The document discusses the interpretation and management of CTG (cardiotocography). It describes the steps to interpret a CTG tracing, including evaluating the fetal heart rate baseline, variability, accelerations, decelerations and their correlation with uterine contractions. It provides a structured DR C BRA VADO method to categorize CTG tracings as normal, suspicious or pathological. The management strategies for each category are then outlined, such as continued monitoring, additional tests like fetal scalp blood pH, or expedited delivery depending on the severity of the CTG abnormalities. Specific situations like the second stage of labor, placental abruption or fetal abnormalities are also addressed.
Sickle cell anemia is an autosome linked recessive trait that can be transmitted from parents to the offspring when
both the partners are carrier for the gene (or heterozygous). The disease is controlled by a single pair of allele, HbA
and HbS. Out of the three possible genotypes only homozygous individuals for HbS (HbS, HbS) show the diseased phenotype. The ability to predict the clinical course of SCD during pregnancy is difficult. It is mandatory to follow up the patient closely from the very beginning i.e. from preconception to antenatal till labor. SCD is associated with both maternal and fetal complications and is associated with an increased incidence of perinatal mortality, premature
labor, fetal growth restriction and acute painful crises during pregnancy.
Prelabour Rupture of Membrane (PROM) by Sunil Kumar Dahasunil kumar daha
Please find the power point on Prelabour Rupture of Membrane (PROM). I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Sickle cell anemia is an autosome linked recessive trait that can be transmitted from parents to the offspring when
both the partners are carrier for the gene (or heterozygous). The disease is controlled by a single pair of allele, HbA
and HbS. Out of the three possible genotypes only homozygous individuals for HbS (HbS, HbS) show the diseased phenotype. The ability to predict the clinical course of SCD during pregnancy is difficult. It is mandatory to follow up the patient closely from the very beginning i.e. from preconception to antenatal till labor. SCD is associated with both maternal and fetal complications and is associated with an increased incidence of perinatal mortality, premature
labor, fetal growth restriction and acute painful crises during pregnancy.
Prelabour Rupture of Membrane (PROM) by Sunil Kumar Dahasunil kumar daha
Please find the power point on Prelabour Rupture of Membrane (PROM). I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
CTG - Cardiotocography or Non stress test
A nonstress test is a screening test used in pregnancy to assess fetal status by means of the fetal heart rate and its responsiveness.
A cardiotocograph is used to monitor the fetal heart rate and presence or absence of uterine contractions. The test is typically termed "reactive" or "nonreactive".
Undergraduate course lectuers in Obstetrics&Gynecology
Prepared by DR Manal Behery
Assistant Professor in OB&GYNE ,Faculty of medicine,Zagazig University
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
2. INTERPRETATION
Steps
1. Evaluate tracing.
Do you have enough of a continuous strip for
interpretation
2. Identify
FHR baseline
BBV
absent, minimal, moderate or marked
Accelerations or
Decelerations.
Aboubakr Elnashar
3. 3. Evaluate contractions
regularity, rate, intensity, duration of
contractions.
4. Correlate accelerations and decelerations with
uterine contractions and identify the pattern.
5. Determine whether the FHR recording is
reassuring, non reassuring or ominous.
6. Document interpretation of
FHR
clinical conclusion
plan of care.
Aboubakr Elnashar
4. STRUCTURED METHOD
The most popular structure can be remembered using the
acronym:
DR C BRA VADO = 7 items
1. Demographics of the patient
2. indication of CTG
3. Any obvious abnormalities
DR - Define Risk ? PET, diabetes, IUGR, smoker
C - Contractions - Frequency, duration, intensity, resting
tone
BRA - Baseline rate - 110-160bpm
V - Variability - 5-25 beats
A - Accelerations - 2 in 20 minutes
D - Decelerations - abnormal
O - Overall risk assessmentAboubakr Elnashar
5. Define Risk
• You first need to assess if this pregnancy is high or low risk
• This is important as it gives more context to the CTG
reading. e.g. If the pregnancy is high risk, your threshold for
intervening may be lowered
• Maternal medical illness
Gestational diabetes
Hypertension
Asthma.
• Obstetric complications
Multiple gestation
Post-date gestation
Previous cesarean section
Intrauterine growth restriction
Premature rupture of the membranes
Congenital malformations
Oxytocin induction/augmentation of labor
Pre-eclampsia. Aboubakr Elnashar
6. O – Overall assessment
Once you have assessed all aspects of the CTG
you need to give your overall impression
Aboubakr Elnashar
11. II. MANAGEMENT
FIGO Fetal HR Pattern Classification
Normal means fetal health
• Suspicious means
- continue observation
- additional tests
• Pathological means
- additional test
- intervention
1. Normal/Reassuring
risk of fetal hypoxia in spontaneous labour is low:
Manage normally.
