This document discusses various nerve block techniques used in dentistry. It describes the areas anesthetized, indications, contraindications, landmarks, and complications for different types of nerve blocks including:
- Maxillary nerve blocks (supraperiosteal, posterior superior alveolar, anterior superior alveolar)
- Palatal nerve blocks (greater palatine, nasopalatine)
- Mandibular nerve blocks (inferior alveolar, Gow-Gates, mental, lingual)
- Extraoral techniques for maxillary and mandibular nerve blocks
It also defines complications of local anesthesia as any deviation from the normal expected outcome, classifying them as local, systemic,
Classification of Impaction and Methods & Techniques of Third molar/Manidibular impaction removal,Flap designs of impaction removal techniques and more
Classification of Impaction and Methods & Techniques of Third molar/Manidibular impaction removal,Flap designs of impaction removal techniques and more
Dr, Kathirvel Gopalakrishnan
M.D.S (OMFS)
Presentation on Maxillary nerve block which helps for a quick refresh.
Applied aspects described well and slides contains images for easy understanding of the subject.
Extraction instruments | Dental surgery | by Dr.mohammad nameerDenTeach
Learn about Extraction instruments - including forceps and elevators types used in general dentistry in any dental clinic.
Powerpoint shared by: Dr.mohammad nameer
You can watch dental videos and read in dentistry on:
www.denteach.com
Dr, Kathirvel Gopalakrishnan
M.D.S (OMFS)
Presentation on Maxillary nerve block which helps for a quick refresh.
Applied aspects described well and slides contains images for easy understanding of the subject.
Extraction instruments | Dental surgery | by Dr.mohammad nameerDenTeach
Learn about Extraction instruments - including forceps and elevators types used in general dentistry in any dental clinic.
Powerpoint shared by: Dr.mohammad nameer
You can watch dental videos and read in dentistry on:
www.denteach.com
INTRODUCTION
TEMPORAL FOSSA
Borders
Clinical correlation
Contents
Temporalis and surgical aspects
Temporal fascia and surgical aspects
Deep temporal nerves and vessels, auriculotemporal nerve, superficial temporal artery
TEMPORAL BONE AND TEMPORAL BONE FRACTURES
CORONAL OR BI-TEMPORAL APPROACH
TEMPORAL (GILLIES) APPROACH
INFRATEMPORAL REGION
Borders
Contents
LOCAL ANESTHESIA AND THE INFRATEMPORAL FOSSA
INFECTION OF THE INFRATEMPORAL FOSSA REGION AND ITS SPREAD
SURGICAL APPROACHES TO THE INFRATEMPORAL FOSSA
PTERYGOPALATINE FOSSA / SPHENOPALATINE FOSSA
Contents
Relations
Communications
Clinical aspects
Applied anatomy of external ear with emphasis on divisions of external ear - pinna, external auditory canal, tympanic membrane with their structure and clinical aspects with mention about otoscopy
Mandibular Nerve Block - By Dr Saikat Saha Dr Saikat Saha
Mandibular nerve block techniques in short for Dental Surgeons. Mandibular nerve blocks are very important for all dental surgeons as it becomes a part and parcel of all dental and oral surgeons. This presentation will be useful for students of dentistry and doctors.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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ABDOMINAL TRAUMA in pediatrics part one.drhasanrajab
Abdominal trauma in pediatrics refers to injuries or damage to the abdominal organs in children. It can occur due to various causes such as falls, motor vehicle accidents, sports-related injuries, and physical abuse. Children are more vulnerable to abdominal trauma due to their unique anatomical and physiological characteristics. Signs and symptoms include abdominal pain, tenderness, distension, vomiting, and signs of shock. Diagnosis involves physical examination, imaging studies, and laboratory tests. Management depends on the severity and may involve conservative treatment or surgical intervention. Prevention is crucial in reducing the incidence of abdominal trauma in children.
DISSERTATION on NEW DRUG DISCOVERY AND DEVELOPMENT STAGES OF DRUG DISCOVERYNEHA GUPTA
The process of drug discovery and development is a complex and multi-step endeavor aimed at bringing new pharmaceutical drugs to market. It begins with identifying and validating a biological target, such as a protein, gene, or RNA, that is associated with a disease. This step involves understanding the target's role in the disease and confirming that modulating it can have therapeutic effects. The next stage, hit identification, employs high-throughput screening (HTS) and other methods to find compounds that interact with the target. Computational techniques may also be used to identify potential hits from large compound libraries.
