This document discusses abdominal compartment syndrome (ACS). It defines ACS as a sustained intra-abdominal pressure (IAP) above 20 mmHg associated with new organ dysfunction. Normal IAP is 5-7 mmHg. Intra-abdominal hypertension is defined as IAP above 12 mmHg and is graded. ACS can result from primary abdominal causes or secondary extra-abdominal causes and leads to organ dysfunction through reduced blood flow. Accurate IAP monitoring via bladder pressure is important for early detection and treatment to prevent organ failure.
Whipple's procedure - Indications, Steps, ComplicationsVikas V
Whipple's Procedure - Explaining the History of Whipple's Procedure, Indications, Contraindications, Step wise detailed procedure, Complications, Perioperative Management.
The Presentation Includes Detailed Step wise approach to the procedure assisted with Pictorial Representation of The steps
Whipple's procedure - Indications, Steps, ComplicationsVikas V
Whipple's Procedure - Explaining the History of Whipple's Procedure, Indications, Contraindications, Step wise detailed procedure, Complications, Perioperative Management.
The Presentation Includes Detailed Step wise approach to the procedure assisted with Pictorial Representation of The steps
OPEN INGUINAL HERNIA REPAIR- OPERATIVE SURGERY
#surgicaleducator #operativesurgery #openinguinalherniarepair #usmle #babysurgeon #surgicaltutor
Dear viewers,
• Greetings from “Surgical Educator”
• Because of the popular demand by viewers of the YouTube channel “Surgical Educator”, I have decided to create and upload videos on common surgeries.
• I have already uploaded videos on open and Laparoscopic Appendicectomy, Thyroidectomy, Modified Radical Mastectomy and open and
Laparoscopic Cholecystectomy
• In this video today, I have discussed Open Inguinal Hernia Repair.
• However, these videos are not real surgeries but the theoretical aspect of operative surgery like going through an atlas of operative surgery.
• Along with these videos, I recommend you to watch real operative surgery videos as well and I will give a link for each surgery in the end of the video as end-cards, which I think will be very useful.
• This will give a very good opportunity for the surgical trainees to mentally rehearse various surgical steps in a sequential manner prior to actual surgery. You can watch the video in the following links:
• surgicaleducator.blogspot.com
• youtube.com/c/surgicaleducator
• Thank you for watching the videos.
OPEN INGUINAL HERNIA REPAIR- OPERATIVE SURGERY
#surgicaleducator #operativesurgery #openinguinalherniarepair #usmle #babysurgeon #surgicaltutor
Dear viewers,
• Greetings from “Surgical Educator”
• Because of the popular demand by viewers of the YouTube channel “Surgical Educator”, I have decided to create and upload videos on common surgeries.
• I have already uploaded videos on open and Laparoscopic Appendicectomy, Thyroidectomy, Modified Radical Mastectomy and open and
Laparoscopic Cholecystectomy
• In this video today, I have discussed Open Inguinal Hernia Repair.
• However, these videos are not real surgeries but the theoretical aspect of operative surgery like going through an atlas of operative surgery.
• Along with these videos, I recommend you to watch real operative surgery videos as well and I will give a link for each surgery in the end of the video as end-cards, which I think will be very useful.
• This will give a very good opportunity for the surgical trainees to mentally rehearse various surgical steps in a sequential manner prior to actual surgery. You can watch the video in the following links:
• surgicaleducator.blogspot.com
• youtube.com/c/surgicaleducator
• Thank you for watching the videos.
Intra-abdominal hypertension and abdominal compartment syndromeNicholas Leary
Intra-abdominal hypertension and abdominal compartment syndrome is equally as prevalent in the medical population as the trauma population. Learn about the pathophysiology, how to monitor, and treatment for IAH and ACS.
