Continue CPR immediately after shock for 2
minutes before reanalysis
Operator: Thank you for the reminder. Yes, CPR should be
resumed immediately after shock delivery for 2 minutes
before reanalysis.
Advanced Cardiovascular Life Support (ACLS) is the pre-eminent resuscitation course for the recognition and intervention of cardiopulmonary arrest or other cardiovascular emergencies.
A "bundle" is a
group of evidence-based care components
for a given disease that, when executed together, may result in better outcomes than if implemented individually.
The CVP catheter is an important tool used to assess right ventricular function and systemic fluid status. Normal CVP is 2-6 mm Hg. CVP is elevated by : overhydration which increases venous return.
Advanced Cardiovascular Life Support (ACLS) is the pre-eminent resuscitation course for the recognition and intervention of cardiopulmonary arrest or other cardiovascular emergencies.
A "bundle" is a
group of evidence-based care components
for a given disease that, when executed together, may result in better outcomes than if implemented individually.
The CVP catheter is an important tool used to assess right ventricular function and systemic fluid status. Normal CVP is 2-6 mm Hg. CVP is elevated by : overhydration which increases venous return.
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
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Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
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1. RAKHI DAS
I YEAR MSc NURSING
JUBILEE MISSION COLLEGE OF NURSING
THRISSUR, KERALA, INDIA
2. INTRODUCTION
THE PROVISION OF BASIC LIFE SUPPORT –
AIRWAY MAINTENANCE, VENTILLATORY
ASSISTANCE AND EXTERNAL CHEST
COMPRESSION ARE THE FIRST STEPS TOWARD
FACILITATING SURVIVAL FOR A VICTIM OF
CARDIOPULMONARY ARREST. THE VICTIM ONCE
STABILIZED WITH THESE MANEUVERS MAY NEED
THE SUPPORT OF ADVANCED LIFE SUPPORT.
RESUSCITATION APPARENTLY MEANS –
“BRINGING BACK TO LIFE” OR “REVIVE TO
SUSTAIN”
3. DEFINITION
CARDIOPULMONARY RESUSCITATION (CPR) IS AN
EMERGENCY PROCEDURE THAT COMBINES
CHEST COMPRESSIONS OFTEN WITH ARTIFICIAL
VENTILATION IN AN EFFORT TO MANUALLY
PRESERVE INTACT BRAIN FUNCTION UNTIL
FURTHER MEASURES ARE TAKEN TO RESTORE
SPONTANEOUS BLOOD CIRCULATION AND
BREATHING IN A PERSON WHO IS IN CARDIAC
ARREST
4. DEFINITION
ADVANCED CARDIAC LIFE SUPPORT
(ACLS) OR ADVANCED CARDIOVASCULAR LIFE
SUPPORT (ACLS) REFERS TO A SET OF CLINICAL
INTERVENTIONS FOR THE URGENT TREATMENT
OF CARDIAC ARREST, STROKE AND OTHER LIFE-
THREATENING CARDIOVASCULAR EMERGENCIES,
AS WELL AS THE KNOWLEDGE AND SKILLS TO
DEPLOY THOSE INTERVENTIONS.
5. Difference between BLS and ACLS
BLS
Non- invasive
No medications
administered
Can be done by anyone
trained
No advanced equipments
Two person/ technician
suffice
No right to give basic
treatment
ACLS
Invasive maneuvers
Along with medications
Medical personnel
specialized
Defibrillator, cardiac
monitors used
Team of workers- doctors,
nurses, paramedic,
emergency medical
technician
Basic treatments for cuts
and injuries can be given
7. PRE REQUISITES FOR PERFORMING
ACLS
ONLY QUALIFIED HEALTH CARE PROVIDERS CAN
PROVIDE ACLS, AS IT REQUIRES
ABILITY TO MANAGE THE PERSON'S AIRWAY
INITIATE INTRAVENOUS (IV) ACCESS
READ AND INTERPRET ELECTROCARDIOGRAMS
UNDERSTAND EMERGENCY PHARMACOLOGY
8. The resuscitation team
ROLES PLANNED IN ADVANCE
IDENTIFY TEAM LEADER
IMPORTANCE OF NON- TECHNICAL SKILLS
TASK MANAGEMENT
TEAM WORKING
SITUATIONAL AWARENESS
DECISION MAKING
STRUCTURED COMMUNICATION
DEBRIEF AFTER EVENT
12. SCENARIO EXAMPLE
YOU FIND AN ADULT LYING ON THE GROUND –
CHECK RESPONSIVENESS
IF UNRESPONSIVE – CALL + POSITION (1PERSON,
2PERSON)
CIRCULATION – ADULT (CAROTID), INFANT
(FEMORAL/BRACHIAL)
IF PULSE- airway and rescue breathing
1 BREATH / 10 SECOND, RECHECK PULSE EVERY 10
MINUTES
13. SCENARIO EXAMPLE
NO PULSE – START CPR
100 to 120 compressions per minute
30 compressions every 15 to 18 seconds
Place palms midline, one over the other, on the lower
1/3 of the patient’s sternum between the nipples- lock
arms
to 2.4 inches (5-6cm) or more – ADULT
1.5 inches or 1/3 the depth – INFANT
Press hard and fast
14. Allow for full chest recoil with each compression
Swap CPR provider(two men) every 2 minutes
Allow for only minimal interruptions to chest
compressions
The compression-ventailation ratio is 30:2
CPR should continue between rhythm checks and
shocks, and until the ACLS team arrives or the victim
shows signs of movement.