Following hit identification, the hits are optimized to improve their efficacy, selectivity, and pharmacokinetic properties, resulting in lead compounds. These leads undergo further refinement to enhance their potency, reduce toxicity, and improve drug-like characteristics, creating drug candidates suitable for preclinical testing. In the preclinical development phase, drug candidates are tested in vitro (in cell cultures) and in vivo (in animal models) to evaluate their safety, efficacy, pharmacokinetics, and pharmacodynamics. Toxicology studies are conducted to assess potential risks.
Before clinical trials can begin, an Investigational New Drug (IND) application must be submitted to regulatory authorities. This application includes data from preclinical studies and plans for clinical trials. Clinical development involves human trials in three phases: Phase I tests the drug's safety and dosage in a small group of healthy volunteers, Phase II assesses the drug's efficacy and side effects in a larger group of patients with the target disease, and Phase III confirms the drug's efficacy and monitors adverse reactions in a large population, often compared to existing treatments.
After successful clinical trials, a New Drug Application (NDA) is submitted to regulatory authorities for approval, including all data from preclinical and clinical studies, as well as proposed labeling and manufacturing information. Regulatory authorities then review the NDA to ensure the drug is safe, effective, and of high quality, potentially requiring additional studies. Finally, after a drug is approved and marketed, it undergoes post-marketing surveillance, which includes continuous monitoring for long-term safety and effectiveness, pharmacovigilance, and reporting of any adverse effects.
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NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
These lecture slides, by Dr Sidra Arshad, offer a quick overview of the physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar lead (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
6. Describe the flow of current around the heart during the cardiac cycle
7. Discuss the placement and polarity of the leads of electrocardiograph
8. Describe the normal electrocardiograms recorded from the limb leads and explain the physiological basis of the different records that are obtained
9. Define mean electrical vector (axis) of the heart and give the normal range
10. Define the mean QRS vector
11. Describe the axes of leads (hexagonal reference system)
12. Comprehend the vectorial analysis of the normal ECG
13. Determine the mean electrical axis of the ventricular QRS and appreciate the mean axis deviation
14. Explain the concepts of current of injury, J point, and their significance
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
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share - Lions, tigers, AI and health misinformation, oh my!.pptxTina Purnat
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• Assessing risk and mitigating harm from adverse health narratives in communities, health workforce and health system
2. Techniques of Injection
• Basic points-
Use a Sterile Sharp Needle
Check The flow of Solution
Position the patient
Dry the tissue/ wipe once.
Apply topical anesthetic
3. Topical antiseptic /optional
Communicate with patient apply firm hand rest
Inject few drops of soln, communicate with
patient,
Advance to the target slowly ,aspirate , inject
Withdraw the needle slowly
Observe the patient & check for anesthetic
symptoms
4. Technique for Maxillary Block
• Supra periosteal injection:
– Anaesthetize buccal soft tissue & hard tissue
– Nerves anaesthetized – large terminal branches
– Indication :
• 1 or 2 teeth need to be anaesthetized / small area
5. – Contra-indication :
• Infection
• Dense bone covering
– Target area :
• Behind apices of tooth
– Landmarks :
• Muco-buccal fold
• Crown & root length
6.
7. • Posterior Superior Alveolar Nerve Block
– Area anaesthetized:
• Maxillary 3rd, 2nd & 1st molar (except mesio-
buccal root of 1st molar
• Bone & periodontium over these
– Indication:
• Treatment of 2 or more molars required
• Supra-periosteal injection – ineffective
• Acute inflammation
8. – Contra-indication:
• Pt with bleeding disorders
– Disadvantage:
• More of soft tissue landmarks used
• 2nd injection for 1st molar
– Landmarks:
• Mucobuccal fold
• Zygomatic process of maxilla
• Infratemporal surface of maxilla
• Anterior border and coronoid process of mandible
• Tuberosity of maxilla
11. • Anterior superior alveolar nerve block
– Areas anaesthetized
• Pulp of maxillary C.Is – Canine
• Buccal periodontium, lower eyelid, lateral aspect of nose
• Upper lip
– Indications
• More than 2 anterior teeth
– Contraindications
• Discreet treatment areas
• Hemostasis of localized area – not adequately achieved
14. Palatal Anaesthesia
• Pressure Anaesthesia
– Slow deposition
– Small quantity
– Effect only a very small area
• Greater palatine nerve block
– Areas anaesthetized
• Palatal soft tissue – posterior aspect
• Palatal hard tissue
15. – Indication
• Surgical procedures posterior portion of hard
palate
• Palatal Anaesthesia in conjunction with
posterior superior alveolar nerve block.
