1. The document discusses shoulder pain and provides a differential diagnosis. It covers anatomy of the shoulder joint, muscles, nerves and bursae.
2. Common causes of shoulder pain discussed include degenerative arthritis, rotator cuff tears, shoulder instability, adhesive capsulitis, bursitis and tendonitis.
3. The differential diagnosis section covers the history, examinations, tests and investigations for various shoulder conditions. It describes conditions like rotator cuff tears, shoulder dislocations, impingement syndrome and frozen shoulder.
Case of Prolapse intervertebral Disc, lumbar disc prolapse, case, physiotherapy management, Assessment, recent Advance, orthopaedic case presentation, musculoskeletal physiotherapy case presentation, orthopaedic physiotherapy, case of a low back pain patient, lumbar radiculopathy, final year,
Case of Prolapse intervertebral Disc, lumbar disc prolapse, case, physiotherapy management, Assessment, recent Advance, orthopaedic case presentation, musculoskeletal physiotherapy case presentation, orthopaedic physiotherapy, case of a low back pain patient, lumbar radiculopathy, final year,
Evidence-based Interventional Pain Medicine
according to Clinical Diagnoses
13. Sacroiliac Joint Pain
Pascal Vanelderen, MD, FIPP*,†; Karolina Szadek, MD‡; Steven P. Cohen, MD§;
Jan De Witte, MD¶; Arno Lataster, MSc**; Jacob Patijn, MD, PHD††;
Nagy Mekhail, MD PhD, FIPP‡‡; Maarten van Kleef, MD, PhD, FIPP††;
Jan Van Zundert, MD, PhD, FIPP*,††
The presentation investigates the following characteristics of the meniscus;
Role of the Meniscus
Material Properties
Structural Limitations / Failure Limits
Mechanism & Treatment of Injuries
Evidence-based Interventional Pain Medicine
according to Clinical Diagnoses
13. Sacroiliac Joint Pain
Pascal Vanelderen, MD, FIPP*,†; Karolina Szadek, MD‡; Steven P. Cohen, MD§;
Jan De Witte, MD¶; Arno Lataster, MSc**; Jacob Patijn, MD, PHD††;
Nagy Mekhail, MD PhD, FIPP‡‡; Maarten van Kleef, MD, PhD, FIPP††;
Jan Van Zundert, MD, PhD, FIPP*,††
The presentation investigates the following characteristics of the meniscus;
Role of the Meniscus
Material Properties
Structural Limitations / Failure Limits
Mechanism & Treatment of Injuries
Describing some of the most important disorders of the shoulder area: frozen shoulder, biceps tenosynovitis, biceps tendon tear, rotator cuff tear, impingement syndrome, Rotator Cuff Calcified Tendonitis
examination,impingement syndrome,rotator cuff injury,shoulder,shoulder instability
All about orthopaedic shoulder examination. comprehensive ppt with all tests arranged symptom wise
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Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
CDSCO and Phamacovigilance {Regulatory body in India}NEHA GUPTA
The Central Drugs Standard Control Organization (CDSCO) is India's national regulatory body for pharmaceuticals and medical devices. Operating under the Directorate General of Health Services, Ministry of Health & Family Welfare, Government of India, the CDSCO is responsible for approving new drugs, conducting clinical trials, setting standards for drugs, controlling the quality of imported drugs, and coordinating the activities of State Drug Control Organizations by providing expert advice.
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In India, pharmacovigilance activities are monitored by the Pharmacovigilance Programme of India (PvPI), which works closely with CDSCO to collect, analyze, and act upon data regarding adverse drug reactions (ADRs). Together, they play a critical role in ensuring that the benefits of drugs outweigh their risks, maintaining high standards of patient safety, and promoting the rational use of medicines.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
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Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
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These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
6. Glenohumeral joint
• Main joint of the shoulder : Ball and socket joint
(synovial)
– humerus fits loosely in joint >
Very mobile (Price: instability)
– 45% of all dislocations
(m/c in body : lack of bony stability)
– Passive stability/ Static joint stability
• Glenoid labrum (50%)
• Joint capsule
• Ligaments
• Bony restraints
• Surrounding musculatature
– dynamic stability
• rotator cuff muscles
• the scapular rotators
6
7. Muscles of shoulder:
Stabiliser muscles
mainly responsible for
stabilization and
rotation of the scapula
• Trapezius
• serratus anterior,
• levator scapulae,
• rhomboid muscles
7
8. Muscles of shoulder:
Extrinsic muscles
• biceps,
• triceps, and
• deltoid muscles
• several actions of
the glenohumeral
joint.
