Peptic ulcers develop in the stomach, esophagus, or duodenum (upper small intestine) and are usually caused by H. pylori bacteria or nonsteroidal anti-inflammatory drugs (NSAIDs) like aspirin. Symptoms include abdominal pain, nausea, vomiting, weight loss, and bloody stools. Diagnosis involves blood tests, breath tests, stool tests, endoscopy, or imaging. Treatment involves antibiotics to kill H. pylori, proton pump inhibitors to reduce acid, and medications to protect the stomach lining. Complications can include bleeding, perforation, and scarring.
Peptic ulcers are sores that develop in the lining of the stomach, lower esophagus, or small intestine. They're usually formed as a result of inflammation caused by the bacteria H. pylori, as well as from erosion from stomach acids. Peptic ulcers are a fairly common health problem.
The most definite way to diagnose peptic ulcer is by a barium x-ray or upper endoscopy. Sometimes, if you have very convincing symptoms of an ulcer, I may test you for the H pylori bacteria and may treat you with ulcer medications before doing any other tests. For More details you camn visit at http://sripathikethumd.com/digestive-conditions/peptic-ulcer/
Peptic ulcers are sores that develop in the lining of the stomach, lower esophagus, or small intestine. They're usually formed as a result of inflammation caused by the bacteria H. pylori, as well as from erosion from stomach acids. Peptic ulcers are a fairly common health problem.
The most definite way to diagnose peptic ulcer is by a barium x-ray or upper endoscopy. Sometimes, if you have very convincing symptoms of an ulcer, I may test you for the H pylori bacteria and may treat you with ulcer medications before doing any other tests. For More details you camn visit at http://sripathikethumd.com/digestive-conditions/peptic-ulcer/
This PPT covers the Pathophysiology of Peptic ulcer. It includes factors causing peptic ulcer, factors causing peptic ulcer, diagnosis and complications of peptic ulcer.
Peptic ulcer ( a medical study)- definition, causes, pathophysiology etc martinshaji
Peptic ulcers include:
Gastric ulcers that occur on the inside of the stomach
Esophageal ulcers that occur inside the hollow tube (esophagus) that carries food from your throat to your stomach
Duodenal ulcers that occur on the inside of the upper portion of your small intestine (duodenum)
It's a myth that spicy foods or a stressful job can cause peptic ulcers. Doctors now know that a bacterial infection or some medications — not stress or diet — cause most peptic ulcers.
this is a detailed study on peptic ulcer
please comment
thank you
This ppt is suitable for b.pharma students. This ppt is prepared according to b.pharma IInd semester syallbus. In this ppt we provide all topics related to pathophysiology of peptic ulcer. In this ppt we covered introduction, types, sign & symptoms, pathophysiology, diagnosis, complications and treatments.
THIS presentation gives a brief overview of what peptic ulcer is and how h. pylori, NSAID's and stress cause peptic ulcer.
it is presented in a diagrammatic view which clears the concept for its causes.
It will be helpful for the people studying pathophysiology of diseases.
This is a presentation about gastrointestinal tract disorders concerning a medical informations about an important disorders that affect GIT of human being.
This PPT covers the Pathophysiology of Peptic ulcer. It includes factors causing peptic ulcer, factors causing peptic ulcer, diagnosis and complications of peptic ulcer.
Peptic ulcer ( a medical study)- definition, causes, pathophysiology etc martinshaji
Peptic ulcers include:
Gastric ulcers that occur on the inside of the stomach
Esophageal ulcers that occur inside the hollow tube (esophagus) that carries food from your throat to your stomach
Duodenal ulcers that occur on the inside of the upper portion of your small intestine (duodenum)
It's a myth that spicy foods or a stressful job can cause peptic ulcers. Doctors now know that a bacterial infection or some medications — not stress or diet — cause most peptic ulcers.
this is a detailed study on peptic ulcer
please comment
thank you
This ppt is suitable for b.pharma students. This ppt is prepared according to b.pharma IInd semester syallbus. In this ppt we provide all topics related to pathophysiology of peptic ulcer. In this ppt we covered introduction, types, sign & symptoms, pathophysiology, diagnosis, complications and treatments.
THIS presentation gives a brief overview of what peptic ulcer is and how h. pylori, NSAID's and stress cause peptic ulcer.
it is presented in a diagrammatic view which clears the concept for its causes.
It will be helpful for the people studying pathophysiology of diseases.
This is a presentation about gastrointestinal tract disorders concerning a medical informations about an important disorders that affect GIT of human being.
Ulcers range from small, painful sores in the mouth to bedsores and serious lesions of the stomach or interstine
Gastric ulcers :are peptic ulcers in the stomach.