This presentation deals with the causes, signs, symptoms and management of compartment syndrome. It also briefly deals with abdominal comaprtment syndrome
CIB W78 2007 - Comparison of distance learning coursesRobert Klinc
Even though advances in information and communication technologies (ICT) significantly changed the way professionals in building and construction (BC) industry work, the dominant training method is still the traditional classroom lecture with all its drawbacks.
In response to the demands from the AEC sector to improve and broaden the competence of engineering students in using new technologies while solving specific problems, in 1993 University of Stanford (USA) started an ICT supported distance learning course named Architecture/Engineering/Construction Computer Integrated Global Teamwork Course (AEC Global Teamwork). The mission of the program is to educate the next generation of professionals to be able to work in multi discipline collaborative environments and to take advantage of information technologies to produce high quality products in faster and more economic way.
Positive feedback of the AEC Global Teamwork encouraged other institutions to introduce their own BC oriented distance learning courses, one of them being ITC Euromaster. In autumn 2001, nine European universities started the project in order to develop an inter university postgraduate programme in information technology in construction (ITC).
This paper describes similarities and differences of both approaches, presents results of the survey carried out among participants of both courses, and compares both of them from the students’ point of view.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
2. ? What it is ?
A disease process that dramatically increases
organ failure and death for medical and surgical
ICU patients
3. What is a normal intra-abdominal
pressure
or
IAP
This is the pressure within the abdominal cavity
5 – 7 mmHg is normal in a critically ill adult
4. Intra – abdominal Hypertension
IAH
• Defined as sustained or repeatedly elevated
abdominal pressure
• >12 and is graded
5. Grades of IAH
• Grade I 12 – 15 mmHg
• Grade II 16 – 20 mmHg
• Grade III 21 – 25 mmHg
• Grade IV >25 (ACS)
6. IAH
• Sustained pressure, >12 that has significant
effects on abdominal organs and cardiac output
with subsequent dysfunction of both abdominal
and extra-abdominal organs
7. Understanding Abdominal Compartment Syndrome
• APP – Abdominal perfusion pressure
• MAP – Mean arterial pressure
• IAP – Intra-abdominal pressure
• APP = MAP – IAP
• A critical IAP that leads to organ failure is
variable by patient & a single threshold cannot
be applied globally to all patients
• APP is superior to IAP, arterial pH, base deficit
& lactate in predicting organ failure & patient
outcomes
8. Definition of ACS
• A sustained IAP > 20 mmHg (with or without an
APP of <60 mmHg) that is associated with new
organ dysfunction/failure
• Adverse physiological effects caused by massive
interstitial and retroperitoneal swelling which
leads to organ or multi-organ failure
• Historically IAPs as high as 40 mmHg had been
acceptable; therefore, most clinicians are
concerned when IAP reaches 20 – 25 mmHg
9.
10. Abdominal Compartment Syndrome
• Primary ACS – associated with injury or disease
in abdomen/pelvis requiring early surgical or
interventional radiological screening
• Secondary ACS is from conditions not
originating in the abdomen/pelvis
• Recurrent ACS is the redevelopment of ACS
following previous surgical or medical treatment
of primary or secondary ACS
16. Pathophysiological Consequences
of ACS
• Cardiovascular
– Reduced Cardiac Output
• Compression of the
inferior vena cava and
portal vein
• Reduced blood return to
the heart
• Afterload increased from
mechanical compression of
vascular beds and
vasoconstriction
17. Pathophysiology
• Cardiovascular
– Reduced Stroke volume
– Tachycardia
– Increased pressure on
great vessels making
hemodynamic monitoring
challenging with falsely
elevated and misguiding
pressures
– Increased risk for
thromboembolic events
secondary to venous stasis
18. Pathophysiology
Pulmonary
– Reduced lung compliance
secondary to diaphragmatic
elevation leads to
– Hypoventilation and
ventilation-perfusion
mismatch
– Increased work of
breathing
– Hypoxia and hypercarbia
– Respiratory failure
20. Pathophysiology
• Renal
– Increased IAP leads to
decreased renal blood flow
and decreased glomerular
filtration
– Oliguria may be observed
with IAP of 15 - 20
– An IAP of >30 leads to
anuria
– Increase of antidiuretic
hormone and activation of
renin-angiotensin-
aldosterone system
– Increased water retention
22. Pathophysiology
• Central Nervous System
– Increased thoracic and
central venous pressure
leads to
– Decreased cerebral outflow
of blood
– Increased intracranial
pressure which leads to
decreased cerebral
perfusion pressure
24. Importance of accurate measurement
• Physical examination yields low levels of
detection of IAH/ACS
• Early detection and intervention reduces
morbidity and mortality.
