tunica
  vaginalis
 Divided into congenital & acquired
  (further divided into primary and
  secondary )
   Congenital
   -patent connection with peritoneal cavity via patent
    processus vaginalis

   Acquired
Primary:
-Idiopathic
-Can reach very large size with no pain

Secondary:
-Trauma/infection/tumor
-Small size. Tender if underlying testis tender
PE
 Usually bilateral
 Translucent
 Testis impalpable
complication
   Rupture
   Hematocele
   Infection
   Hernia of hydrocele sac
   Sac wall calcification
   Testic atrophic
hamatocele
   Collection of blood within tunica
    vaginalis

   Due to trauma or underlying malifnant

   Not translucent (distinguished from
    hydrocele)
varicocele
   Dilated, tortuous & elongated veins of
    pampiniform plexus of spermatid vein
    (varicose vein in spermatid cord)

   90% on the left because Lt testicular vein drain
    into high pressure renal vein where the Rt
    testicular vein drains directly into IVC

   Usually asymptomatic but pt usually infertile as
    it increases scrotal temperature which affect
    normal sperm function
Spermatocele & epididymal cyst
   Testis are palpable

 Cant distinguished clinical. Only by
  aspiration.
-Spermatocele: slightly grey, opaque fluid
  containing spermatozoa
-Epidydymal cyst: clear fluid
Testicular tumor
 20-40 years old
 >90% are derived from germ cells


 Most common
-Seminomas: derived from spermatocyte
-Teratoma: dereved from 3 germ cell layer
  ectoderm/mesoderm/endoderm
Presentation

-solid testicular lump
- painless
- may cause secondary hydrocele
spread
 Spread to para-aortic LN > thoracic duct
  > supraclavicular LN
 Inguinal LN are not involved unless
  spread to scrotal skin
Investigation
   USG for scrotal content

   Chest X-ray for lung secondaries

   Tumour marker
o   B-HCG
o   AFP
o   LDH

   CT for staging
staging
 I: confined to testis
 II: retroperitoneal LN
 III: metastasis above diaphragm
  confined to LN
 IV: extralymphatic metastasis
treatment
 orchidectomy
 Radiotherapy
 Chemotherapy
 LN dissection
Acute epidydymo- orchitis
   Primarily an infection of the epididymis
    but then spread into testis

   Organism : chlamydia/gonococcus/
    E.coli

   May be assoc with UTI
Presentation
 Acute severe testicular pain
 Pain is decrease by raising the testis
 Scrotal skin red, hot & edematous
Aetiology and pathological
features
 Rare,except a/w mumps
 Blood-borne infection
 Surgical procedure on the lower urinary
  tract,e.g. TUR
 Organism: Neisseria gonorrhoeae,
    Escherichia coli and Chlamydia. In young man,
    the commonest is Chlamydia
   Tuberculosis
Clinical features
 Preceding Hx of an operation or of
  dysuria, frequency and heamaturia
 Acute pain in scrotum,swelling
 Epididymis:acutely tender and
  enlarged(although it maybe difficult to
  differentiate from the equally tender
  testis)
 Overlying redness and oedema maybe
  present
Investigation
   FBC: leucocytosis
   Blood culture: helpful to direct antibiotic
    treatment
   Urinalysis: pyuria, organism maybe
    revealed by culture
   Aspiration of the epididymis
   USG: increased blood flow
Management
 Bed rest,scrotal elevation
 Tetracycline or erthromycin
 Other antiobiotic refer to culture
 Partner should also be investigated and
  treated
Epidemiology
 Both testes are undescend in 30% of
  premature infants
 Term:3%
 One year:1%
 Spontaneous descent after one year is
  rare
Aetiology
 Failure of migration along the normal
  line of descent
 Ectopic testis:testicle deviates away
  from the line and lie in front of the penis
  in the superficial inguinal pouch,in the
  perineum or in the thigh.(reason
  unknown)
Risk factor
   Prematurity
   Low birth weight
   Twin gestation
   Down syndrome(fetus) or other chromosomal
    abnormality
   Gestational diabetes mellitus
   Prenatal alcohol exposure
   Hormonal abnormalities (fetus)
   Toxic exposures in the mother
   Mother younger than 20
   A family history of undescended testes
Clinical features
An empty scrotal sac or hemiscrotum at 1 year
  indicates:
 Proximal to the external inguinal
  ring(undescended)
 Truly absent
 Retractile-the cremaster muscle reflexly pulls the
  organ up towards the inguinal canal
 Ectopic
Complication
   Infertility:inevitable in bilateral and
    common in unilateral
    undescent,frequent in those who are
    undescent treated.
   Torsion
   Trauma
   Inguinal hernia
   Malignant disease
Investigation
   USG,CT and laparoscopy