8
9. Intrinsic muscles : Rotator cuff muscles
SITS : (Supraspinatus, Infraspinatus, Teres Minor, Subscapularis, )
Function: IR & ER of the glenohumeral joint, along with humeral abduction.
9
10. • The rotator cuff muscles
depress the humeral head
against the glenoid.
• With a poorly functioning
(torn) rotator cuff, the
humeral head can migrate
upward within the joint
because of an opposed
action of the deltoid
muscle.
10
15. • Axillary Nerve - supplies the Deltoid muscle. Most
commonly stretched with shoulder dislocations.
• Long Thoracic Nerve - supplies Serratus Anterior muscle
and can cause Winging of the Scapula
• SuprascapularNerve supplies supraspinatus and infras
pinatus muscles and can be entrapped or diseased
• Musculocutaneous Nerve - supplies the Biceps muscle
and is injured rarely.
15
20. 1.Degenerative arthritis of the
shoulder
• M/c – OA
• RA – morning stiffness improving with
activity.
• Post traumatic
• Collagen vascular
disease : SLE, scleroderma, RA
• Intense inflammation, swelling ,
erythema : gout, septic arthritis
• Joint effusion : OA, RA, Septic arthritis
20
21. OA of Shoulder Joint
• Constant, Aching PAIN +stiffness.
• Activity makes the pain worse.
• rest and heat provide some relief
• Pain is present at rest and may interfere
with sleep.
• grating or popping sensation, swelling
of joint
• crepitus may be present on physical
examination.
21
24. 1.Downward traction of arm increases
pain : chin adduction test
2.Cross arm test- raise arm and actively
adduct - increased pain
Signs:
24
25. AC Shear Test
- Interlock fingers with hand on
distal clavicle and spine of
scapula
- Pain in A-C joint when hands
squeezed together = (+) test
25
History / Maneuver Sens
(%)
Spec
(%)
AC
Active
compression
100 97
26. Injection – diagnostic as well as
therapeutic
1” medial to tip of acromion
process
MRI
Investigations:
26
27. 3.Shoulder instability
• Young (<35/40)
• May be hypermobile & asymptomatic, Often sport related
• AGE best prognostic factor:
– <20 yrs 90% recurrence
– 20-40 yrs 60% recurrence
– >40 yrs 10% recurrence
• Dislocations or subluxations- recurrent (GHJ/ACJ/SCJ)
• Pathology: lax capsule, labral tear, #glenoid
• Most common is anterior instability (97%)
• Can lead to cuff degeneration & impingement
27
28. Shoulder instability
• Anterior Glenohumeral
Instability: Most common
– Apprehension test
• sense of instability and
apprehension with arm placed
at 90 deg abduction and 90 deg
ER.
– Relocation test
• Examiner applies posterior force on
proximal humerus while externally
rotating patient’s arm
• Positive test = patient expresses relief
28
Test
Instability
Sens
(%)
Spec
(%)
Relocation 57 100
Apprehension 68 100
29. Sulcus sign : Inferior laxity
(+) test - sulcus at infra-acromial area
– compare to unaffected side
Load & shift sign :
Anterior and
posterior laxity
(+) if greater than
50% displacement
29
33. Disorders of the Intrinsic muscles:
Rotator cuff dysfunction
37
•Helps to lift and rotate arm and to stabilize the ball of shoulder with the joint
34. Three stages of rotator cuff disease [Neer]
Stage I <25yrs edema and hemorrhage of the
tendon and bursa
Stage II 25-40 yrs tendinitis and fibrosis of the
rotator cuff
Stage III >40 yrs tearing of the rotator cuff
(partial or full thickness)
38
2 main causes of rotator cuff dysfunction:
1. age related degeneration(chronic) - more common, > 40 yr age
2. Sports injury or trauma (acute)- less common, mostly in young
pts.
35. Rotator cuff Tears can be:
Partial thickness Full thickness Full thickness
with humoral
detachment
often appear as
fraying of an
intact tendon.
"through-and-through".
These tears can be small pin point, larger
button hole, or involve the majority of
he tendon where the tendon still remains
substantially attached to the humeral head and
thus maintains function
and may result in
significantly
impaired
shoulder motion
and function
39
36. Inability to elevate the arm above the level of
the shoulder without the help of the opposite
arm is the hallmark of rotator cuff
disturbance.
Patients often have lesser ROM Actively than Passively due to weakness
40
37. • commonly, the onset is gradual and may be caused by repetitive
overhead activity or by wear and degeneration of the tendon.