Aboubakr Elnashar
12. 2. Suspicious/Equivocal/ Non reassuring
continue EFM
Amniotomy should be performed
+/- fetal scalp blood pH if meconium stained
liquor is present.
Aboubakr Elnashar
13. Initial Evaluation and Treatment of Nonreassuring
Fetal Heart Rate Patterns
Discontinuation of any labor stimulating agent
Cervical examination:
umbilical cord prolapse or
rapid cervical dilation or
descent of the fetal head
Changing maternal position
left or right lateral recumbent position:
reducing compression of the vena cava and improving
uteroplacental blood flow
Aboubakr Elnashar
14. Monitoring BP
for evidence of hypotension, especially in those with
regional anesthesia
if present: treatment with ephedrine or phenylephrine
may be warranted
Assessment of patient for uterine hyperstimulation
by evaluating uterine contraction frequency and
duration
In the presence of abnormal FHR patterns and
uterine hypercontractility not secondary to oxytocin
infusion: tocolysis
subcutaneous terbutaline 0.25 milligrams
Aboubakr Elnashar
15. In cases of suspected or confirmed acute fetal
compromise:
delivery should be accomplished as soon as
possible, accounting for the severity of the FHR
abnormality and relevant maternal factors
The accepted standard has been that ideally this
should be accomplished within 30 minutes.
Aboubakr Elnashar
16. Maternal facial oxygen therapy
Prolonged use of maternal facial oxygen therapy
may be harmful to the fetus and should be avoided.
There is no research evidence evaluating the
benefits or risks associated with the short-term use
of maternal facial oxygen therapy in cases of
suspected fetal compromise .•C
Aboubakr Elnashar
17. 3. Abnormal/Pathological
Amniotomy
Fetal scalp blood pH if meconium stained
liquor to determine subsequent management
or
Deliver if clinically indicated.
Deliver if fetal scalp pH required but not
obtainable i.e. if cervix not sufficiently dilated
or equipment not available.
Aboubakr Elnashar
18. III. SPECIAL SITUATIONS
Second Stage of Labour
Signs of hypoxia:
• Tachycardia
• ↓ variability between and during decelerations
• Late decelerations
• Failure to return to baseline (or > 100 bpm)
after decelerations
• Prolonged bradycardia
* Delay of 20 min → asphyxiated infant
Aboubakr Elnashar
19. Placental abruption
• Uterine irritability shown by frequent
contractions of low amplitude
• FHR trace:
initially tachycardia
± decelerations
no accelerations
↓ variability.
Bradycardia is a late and danger sign of
severe asphyxia
Aboubakr Elnashar
20. Fetal abnormality
• CNS abnormality: →
↓ baseline variability
low baseline rate
• Discrepancy between CTG (abnormal) and
biophysical profile (within normal) suggest
chromosomal abnormalities, especially if bony
measurements are reduced/slight IUGR
Aboubakr Elnashar
26. Category Definition
Normal All four reassuring
Suspicious 1 non-reassuring
Rest reassuring
Pathological 2 or more non-reassuring
1 or more abnormal
Aboubakr Elnashar
27. b)Abnormal/Pathological Trace
- Baseline FHA> 150 bpm + silent pattern and/or repeated late or
variable decelerations
- Silent pattern for >90 minutes
- Complicated variable decelerations (depth >60 bpm for >60
seconds, changes in shape: over-shoot, decreased or
increased baseline FHR following the decelerations, or absence
of baseline variability in or between decelerations, slow
recovery)
- Combined/biphasic decelerations (variable followed by late)
- Prolonged bradycardia in a suspicious trace
- Prolonged bradycardia> 10 minutes with no signs of recovery
- Repeated late decelerations
- Pronounced loss of baseline variability regardless of baseline
FHR with shallow late decelerations
- Sinusoidal pattern with no accelerations
Aboubakr Elnashar
29. a)Normal/Reassuring Trace
- At least two accelerations (> 15 beats per minute
for >15 seconds) in 20 minutes
- Baseline heart rate: 110-150 bpm
- Baseline variability: 5-25 bpm
- Early decelerations (in late first stage of labour)
Aboubakr Elnashar
30. b)Suspicious/Equivocal Trace
- Absence of accelerations for >40 minutes
(non reactive)
- Baseline heart rate: 150-170 bpm or 100-110 bpm
(normal variability, no decelerations)
- Silent pattern (<5 bpm for >40 minutes) although
normal baseline (110-150 bpm), no
decelerations
- Baseline variability >25 bpm in the absence of
accelerations
- Variable decelerations (depth <60 bpm, duration
<60 seconds)
- Occasional transient prolonged bradycardia if
FHR drops to <80 bpm for >2 minutes or
<100 bpm For >3 minutes
Aboubakr Elnashar