– Landmarks
• Greater palatine foramen – junction of the
maxillary alveolar process & palatine bone
• Between the 2nd & 3rd molars – 1-1.5cms away
from gingival margin
16.
17. • Nasopalatine nerve block
– Areas anaesthetized
• Anterior portion of Hard palate and over lying structures
back to the bicuspid area.
– Indications
• Anterior palatal procedures supplementing infraorbital
nerve blocks
• Anaesthesia of nasal septum
– Landmarks
• Central incisor & incisive papilla
18. – Complications
• Hematoma
• Necrosis
– Technique
• Single needle penetration
• Multiple needle penetration
Usually most discomforting block for patient – very painful
19. • Maxillary nerve block
– Areas anaesthetized
• Pulpal Anaesthesia
• Maxillary teeth – 1 side
• Periodontium / soft tissue – 1 side
– Indications
• Extensive oral / periodontal / endodontal procedures
• Other regional nerve blocks not possible
• Therapeutic procedure to diagnose neuralgias
21. – Complications
• Hematoma
• Penetration into orbit
– Volume – displaces orbital structures, periorbital
swelling, proptosis, 6th nr block – diplopia, transient
loss of vision, optic nerve blocked, retrobulbar block
/ hemorrhage, opthalmoplegias (common)
• Penetration into nasal cavity
– Patient complains – LA running down the throat – to
prevent keep mouth wide open
– Technique
• High tuberosity approach
• Greater palatine canal approach
22.
23. • Maxillary nerve block – Extra Oral
– Areas anaesthetised
• Anterior temporal & zygomatic region
• Lower eyelid
• Side of nose
• Anterior cheek
• Upper lip
• Maxillary teeth / alveolar bone & overlying structures –
1side
• Hard & soft palate
• Tonsils – parts of pharynx
• Nasal septum – floor of nose
24. – Indications
• Extensive surgery – 1 half of maxilla
• Others blocks not possible
• Therapeutic purposes
– Technique
• mid point of zygomatic process
• Needle gently contact lateral pterygoid plate
• Maximum length of 4.5cms directed slightly upward & forward
– Note:
• In final position – internal maxillary artery – inferior to needle
• Temporal vessels on either sides
• Posteriorly foramen ovale with mandibular nerve & foramen
spinosum with middle meningeal artery
• Anteriorly pterygomaxillary fissure
28. • Anatomical structures - final position
• Superiorly –
– Inferior alveolar nerves & vessels
– Insertion of medial pterygoid
– Mylohyoid nerves & vessels
• Anteriorly –
– Deep part of parotid gland
• Laterally –
– Lingual nerve
– Internal pterygoid
– Spehnomandibular ligament
• Medially- ramus of mandible.
29.
30.
31. • Closed mouth/ Akinosis technique (1977)—
– Nerves anesthetized -
– Area anesthetized
• one half of mandible upto mid line including lingual tissue and
inferior portion of the ramus of the mandible.
– Land mark-
• occluding plane of the teeth.
• Muco gingival junction maxillary teeth.
• Antr border of ramus.
• Orientation of bevel must be oriented away from the bone of
mandibulaar ramus (bevel faces toward mid line).
– More popular now
• Land marks easy
• One prick – mandibular, buccal, lingual n anesthetised.
• Patient more comfortable.
32.
33. • Advantages
Atraumatic,
pats. with restricted mouth opening.
fewer post op complications.
Disadvantages
Difficult to visualize the path of needle and
depth of insertion.
Complications
hematoma, transient facial n. paralysis.
34. • Gow gates technique– 1973 (mandibular n.
block)
Nerves anaesthetised – inferior alveolar, mental,
incisive, lingual, mylohyoid, auriculotemporal and
buccal.
– Area –all mandibular hard and soft tissue Upto
mid line.
– Indications- multiple procedures on mandibular
teeth, buccal soft tissue anaesthesia from third
molar to midline, conventional inf. alv. n. block is
unsuccessful.
– Contraindications – infection or acute inflammation
in the area of infection, pats. with restricted
mouth opening.
35. – Land marks-
– Extraoral- corner of mouth, lower border of the tragus,
intertragic notch
– Intraoral – height of injection established by
placement of needle tip just below the mesiolingual cusp
of max. 2nd molar, penetration of soft tissue distal to 2nd
molar at the same height.