– Workers who do overhead activities such as painting, stocking shelves
or construction
– Athletes such as swimmers, pitchers and tennis players
• Pt may feel pain in the front of your shoulder that radiates down the
side of the arm.
• Other symptoms may include stiffness and loss of motion.
• may have difficulty using arm to reach overhead to comb hair or
difficulty placing behind your back to fasten a button.
• When the tear occurs with an injury, there may be sudden acute
pain, a snapping sensation and an immediate weakness of the arm.
41
38. It should be remembered that with
rotator cuff tears,
passive range of motion is normal, but
active range of motion is limited
VS
frozen shoulder
both passive and active ranges
of motion are limited
42
39. Examination
Supraspinatus:
Function: abduction of shoulder for 1st 10-15 degrees of
the arc
Jobe test/empty can test
Both arms are abducted to 90 degrees in the scapular plane and then are fully
pronated to point the thumbs toward the ground :
to abduct arms against the examiner's resistance 43
40. • Infraspinatus: external rotation (90%
force)
• Teres minor: external rotation (10%
force)
Test for both:
Arm at 0 deg abduction, elbow at 90 deg,
Externally rotate-pain
Isolated test for teres minor:
Hornblower test: 90 abd, 90
flexion, full ext rot, hold
position 44
42. Drop arm test: Purpose: tears in the rotator cuff, primarily
supraspinatus muscle
46
• A patient with a complete rotator cuff tear will be unable to hold the arm in the
abducted position, and it will fall to the patient's side
•May be able to lower arm slowly to 90° (this is mostly deltoid function)
•If the athlete is able to hold the arm at 90º, pressure on the wrist will cause the arm to
fall.
43. Shrug Sign : massive rotator cuff tears
The patient will often shrug or hitch the shoulder forward
to use the intact muscles of the rotator cuff and the
deltoid to keep the arm in the abducted position.
47
44. Rotator Cuff Tear
History / Maneuver Sens
(%)
Spec
(%)
History of trauma 36 73
Night pain 88 20
Painful arc 33 81
Empty can test 84
89
50
58
Drop arm 21 100
45. X-RAY
A complete tear of the
supraspinatus resulting in a shift
upwards of the head of the
humerus d/t action of deltoid.
USG
showing rotator cuff tear
Investigations
49
46. Normal Rotator cuff full thickness tear
MRI and ultrasound are comparable in efficacy
MRI
50
47. 51
Treatment Options
• pain relief and improve the function of shoulder.
– Anti-inflammatory medication/Steroid injection
– Strengthening exercise and physical therapy
• It may take several weeks or months to restore
the strength and mobility to ones shoulder.
• Rest and limited overhead activity / Use of a sling
• Surgery for tears
49. 1.Adhesive Capsulitis/ Frozen shoulder
53
The causes of frozen shoulder are not
fully understood.
The process involves thickening and
contracture of the capsule surrounding
the shoulder joint.
Frozen shoulder occurs much more commonly in individuals with diabetes, affecting 10-
20% of these individuals.
Other Risk factors : hypothyroidism, hyperthyroidism, lung disease, RA, Parkinson's disease,
stroke and cardiac disease or surgery.
Frozen shoulder can develop after a shoulder is injured or immobilized for a period of time.
50. Frozen shoulder
• Age : 30-55
• STIFF & PAINFUL JOINT
• Worse at night & in cold weather
• Severely restricted ROM
both passive and active ranges
of motion are limited
Risk factors:
• diabetes, stroke, lung
disease, rheumatoid arthritis,
heart disease
54
51. Stage 1 "freezing" or
painful stage
6-9 months slow onset of pain. As
the pain worsens, the
shoulder loses motion
Stage 2 The "frozen" or
adhesive stage
4-9 months slow improvement in
pain but the stiffness
remains
Stage 3 "thawing" or
recovery
5-26 months when shoulder motion
slowly returns toward
normal
Frozen shoulder :
Stages
55
52. 56
Treatment
• Prevention : Attempts to prevent frozen shoulder include early motion of
the shoulder after it has been injured.
• The first goal is pain control >>>> restore motion, physical therapy.
• nerve blocks :Suprascapular nerve block
• Surgical : manipulation under anesthesia and shoulder arthroscopy
– After surgery, physical therapy is important to maintain the motion
that was achieved with surgery.
53. 2.Supraspinatus tendonitis
Acute
• younger pts with inciting event include
– carrying heavy loads in front and away from the body,
– throwing injuries, or
– vigorous use of exercise equipment.