19. BREATHING
Scan and Watch
breathing adequately - maintain a patent airway and
place the patient in the recovery position
not breathing or is breathing inadequately – pulse
present - rescue breaths
no pulse - Begin CPR
22. CHOKING
If partial airway obstruction:
Do not attempt Heimlich maneuver
If complete airway obstruction:
Send someone to call the emergency response team
If alone:
immediately call the emergency response team.
Attempt Heimlich maneuver
27. DEFIBRILLATE
Arrival of the AED (Automated External Defibrillator)
Turn AED On
early defibrillation is the single most important
therapy for survival of cardiac arrest and should be
done as soon as it arrives
Follow verbal AED prompts
28. DEFIBRILLATE
Attachment
Analyze
If the rhythm is not shockable
If the shock is indicated-
Assure no one is touching the patient or is in mutual
contact of a good conductor of electricity by yelling
“Clear, I’m Clear, you’re Clear!” prior to delivering a
shock
Press the shock button when the providers are clear of
the patient
Resume 5 cycles of CPR
29. Defibrillation
In ventricular fibrillation, a precordial thump is
employed by giving sternum to a height of 8-12
inch
If rhythm reverts – inj lidocaine 0.2% is given IV
If VF persists, proceed to BLS and defibrillation
30. Electrical defibrillation
Passing of electric current through a fibrillating heart
allowing for uniform depolarization and organized
cardiac activity
Conductive jelly should be applied before placing the
paddles
Energy level is selected in joules (100-300)
Clear the area in contact with patient or cot
Evaluate effectiveness and administer Inj. Epinephrine
and Sodium bicarbonate
31. Electrocardiograph (ECG)
essential in resuscitation
defibrillators have in-built monitor circuit to assess ECG
Patient ECG pattern are sensed and command follow for
shock
For continuos monitoring, standard ECG monitors are
used
32. THE EFFECTIVENESS OF EMERGENCY
CARDIOVASCULAR CARE (ECC)
CHAIN OF SURVIVAL
33. links in Chain of Survival
early access
early CPR
early defibrillation
early ACLS
34. CORRECTION OF ACIDOSIS AND
FLUID REPLACEMENT
Proper venous access
Peripheral veins may be convenient – if
vasoconstriction and venous collapse; central venous
access made
Correction of by adequate alveolar ventilation
Initial dose of sodium bicarbonate is 1mEq/kg slow IV.
Further determined by ABG analysis; formula is 0.3 x
wt (kg) x based deficit
Volume replacement – isotonic crystalloid for rapid
expansion of circulatory blood volume. Ringer lactate
or Normal Saline
36. POST-RESUSCITATION SUPPORT
Transition from emergency service to critical care unit
thorough assessment and examination
Diagnostic studies should be completed
Ventilatory support is continued at optimal level
Cardiac support with minimal cardiac work is maintained
with appropriate drugs
Transportation
accompanied by a nurse and a physician with adequate
equipment
Portable ventilators
If not -AMBU bag with an oxygen source
37.
38. Journal Abstract
Kah MC, Eric HC, Chih-Wei Y, Hui-
Chih W, Edward P, Yen-Pin C et al.