Duodenal ulcers :are peptic ulcers in the duodenum
Gastrointestinal Diseases
Group 5:
Leticia Bernal Leon
Daydig Rodriguez
Maria Rodriguez
Karina Silveira
Instructor:
Dr. Alain Llanes Rojas, DNP, APRN, FNP-BC
Miami Regional University
Diagnosis, Symptoms & Illness Management
MSN5600
Gastroesophageal Reflux
Gastroesophageal reflux that does not cause symptoms is known as physiologic reflux. In nonerosive reflux disease (NERD), individuals have symptoms of reflux disease but no visible or minimal esophageal mucosal injury
Gastroesophageal reflux disease (GERD) is the reflux of acid and pepsin or bile salts from the stomach to the esophagus that causes esophagitis. The severity of the esophagitis depends on the composition of the gastric contents and esophageal mucosa exposure time.
Definition & Classification
Gastroesophageal Reflux
Causes
GERD can be caused by abnormalities or alterations in
1. Lower esophageal sphincter function
2. Esophageal motility
3. Gastric motility or emptying
Esophageal function studies include the following:
Determination of the lower esophageal sphincter (LES) pressure (manometry)
Graphic recording of esophageal swallowing waves, or swallowing pattern (manometry)
Detection of reflux of gastric acid back into the esophagus (acid reflux)
Detection of the ability of the esophagus to clear acid (acid clearing)
An attempt to reproduce symptoms of heartburn (Bernstein test)
Gastroesophageal Reflux
Risk Factors
Obesity
Hiatal hernia
Use of drugs or chemicals that relax the LES (anticholinergics, nitrates, calcium channel blockers, nicotine)
Cigarette smoke.
Trigger Factors
Coughing
Vomiting
Straining at stool
Asthma
Chronic cough
Sinusitis.
Gastroesophageal Reflux
Common Symptoms
Heartburn that occurs 30 to 60 minutes after meals and when the patient bends over or lies down.
Regurgitation of sour or bitter gastric contents
Belching, and fullness of the stomach
Upper abdominal pain within 1 hour of eating.
Atypical Symptoms
chronic cough
asthma attacks
chronic laryngitis
sinusitis
discomfort during swallowing.
Noncardiac chest pain.
Dysphagia
Gastroesophageal Reflux
Clinical manifestations are related to mucosal injury from acid regurgitation and the frequency and duration of reflux events.
The symptoms worsen if the individual lies down or if intraabdominal pressure increases because of coughing, vomiting, or straining at stool.
Uncomplicated GERD that is responsive to first-line therapy does not require an endoscopy.
Patients who do not respond to therapy and those with suspected complications should undergo an endoscopic examination
Management & Evaluation
Differential diagnosis
Gastritis
Peptic ulcer
Gastric cancer
Cholelithiasis
Angina pectoris.
Gastroesophageal Reflux
Diagnosis of GERD is based on the history and clinical manifestations.
An upper endoscopy with biopsy is the standard diagnostic procedure for GERD. It confirms the diagnosis and documents the type and extent of tissue damage.
Esophageal endoscopy: shows hyperemia ...
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
4. Peptic ulcers are sores that
develop in the lining of the
stomach, lower esophagus, or
small intestine.
They’re usually formed as a result
of inflammation caused by the
bacteria H. pylori, as well as from
erosion from stomach acids.
6. a) DEPENDING ON THE
SITE
Gastric Ulcers: ulcers that
develop inside the stomach.
Esophageal Ulcers: ulcers that
develop inside the esophagus.
Duodenal ulcers: ulcers that
develop in the upper section of
the small intestines, called the
duodenum.
7. 1. Gastric ulcers that occur on
the inside of the stomach.
Gastric ulcers are less
common than duodenal
ulcers.
They affect older age
group.
NSAID’S are common
cause of gastric ulcers.
8. 2. Duodenal ulcers that occur
on the inside of the upper
portion of small intestine
(duodenum).
All duodenal ulcers occur in
duodenal bulb. This part of
duodenum is in the direct
path of the acid contents of
stomach.
They tend to occur in
younger patients and more
common in men than in
women.
9. 3. ESOPHAGEAL ULCER is a painful sore
located in the lining of the lower part of the
esophagus.
Esophageal ulcers usually form as a result
of an infection with a bacterium
called helicobacter pylori.
10.
11.
12. Although the prevalence of peptic
ulcer is decreasing in many
western countries it still affects
approximately 10% of all adults at
some time in their lives. The male
to female ratio for duodenal ulcers
varies from 5:1 to 2:1 while for
gastric ulcers is 2:1 or less.
17. Burning Stomach Pain.
Feeling Of Fullness, Bloating.
Fatty Food Intolerance.
Heartburn.
Nausea.
Pain Worse Between Meals And At Night.
Ulcers May Cause Severe Signs Or
Symptoms Such As:
Vomiting Or Vomiting Blood — Which May
Appear Red Or Black.
Dark Blood In Stools.
Trouble Breathing.
Feeling Faint.
Unexplained Weight Loss.
Appetite Changes.
18. CAUSES
Rare cancerous and non-cancerous tumors
in the stomach, duodenum, or pancreas—
as Zollinger-Ellison syndrome
An infection
with Helicobac
ter pylori (H.
pylori)
Long-term use
of NSAIDs,
as aspirin and
ibuprofen.