Chronic
• older pts,insiduous
57
54. • Pain felt primarily in deltoid
region
• Constant, severe, sleep
disturbances+
• Awaken on night when turned
onto the affected shoulder
• To relieve pain, patients splint
shoulder : “shrugging
appearance”
• Point tenderness over greater
tuberosity
58
55. • Gradual loss of ROM
shoulder
• hair combing, or reaching
overhead quite difficult
• Painful arc of abduction
• catch or sudden onset of
pain in the midrange of
the arc caused by
impingement of the
humeral head onto the
supraspinatus tendon.
59
56. Positive Dawbarn sign
• pain to palpation over the greater tuberosity of the humerus
when the arm is hanging down that disappears when the
arm is fully abducted.
.
• With continued disuse, muscle wasting may occur and a
frozen shoulder may develop
• May be a/w bursitis
60
58. Tests
Speed test
Elbow extended, supinated forearm
Pt tries to flex shoulder against resis:
Positive test = tender in bicipital
groove
Yergason test
Forearm flexed 90
Supinate against resistance >>
Tenderness along the bicipital
groove
62
59. 4.Subdeltoid bursitis
• Pain with any movement of the
shoulder, specially abduction
• Pain in subdeltoid area going upto
upper humerus
• 1st awakening- abduct shoulder-
sudden pain increased
O/E
• Tender acromion
• Swelling of bursa
• Passive elevation & medial rotation
reproduces pain
63
62. -subacromial spurs (bony
projections from the
acromion),
-osteoarthritic spurs
-variations in the shape of
the acromion.
-Thickening or
calcification of the
coracoacromial ligament.
66
63. Symptoms
• Pain, Weakness
• Loss of movement at affected
shoulder
• Worsened by overhead
abduction (60-120 deg)
• May occur at night if patient
turns over that shoulder
• Grinding/ popping sensation
+-
67
64. Neer Sign- pronation +
forced flexion leading to pain
Hawkin sign- more sensitive, performed
by elevating the patient's arm forward to 90
degrees while forcibly internally rotating the
shoulder .
Impingement test- injecting lignocaine into the sub-acromial
bursa-relief of pain
68
Test
Impingement
Sens
(%)
Spec
(%)
Hawkin’s 89 60
66. • Deep poorly circumscribed pain
• Posterior and lateral aspect of the
shoulder
• Muscle atrophy may be +
• (supra & infraspinatus)
O/E
• Deep pressure over suprascapular
notch-painful
• Cross body adduction test+
because of the stretching of the
nerve
70
67. Referred Pain to the shoulder
Referred from viscera
– Liver & Gall bladder
– Lung
– Heart (angina)
– Diaphragmatic irritation
Cervical Radicular Pain *C5
Cervical facet pain
Sympatheticlally mediated pain : CRPS
71
68. Red flags
• Unexplained deformity or swelling
• Significant weakness not due to pain
• Suspected malignancy
• Fever/malaise
• Significant/unexplained sensory or motor deficit
• Pulmonary or vascular compromise
72
69. Urgent referral
• Displaced or unstable fractures.
• Failed attempted reduction of dislocated
shoulder.
• Massive cuff tear.
• Severe dislocation of ACJ or SCJ.
• Undiagnosed severe pain.
73
70. DIAGNOSIS POSITIVE FINDINGS IMAGING
AC JOINT ARTHRITIS Pain at AC joint
+ve cross-body adduction test
h/o trauma/weight lifting
X-ray :OA at AC joint
AC separation
ADHESIVE CAPSULITIS Age >40yrs
Restricted active and passive motion
h/o DM/thyroid d/s
X-ray:Normal
GLENOHUMERAL
INSTABILITY
Age<40yrs,h/o subluxation/dislocation
+ve apprehension test
X-ray:Normal
GLENOHUMERAL OA Age >50yrs
Progressive pain
Crepitus with ROM
Xray :narrowing of joint
space, spurring,
osteophytes
SHOULDER
IMPINGEMENT
SYNDROME
Age >40 yrs
Pain with overhead activity/PAINFUL
ARC, Night pain/weakness
+ve hawkin’s test
X ray:Acromial spur,
humeral head sclerosis
or cyst, loss of acro-
humeral interval
FULL-THICKNESS
ROTATOR CUFF TEARS
bit older than impingement pts,
Pain rolling on shoulder in bed/night
pain ,Weakness ,Lateral arm pain,
Passive motion typically greater than
active motion
MRI to document extent
of tear
71. Remember:
• Polyarthralgia- examine all other joints,
work up for connective tissue disorders.
• Systemic features like fever/malaise- alert .
Rule out septic joint.
• Perform Neurovascular examination of UL
75