Advanced Cardiac Life Support
(ACLS) Is All About Airway-
Circulation-Leadership-Support (A-
C-L-S): A Novel CPR Teamwork
Model. Originally published 27 Mar
2018 Circulation. 2018; 130:A265
41. CENTRAL OBJECTIVE
By the end of the class, the learner will acquire
adequate technical knowledge and skill for using
defibrillator and will be able to apply this knowledge in
clinical areas skillfully and aid in nursing research with
a positive attitude
42. SPECIFIC OBJECTIVES
Understand meaning of defibrillator and cardioversion
Focus on indications and contraindications of defibrilation
Learn various types of defibrillator and cardioversion
Enumerate equipments needed for defibrilation
Identify shockable and non- shockable rhythms
Acquire skill in performing advanced life support and
defibrillation
Discuss about nurses responsibilities in preparation of
patient and performing defibrillation
Demonstrate the procedure of defibrilation
Incorporate evidence based nursing skills in practice of
Defibrilation and cardioversion
43. Definition- Defibrillation
Defibrillation is non synchronized random
administration of shock during a cardiac
cycle performed to correct life-threatening
arrhythmias of the heart including
ventricular fibrillation and pulseless
ventricular tachycardia
44. Definition - Cardioversion
Cardioversion is a synchronized administration of shock
during the R waves or QRS complex of a cardiac cycle
During defibrillation and cardioversion, electrical
current travels from the negative to the positive
electrode by traversing myocardium
heart cells to contract
interrupts and terminates abnormal electrical rhythm
allows the sinus node to resume normal pacemaker
activity
58. Types of Cardioversion
Chemical cardioversion
Electrical cardioversion
Other considerations
Internal cardioversion
Cardioversion in patients with digitalis toxicity
Cardioversion in patients with permanent
pacemakers/ICDs
Cardioversion during pregnancy
59. NURSES RESPONSIBILITIES
Patient preparation and sedation
General instructions
Patient preparation for elective procedures
Paddle placement
Predefibrillation care
(preparation of defibrillation)
Post defibrillation care
60. PATIENT PREPARATION AND
SEDATION
emergent maneuver - promptly performed in
conjunction with or prior to administration of
induction or sedative agents
no preparation for emergency
ACLS measures - obtaining intravenous access and
preparing airway management equipment, sedative
drugs, and a monitoring device
Cardioversion - under induction or sedation (short-
acting agent such as midazolam)unless patient is
hemodynamically unstable or cardiovascular collapse
is imminent
61. GENERAL INSTRUCTIONS
Prevent potential complications while using defibrillator
such as:
Burns to the patient
Shock to operator/ other personnel
Fire / sparks
Arrhythmias
Ineffective shock
62. Patient preparation for elective
procedures
Nil per os (NPO) for 8 hours prior to the procedure
Stop digoxin 48 hours prior to the procedure
Continue medications on the morning of the
procedure under the direction of the physician
63. Paddle placement
2 conventional positions:
Anterolateral
a single paddle is placed on the left fourth or fifth
intercostal space on the midaxillary line.
The second paddle is placed just to the right of the sternal
edge on the second or third intercostal space
more effective for persistent atrial fibrillation
Anteroposterior
a single paddle is placed to the right of the sternum
other paddle is placed between the tip of the left scapula
and the spine
preferred in patients with implantable devices
64. Predefibrillation care (preparation
of defibrillation)
Explain procedure, if patient is conscious or to the
relatives
Position in supine without any pillow for head
Confirm cardiac arrest (VT or VF by checking
patient’s clinical condition)
ensure the cardiac arrest team is alerted; get ready a
defibrillator and cardiac arrest trolley
Commence cardiopulmonary resuscitation (CPR) at 30
compressions to two ventilations
65. Predefibrillation care
As soon as the defibrillator arrives, switch it on and prepare
the patient’s chest if necessary. Ensure CPR continues
Ensure 'synchronize’ mode is off
Apply self-adhesive defibrillation electrodes to the patient’s
bare chest following the manufacturer’s recommendations.
Ensure CPR continues. If paddles are used, apply
conductive gel to paddles.
Once the electrodes are in place and are connected to the
defibrillator, ECG analysis can start; depending on the
defibrillator, this will begin automatically or the user will
be prompted to press an ECG analysis button. Briefly stop
CPR
66. Predefibrillation care
Keep one paddle anteriorly at 2nd intercostals space right
of sternum and another paddle laterally at 5th intercostals
space to left of sternum at midaxillary line or at cardiac
apex. Ensure there is 10 cm distances between paddles
If a shockable rhythm is detected, ensure the appropriate
shock energy has been selected. Some advisory
defibrillators will do this automatically while others require
the manual check. Most advisory defibrillators will charge
up automatically, while some – typically the older models –
require the operator to press a “charge button”
Discontinue oxygen inhalation to prevent fire hazards.