Management
   Target is to bring the testicle with its blood supply
    into the scrotum as early as possible
 Orchidopexy:should be done beyong puberty
   Testicular prosthesis can be placed in the
    scrotum
1 Epididymis
2 Head of epididymis
3 Lobules of epididymis
4 Body of epididymis
5 Tail of epididymis
6 Duct of epididymis
7 Deferent duct (ductus deferens or vas
deferens)
Testicular torsion
   Testicular torsion occurs when the spermatic
    cord(from which the testicle is suspended)
    twists, cutting off the testicle's blood
    supply(ischemia)
   Cause: recognised complication of testicular
    maldescent wherein the testis is inadequately
    affixed to the scrotum allowing it to move freely
    on its axis and susceptible to induced twisting of
    the cord and its vessels.
   Occurs most probably between birth and early
    adolescence
Twist VS Untwist
 Twist deprives the organ of its blood
  supply
 If untwist does not take place within 6
  hours,ischaemia is irreversible,gangrene
  develops and the testis either
  suppurates or atrophies
Presentation & Finding
   Acute severe testicular pain(affected side)
   Testis is tender,swollen and hang higher
    up(compared to other side)
   Poorly localized central abdo pain
   Vomitting(sometimes)
   Scrotal skin become red,hot and edematous in
    later stage
   Palpation may feel the twisted cord

Pain is increase or no improvement by raising the
  testis
Investigation
 Urinalysis:sterile,acellular urine
 USG:absence of blood supply to the
  affected testicle
Management
 Surgical emergency
 Non-operative
    Maybe possible to de-rotate the testis
   Surgical
    Failure of non-operative reduction require
     emergency operation
    The testis is de-rotated and fixed
    The gangrenous testis is removed
Dignosis of lumps in the scrotum
1.   Can u get above it? : if not, mostlikely is an
     inguinoscrotal hernia.(or a hydrocele extending
     proximally)
2.   Is it separate from the testis?
3.   Is it cystic or solid?
    Separate and cystic - epididymal cyst or
     spermatocele
    Separate and solid - epididymitis (may also
     orchitis)
    Testicular and cystic – hydrocele
    Testicular and solid – tumour, orchitis
Other scrotal swelling by Dr. Teo