19. H.PYLORI
H. Pylori are gram-
negative, spiral & have
multiple flagella at one
end. Transmitted from
person-to-person by oro–
oral or feco-oral spread .
H. Pylori bacteria can
cause peptic ulcer disease
by damaging the mucous
coating that protects
lining of stomach and
duodenum.
20. NSAID
Nonsteroidal anti-
inflammatory drugs
reduce pain, fever &
inflammation, or swelling.
NSAIDS blocks or reduce
amount of prostaglandin
that body makes.
These drugs also generate
oxygen-free radicals &
products of the
lipoxygenase pathway that
may contribute to
ulceration.
21. ZOLLINGER ELLISON
SYNDROME
Zollinger-Ellison
syndrome is a rare
disorder.
The tumors release large
amounts of gastrin, a
hormone that causes
your stomach to produce
large amounts of acid.
The extra acid causes
peptic ulcers to form in
your duodenum and in
the upper intestine.
22.
23. To help diagnose a peptic
ulcer, your doctor will ask you
questions about your medical
history, symptoms, and
medicines you take.
Be sure to mention medicines
that you take without a
prescription, especially
NSAIDS such as
ASPIRIN (bayer aspirin)
IBUPROFEN (motrin, advil)
MEDICAL HISTORY
24. If you have a (H. Pylori) infection,
your doctor will order these
tests:
1. BLOOD TEST:
A blood test involves drawing a
sample of your blood at lab.
A health care professional tests
the blood sample to see if the
results fall within the normal
range for different disorders or
infections.
25. 2. UREA BREATHE TEST:
For a urea breath test, you will
drink a special liquid that
contains urea,.
If H. pylori are present, the
bacteria will change this waste
product into carbon dioxide—a
harmless gas.
If your breath sample has
higher levels of carbon dioxide
than normal, you have H.
pylori in your stomach or small
intestine.
26. 3. STOOL TEST:
Doctors use a stool test to study a
sample of your stool.
A doctor will give you a container for
catching and storing your stool at home.
You return the sample to the doctor or a
commercial facility, who then sends it to
a lab for analysis. Stool tests can show
the presence of H. Pylori.
27. 4. UPPER GI ENDOSCOPY:
More reliable method and visual diagnosis.
An intravenous (IV) needle will be placed in
your arm to provide a sedative. Sedatives
help you stay relaxed and comfortable
during the procedure.
The doctor may perform a biopsy with the
endoscope by taking a small piece of tissue
from the lining of your esophagus.
28. 5. UPPER GI SERIES:
During the procedure, you’ll stand or sit
in front of an x-ray machine and drink
barium, a chalky liquid. Barium coats
your esophagus, stomach, and small
intestine so your doctor can see the
shapes of these organs more clearly on
x-rays.
29. 6. H.PYLORI TESTING:
Invasive tests using an
endoscopy procedure are less
frequently performed than
non-invasive tests as they
require a tissue biopsy
collection. Tests include:
Histology – examination of
tissue under a microscope.
Rapid urease testing – detects
urease, an enzyme produced
by H. Pylori which lead to
change a ph.
Culture – growing H.
Pylori in/on a nutrient
solution.
33. Treatment goals
in acute setting
are relief of
discomfort and
protection of the
gastric mucosal
barrier to
promote
healing.
Anti-ulcer drugs
are classified as:
ANTI-ULCER DRUGS
H2 RECEPTOR
BLOCKER
PROTON PUMP
INHIBITOR (PPI)
ANTACID
ULCER
PROTECTIVE
AGENTS
ANTI H.PYLORI
DRUGS
39. Constipation (caused by
aluminum-containing
antacids).
Diarrhea (caused by
magnesium-containing
antacids).
SIDE EFFECTS
DRUG
INTERACTION
Certain medications may cause complications
when taken with antacids or acid reducers,
particularly those that require an acidic
environment in the stomach for absorption
like aspirin.
45. The treatment regimens are omeprazole,
amoxicillin, and clarithromycin (OAC)
for 10 days.
Bismuth subsalicylate, metronidazole,
and tetracycline (BMT) for 14 days.
TREATMENT
REGIMEN
46. A. PERFORATION:
A hole develops in
the lining of the
stomach or small
intestine and
causes an infection.
48. B. INTERNAL
BLEEDING:
Bleeding ulcer can
result in significant
blood loss and thus
require
hospitalization.
Signs of a bleeding
ulcer include
lightheadedness,
dizziness, and black
stools.
49. C. SCAR TISSUE:
This is thick tissue that
develops after an injury.
This tissue makes it
difficult for food to pass
through your digestive
tract.
Signs of scar tissue
include vomiting
and weight loss.
50. D. GASTRIC OUTLET
OBSTRUCTION:
They can also cause scar
tissue, swelling or scar
tissue can obstruct the
normal passage of food
through digestive tract.
This obstruction can
cause reported episode
of vomiting .