• Diagnosis is dependent on frequent and accurate
measurement of IAP (watching trends)
• Cost effective, safe and accurate
25. Assessment Guidelines
• New ICU admission
• Evidence of clinical deterioration
• Pt has two risk factors for IAH/ACS
– Decreased abdominal wall compliance
– Increased intra-luminal contents
• ileus, gastroparesis, obstruction
– Increased abdominal contents
• Pneumoperitoneum, hemoperitoneum, ascities, liver
dysfunction
– Capillary Leak/fluid resuscitation
26. IAH/ACS Assessment algorithm from
World Society of Abdominal Compartment
Syndrome (WSACS)
www.wsacs.org
Excellent references
27. Types of Measurements
• Direct Pressure via intraperitoneal catheters
• Indirect Pressure
– Gastric Measure
– IVC
– Rectal
– Urinary bladder pressure – Gold Standard
28. Urinary Bladder Pressure
Most technically reliable
Correlate closely with pressures measured directly
in the abdominal cavity
Reliably reproducible
Transduced through a Foley catheter
31. Equipment needed for open measurement
• Disposable transducer
• 12” pressure monitoring
tubing
• 4-way stopcock
• Red dead-ender
• 60 cc, lure-lock syringe,
sterile
• Sterile normal saline
• Clamp, non-sterile
• Level
32. Procedure for open, intermittent monitoring
• Collect and gather all
supplies
• Attach stopcock to end of
sterile transducer
• Important to maintain
sterile technique to avoid
contamination and
potential infectious
process
33. Procedure for open, intermittent monitoring
• Attach pressure tubing to
the remaining end of the
transducer
34. Procedure for open, intermittent monitoring
• Fill 60 cc syringe with 40
cc of sterile normal saline
• Attach syringe to side
port of the stopcock
• Flush stopcock, pressure
tubing and transducer
with the normal saline
ensuring all air is removed
35. Procedure for open, intermittent monitoring
• Clamp the urinary drain tubing distal to the sampling port
• Cleanse the sampling port with alcohol
• Using sterile technique attach the pressure tubing to
the LuerLok connecting sampling port of the urinary
catheter
36. Procedure for open, intermittent monitoring
• Instill 25 cc of sterile normal saline into urinary
catheter via the sampling port (Larger vol. of NS can result in
falsely elevated IAP measurements)
• Briefly release the clamp to allow fluid from the bladder
to fill tubing and reclaim
• Read the IAP as a mean pressure at end expiration 30 –
60 seconds after instillation.
• Perform with patient supine
• Notify MD for sustained IAP greater than 12 mmHg
unless otherwise ordered.
37. Disadvantages with open, intermittent monitoring
• Collecting a number of items
• Correct assembly
• Risk of infection every time system is accessed
38. Closed Monitoring
• AbViser, Wolfe Tory Medical, SLC, UT
• Pre-assembled kit
• Adapts to Foley catheter and any transducer
• Reduces risk of infection
• Readily available, easily assessable data
41. Management Considerations
Early detection via frequent monitoring of at risk
patients
Screen for IAH/ACS in new ICU admissions with
new or progressive organ failure
Look for trends of increasing abdominal pressures
Preserve organ perfusion and treat clinical
conditions with grades I & II
42. Management Considerations
Early surgical consultations for at risk patients
Early intervention for ACS or Grade III
Anticipate emergent surgical interventions to
prevent tissue damage/death
43. Management Considerations
• Anticipate patient to return with an alternative
surgical closure or “open” abdomen.