– Final position needle is just inferior to condyle and insertion
of lateral pterygoid.
Gained popularity – single needle penetration, relies on soft
tissue landmarks – differ from patient to patient
36.
37. • Lingual nerve block –
– Area anaesthetised –
• Anterior 2/3rd tongue, floor of mouth, lingual
mucoperiosteum
Only used singly to operate on tongue, floor of mouth
• Buccinator / long buccal nerve block
– Area anaesthetised –
• Buccal mucosa & mandibular molar – mucoperiosteum
– Land marks
• External oblique ridge, retromolar triangle
38.
39. • Mental nerve block
– Areas anaesthetised
• Lower lip, mucous membrane – anterior to mental
foramen
– Landmarks
• Mandibular bicuspids
– Indications
• Surgery of lower lip or mucous membrane
40.
41. Extra Oral Technique
• Mandibular nerve
– Area anaesthetised
• Temporal region with auricle of ear & external auditory
meatus
• TMJ, salivary glands
• Anterior 2/3rd of tongue
• Mandible – hard & soft tissue – midline
– Landmarks
• mid point of zygomatic arch
• Zygomatic notch
• Cornoid process of mandible
• Lateral pterygoid plate
42. – Indications
• When need to anaesthetise entire mandibular nerve
• Infection / trauma – makes terminal anaestheisa not
possible
• Diagnostic / therapeutic
The needle is pointed posteriorly & to a greater
depth of 5 cms
43.
44. • Mental & Incisive nerve block
– Area anaesthetised
• Mandibular hard & soft tissue – labial aspect with lower
lip
– Landmarks
• Bicuspid teeth, lower ridge of body of mandible
• Supra & infra orbital notch
• Pupil of the eye
2 inch 22 gauge needle used & introduced slightly
anteriorly & downwards
46. Complications
• Definition
– An anaesthetic complication may be defined as
any deviation from the normal expected pattern
during or after securing regional anaesthesia
– 2 types
• Local
• Systemic
47. • LOCAL COMPLICATIONS
– Needle breakage
– Pain on injection
– Burning on injection
– Persistent anaesthesia or paresthesia
– Trismus
– Hematoma
– Sloughing of the tissue / soft tissue injury
– Facial nerve paralysis
49. • Classification
– Primary / secondary
• Primary – caused & manifested at time of anaesthesia
• Secondary – manifested later
– Mild / severe
• Mild – exhibit slight change from normal expected
pattern
- reverses itself without treatment
• Severe – manifests itself – pronounced deviation
- requires specific treatment
50. – Transient / permanent
• Transient – is one that is severe at occurrence – no
residual effects
• Permanent – residual effect; lasts for a life time even
though it is mild
Complications could be a combination of any of the above
mentioned types
Majority are either Primary Mild & Transient or Secondary
Mild & Transient
51. • Complications
– Attributed to solutions – toxicity, allergy,
idiosyncrasy, anaphylactoid reaction, local irritation
– Attributed to technique / needle – syncope,
muscle trismus, pain, edema, hematoma
52. Needle breakage
• Cause –
– Unexpected movement – patient (if patient
movement is opposite to path of needle insertion)
– Multiple used needle
– Defective manufacture of needles/barbed needles
– smaller gauge – more likely to break
53. • Prevention
– Correct gauge – 25 gauge
– Long needles – prevent penetration till hub
– Not to redirect when in tissue
• Management
– Patient – not to move – hand in the mouth –
mouth open
– Fragment visible – remove it
– Fragment not visible – inform patient – not
necessary for intervention immediately –
Radiograph suggested
54. • Precautions
– Avoid bony contact
– Avoid heavy pressure
– Avoid movement of needle and patient
56. Burning on injection
• Causes
– Due to pH of solution 5 (LA) – 3 (LA+VC)
– Rapid injection
– Contamination
– Warm solution
• Problems
– pH disappears upon LA action – no residual
effect
– Contaminated solution other complications –
trismus, edema, paraesthesia
57. • Prevention
– Slow injection – 1ml / minute
– Cartridge stored at room temperature – away from
containers with alcohol / other agents
58. Persistent anaesthesia / paresthesia
• Causes
– Direct trauma to nerve – bevel of needle
– LA solution containing neurotoxic substance –
alcohol
– Injection of wrong solution
– Hemorrhage / infection – near to nerve
• Problem
– Persistent anaesthesia – usually rare
– Biting / thermal / chemical insult – without patient
awareness
– When lingual nerve is involved – taste impaired
59. • Prevention
– Proper care & handling of dental cartridge
– Adherence to injection protocol
• Management
– Usually resolve in 8 weeks
– Periodic recall & check up of patients
– Persistence – consult neurosurgeon
– TENS
– Recall patient every 2 months for check up
60. Trismus
• Definition
– “difficulty in opening the jaws due to muscle spasm”
• Causes
– Trauma – muscle / blood vessel
– Irritating solution
– hemorrhage
– Infection
– Multiple needle punctures
– LA have been known to have slight myotoxicity
– Excessive volume – distension of tissues
• Problems
– Pain / hypomobility
61. • Prevention
– Use of sharp, sterile, disposable needle
– Aseptic technique
– Practice atraumatic methods
– Avoid repeated injections
– Use minimum volume
– Control infection
62. • Management
– Heat therapy
• Warm saline rinses, moist hot packs
– Analgesics
• Aspirin, Codeine (30-60mg), muscle relaxants
– Initial physiotherapy
• Thrice a day
– Antibiotic regime
• Possibility of infection
63. Hematoma
• “effusion of blood into extra-vascular spaces”
• Causes
– Arterial & venous puncture – common in PSA & Inf. Alv.