67. Predefibrillation care
Shout “stand clear” and perform a quick visual check
of the area to ensure that all people are clear
Apply pressure of 25 pounds per paddle. Do not lean
forward.
Press the shock button to discharge the shock
Reassess cardiac monitor to determine rhythm and
subsequent action, while paddles are still on chest.
If VF / VT is still present, reset and increase energy at
200 to 300 J and deliver
68. Predefibrillation care
If VF/VT is not revered, deliver 360 J and reassess
cardiac rhythm
When VF/VT persist, administer emergency drugs, e.g.
inj. adrenaline and atropine and give cardiopulmonary
resuscitation (CPR) for 1 minute
Repeat defibrillation at 360 J for 3 times as ordered
Discontinue procedure
Clean and replace paddles for next use
69. Post defibrillation Care
Assess patient responsiveness/sensorium.
Check airway, breathing and circulation.
Monitor cardiac rhythm continuously and assess Vital
signs including BP half hourly until stable.
Maintain oxygenation.
Detect arrhythmias and side effects of drugs used
during emergency
Provide comfort and psychological support to patient
and family
70. Post defibrillation Care
Administer analgesic as ordered if patient experiences
pain over defibrillation site.
Document Joules, number of shocks and response of
patient.
If defibrillation is unsuccessful, explain situation to
family with the help of doctor
Instruct After the procedure, do not drive, operate
machinery, or sign important documents for 24 hours
and/or until sedation has worn off
71. Complications
The most common complications are harmless
arrhythmias, such as atrial, ventricular, and junctional
premature beats.
Serious complications include ventricular fibrillation
(VF) resulting from high amounts of electrical energy,
digitalis toxicity, severe heart disease, or improper
synchronization of the shock with the R wave.
72. Complications
Thrombo embolization is associated with cardioversion,
especially in patients with atrial fibrillation who have not
been anticoagulated prior to cardioversion. American
Heart Association (AHA) guidelines recommend to
anticoagulate for 3-4 weeks before and after cardioversion.
Myocardial necrosis can result from high-energy shocks. ST
segment elevation can be seen immediately and usually
lasts for 1-2 minutes.
Myocardial dysfunction due to an absence of cardiac
output and coronary blood flow during arrest, resulting in
ischemia.
73. Complications
Pulmonary edema is a rare complication of
cardioversion. It is probably due to transient left atrial
standstill and left ventricular systolic dysfunction. It is
more common in atrial fibrillation due to valvular
heart disease or left ventricular systolic dysfunction.
Allergic reaction to sedation medication.
74. Complications
Painful skin burns can occur after cardioversion or
defibrillation; they are moderate to severe in 20-25% of
patients. They most likely are due to improper
technique and electrode placement. It occurs less with
use of biphasic waveform defibrillators and use of gel-
based pads. Prophylactic use of steroid cream or
topical ibuprofen reduces pain and inflammation.
75. JOURNAL ABSTRACT
Philip W, Kodoth V, McEneaney D, Rodrigues P, Jose
V, Waterman N et al. Towards Low Energy Atrial
Defibrillation. PMCID 2015 Sep; 15(9): 22378–22400
76. BIBLIOGRAPHY
Sharon L. Lewis , Shannon Ruff Dirksen Margaret
McLean Heitkemper , Linda Bucher, Medical-Surgical
Nursing: Assessment and Management of 9thEdition
Smeltzer S C, Bare B , Brunner &suddarth’s Medical
surgical nursing, edition 10th, ( 2000), Westline
Industrial drive, Missouri.
Joyce Black , Jane Hokanson Hawks, Esther
Matassarin-JacobsMedical-Surgical Nursing: Clinical
Management for Positive Outcomes, 7th Edition
77. BIBLIOGRAPHY
Susan Woods, Erika S Sivarajan, Sandra Underhill,
Elizabeth J Bridges, Cardiac Nursing, 5th Edition,
Lippincott William & Wilkins
Clement I. Basic concepts of Nursing procedures.
Second edition. New Delhi: Jaypee brothers; 2006
Johnson Priyadarshini. Clinical Nursing Procedure
manual. Chennai: KVMathew BI publications
Soni S. Textbook of Advanced Nursing Practice. New
Delhi: Jaypee Brothers
78. BIBLIOGRAPHY
Philip W, Kodoth V, McEneaney D, Rodrigues P, Jose
V, Waterman N et al. Towards Low Energy Atrial
Defibrillation. PMCID 2015 Sep; 15(9): 22378–22400
Ventricular Fibrillation and Pulseless Ventricular
Tachycardia
https://acls-algorithms.com/vfpulseless-vt/