Other scrotal swelling by Dr. Teo

  • 1.
  • 2.
    Content  Hydrocele  Hematocele  Spermatocele & eppidydymal cyst  Varicocele  Testicular tumor  Testic torsion  Epidydymo-orchitis Undescended testis
  • 6.
    hydrocele  Excessive collectionof fluid within tunica vaginalis  Divided into congenital & acquired (further divided into primary and secondary )
  • 7.
    Congenital  -patent connection with peritoneal cavity via patent processus vaginalis  Acquired Primary: -Idiopathic -Can reach very large size with no pain Secondary: -Trauma/infection/tumor -Small size. Tender if underlying testis tender
  • 8.
    PE  Usually bilateral Translucent  Testis impalpable
  • 9.
    complication  Rupture  Hematocele  Infection  Hernia of hydrocele sac  Sac wall calcification  Testic atrophic
  • 11.
    hamatocele  Collection of blood within tunica vaginalis  Due to trauma or underlying malifnant  Not translucent (distinguished from hydrocele)
  • 12.
    varicocele  Dilated, tortuous & elongated veins of pampiniform plexus of spermatid vein (varicose vein in spermatid cord)  90% on the left because Lt testicular vein drain into high pressure renal vein where the Rt testicular vein drains directly into IVC  Usually asymptomatic but pt usually infertile as it increases scrotal temperature which affect normal sperm function
  • 14.
    Spermatocele & epididymalcyst  Testis are palpable  Cant distinguished clinical. Only by aspiration. -Spermatocele: slightly grey, opaque fluid containing spermatozoa -Epidydymal cyst: clear fluid
  • 15.
    Testicular tumor  20-40years old  >90% are derived from germ cells  Most common -Seminomas: derived from spermatocyte -Teratoma: dereved from 3 germ cell layer ectoderm/mesoderm/endoderm
  • 16.
    Presentation -solid testicular lump -painless - may cause secondary hydrocele
  • 17.
    spread  Spread topara-aortic LN > thoracic duct > supraclavicular LN  Inguinal LN are not involved unless spread to scrotal skin
  • 18.
    Investigation  USG for scrotal content  Chest X-ray for lung secondaries  Tumour marker o B-HCG o AFP o LDH  CT for staging
  • 19.
    staging  I: confinedto testis  II: retroperitoneal LN  III: metastasis above diaphragm confined to LN  IV: extralymphatic metastasis
  • 20.
  • 21.
    Acute epidydymo- orchitis  Primarily an infection of the epididymis but then spread into testis  Organism : chlamydia/gonococcus/ E.coli  May be assoc with UTI
  • 22.
    Presentation  Acute severetesticular pain  Pain is decrease by raising the testis  Scrotal skin red, hot & edematous
  • 24.
    Aetiology and pathological features Rare,except a/w mumps  Blood-borne infection  Surgical procedure on the lower urinary tract,e.g. TUR  Organism: Neisseria gonorrhoeae, Escherichia coli and Chlamydia. In young man, the commonest is Chlamydia  Tuberculosis
  • 25.
    Clinical features  PrecedingHx of an operation or of dysuria, frequency and heamaturia  Acute pain in scrotum,swelling  Epididymis:acutely tender and enlarged(although it maybe difficult to differentiate from the equally tender testis)  Overlying redness and oedema maybe present
  • 26.
    Investigation  FBC: leucocytosis  Blood culture: helpful to direct antibiotic treatment  Urinalysis: pyuria, organism maybe revealed by culture  Aspiration of the epididymis  USG: increased blood flow
  • 27.
    Management  Bed rest,scrotalelevation  Tetracycline or erthromycin  Other antiobiotic refer to culture  Partner should also be investigated and treated
  • 29.
    Epidemiology  Both testesare undescend in 30% of premature infants  Term:3%  One year:1%  Spontaneous descent after one year is rare
  • 30.
    Aetiology  Failure ofmigration along the normal line of descent  Ectopic testis:testicle deviates away from the line and lie in front of the penis in the superficial inguinal pouch,in the perineum or in the thigh.(reason unknown)
  • 31.
    Risk factor  Prematurity  Low birth weight  Twin gestation  Down syndrome(fetus) or other chromosomal abnormality  Gestational diabetes mellitus  Prenatal alcohol exposure  Hormonal abnormalities (fetus)  Toxic exposures in the mother  Mother younger than 20  A family history of undescended testes
  • 32.
    Clinical features An emptyscrotal sac or hemiscrotum at 1 year indicates:  Proximal to the external inguinal ring(undescended)  Truly absent  Retractile-the cremaster muscle reflexly pulls the organ up towards the inguinal canal  Ectopic
  • 33.
    Complication  Infertility:inevitable in bilateral and common in unilateral undescent,frequent in those who are undescent treated.  Torsion  Trauma  Inguinal hernia  Malignant disease
  • 34.
    Investigation  USG,CT and laparoscopy Management  Target is to bring the testicle with its blood supply into the scrotum as early as possible  Orchidopexy:should be done beyong puberty  Testicular prosthesis can be placed in the scrotum
  • 35.
    1 Epididymis 2 Headof epididymis 3 Lobules of epididymis 4 Body of epididymis 5 Tail of epididymis 6 Duct of epididymis 7 Deferent duct (ductus deferens or vas deferens)
  • 36.
    Testicular torsion  Testicular torsion occurs when the spermatic cord(from which the testicle is suspended) twists, cutting off the testicle's blood supply(ischemia)  Cause: recognised complication of testicular maldescent wherein the testis is inadequately affixed to the scrotum allowing it to move freely on its axis and susceptible to induced twisting of the cord and its vessels.  Occurs most probably between birth and early adolescence
  • 37.
    Twist VS Untwist Twist deprives the organ of its blood supply  If untwist does not take place within 6 hours,ischaemia is irreversible,gangrene develops and the testis either suppurates or atrophies
  • 38.
    Presentation & Finding  Acute severe testicular pain(affected side)  Testis is tender,swollen and hang higher up(compared to other side)  Poorly localized central abdo pain  Vomitting(sometimes)  Scrotal skin become red,hot and edematous in later stage  Palpation may feel the twisted cord Pain is increase or no improvement by raising the testis
  • 39.
    Investigation  Urinalysis:sterile,acellular urine USG:absence of blood supply to the affected testicle
  • 40.
    Management  Surgical emergency Non-operative Maybe possible to de-rotate the testis  Surgical Failure of non-operative reduction require emergency operation The testis is de-rotated and fixed The gangrenous testis is removed
  • 41.
    Dignosis of lumpsin the scrotum 1. Can u get above it? : if not, mostlikely is an inguinoscrotal hernia.(or a hydrocele extending proximally) 2. Is it separate from the testis? 3. Is it cystic or solid?  Separate and cystic - epididymal cyst or spermatocele  Separate and solid - epididymitis (may also orchitis)  Testicular and cystic – hydrocele  Testicular and solid – tumour, orchitis