nerve blocks
– Patients with bleeding disorders
• Problem
– Bruise – may / may not be visible extra-orally
– Complications – pain & trismus
– Swelling & discoloration
• Prevention
– Knowledge of normal anatomy – proper technique
– Shorter needle – PSA, minimize the number of penetration
– Discard defective needles- barbed needles
64. • Management
– Immediate – apply firm pressure 5-10minutes
• Inf. Alv. Nr. Block – medial aspect of ramus
• Infra orbital, Mental, Incisive block – directly over
foramen
• PSA – pressure on soft tissue with finger as
posteriorly as tolerated by patient – medial superior
direction
• Patient to be reviewed after 24 hours, advice
analgesics, cold application upto 4-6 hours, warm-
pack application next day
65. Infection
• Comparitively rare complication
• Instrument needle solution to be as aseptic as
possible
• Area & operative hands – cleaned
• Avoid passing needle through infected area
• Use disposable syringes
67. • Prevention
– Proper care & handling of armamentarium
– Atraumatic injection technique
– Complete medical evaluation prior to injection
• Management
– Trauma – resolve in few days without therapy
– Hemorrhage – resolve slowly 7-14 days
– Allergy – life threatening, airway impairment –
basic life support, call medical help, Epinephrine –
0.3mg, Antihistamine, Corticosteroids
– Total airway obstruction – Tracheostomy /
Cricothyroidectomy
68. Sloughing of tissue
• Causes
– Epithelial desquamation – topical anaesthesia –
long time, heightened sensitivity to LA
– Sterile abscess – secondary to prolonged ischemia
– VC in LA site – hard palate
• Problems
– Pain & infection
• Prevention
– Topical – for not more than 1-2 minutes
– VC – minimal concentration in solution
69. • Management
– Symptomatic – pain – analgesia
– Epithelial desquamation – resolve few days
– Sterile abscess resolve 7-10 days
70. Soft tissue injury
• Causes
– Trauma occurs – frequently mentally / physically challenged
children
– Primary cause – significantly longer duration of action
• Problem
– Pain & swelling
– Infection of soft tissue
• Prevention
– Cotton roll between lip & teeth
– Patient – guarded against eating / drinking
– Warning sticker
71. Facial nerve paralysis
• Cause
– LA solution into parotid gland – usually while
giving Inf Alv Nr. Block, Akinosis technique
• Problem
– Ipsilateral loss of motor control – Buccinator
muscle
– Inability to raise the corner of Mouth, close Eye lid
• Prevention
– Needle tip to contact bone, redirection of needle
to be done only after complete withdrawal
72. • Management
– Reassure the patient
– Resolves after action of LA is over
– Eye patches to the affected – eye drops
– Contact lenses if any – removed
73. Systemic complications
• Toxicity / toxic overdose
– “Signs and symptoms that result from an overly high blood
level of a drug in various target organs and tissues”
– Predisposing factors
• Age – any age
• Weight – greater the body weight greater is the amount of dose
tolerated before overdose reaction
• Sex – during pregnancy – renal function disturbed – females more
affected at this time
• Diseases – hepatic & renal dysfunction reduced breakdown
• Congestive heart failure – less liver perfusion
• Genetics – pseudocholinesterase deficient – toxicity - Ester LA
74. • Drug factors – Vasoactivity – vasodilation – increase in
blood concentration
• More concentration – greater risk
• Dose- smaller dose should always be preferred
• Route of Administration – Intravascular – increased
toxicity
• Rate of injection – slower rate preferred
• Vascularity of injection site – more vascular – greater
absorption
• Presence of Vasoconstrictor – with VC less absorption
75. – Causes of toxicity –
• Biotransformation usually slow
• Drug – slowly eliminated by kidney
• Too large a total dose
• Absorption from injection site - rapid