• The abdominal contents will not be sutured into
the abdominal cavity
• Alternative closures vary from surgeon to
surgeon
Examples:
The “Bogata Bag” – A 3 L IV bag, open and
sterilized and applied to the abdominal opening
44.
45. Management Considerations
• KCI Vac Pac
• Sponge overlies abd.
Dressing/contents
• Attached to
continuous suction
canister
• Covered over with
occlusive dressing
46. Management Considerations
• Ioban Dressing
• An occlusive dressing
with iodine
impregnation
• Surgical towels will
overlie abdominal
contents with JP
drains – Ioban
overlies abdomen
48. Conclusion
• Know the difference between IAH and ACS
– IAH = Abdominal pressure >12 and graded via severity
– ACS = Abdominal pressures > 20 – 25
Identify At risk patient populations
abdominal trauma/major burns
Pancreatitis
Ruptured AAA
abdominal obstructions/ischemia
ect….
49. Conclusion
• Understand the pathophysiology
– Ischemia/inflammation – inflammatory response –
capillary leak + fluid resuscitation = tissue edema in an
uncompromising cavity = ACS = tissue/cell death = bad
Perform an accurate assessment of abdominal pressure
using Abdominal bladder pressure monitoring via Foley
catheter or AbViser – Wolfe Torey Medical
Anticipate patient interventions/outcomes
Support/educate family
50. Case Study - 63 Y.O. male pt with pancreatitis is admitted
to the ICU. Pt has history of gallbladder disease, COPD
and ETOH abuse. He has been without ETOH reportedly
for approximately 24 hrs. VS upon admission are T 38.0,
HR 130, BP 90/62, MAP 61, RR 30 – 34 & O2 sat of 91%
on 100% NRB, wt approximately 125 kg. His breathing is
labored and he has c/o SOB. He is also mildly agitated &
resistive to O2 therapy with Bi-Pap. His lung sounds are
diminished bilaterally. Denies recent increase in cough.
His abdomen is firm and distended. States unknown last
BM but + for N/V.
51. • He has a Foley catheter in place with
approximately 100 cc of dark, amber urine in the
collection chamber. Lab values show H&H of
10.2/31.0, wbc 20, K 5.0, Na 142, Foley was
placed approximately 4 hours ago in the ED. His
peripheral arterial pulses are weak and thready
and his BLE show signs of PVD. He is currently
receiving bolus # 3 of NS.
52. Does this patient need IAP monitoring?
Is he at risk?
What could you use as a reference if you were
unsure?
53. After consulting with your attending MD, it is
decided that a baseline ABP reading would be
appropriate for this patient. Your initial ABP is
15mmHg.
54. Does this value represent intra-abdominal
hypertension or abdominal compartment
syndrome?
What is his APP based on his MAP and IAP?
55. What grade would you give this value?
Why is this patient at risk?
How would you proceed?
56. After reporting the findings to the resident,
serial ABP readings are ordered Q6 HR. His SBP
continues to remain low with a map consistently <
65 & his respiratory status continues to
deteriorate. The resident also orders another
fluid bolus.
• With what you have learned about IAH /ACS
management, what clinical suggestions could you
collaborate on to advocate for your patient?
•
57. After collaboration with the medical team the decision is
made to intubate as his O2 sats continue to drop and RR
rate cont. to increase. After intubation and appropriate
sedation, the patient continues to have an increasingly
firm abdomen, increased HR and decreased SBP and map
<60 despite added norepinephrine. He is also now vented
with a respiratory rate of 24 – 30 and has become
increasingly agitated. His urine output for the last 2
hours is 30 ml. You repeat the ABP prior to the 4 hr
interval and you notice that his ABP value has risen to 20
after two separate measurements. What could you
expect at this point?