• Accidental intra-vascular injection
– Symptoms –
• CNS – cerebral cortical stimulation – talkative, restless,
apprehensiveness, convulsions
• Cerebral cortical depression – lethargy, sleepiness,
unconsciousness
• Medullary stimulation – increased B.P, Pulse rate,
Respiration
76. – Medullary depression – mild fall in B.P– severe cases drops to
0 , Pulse , Respiration – similar effect
• Treatment
– Mild overdose reaction – slow onset reaction – > 5 mins
administer Oxygen (prevent acidosis), monitor vital signs, in
case of convulsions – anti-convulsants (diazepam/midazolam
infusion)
– Slower onset - >15 mins – same procedure
– Severe overdose reaction – rapid onset – 1 minute –
unconsciousness with or without convulsion, patient in supine
position, convulsions – protect hand, leg, tongue, BLS,
administer anti-convulsant,use of vasopressor(phenyl ephrine)
i.m if hypotensiom presists.
– post seizure – CNS depression usually present
77. Idiosyncrasy
• “It is an adverse response that is neither an
overdose nor an allergic reaction”
• Common cause – some underlying
pathology/psychological /genetic mechanism
• Psychotherapy may be helpful
• Treatment – symptomatic ..remember ABC’s!
78. Syncope
• “transient loss of consciousness that is caused due to cerebral
ischemia (neurogenic shock)”
• Anxiety – increased blood supply to muscles, sitting position
2mm Hg, less pressure – cerebral arteries
• Clinically pallor, light headedness, dizziness, tachycardia &
palpitation – may further lead to Unconsciousness
• Treatment – discontinue procedure, supine position-
(trendelenburg position), deep breathing, O2 administration if
required, BLS
79. Allergy
• “hypersensitive state acquired through exposure to a particular
allergen reexposure to which produces a heightened capacity to
react”
• 1 % of all reaction in LA is allergy
• Predisposing factors
– Hyper sensitivity to ester more common-procaine
– Most of patients allergic to methyl paraben
– Recently allergy to sodium meta bi sulfide is also increasing
Precautions---
Ho of allergy to be recorded
Ho any asthmatic attack to be noted.
Always better to test the patient for allergy before treatment.
80. – Consultation and allergy testing
• Refer doubtful cases for allergic skin test – sub cutaneous
test most sensitive.
• Informed consent that includes cardiac arest end death to
be included.
– Signs and symptoms of allergy.
• Dermatological------ urticaria –wheal and smooth elevated
patch seen, ------angio oedema—localised swelling – face
hands, common
• Respiratory– broncho spasm, respiratory distress,
– dysnea, wheezing, flushing, tachycardia etc.
81. –Laryngeal edema – type of angio
neurotic oedema- life threating.
•Edema upper air way – laryngeal edema
• Lower air way affect bronchioles- small.
82. –Management
•skin reactions-
–Delayed – non life threatening - oral
histamine blockers- 50 mg diphenhidramine,10
mg chlorpheniramine 3-4 days.
–Immediate reaction—with conjunctivitis
rhinitis- vigorous management.
– 0.3 mg epinephrine. IM
– 50 mg diphenhydramine Im
– medical help summoned.
83. – Observe patient for minimum of 60 min
– Oral histamine blockers for 5 days.
– Respiratory reaction –
• patient in comfortable position.
• administer - oxygen
• Admn epinephrine- bronchodilator
• Observe for 60 min , advise anti histamines to prevent relapse.
• Histamine blockers Im
– Laryngeal edema-
• Patient position ,oxygen, broncho-dilator, iv anti histamines.
• If condition not improving cricothyrotomy - achieve patent air
way if necessary give artificial ventilation.
84. • Patient with confirmed allergy status-
– if patient allergic to any one type of anesthetic
ester / amide use the other.
– Use histamine blocker like diphenhydramine as
anesthetic.
– General anesthesia
– alternative method of pain control –
• electric anesthesia / hypnosis.